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1 lack a gut (Porifera and Placozoa) or have a sac-like gut (Ctenophora and Cnidaria) and that a throug
3 The perception that ctenophores possess a sac-like blind gut with only one major opening remains a
4 mited growth conditions, sulfur acclimation (sac) mutants, which are more severely defective for accl
10 : a dense, thick and downturned rostrum; air sac fossae; cranial asymmetry; and exceptionally broad m
13 custom-built fast valve connected to the air sac system, we achieved partial or total silencing of sp
21 model, termed the post-implantation amniotic sac embryoid (PASE), that recapitulates multiple post-im
22 model, termed the post-implantation amniotic sac embryoid, to recapitulate early embryogenic events o
23 ial to embryogenesis and pregnancy, amniotic sac development in humans remains poorly understood.
25 UC 0.857, 95% CI: 0.755-0.928, p<0.0001) and sac-to-neck ratio (AUC 0.817, 95% CI: 0.708-0.898, p<0.0
28 r and volume of an abdominal aortic aneurysm sac can be used for temporal monitoring after endovascul
29 ine; volume of the abdominal aortic aneurysm sac; and volume from the lowest renal artery to the aort
32 showed the strongest indicators for aneurysm sac enlargement were complex IMA-LA type II endoleak (od
34 pe II endoleaks with an increase in aneurysm sac size and in patients with type I or III endoleaks.
36 as mainly attributable to secondary aneurysm sac rupture (13 deaths [7%] in EVAR vs two [1%] in open
37 ree of the 56 patients (41%) showed aneurysm sac enlargement during follow-up (mean follow-up, 3.0 ye
39 largest cross-sectional area of the aneurysm sac was measured using a curved multiplanar reconstructi
40 rigin of the IMA in relation to the aneurysm sac, diameter of the IMA, the cross-sectional area of th
43 supports the coil mass inside the aneurysmal sac, and furthermore, has an effect on local hemodynamic
45 crovesicles are heterogeneous membrane-bound sacs that are shed from the surfaces of tumor cells into
49 adhesive matrices placed in the conjunctival sac can enhance drug delivery by increasing precorneal r
51 50 mul of sterile saline to the lower cul-de-sac of each eye and using capillary action microcaps to
54 vs extracapsular), ascites beyond the cul-de-sac, peritoneal implants, ipsilateral pleural effusion,
55 , rebounding off the leaflet into the cul-de-sac, was noted in 82% of the obstructed HCM, 9% of nonob
57 sults, but rather than being climatic cul-de-sacs, many mountain streams appear poised to be redoubts
59 n, namely in pollen tube guidance and embryo sac fertilization, pathogen defense, and responses to ab
62 most similar to the early aposporous embryo sac transcriptome when comparing known functional annota
67 nuclei at the stage of four-nucleate embryo sac and delay in the progression of embryo development,
69 stically established in the syncytial embryo sac by spatially restricted CKI1 expression, followed by
71 iosperms, double fertilization of the embryo sac initiates the development of the embryo and the endo
74 unction is to bear and to nurture the embryo sac/female gametophyte and pollen, in which the egg and
76 ies (ROS) accumulation in anthers and embryo sacs, as evidenced by nitroblue tetrazolium staining.
77 th undivided nuclei, early aposporous embryo sacs containing two to four nuclei, and random groups of
81 ecedes fertilization, suggesting that embryo sacs sense the imminent arrival of pollen tubes and resp
82 pomeiosis, the formation of unreduced embryo sacs derived from nucellar cells of the ovary and, by pa
83 stoperative complications, type II endoleak, sac expansion, and additional interventions after EVAR.
87 ession in the developing mouse endolymphatic sac is required for acquisition of normal inner ear stru
90 epithelium, a precursor of the endolymphatic sac (ES) and duct (ED), which mediate endolymph homeosta
92 analysis of pre- and postnatal endolymphatic sacs demonstrates two types of differentiated cells.
