1 ne) is a formerly used and highly neurotoxic rodenticide.

2  for the Federal Insecticide, Fungicide, and Rodenticide Act, and recommended for the Toxic Substance

3 y, effects of low-level exposure to multiple rodenticides, and quantitative data on the magnitude of

4                                        These rodenticides are readily available household environment

5 e, routine application of some anticoagulant rodenticides (ARs) may be at a crossroad due to new regu

6 poisoning with the long-acting anticoagulant rodenticide, brodifacoum.

7 e, mosquito repellents and the superwarfarin rodenticides can help protect children from significant

8 fect that local selection with anticoagulant rodenticides has had on microsatellite marker variation

9 ntrogressed allele after the introduction of rodenticides in the 1950s, we found signatures of select

10 ing of genotypes expressing an anticoagulant-rodenticide-insensitive vitamin K 2,3-epoxide reductase

11 ding the Vkorc1 gene, which is implicated in rodenticide resistance, and olfactory receptor genes.

12  pest management consisted of application of rodenticide, sealing of holes that could serve as entry

13 219 and the genomic background revealed that rodenticide selection has overwhelmed drift-mediated pop

14 o are recent (one presumably due to warfarin rodenticide selection).

15 ticus) can cause resistance to anticoagulant rodenticides such as warfarin [1-3].

16 rin, warfarin-like anticoagulants, or potent rodenticides ("superwarfarins"), such as brodifacoum.

17  Use of the highly toxic and easily prepared rodenticide tetramethylenedisulfotetramine (TETS) was ba

18 nformation on common mosquito repellents and rodenticides used in households and discusses their toxi

19 nsecticides permethrin and carbaryl, and the rodenticide warfarin.