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1 arge lesion of the sensorimotor cortex, some rubrospinal and reticulospinal neurons are labeled with
2                                              Rubrospinal and reticulospinal tract axons also did not
3  descending brainstem pathways including the rubrospinal and reticulospinal tracts, or into the L5 do
4 etely abolished, whereas others, such as the rubrospinal and vestibuospinal tracts, are only partiall
5  cord bilaterally that remove corticospinal, rubrospinal, and cerulospinal projections.
6 pinal tracts (i.e., lateral vestibulospinal, rubrospinal, and corticospinal).
7 ast grafts, including dorsal column sensory, rubrospinal, and nociceptive axons.
8 y found full remyelination of spared, intact rubrospinal axons caudal to the lesion in chronic mouse
9 erogradely labeled, intact corticospinal and rubrospinal axons throughout the extent of the lesion.
10 hat for comparable growth of raphespinal and rubrospinal axons.
11 ures of locomotion, and affect plasticity of rubrospinal circuitry known to support adaptive behavior
12                                              Rubrospinal connections were assessed following Fluoro-G
13 ng NgR, regeneration of some raphespinal and rubrospinal fibers does occur.
14                                              Rubrospinal motoneurons (RSMN) represent a population of
15                                              Rubrospinal motoneurons (RSMN) represent a population of
16 g, we show that innervation of cerebellum by rubrospinal neuron collaterals is remarkably selective f
17 rment, exacerbated lesion pathology, greater rubrospinal neuron loss, increased intraspinal T-cell ac
18 prised of feedback collaterals from premotor rubrospinal neurons can directly modulate IN output inde
19 eled neurons in the turtle were used to fill rubrospinal neurons in 150-200-microm-thick fixed sectio
20 ellular distribution of cerebellar inputs on rubrospinal neurons is similar between the rat and turtl
21 ggest that the basic dendritic morphology of rubrospinal neurons may have been established early in p
22 ures of the soma and dendritic morphology of rubrospinal neurons that receive cortical input, as in r
23 n was also examined after central axotomy of rubrospinal neurons, which constitutively show alpha-int
24 e functional and anatomical integrity of the rubrospinal pathway also was analyzed using the inclined
25 natally in cats, nothing is known about when rubrospinal projections could support motor functions or
26 extensive sprouting of intact rubrofugal and rubrospinal projections with the emergence of a de novo
27 xpressed by neurons of the corticospinal and rubrospinal projections, and by intrinsic neurons of the
28 1, supporting our hypothesis of preferential rubrospinal rather than corticospinal control for early
29 MC neurons were identified antidromically as rubrospinal (RMC-spinal) cells by stimulation of the con
30                        The results show that rubrospinal soma size is slightly larger in the rat than
31 he development of the motor functions of the rubrospinal system and the corticospinal system, the oth
32                  We show that the developing rubrospinal system competes with the corticospinal syste
33 wo systems helps predict the response of the rubrospinal system following corticospinal system develo
34 established that, as in the rat, the hamster rubrospinal system is essentially crossed and that injur
35 he cat to investigate whether the developing rubrospinal system is shaped by activity-dependent inter
36     Our findings suggest that the developing rubrospinal system is under activity-dependent regulatio
37                        The corticospinal and rubrospinal systems function in skilled movement control
38 ons between the developing corticospinal and rubrospinal systems, two key systems for skilled limb mo
39 r functions and, in turn, if the red nucleus/rubrospinal tract (RN/RST) compensates for developmental
40                     The red nucleus (RN) and rubrospinal tract (RST) are important for forelimb motor
41   In the present study, we found that spared rubrospinal tract (RST) axons of passage traced with act
42 ved neurotrophic factor (BDNF) would promote rubrospinal tract (RST) regeneration in adult rats.
43 nt, SKP-SC-transplanted rats showed enhanced rubrospinal tract (RST) sparing/plasticity in the gray m
44 n establishing the red nucleus motor map and rubrospinal tract connections.
45                                 Further, the rubrospinal tract is critical in the normal cat during s
46          The red nucleus (RN), source of the rubrospinal tract, has been implicated in the production
47                                        After rubrospinal tractotomy, alpha-internexin immunoreactivit
48 f descending motor tracts (corticospinal and rubrospinal) was analyzed by spinal surface recording el

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