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1 tor VIII protects cells from H2O2 similar to ropinirole.
2 on) and prevented the protection mediated by ropinirole.
3 sonian effects of the D2/D3 receptor agonist ropinirole.
4               At about 5 months post-lesion, ropinirole (0.2 mg/kg, i.p.) was administered daily for
5  oxidative stress and protection provided by ropinirole, a D2 receptor agonist in PC12 cells and prim
6 in the Btbd9 mutant mice were relieved using ropinirole, a dopaminergic agonist widely used for RLS t
7 veal whether their over-expression modulated ropinirole activity.
8 ith dopamine agonists such as pramipexole or ropinirole allows levodopa to be delayed, but once levod
9  the role of dopamine agonists, particularly ropinirole and pramipexole, and management of motor and
10 including dopamine agonists (pramipexole and ropinirole) and alpha2 delta ligands (gabapentin).
11 nt targets for locally perfused pramipexole, ropinirole, and gabapentin, which significantly countera
12 lometazoline, oxymetazoline, fenoldopam, and ropinirole) are approved medications.
13 g-like" dual acting molecules, incorporating ropinirole as a dopamine D2 receptor agonist and ZM 2413
14 chlorperazine) and a lower-affinity agonist (ropinirole) exhibited reduced temporal dissociation betw
15      In contrast, the subtherapeutic dose of ropinirole generated only modest motor effects in lesion
16 o be significantly (one third) slower in the ropinirole group compared with that of the L-DOPA group.
17                               Patients given ropinirole had significantly reduced loss of striatal up
18                   The subtherapeutic dose of ropinirole improved the use of previously akinetic forel
19 upport the use of the subtherapeutic dose of ropinirole in a chronic model of PD.
20                                              Ropinirole-induced protection was associated with phosph
21 hty-six PD patients were randomized (1:1) to ropinirole or L-DOPA therapy, and (18)F-DOPA PET was per
22  patients taking either the dopamine agonist ropinirole or L-DOPA.
23 e and duloxetine (RR = 1.94); loratadine and ropinirole (RR = 3.21); and promethazine and tegaserod (
24                                              Ropinirole treatment was associated with rapid transloca
25 2 and 3 (D2R and D3R) as possible targets of ropinirole, we over-expressed these dopamine receptor ge
26 g analysis of the pEC(50) data revealed that ropinirole (which is not associated with valvulopathy) w
27                                              Ropinirole, which is a non-ergot dopamine agonist deriva

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