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1 (95% CI, 64%-93%) against moderate to severe rotavirus.
2 t protection through reduced transmission of rotavirus.
3 other enteropathogens, with the exception of rotavirus.
4 hea, and controls were children negative for rotavirus.
5 000 for Cryptosporidium, and 1 in 118000 for rotavirus.
6 thogens, which include influenza viruses and rotaviruses.
7 nual changes in hospitalizations for AGE and rotavirus; 2012 was excluded as a transition year.
8                                              Rotavirus, a leading cause of severe gastroenteritis and
9                    In the Africa Region, the rotavirus AF declined from 54.8% (95% CI, 48.3%-61.5%) i
10 tions and emergency department visits due to rotavirus AGE were reduced by a median of 67% overall an
11  birth cohort, it will avert 54 000 cases of rotavirus and 281 deaths in children aged <5 years.
12 assess the impact of vaccine introduction on rotavirus and acute gastroenteritis (AGE) hospitalizatio
13 cination on hospitalizations and deaths from rotavirus and all-cause acute gastroenteritis (AGE) duri
14 irus vaccine introduction, seasonal peaks of rotavirus and all-cause AGE were dwarfed.
15 ubstantially decreased hospitalizations from rotavirus and all-cause AGE.
16        Knowledge of innate susceptibility to rotavirus and norovirus can lead to improved understandi
17 Mirpur, Dhaka) as part of the performance of rotavirus and oral polio vaccines in developing countrie
18                       Stools were tested for rotavirus and sera for antirotavirus immunoglobulins by
19 ed on enterocytes are proposed receptors for rotaviruses and can be measured in saliva.
20 haracterization of the confrontation between rotaviruses and their host cells has allowed us to learn
21 ubella, yellow fever, Japanese encephalitis, rotavirus, and invasive bacterial diseases.
22  among children whose stool was positive for rotavirus antigen (cases) and children whose stool was n
23  was made by enzyme immunoassay detection of rotavirus antigen in stool specimens.
24 talized for AGE have stool sample tested for rotavirus antigen, was used to assess trends in rotaviru
25                                Norovirus and rotavirus are prominent enteric viruses responsible for
26                                              Rotaviruses are known to recognize human histo-blood gro
27 athogens Campylobacter, Cryptosporidium, and rotavirus as conservative risk proxies for infections vi
28 sing children with AGE who test negative for rotavirus as controls for the rotavirus-positive cases.
29 e gastroenteritis (AGE) hospitalizations and rotavirus-associated hospitalizations during the prevacc
30  We studied trends in admissions specific to rotavirus at one hospital that had undertaken active rot
31                                        Using rotavirus binding and blocking assays, this study elucid
32              Fecal specimens were tested for rotavirus by enzyme immunoassay and genotyped.
33 GE had their stools collected and tested for rotavirus by enzyme immunoassay.
34 garis (PV), as well as B cells responding to rotavirus capsid protein VP6.
35 t per DALY averted was $10, and the cost per rotavirus case averted was $1.
36                                 Based on 204 rotavirus case patients and 601 test-negative controls a
37 rmed a case-control study of VE by comparing rotavirus case patients with test-negative controls.
38 ased diarrheal surveillance used to identify rotavirus cases but who test negative for rotavirus (tes
39       Vaccination among laboratory-confirmed rotavirus cases was compared with rotavirus-negative AGE
40 fectiveness was 59% (95% CI, 4%-83%) against rotavirus caused by G2P, the most common (37%) circulati
41 ore susceptible to infections from P[8]-type rotaviruses compared with nonsecretors (95% CI, 8.3-85.0
42 nt for more than half the global deaths from rotavirus, concerns remain about the performance of oral
43 confidence interval, 74.3%, 83.0%) >/=2-dose rotavirus coverage among participants eligible for publi
44 or approximately half (49%) of all estimated rotavirus deaths in 2013.
45    Globally, we estimated that the number of rotavirus deaths in children <5 years of age declined fr
46                 In 2013, an estimated 47 100 rotavirus deaths occurred in India, 22% of all rotavirus
47 tavirus deaths occurred in India, 22% of all rotavirus deaths that occurred globally.
