ライフサイエンスコーパス: rose bengal

1 xposed to visible light and photosensitizer (rose bengal).

2 nglet oxygen sensitizer ([Ru(bpy)(3)](2+) or Rose Bengal).

3 ated photodynamically by the photosensitizer Rose Bengal.

4 s, which do not express mucins, stained with rose bengal.

5 of islands of stratified cells that excluded rose bengal.

6 ere varied to alter the fraction of adsorbed rose bengal.

7 irradiation); and Group 5, rose bengal PDT (rose bengal + 518 nm irradiation).

8 we show the in vitro photodynamic effect of rose bengal activated by intracellular generation of lig

9 d time to carotid artery occlusion following Rose Bengal administration and laser-induced arterial in

10 Separate measurements performed with rose bengal adsorbed on p-GaP surfaces pretreated with (

11 groups: Group 1, no treatment; Group 2, 0.1% rose bengal alone; Group 3, 518 nm irradiation alone; Gr

12 Rose bengal and chlorin e6, photosensitizers (PSs) that

13 ts show that the strong affinity of PPI-5 to Rose Bengal and erythrosine B is attributed to the good

14 elayed carotid artery occlusion times on the rose bengal and ferric chloride thrombosis models.

15 ation of the mechanism of the ocular surface rose bengal and fluorescein staining that occurs in this

16 l retinal artery by intravenous injection of rose bengal and green laser irradiation of the artery.

17 hosphorescence decay of the triplet state of rose bengal and its quenching by ferricyanide.

18 Retinal vascular occlusion was introduced by rose Bengal and laser photocoagulation on chimeric mice

19 It was also noted that both rose bengal and lissamine green B treatments slightly pr

20 tures were incubated for 5 minutes with 0.1% rose bengal and photographed.

21 noparticles was developed, which showed that Rose Bengal and Rapa have high non-specific encapsulatio

22 ravenous injection of the photosensitive dye rose bengal and skull irradiation with a beam of focused

23 43 nm were prepared and functionalized with rose bengal and sulforhodamine B by a ligand-exchange pr

24 In contrast, rose bengal and the ATP-regulated potassium channel anta

25 et oxygen yield of a common photosensitizer (Rose Bengal) and the theoretical electric field enhancem

26 itive to peroxide but not methyl viologen or Rose Bengal, and GPXs, APX, and MSRA2 genes (encoding gl

27 Vital dyes such as fluorescein, rose bengal, and lissamine green B, among others, have b

28 ein mouse models with the photosensitive dye Rose bengal, and monitored plaque formation in real time

29 synthetic photosensitizers (methylene blue, rose bengal, and nitrite) and two model natural photosen

30 les for the photosensitizers methylene blue, Rose Bengal, and tetraphenylporphine.

31 n the DG, the VGLUT inhibitors Congo Red and Rose Bengal, and the mGlu2/3 agonist LY354740, also redu

32 Rose bengal- and riboflavin-mediated photodynamic therap

33 0.1% rose bengal; Group III, MRSA with 0.03% rose bengal; and Group IV, MRSA with 0.1% riboflavin.

34 on products of SQ incubated in solution with Rose Bengal as a photooxidizer were isolated by semiprep

35 In one type of particle, Rose Bengal as an efficient singlet oxygen ((1)O2) produ

36 idines with alcohols has been achieved using rose bengal as an organic photoredox catalyst at room te

37 presence of the photooxidants riboflavin and Rose Bengal as well as the diffusible one-electron oxida

38 y transfer between excited- and ground-state rose bengal at the C-18 silica/solution interface.

39 chronically contused rat spinal cord using a rose Bengal-based phototoxic method.

40 n the targeted artery, we photoactivated the rose bengal by illuminating the longitudinal hippocampal

41 tion include the use of metal-free, low-cost Rose Bengal catalyst and practical operation (ambient te

42 h MRSA strains was demonstrated (1) for both rose bengal concentrations under ambient and green LED i

43 aggregation inhibition by ERB analogs except rose bengal correlated well to the inhibition of Abeta c

44 n, as indicated by increased permeability to rose bengal diagnostic dye.

45 In vivo, the ophthalmic dye rose bengal displays profound antiviral effects against

46 The adsorption/desorption kinetics of rose bengal, distributed between a C-18 derivatized poro

47 ed manner using photodynamic thrombosis with rose bengal dye and thermal burns from an argon laser wi

48 ated HCLE cells showed significantly reduced rose bengal dye exclusion.

49 neas were treated on the incision walls with rose bengal dye followed by exposure to 514-nm laser rad

50 on surface barrier function were measured by rose bengal dye penetrance.

51 N ischemia was generated using laser-coupled rose Bengal dye photoactivation, and the infarct localiz

52 ity laser illumination of mice injected with Rose Bengal dye to induce photochemical injury in the re

53 HCLE cells were incubated with rose bengal dye to measure the role of MUC16 in ocular s

54 ed in adult rats by intravenous injection of Rose Bengal dye, followed by argon green laser treatment

55 up I, MRSA control; Group II, MRSA with 0.1% rose bengal; Group III, MRSA with 0.03% rose bengal; and

56 olerance against stress induced by Paraquat, Rose Bengal, heavy metal, and the synthetic auxins 1-nap

57 ess conditions created by high light levels, rose bengal, high salt levels, and osmotic shock.

58 The 0.03% rose bengal in dark conditions showed complete inhibitio

59 green LED irradiation, and (2) for the 0.1% rose bengal in the dark.

60 pocampal artery in urethane anesthetized and rose bengal-injected mice.

