ライフサイエンスコーパス: salicylic acid

1 nology to manufacture anti-acne devices with salicylic acid.

2 efense signals, hydrogen peroxide (H2O2) and salicylic acid.

3 w research questions about the plant hormone salicylic acid.

4 cluding those regulated by jasmonic acid and salicylic acid.

5 ES1 or the local or systemic accumulation of salicylic acid.

6 h reinstated signaling by the immune hormone salicylic acid.

7 icantly improve electrochemical responses of salicylic acid.

8 ic reaction was induced by additional acetyl-salicylic acid.

9 riggered immunity (ETI) but independent from salicylic acid.

10 hese were correlated with elevated levels of salicylic acid.

11 ing to whether they were administered acetyl salicylic acid.

12 s syringae and in response to treatment with salicylic acid.

13 y lithium borates fusion in combination with salicylic acid.

14 s sucrose, major amino acids, shikimate, and salicylic acid.

15 ber of soybean WRKY genes were responsive to salicylic acid.

16 acid (ABA), ethylene, jasmonic acid (JA) and salicylic acid.

17 g loading in the FPLA-salicylic acid and PCL-salicylic acid 3D printed patches was 0.4% w/w and 1.2%

18 plants were compromised in pathogen-induced salicylic acid accumulation and disease resistance.

19 ulescens on pathogen-induced ROS production, salicylic acid accumulation and downstream defence respo

20 al flagellin (flg22), and pathogen-inducible salicylic acid accumulation from PEN3 activity in extrac

21 Salicylic acid accumulation was triggered by high-growth

22 ns, high defence marker gene expression, and salicylic acid accumulation.

23 lutathione oxidation influences redox-driven salicylic acid accumulation.

24 We sought to determine whether acetyl salicylic acid administration at the time of development

25 the 970 patients with proven sepsis, acetyl salicylic acid administration was associated with a lowe

26 Propensity matching also found that acetyl salicylic acid administration was associated with increa

27 ted topical emollients, corticosteroids, and salicylic acid along with oral retinoids, methotrexate,

28 e compromised in immune responses induced by salicylic acid and by microbe-associated molecular patte

29 Hydrogen peroxide (H2 O2 ), salicylic acid and camalexin (a phytoalexin) levels were

30 erential expression of the genes involved in salicylic acid and defense mechanisms were the earliest

31 ant showed elevated basal as well as induced salicylic acid and ethylene accumulation.

32 it shows variations in water use efficiency, salicylic acid and hydrogen peroxide concentrations, pho

33 ngae, which correlated with higher levels of salicylic acid and hydrogen peroxide levels in pathogen-

34 precursor aminocyclopropanecarboxylic acid), salicylic acid and jasmonic acid (applied as methyl jasm

35 and herbivory-responsive pathways including salicylic acid and jasmonic acid also were up-regulated

36 MAMPs-induced signaling to regulate ALMT1 is salicylic acid and JASMONIC ACID RESISTANT1 (JAR1)/JASMO

37 Salicylic acid and jasmonic acid signaling pathway compo

38 ave elevated levels of two defense hormones: salicylic acid and jasmonic acid, and show increased res

39 s the action of the classic defense hormones salicylic acid and jasmonic acid.

40 NaCl, 1-aminocyclopropane-L-carboxylic acid, salicylic acid and methyl jasmonate) on the phytochemica

41 Drug loading in the FPLA-salicylic acid and PCL-salicylic acid 3D printed patches

42 nthesize the phytoalexin camalexin, Pip, and salicylic acid and primes plants for early defense gene

43 how the host integrates signals to activate salicylic acid and reactive oxygen pathways that orchest

44 dy shows a strong association between acetyl salicylic acid and survival in intensive care unit syste

45 ory effect to some therapies, such as acetyl salicylic acid and the statins, none of the currently ap

46 function as a receptor of the plant hormone salicylic acid and to mediate proteosomal degradation of

47 pathogens would induce flavones to decrease salicylic acid and, hence, increase susceptibility.

48 n, coumarin, sulfadymethoxine, warfarin, and salicylic acid) and HSA molecules.

