ライフサイエンスコーパス: round cell

1 large multipolar cells and a group of small round cells.

2 Rac1 interfered with the ability of ExoT to round cells.

3 yer generates rapid shape transformations in rounded cells.

4 hout MreB exhibit an instability that favors rounded cells.

5 in severely stunted plants composed of small rounded cells.

6 l cell morphology, resulting in larger, more rounded cells.

7 s in this pathway lead to bent, branched, or round cells [2].

8 ) were included: 23 dendritic cell (40%), 21 round cell (37%), 2 dermal nests (4%), 2 combined (4%),

9 ic lbc and p115, increased the percentage of rounded cells 4-5-fold, and this was inhibited by C3.

10 of endothelial markers and the formation of round cells, a morphologic change characteristic of EHT.

11 atum basale, having one layer of columnar to round cells, a relatively flattened stratum spinosum and

12 y; pagetoid infiltration of dendritic and/or round cells; a nonspecific architectural pattern at the

13 Deleting these genes results in round cells and diminishes bud elongation in mutants tha

14 ar behavior, shifting back and forth between round cells and multibodied proplatelets.

15 hat VASP was localized at the cell cortex in round cells and redistributed to focal adhesions at the

16 ell division, resulting in plants with small rounded cells and fewer cells.

17 -differentiated/dedifferentiated, (b) myxoid/round cell, and (c) pleomorphic, based on morphologic fe

18 dedifferentiated, 144 (18%) myxoid, 81 (10%) round cell, and 64 (8%) pleomorphic histology.

19 ls, stellate cells, giant cells, radiate (or round) cells, and a variety of small cells.

20 0023, caused bulged cell poles, formation of rounded cells, and defects in polar localization of Wag3

21 Consequently, the rounded cells are suddenly presented with a several-fold

22 ic cells (especially roundish); and atypical round cells at the DEJ.

23 del, cell types strongly sort by shape, with round cells at the top of the colony and rod cells domin

24 read underwent osteogenesis, while unspread, round cells became adipocytes.

25 ng basal cells, with almost all cells having round cell bodies similar to rodent globose basal cells.

26 They have rounded cell bodies, have prominent nucleoli, attach poo

27 med long thin processes that extended from a round cell body.

28 four primary dendrites, a thick axon, and a rounded cell body and likely are made up of multiple typ

29 d mixed colonies consisting of diffuse small round cells characteristic of immature NK cells and othe

30 morphologic classification: dendritic cell, round cell, dermal nest, combined, and nonclassifiable t

31 esylation, prevented lovastatin-induced cell rounding, cell detachment and apoptosis, and stimulated

32 of FAK and AKT, sarcomeric disassembly, cell rounding, cell detachment from underlying matrix, and mo

33 In a multivariate analysis, only round-cell differentiation and low expression of p27 ind

34 posarcomas, even in neoplasms with few or no round-cell differentiation.

35 ells undergo the normal processes of mitotic rounding, cell elongation, polar ruffling, furrow ingres

36 Large, round cells expressing bone morphogenetic protein recept

37 en and thicken uniformly, with the resultant rounded cell finally bursting.

38 s, YW_SL and LL_RQ, severely impaired single-round cell-free infectivity 10-fold or more relative to

39 re smaller size (HR = 0.7, P = 0.01), myxoid/round cell histologic subtype (HR = 0.3, P = 0.03) and t

40 mutant failed to completely demonstrate the round-cell histology that is seen in both Ewing's tumor

41 sed by coelomocytes in the nephridium and in round cells in the epidermis and in eggs.

42 d spermatids, and the appearance of numerous round cells in the epididymal lumen.

43 longated cells, it increased the motility of rounded cells in the tumor.

44 CD, SCs do not differentiate and appear as "rounded" cells in contact with axons.

45 se-activating protein-deficient form of ExoS rounded cells, indicating the ADP-ribosyltransferase dom

46 trol of division planes by the Min system in round cells is proposed.

47 Round cells lacking MinCDE were viable, but growth, morp

48 Myxoid round cell liposarcoma (MRCLS) is a common liposarcoma s

49 ces apoptosis in dedifferentiated and myxoid/round cell liposarcoma cell lines, but not in either adi

50 myogenic and with neural differentiation and round cell liposarcoma, respectively.

51 gnostic factor in the spectrum of myxoid and round-cell liposarcomas has not been examined.

52 Forty-seven cases of myxoid and round-cell liposarcomas were examined.

53 predicts the clinical behavior of myxoid and round-cell liposarcomas, even in neoplasms with few or n

54 phic liposarcomas) and PIK3CA (18% of myxoid/round-cell liposarcomas, or MRCs).

55 4d were ablated the embryo developed into a rounded cell mass covered with an epithelial cell sheet.

56 being associated with chronic sun damage and round cell melanoma with early age at onset and phototyp

57 Round cell melanomas were identified more often in the f

58 vels of YAP activation with smaller and more rounded cell morphologies were induced in hMSCs.

59 We introduce three complementary models of rounded cell morphologies with a prescribed excess surfa

60 ntiated, 9 (5%) with myxoid, and 4 (2%) with round cell morphology.

61 s with H218 or edg-3, but not edg-1, induces rounded cell morphology in the presence of serum, which

62 moeboid cells (poor collagenolytic activity, rounded cell morphology), were 5-fold more invasive in t

63 fficient to predict the location of blebs in rounded cells moving in a highly resistive environment.