61 Rose bengal is an organic anionic dye used to assess dam

62 d to these stratified cells, indicating that rose bengal is excluded from cells that lack negative ch

63 We measured the binding of one inhibitor, rose bengal lactone (RBL), to kinesin (dissociation cons

64 2 carotid artery injury models (FeCl(3) and Rose Bengal/laser), fXII-deficient mice are more resista

65 Rose bengal-mediated PDT successfully inhibited the grow

66 Rose bengal-mediated PDT successfully inhibited the grow

67 Using focal photothrombosis via the Rose Bengal method, as well as excitotoxic NMDA lesions,

68 intervals up to 16 days post infection (dpi) rose bengal or lissamine green B was instilled in the le

69 ous HSV-1 and viral DNA in eyes treated with rose bengal or lissamine green B.

70 amples that are culture negative and contain rose bengal or lissamine green B.

71 n of dGuo produced 5-Lys-Sp exclusively when Rose Bengal or methylene blue was used to photochemicall

72 g antibody were the negative controls, while rose bengal or protoporphyrin IX with visible light were

73 boflavin + 375 nm irradiation); and Group 5, rose bengal PDT (rose bengal + 518 nm irradiation).

74 r apical surfaces provide protection against rose bengal penetrance in vitro and suggest a role for m

75 as severe focal stroke injury was induced by Rose Bengal photosensitization.

76 cell concentrations (8%RB and 3%RB), French rose bengal plate test with 4.5% cell concentration (4.5

77 id automated presumptive test (RAP), Mexican rose bengal plate tests with 8 and 3% cell concentration

78 We evaluated collagen cross-linking by rose bengal plus green light (RGX) in rabbit eyes and in

79 agreement with a model photosensitization by rose bengal (RB(2-)) in deoxygenated aqueous solutions r

80 The photosensitizers rose bengal (RB) and acridine orange (AO) were localized

81 d a phospholipid coating functionalised with Rose Bengal (RB) and/or 5-fluorouracil (5-FU), were asse

82 ARPE-19 cells preloaded with MC-540 or rose bengal (RB) were sublethally irradiated with green

83 served human amniotic membrane, stained with Rose Bengal (RB), was placed over a full-thickness wound

84 Rose bengal (RB, 0.1%) was applied to deepithelialized c

85 the vesicular glutamate transport inhibitor, Rose Bengal, reduced astrocytic glutamate release, sugge

86 mine whether exclusion of negatively charged rose bengal requires a negative charge at the cell surfa

87 zed separately by six triphenylmethane dyes (rose bengal, rhodamine B, crystal violet, ethyl violet,

88 Bacterial suspensions were mixed with rose bengal, riboflavin, or water according to experimen

89 bilised microbubbles (MBs), decorated with a Rose Bengal sensitiser, for SDT-based treatment of a pan

90 pression; however, HCLE cells incubated with rose bengal showed that exclusion of the dye was signifi

91 In contrast, rose bengal significantly decreased the infectious virus

92 ; however, there was no surface keratin, and rose bengal stain results were negative.

93 nifera (testate protists), including 'live' (Rose Bengal stained) and dead tests, in 5 cores (0-1 cm

94 of the eyes treated with NGF plus DHA showed rose bengal staining 30 days after PRK, compared with 50

95 GK rats had increased rose bengal staining and cornea fragility.

96 ased epithelial proliferation, and decreased rose bengal staining compared with NGF, DHA, or vehicle

97 The current study was undertaken to evaluate rose bengal staining in a human corneal-limbal epithelia

98 The effect of hyperosmotic stress on rose bengal staining in vitro was evaluated by increasin

99 at mucins have a protective role, preventing rose bengal staining of normal ocular surface epithelial

100 tear production, tear film breakup time, and rose bengal staining score were determined.

101 eakup time were significantly decreased, and rose bengal staining was significantly increased.

102 ye diagnosis (Lactoplate, Schirmer test, and Rose Bengal staining), even when the other test measures

103 chirmer testing with and without anesthesia, rose bengal staining, central corneal sensitivity, nucle

104 STZ rats showed stronger Rose Bengal staining, decreased tear secretion, slightly

105 All parameters, except rose bengal staining, deteriorated significantly after s

106 mucin layer were assessed via LC-biotin and Rose Bengal staining, respectively.

107 covery after surgery in goblet cell density, rose bengal staining, Schirmer test values without anest

108 The starry sky pattern correlated with rose bengal staining.

109 ith dry eye, may cause MAM release, allowing rose bengal staining.

110 silica nanoparticles with a photosensitizer, Rose Bengal, tethered to their surface.

111 Cores were sliced and stained with rose Bengal to detect live specimens of foraminifera.

112 A total of 44% and 78% of the rose bengal-treated and untreated eye samples, respectiv

113 itive than culture for detection of HSV-1 in rose bengal-treated eyes, in that 74% of rose bengal-tre

114 in rose bengal-treated eyes, in that 74% of rose bengal-treated samples were positive by PCR compare

115 e presence of the nonhazardous photocatalyst Rose Bengal under irradiation of visible light.

116 the ion concentration (Ca+2 and Mg+2) in the rose bengal uptake assay.

117 reduced the cellular area protected against rose bengal uptake.

118 Rose bengal was also effective in dark and ambient condi

119 rescein analogs (ethyleosin, phloxine B, and rose bengal) were relatively potent inhibitors of bindin

120 tors, Erythrosin B, Eosin Y, Phloxine B, and Rose Bengal, were determined.

121 ia, only methylene blue with E. coli K12 and rose bengal with C. jejuni showed an enhancing effect.