48 of children with AGE who tested positive for rotavirus declined from 53% (645/1223) in prevaccine yea
49  VE estimate is consistent with the observed rotavirus decrease and with efficacy estimates from else
50 acity that is characteristic of RNA viruses, rotavirus dedicates substantial resources to avoiding th
51                         The predicted annual rotavirus detection rate from these studies declined sli
52 n less than 2 y of age presenting with acute rotavirus diarrhea (ARD) through March 31, 2011.
53 understand variables contributing to risk of rotavirus diarrhea (RVD).
54  RV5 were similarly effective against severe rotavirus diarrhea caused by a heterotypic strain in Gua
55 o determine the effectiveness of RV1 against rotavirus diarrhea hospitalization using a case-control
56 t of nutritional status on susceptibility to rotavirus diarrhea is not well understood.
57                                      Against rotavirus diarrhea of all severity, the adjusted 2-dose
58 E of 2 RV1 doses against hospitalization for rotavirus diarrhea was 57% (95% confidence interval, 14%
59 patients (children with laboratory-confirmed rotavirus diarrhea) and nonrotavirus "test-negative" dia
60         Assuming a 1% attack rate for severe rotavirus diarrhea, a 3% attack rate for severe nonrotav
61 tavalent rotavirus vaccine (RotaTeq) against rotavirus diarrhea.
62 immunization program to reduce the burden of rotavirus disease (documented to cause 38% of acute gast
63 Rotavirus vaccine is effective in preventing rotavirus disease in Rwandan children who began their ro
64                        In Peru, incidence of rotavirus disease was relatively higher during the secon
65  use, both RV1 and RV5 are effective against rotavirus disease, supporting the World Health Organizat
66 gA responses associated with protection from rotavirus disease.
67 and tended to be greater against more severe rotavirus disease.
68 ding a novel approach to vaccination against rotavirus disease.
69                  We show that binding of the rotavirus-encoded non-structural protein NSP2 to viral s
70  VH1-46 mAbs previously isolated from PV and rotavirus-exposed individuals indicates that cross-react
71 isms prevent the onset of autoimmunity after rotavirus exposure.
72 rcentage of hospital admissions positive for rotavirus fell from 45% in the prevaccine period to 25%
73 rcentage of hospital admissions positive for rotavirus fell from 48% in the prevaccine period to 28%
74 ength of naturally acquired immunity against rotavirus gastroenteritis (RVGE), mirroring vaccine unde
75 first episode of laboratory-confirmed severe rotavirus gastroenteritis (Vesikari score, >/=11) beginn
76 ine, had an efficacy of 66.7% against severe rotavirus gastroenteritis among infants in Niger.
77 cacy analysis, there were 31 cases of severe rotavirus gastroenteritis in the vaccine group and 87 ca
78                                   Each year, rotavirus gastroenteritis is responsible for about 37% o
79 ce interval [CI], 31%-91%) effective against rotavirus gastroenteritis requiring hospitalization or a
80  Serum Institute of India) to prevent severe rotavirus gastroenteritis.
81 al stools, and 344 (19.8%) children ever had rotavirus gastroenteritis.
82 e found 2 significant positive interactions: rotavirus + Giardia (odds ratio (OR) = 23.91, 95% confid
83                                         P[6] rotaviruses have been circulating with a high prevalence
84 wo-dose VE was 79% (95% CI, 62%-88%) against rotavirus hospitalization and 84% (95% CI, 64%-93%) agai
85 ctions of 40%, 46%, and 69% in the number of rotavirus hospitalizations among infants in 2013, 2014,
86         We performed active surveillance for rotavirus hospitalizations at the largest hospital in Za
87 n infants <1 year who accounted for 84.4% of rotavirus hospitalizations prior to vaccine introduction
88  Armenian children and substantially reduced rotavirus hospitalizations shortly after introduction.
89 ough indirect protection; overall in year 3, rotavirus hospitalizations were reduced by 69% among chi
90 ectiveness (VE) of 2 RV1 doses in preventing rotavirus hospitalizations.
91 avirus antigen, was used to assess trends in rotavirus hospitalizations.
92 hat is believed to play an important role in rotavirus host susceptibility and host range.
93 ultures from multiple individuals with human rotavirus (HRV) and assessed the host epithelial respons
94 bella antibody seroconversion and evaluating rotavirus IgA/IgG seroresponses in MR + HRV recipients.
95 odds ratio [OR], 0.77; P = .002) and lack of rotavirus immunoglobulin A (IgA) seroconversion (OR, 1.9
96 rial who were seronegative at baseline, anti-rotavirus immunoglobulin A seroconversion rates after 3
97 mented routine childhood vaccination against rotavirus in 2007.