64 Synovial sarcomas, round-cell/myxoid liposarcomas, clear-cell sarcomas and

65 anced or metastatic dedifferentiated, myxoid/round cell, or pleomorphic LPS incurable by surgery or r

66 The mutant virus induced a round-cell phenotype that accumulated in the G(2)/M comp

67 hibitor of viral DNA synthesis increased the round-cell phenotype.

68 , accompanied by a morphological change to a rounded cell phenotype and down-regulation of vimentin,

69 o maintain a straight sidewall and uniformly rounded cell poles.

70 Accumulation of rounded cells positive for MC markers close to the vesse

71 sarcomas, all high grade with pleomorphic or round cells rather than spindle cells, contained an unid

72 Regardless of their fate in vivo, all the round cells remained negative for smooth muscle markers,

73 e describe a mechanism through which coccal (round) cells rise to the upper surface of a community, l

74 vision planes, we investigated its role in a round-cell rodA mutant.

75 ficiently induced Ewing's sarcoma-like small round cell sarcoma formation by these cells.

76 Ewing sarcoma is a primitive round cell sarcoma with a peak incidence in adolescence

77 improved clinical outcomes in patients with round cell sarcomas.

78 n rhabdomyosarcoma, Ewing sarcoma, and other round cell sarcomas.

79 newly diagnosed and refractory or recurrent round cell sarcomas.

80 s-RbAp48-hi) leads to cell size reduction, a rounded cell shape, decreased cellular protrusions, and

81 focused around the nucleus and resulted in a rounded cell shape.

82 This release is accompanied by cell rounding, cell swelling, and reorganization of the actin

83 ltered cellular morphology, producing small, round cells that closely resemble Cdc42-/- cells.

84 lso contain a third kind of cell: very small round cells that rapidly self-renew.

85 midpoint, prior to the formation of enlarged round cells that showed evidence of compromised cellular

86 Vincristine converted discoid platelets to rounded cells that extended pseudopods when chilled and

87 rs form irregular double or triple layers of rounded cells that lack zonula adherens.

88 In vitro VIP-treated rNSCs changed from round cells to glia-like cells with processes that stain

89 r cell sarcoma (CCS), and desmoplastic small round cell tumor (DSRCT) are soft-tissue malignancies oc

90 Desmoplastic small round cell tumor (DSRCT) is a rare disease of children,

91 Desmoplastic small round cell tumor (DSRCT) is a rare sarcoma in adolescent

92 Desmoplastic small round cell tumor (DSRCT) is an abdominal malignancy in c

93 Desmoplastic small round cell tumor (DSRCT) is defined by a chimeric transc

94 s translocation underlies desmoplastic small round cell tumor (DSRCT), which is noted for the abundan

95 One patient (desmoplastic round cell tumor [DSRCT]) achieved a confirmed partial r

96 ch with hepatoblastoma or desmoplastic small round cell tumor achieved a partial response; eight pati

97 leven of 14 patients with desmoplastic small round cell tumor had primary abdominal involvement.

98 ectodermal tumor, and one desmoplastic small round cell tumor).

99 eristic of intraabdominal desmoplastic small round cell tumor.

100 1 found in the aggressive desmoplastic small round cell tumor.

101 osarcoma protuberans, and desmoplastic small round cell tumor; one sarcoma predisposition syndrome wi

102 descriptively designated desmoplastic small round-cell tumor (DSRCT) are a distinctive, rare, poorly

103 ilable in vitro model for Desmoplastic Small Round Cell Tumors (DSRCT), while having only minor effec

104 ng family tumors (EFT) or desmoplastic small round cell tumors (DSRCT).

105 the diagnosis and therapy of malignant small round cell tumors that affect children, particularly in

106 9 osteosarcomas, 35 of 37 desmoplastic small round cell tumors, 2 of 3 synovial sarcomas, 4 of 4 leio

107 alveolar rhabdomyosarcomas, two desmoplastic round cell tumors, and one each of infantile fibrosarcom

108 euroectodermal tumors and desmoplastic small round cell tumors.

109 omosomal translocation in desmoplastic small round-cell tumors (DSRCT), a rare but aggressive soft ti

110 ated tumors occurring in young people (small round-cell tumors).

111 s sarcoma family of tumours (ESFT) are small round cell tumours characterized by the non-random EWS-E

112 utants lacking CBK1 form large aggregates of round cells under all growth conditions and lack the abi

113 teration in Epa polysaccharide content, more round cells versus oval cells with OG1RF, decreased biof

114 The round cells were characteristic of an abortive viral inf

115 Spindle and round cells were loosely distributed with increased part

116 Sparse round cells were observed 21 days after plating onto dee

117 It was observed that when rounded cells were injected with either the integrin pep

118 e/Dead viability stain revealed that >90% of rounded cells were viable.

119 ation showed a clear change of morphology to round cells, which accompanies detachment from the surfa

120 r, cells depleted of Rhb1 arrested as small, round cells with 1N DNA content, arrested more quickly i

121 In round cells with a long axis, such as those undergoing c

122 ) of neuroectodermal origin formed by small, round cells with limited neural differentiation, which a

123 toskeleton, since rho3 mutants exhibit large rounded cells with an aberrant actin cytoskeleton.

124 crease in the number of multinucleate cells, rounded cells with condensed ball-like nuclei, and cells

125 Wild type HA-p190 induced a phenotype of rounded cells with long, beaded extensions similar to th