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1 ising phase, followed by a larger and slower rising phase.
2 ent that is followed by a slower and delayed rising phase.
3            The OFF Idelta component showed a rising phase.
4 ial components with time constants of 13 ms (rising phase), 70 ms and 350 ms (decaying phase).
5  total Ca2+ to the end-pools accelerated the rising phase and abbreviated the duration of the hump.
6 ibuted in part to lower amplification in the rising phase and in part to faster recovery kinetics.
7 ces are known to generate, respectively, the rising phase and the prolonged plateau phase of cerebell
8 very recording: monophasic EPSCs, with sharp rising phases and monoexponential decays, and multiphasi
9 rhodopsin in transgenic mice accelerates the rising phases and recoveries of flash responses by about
10 ted by two exponentials with means of 45 ms (rising phase) and 4408 ms (decaying phase) components.
11 (2+) signal revealed diverse kinetics of the rising phase, and hence various rates in the release of
12 ude at a given intensity of stimulus, faster rising phase, and shorter latencies than those of ISS-EP
13 ence of extracellular Ca(2+) showed a normal rising phase but a slower decay phase, resulting in long
14 within a few seconds, accompanied during its rising phase by a short burst of action potentials.
15 an approximate 20-y periodicity, including a rising phase during the 1990s that contributed to the pe
16 als, evoked calcium transients showed a fast-rising phase followed by a decay with a time constant of
17 ng sperm entry in vertebrate eggs has a slow rising phase followed by a sustained plateau.
18 iate [Ca2+]o bumps were biphasic with a slow rising phase followed by rapid amplification and inactiv
19 ransients composed of a small-amplitude fast rising phase, followed by a larger and slower rising pha
20  outward current and a slow component to the rising phase of [Ca(2+)](i).
21 ansient tension rise when applied during the rising phase of a twitch contraction, the amplitude of w
22  hillock regions and propagating the initial rising phase of action potentials through axons.
23 iratory component that closely resembled the rising phase of burstlets.
24 ponse to light that suggests a defect in the rising phase of clock protein expression.
25 aP with 5 microm GABA resulted in a biphasic rising phase of current with time constants of 50-60 ms
26 annels; the first peak of the differentiated rising phase of depolarizations - attributed to the infl
27 he activation of NMDA receptors and that the rising phase of each calcium spike is coincident with a
28 4, WD25, WD36, or WD60) selected to span the rising phase of incubation and the transition from low t
29                  The variability between the rising phase of individual sIPSCs was quantified by calc
30                                          The rising phase of LLDs shows characteristics of polyneuron
31 n to those from lactating rats in vitro, the rising phase of male bursts was more rapid, the decay ph
32 ed by fitting an exponential function to the rising phase of responses to supramaximal serotonin to b
33                         From analysis of the rising phase of rod-isolated flash responses we determin
34          Standard deviation functions of the rising phase of simulated IPSCs accurately described dis
35                            Unexpectedly, the rising phase of somatic APs is electrically indistinguis
36                                          The rising phase of such sIPSCs were multi-phasic, composed
37 rial dehydrogenases were synchronized to the rising phase of the [Ca(2+)](c) spikes.
38 iliary beta subunits are responsible for the rising phase of the action potential in cardiac muscle,
39 iliary beta subunits are responsible for the rising phase of the action potential in cardiac muscle,
40 (+) channels (VGSCs) are responsible for the rising phase of the action potential in excitable cells,
41 e was also significant Ca2+ entry during the rising phase of the action potential.
42       This activity also correlated with the rising phase of the arterial blood pressure wave consist
43 llations by allowing Ca(2+) entry during the rising phase of the Ca(2+) spikes and by providing an OF
44                                          The rising phase of the Ca(2+) transient was correlated with
45 dback ( approximately 2-4 microm) shapes the rising phase of the Ca2+ signal by acting to co-ordinate
46 y declined (to about 60% of peak) during the rising phase of the cell-wide Ca2+ transient before incr
47                                          The rising phase of the center response was slower for large
48 ctivation of cone transduction; that is, the rising phase of the cone response per bleached rhodopsin
49          Background light did not affect the rising phase of the dim flash response, a measure of the
50   ERG recordings demonstrated a delay in the rising phase of the ERG b-wave, larger photopic b-wave a
51  of recurrent IPSPs did not occur during the rising phase of the evoked EPSP, but > 3.0 ms after the
52 bserved between monosynaptic g GABAA and the rising phase of the evoked EPSP/EPSC.
53 er BDZ receptor ligand upon the slope of the rising phase of the evoked population excitatory postsyn
54  phase of jumps occurs on average during the rising phase of the firing rate of the looming-sensitive
55  the effect and induced a speeding up of the rising phase of the flash response, similar to the actio
56 mutant subunit was expressed with GluK4, the rising phase of the glutamate steady state concentration
57 ar dendrites, Ret-RGS1 should accelerate the rising phase of the light response of the ON bipolar cel
58 -dependent potentiation and the delay in the rising phase of the mutant ACh response suggest that the
59                             The onset of the rising phase of the photoresponse was significantly dela
60 umber of IP(3)Rs for 19 ms, representing the rising phase of the puff.
61 sets of feedforward IPSPs coincided with the rising phase of the pyramidal cell EPSP in > 80 % of pai
62 "deceleration-acceleration" notch during the rising phase of the response, as a signature of fast int
63 d attention was focused on the initial rapid rising phase of the responses.
64  molecule (Rh*) has a smaller slope than the rising phase of the rod response per Rh*, perhaps becaus
65 e rapid, the decay phase was slower, and the rising phase of the spike after hyperpolarization was fa
66  entry was nearly completely confined to the rising phase of the spike: only approximately 25% more s
67 ted reaction-diffusion of [Ca2+]i during the rising phase of the transient (first 30 ms after initiat
68  colliculus during the middle portion of the rising phase of the whisker movement protraction elicite
69                                 The earliest rising phase of this 'fractional response per unit inten
70                                          The rising phases of transients obtained from global illumin
71 sence of Na-current after, the regenerative (rising) phase of the action potential.
72     The resulting gating current exhibited a rising phase similar to that measured in voltage-depende
73        (2) slIPSCs frequently had an notched rising phase suggestive of summated IPSCs resulting from
74  smaller events exhibited a stammer in their rising phase that is interpreted as a brief pause in por
75 eases of [Ca(2+)](m) characterized by a fast rising phase that was followed by a slow decay.
76  that LCRs impart a nonlinear, exponentially rising phase to the DD later part, which exhibited beat-
77 amp methods were optimized for the fast MEPC rising phase using custom electronics.
78  prolonged in the presence of CPA, while the rising phase was unaffected.
79 tail currents at -40 mV displayed an initial rising phase with tau = 10 msec, followed by a slow mult

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