98 children with diarrhoea testing positive for rotavirus in almost every age group.
99    We assessed vaccine effectiveness against rotavirus in Guatemala, where both the monovalent (RV1;
100  the benefits of routine vaccination against rotavirus in low-income settings.
101  of admissions to hospital for diarrhoea and rotavirus in Rwanda fell substantially after rotavirus v
102  increasing global importance of genotype G9 rotaviruses in humans and pigs.
103 ric viral pathogens, including norovirus and rotavirus, in both preventing and curing infection.
104        In low-resource populations with high rotavirus incidence, large-scale vaccination across a wi
105 te recruitment, and biliary injury in rhesus rotavirus-induced BA.
106 isk of rotavirus infection and all P[8]/P[4] rotavirus infected children were secretors.
107 iral response is specific to the pancreas of rotavirus-infected mice, similar to the autoimmunity ass
108                        The rapid declines in rotavirus infection and AGE in vaccinated and unvaccinat
109 ses were found to correlate with the risk of rotavirus infection and all P[8]/P[4] rotavirus infected
110                                              Rotavirus infection and disease were common, but with si
111 rstand naturally acquired protection against rotavirus infection and RVGE.
112 omposing the incidence rate into the rate of rotavirus infection and the risk of RVGE given infection
113 od of a 12-month increment, and detection of rotavirus infection by enzyme immunoassay in at least 10
114 multicountry birth cohort study, we describe rotavirus infection in the first 2 years of life in site
115                                              Rotavirus infection is one of the most common causes of
116 host genetic susceptibility to norovirus and rotavirus infection may be strain specific.
117   As childhood nutrition improves worldwide, rotavirus infection may remain a public health challenge
118            We find that MDA5 activity limits rotavirus infection not only through the induction of an
119 s crucial for the immune response to enteric rotavirus infection, a proposed etiological agent for T1
120 in intestinal epithelial cells and restricts rotavirus infection.
121 e associated with partial protection against rotavirus infection.
122 er T1D risk exhibit reduced activity against rotavirus infection.
123 estimated the impact on laboratory-confirmed rotavirus infections and hospitalizations for all-cause
124 ed to evaluate potential association between rotavirus infections and human HBGA phenotypes.
125  have been observed for laboratory-confirmed rotavirus infections during the 2014-2015 season.
126 o age-related, antibody-independent risk for rotavirus infections to cause RVGE.
127 ving childhood vaccines for pneumococcal and rotavirus infections while greatly expanding coverage of
128 rstood, but in analogy to Gardia lamblia and rotavirus infections, secondary lactose maldigestion (LM
129 mechanisms that host cells employ to prevent rotavirus invasion and the countermeasures that these vi
130                                              Rotavirus is a leading cause of death due to diarrhea am
131                                              Rotavirus is a leading cause of dehydrating diarrhea and
132 and reduces pathogenicity in mice.IMPORTANCE Rotavirus is the leading cause of diarrhea in humans.
133                                              Rotavirus is the world's leading cause of childhood diar
134                                              Rotaviruses, like most viruses that lack membranes of th
135 ns, and monitor vaccine impact, we estimated rotavirus mortality for children <5 years of age from 20
136                      Continued monitoring of rotavirus mortality rates and deaths through rotavirus s
137 hia coli (EPEC, n = 21), norovirus (n = 21), rotavirus (n = 15), sapovirus (n = 9), and Salmonella (n
138 -confirmed rotavirus cases was compared with rotavirus-negative AGE controls.
139 ine (33/44 [73%] unvaccinated) compared with rotavirus-negative children (81/136 [59%] unvaccinated)
140                                              Rotavirus nonstructural protein 4 (NSP4) is an endoplasm
141                                              Rotavirus, norovirus genogroup II, Cryptosporidium, and
142 ere >88% for Shigellaspp.,Campylobacterspp., rotavirus, norovirus genotype 1/2 (GI/GII), and adenovir
143 health burdens for Giardia, Cryptosporidium, rotavirus, norovirus, and adenovirus infections resultin
144                                    Moreover, rotavirus NSP1 (nonstructural protein 1) employs a pLxIS
145 come countries where >90% global deaths from rotavirus occur.
146 tolerated) to parenteral P2-VP8-P[8] subunit rotavirus or placebo injection.