96 n the lung occurs within alveoli, air-filled sacs composed of type 2 and type 1 epithelial cells (AEC
99 EP of less than 0.5 mL for showing no future sac volume enlargement were 33% (19 of 57), 100% (56 of
100 res are empty uterus and cervix, gestational sac in the anterior part of lower uterine segment with a
101 ere there is implantation of the gestational sac onto the anterior wall of the uterus at the site of
103 livery in a female with a viable gestational sac in the lower uterine segment and elevated B-Hcg leve
106 the chemical composition of individual honey sac contents with the most intensive and complex absorpt
107 mandarin (Citrus unshiu Marc.) nectar, honey sac content and honey were analyzed by FTIR-ATR spectros
108 conversion takes place directly in the honey sac; the average sugar content (w/w) was 17.93% (nectar)
109 anin in two > 160 Ma Jurassic cephalopod ink sacs and to confirm its chemical similarity to the ink o
111 ngth, in vitro binding to everted intestinal sacs and quantitative in vivo uptake; this data suggests
112 ion to include DCR revisions, acute lacrimal sac abscesses, nasolacrimal duct obstructions in patient
113 ipants had acute dacryocystitis and lacrimal sac abscess presenting within 2 weeks of onset, who were
114 ive either percutaneous drainage of lacrimal sac abscess followed by EN-DCR after the acute episode s
117 EN-DCR in acute dacryocystitis with lacrimal sac abscess results in faster resolution compared with s
119 endoleaks (45.1% vs 17.9%; P = .02) and late sac expansion (51.0% vs 21.4%; P = .01) and required mor
120 BRCA-mutant HGSOC, presence of PD in lesser sac (odds ratio [OR] = 2.40) and left upper quadrant (OR
123 tial for the separation of the jugular lymph sac from the cardinal vein and formation of the lymphove
124 elop via sprouting from venous-derived lymph sacs, vessels of lumbar and dorsal midline skin form via
128 We found that endothelial cells of lymph sacs at embryonic day (E)12.5 and tracheal lymphatics at
129 e veins to produce scattered primitive lymph sacs, from which most of the lymphatic vasculature is de
131 d posterolateral CDH covered by a membranous sac who had no features suggestive of BNAR or MOTA syndr
133 s and microvesicles (MV) are cell membranous sacs originating from multivesicular bodies and plasma m
136 of prominent camera-type eyes, paired nasal sacs, possible cranium and arcualia, W-shaped myomeres,
139 tively cover the bare bone around the opened sac, and provide a similar or even better clinical outco
142 statte, Yunnan Province, China, is an ovoid, sac-like metazoan that bears single-element spines on it
146 aphragm development with dilated pericardial sacs and failure of yolk sac remodeling suggestive of ca
156 transplanted either into an isolated venous sac made from lumbar vein or into the portal vein of syn
158 rafted and functioned in the isolated venous sac with ability to restore euglycemia in diabetic rats.
159 luated in animals whether an isolated venous sac would support survival of transplanted islets, along
160 rity and vascular organization of the venous sac was determined by studies of the local microcirculat
162 to float, limits the inflation of his vocal sac, and consequently reduces signal conspicuousness in
163 ysm three dimensions and volume, neck width, sac-to-neck ratio, initial result of embolization, numbe
165 hich revealed a 5-cm tumor that was 95% yolk sac tumor and 5% embryonal carcinoma, and retroperitonea
172 xicity included pericardial, ocular and yolk sac edema, nondepleted yolk, spinal curvature, tail malf
173 an ontogenic process of blood cell and yolk sac endothelial maturation that is required to display f
175 e earlier, that displayed labyrinth and yolk sac-specific defects, but our findings extend those obse
176 ein is a rodent-specific, placenta- and yolk sac-specific member of the tristetraprolin (TTP) family
179 ecent paradigm shift that microglia are yolk sac-derived, not hematopoietic-derived, is reshaping our
185 d angiogenesis and apoptosis in the cKO yolk sac mesoderm, but also restored the epithelial defects o
186 levels of VEGFA are observed in the cKO yolk sac, suggesting a cause for the angiogenesis defects.
188 states characteristic of the definitive yolk sac, HSCs undergoing specification, and definitive HSCs.
190 Several of these mutants also display yolk sac vascular defects, suggesting a role for thrombin sig
192 r/colony-forming cells of the embryonic yolk sac (YS), which are endowed with megakaryocytic potentia
194 lobin was co-expressed in the embryonic yolk sac, but not in the fetal liver; and wild-type beta-glob
195 ification in the dorsal aorta, enhanced yolk sac hematopoiesis, and exuberant cardiac blood island fo
196 ion were confined to the YY1-expressing yolk sac mesoderm indicating that loss of YY1 in the visceral
197 physiologically expressed in the fetal yolk sac, a tissue derived from the extraembryonic endoderm (
199 the allantois that are unavailable for yolk sac or dorsal aorta, and review how this system has been
200 e-resident macrophages are derived from yolk sac erythromyeloid progenitors and fetal liver progenito
201 Resident macrophages are derived from yolk sac precursors and seed the liver during embryogenesis.
202 progenitor pools, microglia arise from yolk sac progenitors and are widely considered to be equivale
203 rly embryogenesis, microglia arise from yolk sac progenitors that populate the developing central ner
204 ively self-renew and can be seeded from yolk sac/foetal liver progenitors with little input from mono
206 SC-derived haematopoiesis, and identify yolk sac EMPs as a common origin for tissue macrophages.
208 ed a cardiac transcriptional program in yolk sac endothelium, leading to the emergence of CD31+Pdgfra
210 ability to roll and adhere on inflamed yolk sac vessels during late fetal development, whereas at ea
211 hesion, and extravasation from inflamed yolk sac vessels is apparent late in development, but that be
212 ifferentiating mouse ES cells and mouse yolk sac cultures, addition of Indian Hh ligand increased hem
215 ge tracing, we identify a first wave of yolk sac (YS)-derived primitive myeloid progenitors that seed
216 e tracing revealed that the majority of yolk sac and many adult hematopoietic cells derive from Mesp1
217 tify, in the fetal liver, a sequence of yolk sac EMP-derived and HSC-derived haematopoiesis, and iden
219 dilated pericardial sacs and failure of yolk sac remodeling suggestive of cardiovascular failure.