49 confirmed that the cardol-cardanol mixture, salicylic acid, and aspirin, all of which lack key funct

50 tohormone signaling pathways, jasmonic acid, salicylic acid, and ethylene (ET), in TuMV-infected Arab

51 t hormones indoleacetic acid, jasmonic acid, salicylic acid, and gibberellic acid or by wounding, tem

52 amounts of active gibberellins, cytokinins, salicylic acid, and jasmonate compared with diploid indi

53 lasers and other agents including bleomycin, salicylic acid, and light-emitting diode have shown some

54 and ethylene and increases the production of salicylic acid, and these differential responses of plan

55 defense response via cell wall modification, salicylic acid- and jasmonic acid-dependent pathways, re

56 itogen-activated protein kinases, as well as salicylic acid- and jasmonic acid-mediated defense signa

57 SCMV showed no obvious correlation with the salicylic acid- and jasmonic acid-related defense signal

58 AtWRKY40 (DMG402007388) that was induced by salicylic acid; and a jasmonate ZIM-domain protein 1 (DM

59 Jasmonate and salicylic acid are positive regulators of leaf senescenc

60 sis in two half-reactions: activation of the salicylic acid as an acyl-adenylate and ligation onto th

61 By quantifying salicylic acid at the level of several nanograms in situ

62 This article explores the use of SDS-salicylic acid based micellar systems for their potentia

63 ere able to perform in situ determination of salicylic acid based on its electrocatalytic oxidation.

64 A salicylic acid-based poly(anhydride-ester) (SA-PAE) prov

65 ise for cancer therapy, and a novel class of salicylic acid-based STAT3 dimerization inhibitors that

66 nstances of protein functional change in the salicylic acid/benzoic acid/theobromine (SABATH) lineage

67 Salicylic acid biosynthesis at low temperature did not c

68 the SAGs, and cytokinin, abscisic acid, and salicylic acid biosynthesis genes.

69 efense amplification, positive regulation of salicylic acid biosynthesis, and priming to guarantee ef

70 quantify selected disinfection byproducts of salicylic acid, bisphenol A, gemfibrozil, naproxen, dicl

71 aprolactone (PCL) filaments were loaded with salicylic acid by hot melt extrusion (HME) (theoretical

72 .g., phenol, 4-chlorophenol, 2-chlorophenol, salicylic acid, catechol, maleic acid, oxalate, and urea

73 f programmed cell death, and accumulation of salicylic acid, closely mimicking phenotypes observed pr

74 Salicylic acid-coated magnetic nanoparticles were prepar

75 gnaling capacity but exhibit hyper-inducible salicylic acid concentrations and deregulated cell death

76 The salicylic acid content increased in low-light-treated sa

77 ato (Pst-AvrRpm1), which was associated with salicylic acid-dependent defense.

78 ants seems to be a combination of priming of salicylic acid-dependent defenses and reduced sensitivit

79 In addition, salicylic acid-dependent gene expression is similar in b

80 of powdery mildew infection, and neither the salicylic acid-dependent nor jasmonate-dependent pathway

81 ic acid (ABA) signaling and thereby suppress salicylic acid-dependent resistance.

82 oxygen species, which act by stimulating the salicylic acid-dependent signaling pathway of the plant

83 s, while an increased ratio of free to total salicylic acid did not convey elevated pathogenesis-rela

84 tal results demonstrated that the amounts of salicylic acid differed statistically in normal, phytoen

85 gibberellic acid, methyl jasmonic acid, and salicylic acid differentially regulate the stability of

86 redox status triggered by the immune signal salicylic acid does not compromise the circadian clock b

87 rystallized from the melt in the presence of salicylic acid either generated from aspirin decompositi

88 tic interactions between the stress hormones salicylic acid, ethylene, jasmonic acid, and abscisic ac

89 Moreover, increased levels of oxylipins and salicylic acid favored closure of stomata in response to

90 as a nose-shape mask by FDM 3DP, but the PCL-salicylic acid filament was not.