147                                   Infectious rotavirus particles are triple-layered, icosahedral asse
148                            The properties of rotavirus particles make it possible to determine molecu
149 the following question: what fraction of the rotavirus particles that penetrate into the cell make ne
150 ted randomly among binding locations, inside rotavirus particles.
151  C viruses, herpes simplex virus, norovirus, rotavirus, parvovirus, and Epstein-Barr virus.
152 e likely to stimulate new research into both rotavirus pathogenesis and endotoxemia.
153 sis underlying inter-segment interactions in rotaviruses, paving the way for delineating similar RNA-
154 evaluate the efficacy of a live, oral bovine rotavirus pentavalent vaccine (BRV-PV, Serum Institute o
155 t negative for rotavirus as controls for the rotavirus-positive cases.
156                                              Rotavirus-positive children were significantly less like
157                                          The rotavirus positivity rate declined from 40.1% (449/1121)
158 est reduction was noted in infants, with the rotavirus positivity rate in this age group declining fr
159                                              Rotavirus prevalence is estimated to decline by 10.5% wi
160 ich are a common beginning in articles about rotaviruses, reflects the fact that these viruses have e
161 ovel insights into the mechanisms underlying rotavirus regulation of different interferons and are li
162                                              Rotavirus remained the leading etiology (overall weighte
163                                              Rotavirus remained the leading etiology of acute watery
164         Among 146 children enrolled in 2015, rotavirus remained the leading etiology of diarrhea requ
165 act of vaccine introduction in this setting, rotavirus remained the leading etiology of diarrhea requ
166 sulted in enhanced susceptibility of mice to rotavirus replication.
167                                             "Rotaviruses represent the most important etiological age
168 ugh the identified human G9P[19] and G9P[13] rotaviruses represented minority strains, the repeated d
169  of 2-3 doses of a rotavirus vaccine against rotavirus requiring emergency department visit or hospit
170 -specific inter-segment interactions between rotavirus RNAs, taking place in a complex RNA- and prote
171                                       Rhesus rotavirus (RRV) can also lead to biliary atresia (a neon
172                     In neonatal mice, rhesus rotavirus (RRV) can induce biliary atresia (BA), a disea
173     The murine model of BA, employing rhesus rotavirus (RRV), parallels human disease and has been us
174                                              Rotavirus (RV) causes significant morbidity and mortalit
175                                              Rotavirus (RV) is the leading cause of diarrhea-related
176 Temperature-sensitive (ts) mutants of simian rotavirus (RV) strain SA11 have been previously created
177                     Hepatitis E virus (HEV), rotavirus (RV), and astrovirus (AstV) are important path
178                                              Rotavirus (RV)-associated infections account for high nu
179                                      Group A rotaviruses (RVA) are a significant cause of pediatric g
180                                              Rotaviruses (RVs) can evolve through the process of reas
181                            The regulation by rotaviruses (RVs) of antiviral pathways mediated by mult
182 pe belongs to the P[II] genogroup of group A rotaviruses (RVs).
183      Declines were most prominent during the rotavirus season (May-October) and among infants <1 year
184 s, with greater declines observed during the rotavirus season compared with non-rotavirus season mont
185 ed mortality and hospitalizations during the rotavirus season months were considerably diminished in
186 uring the rotavirus season compared with non-rotavirus season months.
187  admissions in 2015, as well as delay of the rotavirus season.
188 us, kobuvirus, parechovirus, parvovirus, and rotavirus sequences were frequently detected but were no
189 D45(-) PCs during ageing and the presence of rotavirus-specific clones entirely within the CD19(-) PC
190                                              Rotavirus specifically infects the intestinal epithelial
191  participants who met study criteria and had rotavirus stool testing performed and vaccine status con
192 n both in vitro and in vivo In addition many rotavirus strains are resistant to the actions of differ
193                                  Because >60 rotavirus strains have been reported worldwide, concerns
194 ains, the repeated detection of porcine-like rotavirus strains in Taiwanese children over time justif
195 nding that the sequence is specific to those rotavirus strains that cause obstructive cholangiopathy.
196 s at one hospital that had undertaken active rotavirus surveillance from 2011 to 2014.
