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1 le of glycophorin C as a receptor in P vivax rosette formation.
2 C 4 region of CD236R significantly inhibited rosette formation.
3 g malaria, through the mechanism of enhanced rosette formation.
4 es spanning multiple pairs of cells leads to rosette formation.
5 y affect the frequency and directionality of rosette formation.
6 d tests their ability to inhibit erythrocyte rosette formation.
7 lted in an increase of sialic acid-dependent rosette formation.
8 ity, indicating an essential role for CR1 in rosette formation.
9 ulation and cellular transitions involved in rosette formation.
10 ding defects in the outer limiting membrane, rosette formation and a reduction in functional acuity.
11 ignificant cellular rearrangement, including rosette formation and apical displacement of inner retin
13 We discuss and compare specific models for rosette formation and highlight outstanding questions in
14 i and act to specify edge contraction during rosette formation and to mediate timely rosette resoluti
16 tructures align across multiple cells during rosette formation, and adherens junction proteins assemb
17 s, intra-acinar lymphocytes and eosinophils, rosette formation, and canalicular cholestasis yielded a
18 s, the presence or absence of emperipolesis, rosette formation, and cholestasis in a blinded fashion
19 ional tension, a disruption of multicellular rosette formation, and defective convergent extension.
21 re frequently demonstrated lobular disarray, rosette formation, and hemorrhage than those with choles
22 e podJ gene are nonchemotactic, deficient in rosette formation, and resistant to polar bacteriophage,
24 otably, deficits in proliferation and neural rosette formation are rapidly reversed upon silencing on
25 calized to the leading zone are required for rosette formation, atoh1a expression, and primordium mig
27 in, NR2F1, NR2F2, and IRX2 - in the onset of rosette formation, during spontaneous neural differentia
29 ut inflorescence height, flowering time, and rosette formation have greatly diverged between the nati
36 d this appears to be linked to photoreceptor rosette formation in the rodless (cone-only) Nrl(-/-) re
37 roblast growth factor (Fgf) signals regulate rosette formation in the zebrafish posterior lateral lin
38 to Plasmodium falciparum-infected cells, and rosette formation is associated with severe malaria.
43 h the inhibition of macrophage spreading and rosette formation, MacMARCKS mutant also inhibits integr
44 -coated sheep erythrocytes, and also induced rosette-formation of erythrocytes with human monocytes a
46 macrophages, (iii) is required for podosome rosette formation triggered by Hck, and (iv) is necessar
47 hat OTX2 is highly expressed at the onset of rosette formation, when rosettes comprise no more than 3
49 contrast to the conventional way of studying rosette formation, which involves induction of neuronal
50 binding of infected RBCs to uninfected RBCs (rosette formation), while antibodies targeting STEVOR in
51 of immunized donors were separated following rosette formation with tetanus toxin (TT)-coupled immuno
52 mitive neuroectoderm, which is manifested by rosette formation, with consecutive differentiation into
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