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1 skim milk and 50 g fat from high-oleic acid safflower and canola oils (monounsaturated fatty acid; M
4 ysis demonstrates that natural pigments from safflower are more stable in sugar-based NADES than in w
7 ch: an extract of Carthamus oxyacantha (wild safflower), containing an array of spiro compounds, and
8 category) from other vegetable oils (canola, safflower, corn, peanut, seeds, grapeseed, palm, linseed
9 bsolute and relative sensitivities of 2pg of safflower DNA ( approximately 1.4 DNA copies) and 0.1% o
11 ( approximately 1.4 DNA copies) and 0.1% of safflower in saffron (Crocus sativus L.), respectively.
12 so proposed in the range of 0.1-20% (w/w) of safflower in saffron, which was successfully validated a
13 A; n-9 and n-3 rich), 3) a blend of corn and safflower oil (25:75) (CornSaff; n-6 rich), 4) a blend o
14 H versus TCA cycle flux was decreased in the safflower oil (43 +/- 8%) versus the control (73 +/- 8%,
16 ary oils, conjugated linoleic acid (CLA) and safflower oil (SAF), on body weight and composition in o
17 eatment with 4 g/d of ethyl esters of either safflower oil (SAF; control), eicosapentaenoic acid (EPA
18 oils (no supplementation, JO, fish oil [FO], safflower oil [SO], and arachidonic acid [AA]) were fed
19 were consistent with complete absorption of safflower oil and the nonabsorbability of olestra and ca
20 sumption of triacylglycerol-esterified LA in safflower oil did not increase plasma concentrations of
24 in-stimulated glucose disposal attributed to safflower oil feeding was a consequence of reduced glyco
25 iglyceride (TG) content was increased in the safflower oil group (7.3 +/- 0.8 micromol/g) compared wi
26 n insulin-stimulated glucose disposal in the safflower oil group was associated with a lower rate of
29 trol diet or isocaloric diets containing 27% safflower oil or 27, 13.5, and 8% menhaden fish oil.
30 were fed isocaloric diets containing either safflower oil or fish oil for 2 weeks before the start o
31 When rats were fed a diet containing 10% safflower oil or menhaden fish oil, the level of hepatic
32 igh-fat diets containing 27% fat from either safflower oil or safflower oil with an 8% fish oil repla
34 PPAR-alpha null mice fed the fish oil diet, safflower oil plus fish oil, hepatic insulin resistance
35 tion period a salad dressing containing 21 g safflower oil providing 16 g LA/d was added to the subje
36 s demonstrated that high dietary corn oil or safflower oil rich in omega-6 fatty acids increased the
38 work investigates the extraction process of safflower oil using pressurized ethanol, and compares th
40 .8 and 31.7 +/- 1.9 mg x kg(-1) x min(-1) in safflower oil versus control and fish oil groups, respec
42 taining 27% fat from either safflower oil or safflower oil with an 8% fish oil replacement (fish oil
43 ed by measuring absorption of well-absorbed (safflower oil) and poorly absorbed (olestra; calcium soa
44 (milk-derived) fat, but not polyunsaturated (safflower oil) fat, changes the conditions for microbial
45 tion [HGP], P < 0.002 vs. wild-type mice fed safflower oil), whereas in contrast, in PPAR-alpha null
46 were pair-fed on diets consisting of 1) 59% safflower oil, 2) 59% menhaden fish oil, or 3) 59% carbo
47 n was used to study the effects of palm oil, safflower oil, a mixture of fish and safflower oil, and
48 lm oil, safflower oil, a mixture of fish and safflower oil, and olive oil on postprandial apolipoprot
49 a different fat: menhaden oil, herring oil, safflower oil, canola oil, coconut oil, or cocoa butter.
50 19, and 125 +/- 14 nmol x g(-1) x min(-1) in safflower oil, fish oil, and control, respectively) was
51 ic acid from herring oil, linoleic acid from safflower oil, linolenic acid from canola oil, lauric ac
52 ocaloric diets modified to include corn oil, safflower oil, or DFO (doses ranging from 0.75% to 6.00%
53 d a 5% lipid diet containing lard, lard plus safflower oil, or lard plus linseed oil for 10 weeks.
54 Animals were either untreated, treated with safflower oil, or treated with DMH-11C in safflower oil.
55 loid precursor protein transgenic mice fed a safflower oil-enriched ("Bad") diet used to accelerate p
65 (CornSaff; n-6 rich), 4) a blend of flax and safflower oils (60:40) (FlaxSaff; n-6 and short-chain n-
68 ts in saffron, i.e., Crocus sativus stamens, safflower, turmeric, and gardenia were investigated.
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