197  World Health Organization (WHO)-coordinated rotavirus surveillance network between 2008 and 2013 tha
198 rotavirus mortality rates and deaths through rotavirus surveillance will aid in monitoring the impact
199                      Costs did not differ by rotavirus test result, but were significantly higher for
200                                   The use of rotavirus test-negative controls offers an efficient and
201 fy rotavirus cases but who test negative for rotavirus (test-negative controls) can be considered a s
202 re, we focus on the transmission dynamics of rotavirus, the main diarrheal disease in infants and you
203        We searched PubMed using the keyword "rotavirus" to identify studies that met each of the foll
204  infections (respiratory syncytial virus and rotavirus) to illustrate this problem.
205    Although the potential health benefits of rotavirus vaccination are huge in low-income African cou
206  separately, we propose a design to estimate rotavirus vaccination coverage using controls from a rot
207                  The public health impact of rotavirus vaccination in African settings with a high hu
208 bing the impact and effectiveness of routine rotavirus vaccination in developing countries that were
209      This supports broader implementation of rotavirus vaccination in low-income countries where >90%
210 ts that could result in countries that adopt rotavirus vaccination into their national immunization p
211                   We summarize the impact of rotavirus vaccination on hospitalizations and deaths fro
212 roups within 1 year of introducing an infant rotavirus vaccination program are far greater than expec
213                             The two-dose HRV rotavirus vaccination program significantly reduced medi
214 lowing implementation of the Quebec, Canada, rotavirus vaccination program.
215                  In countries with effective rotavirus vaccination programs, disease due to that path
216 st 2 years of life in sites with and without rotavirus vaccination programs.
217                                              Rotavirus vaccination reduces childhood hospitalization
218 d encourage other countries to adopt routine rotavirus vaccination to reduce the health burden of sev
219 nfants (aged 6 to <8 weeks, without previous rotavirus vaccination).
220                               In addition to rotavirus vaccination, actions to improve nutrition stat
221 ay be required to obtain the full benefit of rotavirus vaccination, including indirect effects.
222 itionally, the effectiveness of programmatic rotavirus vaccination, including possible indirect effec
223 n children associated with implementation of rotavirus vaccination.
224 o before and 7 years after implementation of rotavirus vaccination.
225 ve of the beneficial public health impact of rotavirus vaccination.
226 sed on samples from a country with universal rotavirus vaccination.
227 e significantly less likely to have received rotavirus vaccine (33/44 [73%] unvaccinated) compared wi
228 tiveness of a two-dose schedule of the human rotavirus vaccine (HRV; Rotarix) given early at 6 and 10
229  vaccine effectiveness (VE) of a pentavalent rotavirus vaccine (RotaTeq) against rotavirus diarrhea.
230 a surveillance to evaluate monovalent G1P[8] rotavirus vaccine (RV1) efficacy and understand variable
231           Botswana introduced monovalent G1P rotavirus vaccine (RV1) in July 2012, providing one of t
232 ndomized controlled trial of monovalent oral rotavirus vaccine (RV1).
233  (HEU) African infants receiving pentavalent rotavirus vaccine (RV5) in a clinical trial.
234                                  Pentavalent rotavirus vaccine (RV5) was introduced into the Israeli
235             To facilitate decision making on rotavirus vaccine adoption by countries, help donors pri
236              Effectiveness of 2-3 doses of a rotavirus vaccine against rotavirus requiring emergency
237 declined from 54.8% (95% CI, 48.3%-61.5%) in rotavirus vaccine age-ineligible children to 20.0% (95%
238   Plasma IgA levels specific for antigens in rotavirus vaccine and oral polio vaccine containing poli
239 nd immunogenicity of the P2-VP8-P[8] subunit rotavirus vaccine at different doses in South African to
240            We sought to determine monovalent rotavirus vaccine cost-effectiveness in Malawi, one of A
241 uncan Steele discuss the evidence supporting rotavirus vaccine deployment in Asian countries.