91 FPLA-salicylic acid filament was successfully printed as a no

92 FZL and the defense signaling molecule salicylic acid form a negative feedback loop in defense

93 delic acid, phthalic acid, benzoic acid, and salicylic acid) from aqueous samples.

94 ucoside (SAG), while UGT74F2 forms primarily salicylic acid glucose ester (SGE).

95 rmation of different products: UGT74F1 forms salicylic acid glucoside (SAG), while UGT74F2 forms prim

96 As one type of plant hormones, salicylic acid has recently been found to be one of pivo

97 -H carboxylation reaction of phenols to give salicylic acids has been developed.

98 dodecyl sulphate (SDS) and the phenolic acid salicylic acid have been studied at several temperatures

99 Salicylic acid helps the micellization of SDS, both by i

100 isic acid, or autophagy, but associates with salicylic acid homeostasis and signaling.

101 development, e.g., flagella, prophages, and salicylic acid hydroxylase.

102 tible host (efficient in the accumulation of salicylic acid).IMPORTANCE High diversity of within-host

103 onse syndrome, 2082 were administered acetyl salicylic acid in a 24-hr period around the time of syst

104 ents detected, suggesting a central role for salicylic acid in saul1 senescence and cell death.

105 devices for sensitively in situ detection of salicylic acid in tomato leaves with the sample volume o

106 of plant stress responses (jasmonic acid or salicylic acid) increased seedling MDA levels over 20-fo

107 the med14 mutation significantly suppresses salicylic acid-induced defense responses, alters transcr

108 th the YUC1-overexpression transgene and the salicylic acid induction deficient 2 (sid2) mutation, wh

109 at resistance to GPA is unaltered in an eds1 salicylic acid induction deficient2 (sid2) double mutant

110 resser of PR genes1 [npr1]) or biosynthesis (salicylic acid induction deficient2 [sid2]).

111 s non-expressor of pathogenesis related1 and salicylic acid induction-deficient2 increased hybrid see

112 luding the predominant isoform SUPPRESSOR OF SALICYLIC ACID INSENSITIVE2.

113 e background of the well-known suppressor of salicylic acid-insensitive2 (ssi2-2) mutant to confirm t

114 igh similarity to AtSSI2/FAB2 (Suppressor of Salicylic acid-Insensitivity2/Fatty Acid Biosynthesis2),

115 and is responsible for the incorporation of salicylic acid into the mycobactin siderophores.

116 ependent on, the foliar defense phytohormone salicylic acid is required to assemble a normal root mic

117 ts, suggesting that the hyperaccumulation of salicylic acid is unlikely to be responsible for dwarfis

118 ll three canonical defense hormone pathways (salicylic acid, jasmonate, and jasmonate/ethylene pathwa

119 ty and function in regulating the endogenous salicylic acid level.

120 Despite significantly increased salicylic acid levels and constitutive expression of the

121 Salicylic acid levels are similar in both types of plant

122 ecreased abscisic acid levels, and increased salicylic acid levels at the early stages of infection.

123 Intriguingly, elevated salicylic acid levels did not contribute to the HR-like

124 leaf damage and induced significantly higher salicylic acid levels in irAOX compared with wild-type p

125 Upon fungal challenge, salicylic acid levels were triggered in EV samples, whil

126 tutive hypersensitive response with elevated salicylic acid levels.

127 crease in plant hydrogen peroxide (H2O2) and salicylic acid levels.

128 ic stress that coincides with an increase in salicylic acid levels.

129 flexible personalised-shape anti-acne drug (salicylic acid) loaded devices was demonstrated by two d

130 a strain Witches' Broom protein11 suppresses salicylic acid-mediated defense responses and enhances t

131 In the present study we report salicylic acid-mediated differential elicitation of diff

132 ral folates amenable for enhancement through salicylic acid-mediated elicitation, thereby holding a g

133 ns in diverse biological processes including salicylic acid-mediated immune response.