220 xican-born mothers had a higher risk of yolk sac tumors (HR, 1.46; 95% CI, 0.99-2.17), while children
222 Runx1 is essential for the formation of yolk sac-derived erythroid/myeloid progenitors (EMPs) and hem
223 pulp macrophages, a discrete subset of yolk sac-derived macrophages, were found to be altered in SMA
224 f target transcripts in placenta and/or yolk sac, and that some of these would be important for femal
226 ds to vascular defects in the placenta, yolk sac, and embryo proper, as well as abnormal neural tube
227 nd in other embryonic niches (placenta, yolk sac, and extraembryonic vessels), attempts to detect the
229 different ontogenetic origins: prenatal yolk sac-derived Kupffer cells and peripheral blood monocyte-
230 2(+) macrophages derived from primitive yolk sac, recombination activating gene 1(+) lymphomyeloid, a
233 ciated with worse outcome, whereas pure yolk sac tumor (YST) was associated with better outcome, alth
235 e exocoelomic cavity, and the secondary yolk sac function together as a physiological equivalent.
236 cies indicates that the human secondary yolk sac likely performs key functions early in development,
244 everal embryonic regions, including the yolk sac and dorsal aorta, that undergoes vasculogenesis, the
245 originate during embryogenesis from the yolk sac and enter the CNS quite early (embryonic day 9.5-10
246 f cancer cells after injection into the yolk sac and extravasation of cancer cells into tissues from
248 sualized at embryonic day (E)9.0 in the yolk sac and neuroectoderm; 2) at E10.5, CX3CR1 single-positi
249 from progenitor cells generated in the yolk sac and of 'passenger' or 'transitory' myeloid cells tha
251 sive arterial morphogenesis both in the yolk sac and the embryo proper and disrupted arterial-venous
252 ad markedly deformed vasculature of the yolk sac and the embryo, as well as poorly looped hearts with
253 demonstrate that DPFCs originate in the yolk sac and then rapidly migrate to other extra- and intraem
255 ial in vitro have been described in the yolk sac before emergence of HSCs, and fetal macrophages can
256 Primitive hematopoiesis occurs in the yolk sac blood islands during vertebrate embryogenesis, where
259 GW182, is selectively expressed in the yolk sac endoderm and that gene trap disruption of GW182 lead
260 stricted potential originating from the yolk sac even before the emergence of the first hematopoietic
262 ve paracrine signal, originating in the yolk sac mesoderm, is required to promote normal visceral end
265 (2)(1)(0)Po also accumulates in the yolk sac of the embryo and in the fetal and placental tissues
266 esins can be carefully implanted in the yolk sac of zebrafish embryos and display excellent biocompat
268 he blastoderm begins to spread over the yolk sac, a process involving coordinated epithelial surface
269 p adjacent to blood vessel walls in the yolk sac, aorta-gonad-mesonephros region, embryonic liver, an
270 ythro-myeloid progenitors (EMPs) in the yolk sac, but it decreased the expression of alpha4-integrin
273 identifying the earliest stages in the yolk sac, throughout embryonic development and in all adult t
281 C-(57)CoB12 accumulated in the visceral yolk sac of KO mice where megalin is expressed and provides a
285 or dysplasia and malignant tumors, with yolk sac tumors and embryonal carcinomas positive for alpha-f
286 ave aberrant vasculogenesis in embryos, yolk sacs and placentas, and die between embryonic day 10.5 a
289 ck-out mice: no mature large vessels in yolk sacs, defective angiogenesis in the brain and intersomit
291 (RNA-seq) data for the human and murine yolk sacs and compare those data with data for the chicken.
293 that develop during organogenesis from yolk-sac erythro-myeloid progenitors (EMPs) distinct from hae
295 This study demonstrates that later yolk-sac Chinook larvae (before exogenous feeding) are expose
296 ow that the previously reported lack of yolk-sac hematopoiesis and vascular development in Ldb1(-/-)
298 macrophages are derived from primitive yolk-sac hematopoietic progenitors and exhibit hallmarks of M
299 Genetic fate mapping revealed that yolk-sac and fetal monocyte progenitors gave rise to the majo
300 ctivity in mammalian development is the yolk-sac blood island, which originates from the hemangioblas
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