242 l study at 6 public sector sites to estimate rotavirus vaccine effectiveness (VE) in age-eligible chi
243                        We sought to evaluate rotavirus vaccine effectiveness (VE) in this setting.
244                                              Rotavirus vaccine efficacy is lower in low-income countr
245 MA) to identify observational evaluations of rotavirus vaccine impact among children <5 years of age
246 rotavirus in Rwanda fell substantially after rotavirus vaccine implementation, including among older
247 pilot introduction of the Rotarix live, oral rotavirus vaccine in all public health facilities in Lus
248     Together with the demonstrated impact of rotavirus vaccine in reducing population hospitalization
249 countries in sub-Saharan Africa to introduce rotavirus vaccine into its national immunization program
250 in the Newly Independent States to introduce rotavirus vaccine into its national immunization program
251 ome African country to introduce pentavalent rotavirus vaccine into its routine national immunisation
252  recommendation that all countries introduce rotavirus vaccine into their national immunization progr
253                                          Pre-rotavirus vaccine introduction (2009-2011) and post-rota
254 us vaccine introduction (2009-2011) and post-rotavirus vaccine introduction (2013-2014) periods were
255  of diarrhea requiring hospitalization after rotavirus vaccine introduction in Africa.
256                                    Following rotavirus vaccine introduction, seasonal peaks of rotavi
257 hospitalizations, temporally associated with rotavirus vaccine introduction, was observed in children
258 ins poorly characterized, particularly after rotavirus vaccine introduction.
259 ted to children 7-18 weeks of age at time of rotavirus vaccine introduction.
260 n-hospital morbidity and mortality following rotavirus vaccine introduction.
261 te watery diarrhea despite a clear impact of rotavirus vaccine introduction.
262                                              Rotavirus vaccine is effective in preventing rotavirus d
263                                              Rotavirus vaccine is recommended for routine use in all
264         We aimed to assess the impact of the rotavirus vaccine program and estimate vaccine effective
265                                          The rotavirus vaccine program is highly cost-effective.
266                                 A monovalent rotavirus vaccine remains effective against a broad rang
267 n and immune activation were correlated with rotavirus vaccine responses in 68 human immunodeficiency
268  disease in Rwandan children who began their rotavirus vaccine series from 7 to 18 weeks of age.
269                   Togo introduced monovalent rotavirus vaccine starting 19 June 2014.
270                                       Adding rotavirus vaccine to the national schedule costs Malawi
271      Among 181 Pakistani infants in a G1P[8] rotavirus vaccine trial who were seronegative at baselin
272                       In Bolivia, monovalent rotavirus vaccine was introduced in 2008 and a previous
273                               The monovalent rotavirus vaccine was introduced in Tanzania on 1 Januar
274               Three doses of BRV-PV, an oral rotavirus vaccine, had an efficacy of 66.7% against seve
275                              The oral infant rotavirus vaccine, Rotarix, was introduced in England an
276 ntries that were among the early adopters of rotavirus vaccine.
277                      Despite successes, oral rotavirus vaccines are less effective in developing coun
278                                              Rotavirus vaccines are now globally recommended by the W
279                            Implementation of rotavirus vaccines has substantially decreased hospitali
280                                          Two rotavirus vaccines have been licensed in >100 countries
281 level of protection detected for the current rotavirus vaccines in low-income versus high-income sett
282 t lower effectiveness and waning immunity of rotavirus vaccines in resource-poor populations.
283 oncerns remain about the performance of oral rotavirus vaccines in these challenging settings.
284                        Efficacy of live oral rotavirus vaccines is reduced in low-income compared wit
285                   Parenteral non-replicating rotavirus vaccines might offer benefits over oral vaccin
286                                              Rotavirus vaccines were initially introduced in Australi
287                                   In 2006, 2 rotavirus vaccines were licensed.
288 strain replacement after the introduction of rotavirus vaccines, particularly in developing countries
289                                          Two rotavirus vaccines, Rotarix (RV1) and RotaTeq (RV5), wer
290  protection provided by live attenuated oral rotavirus vaccines.
291 s vaccination coverage using controls from a rotavirus VE test-negative case-control study and to exa
292 nt and cost-effective approach to estimating rotavirus VE through case-control studies.
293 mation of the spatiotemporal distribution of rotavirus VP7 (G) and VP4 (P) genotypes have shown evide
294                       During entry of rhesus rotavirus, VP8* interacts with cell surface gangliosides
295                                     Overall, rotavirus was detected in 5.5% (408/7440) of diarrheal s
296                                              Rotavirus was detected in 75 (28%) of 266 fecal samples
297                                Historically, rotavirus was the most common cause of severe disease in
298 To predict the proportion of diarrhea due to rotavirus, we constructed a multiple linear regression m
299 ed in 75 (28%) of 266 fecal samples and P[8] rotaviruses were found to be the predominant genotype.
300            In this study, new variants of G9 rotaviruses were identified in two children with diarrhe

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