134 o been reported that Ca(2+) signals suppress salicylic acid-mediated plant defense through AtSR1/CAMT

135 CPK5 signaling resulted in enhanced salicylic acid-mediated resistance to the bacterial path

136 self-modulator that can possibly prevent the salicylic acid-mediated runaway defense responses trigge

137 hydrogen peroxide, hypochlorite, dichromate, salicylic acid, melamine, and urea.

138 Salicylic acid modulates colonization of the root by spe

139 with the replacement of aspirin molecules by salicylic acid molecules in the crystal structure is com

140 The manifold effects of acetyl salicylic acid on human physiology potentially may benef

141 athogen-induced PME activity did not require salicylic acid or ethylene signaling, but was dependent

142 In contrast to salicylic acid or its functional analog benzothiadiazole

143 rating enzymes) prevents full priming of the salicylic acid pathway and associated resistance by high

144 ul1 senescence depends on the PAD4-dependent salicylic acid pathway but does not require NPR1 signali

145 Key DEGs including PALs and PR-10 in salicylic acid pathway involved in SAR were significantl

146 are known to induce plant resistance via the salicylic acid pathway, whereas biting-chewing herbivore

147 nd inhibited cat2-triggered induction of the salicylic acid pathway.

148 nce is not due to the hyperactivation of the salicylic acid pathway.

149 des specific synergism between cytokinin and salicylic acid pathways and previously undiscovered aspe

150 o suggest cross talk between the flavone and salicylic acid pathways in Arabidopsis; in this way, pat

151 a pivotal role in regulating senescence via salicylic acid pathways.

152 The study further demonstrates that bicyclic salicylic acid pharmacophores can be used to deliver PTP

153 Salicylic acid plays the role of a stabilizer, and gives

154 th the wild type, the increases in the total salicylic acid pool and camalexin accumulation were redu

155 In addition, salicylic acid pre-treatment enhances the AtRbohD-mediat

156 dified or absent in N. caerulescens, whereas salicylic acid production in response to infection was r

157 ion of the chimera cause the accumulation of salicylic acid, reduced growth, and eventually lead to p

158 nodulation, including phosphorous supply and salicylic acid-related defense response.

159 ome activity, resulting in the inhibition of salicylic acid-related immune responses.

160 ngae DC3000 as well as reduced activation of salicylic acid-responding genes.

161 evealed that various phytohormone (auxin and salicylic acid) response genes are significantly altered

162 sis-Related1 (NPR1), the master regulator of salicylic acid responses, leading to the accumulation of

163 eus and attenuates induction of jasmonic and salicylic acid-responsive genes.

164 StRDR1 was identified and shown to be salicylic acid-responsive.

165 dy was to evaluate the foliar application of salicylic acid (SA) (0.5, 1 and 2mM) or hydrogen peroxid

166 tSR1 is indispensable for the suppression of salicylic acid (SA) accumulation and disease resistance.

167 and assays for the hypersensitive response, salicylic acid (SA) accumulation and reactive oxygen spe

168 we demonstrate that SIZ1-mediated endogenous salicylic acid (SA) accumulation plays an important role

169 ition, and reactive oxygen species (ROS) and salicylic acid (SA) accumulation.

170 ormation of tyloses, whereas treatments with salicylic acid (SA) and 1-aminocyclopropane-1-carboxylic

171 dogenous levels of jasmonates (JAs), but not salicylic acid (SA) and abscisic acid (ABA) increased in

172 onse, ceramide synthesis, JA, ethylene (ET), salicylic acid (SA) and abscisic acid (ABA) signaling.

173 nity that promotes production of the hormone salicylic acid (SA) and activation of defense gene expre

174 lant immunity that promote the production of salicylic acid (SA) and affect the expression of SA-depe

175 t least in part, to negative cross talk with salicylic acid (SA) and gibberellic acid (GA) pathways.

176 In contrast, salicylic acid (SA) and H(2)O(2) treatments had no influ

177 ic plants showed increased concentrations of salicylic acid (SA) and higher levels of resistance to s

178 as to determine the effect of chitosan (CH), salicylic acid (SA) and hydrogen peroxide (H2O2) at diff

179 nduction of SAR requires the signal molecule salicylic acid (SA) and involves profound transcriptiona

180 to the antagonistic interactions between the salicylic acid (SA) and JA defense pathways, efforts to

181 the normally antagonistic defence hormones, salicylic acid (SA) and jasmonic acid (JA) associated wi

182 Expression analysis of salicylic acid (SA) and jasmonic acid (JA) biosynthesis

183 of DELLA on the archetypal defense hormones salicylic acid (SA) and jasmonic acid (JA) in Arabidopsi

184 Antagonism between the defense hormones salicylic acid (SA) and jasmonic acid (JA) plays a centr

185 syringae, ultraviolet-C (UV-C) irradiation, salicylic acid (SA) and jasmonic acid (JA) were investig

186 Here we show that the interconvertible salicylic acid (SA) and methylated SA (MeSA), well chara

187 mutant is associated with enhanced levels of salicylic acid (SA) and mRNA encoding the pathogenesis-r

188 the two immune-regulatory plant metabolites, salicylic acid (SA) and pipecolic acid (Pip), in the est

189 tress defense by the stress response hormone salicylic acid (SA) and the UPR, which is modulated by S

190 ) genes CBF1 and CBF2, and the repression of salicylic acid (SA) biosynthesis at warm temperature.

191 ts after inoculation reveal that several key salicylic acid (SA) biosynthesis genes are significantly

192 ion in the hybrids indicate decreases to the salicylic acid (SA) biosynthesis pathway and increases i

193 ant cells and causes a loss of ICS1-mediated salicylic acid (SA) biosynthesis.

194 We found that the plant immune hormone salicylic acid (SA) can trigger DNA damage in the absenc

195 After observing that salicylic acid (SA) enhanced the accumulation of folates

196 Salicylic acid (SA) has long been implicated in plant re

197 as been shown that the crystal nucleation of salicylic acid (SA) in different solvents becomes increa

198 is identified the increased concentration of salicylic acid (SA) in the SV-treated plants after patho

199 Feeding with benzoic acid (BA) or salicylic acid (SA) increased veratrole emission 2-fold,

200 Here, we provide experimental evidence that salicylic acid (SA) is a critical hormonal signal that r

201 Salicylic acid (SA) is a plant immune signal produced af

202 Salicylic acid (SA) is a signaling molecule utilized by

203 Salicylic acid (SA) is a small phenolic molecule that no

204 Salicylic acid (SA) is a small phenolic molecule with ho

205 Salicylic acid (SA) is an important plant hormone that i

206 Salicylic acid (SA) is central for the defense of plants

207 ignaling mediated by the phenolic metabolite salicylic acid (SA) is critical for the manifestation of

208 The plant hormone salicylic acid (SA) is essential for local defense and s

209 pression of many genes bear the signature of salicylic acid (SA) mediated regulation, the breadth of

210 thus resembling the antagonistic effects of salicylic acid (SA) on JA responses.

211 The effects of salicylic acid (SA) or acetylsalicylic acid (ASA) treatm

212 emically during SAR signaling and locally by salicylic acid (SA) or its functional analog, benzo 1,2,

213 pathway and simultaneous down-regulation of salicylic acid (SA) pathway in guard cells.

214 nctions of defense genes associated with the salicylic acid (SA) pathway, including ENHANCED DISEASE

215 lants recognize insect eggs and activate the salicylic acid (SA) pathway.

216 ough it is well known that the plant hormone salicylic acid (SA) plays an essential role in defense,

217 The plant hormone salicylic acid (SA) plays critical roles in plant defens

218 ochemical evidence supporting CATALASE2 as a salicylic acid (SA) receptor has finally emerged.

219 By contrast, under biological activation of salicylic acid (SA) signaling and hypersensitive PCD, Bi

220 crobe-associated molecular pattern-triggered salicylic acid (SA) signaling and infection-triggered et

221 ding of FB17 invokes abscisic acid (ABA) and salicylic acid (SA) signaling pathways to close light-ad

222 tochondrial succinate dehydrogenase (SDH) in salicylic acid (SA) signaling was analyzed using two mut

223 ent in jasmonic acid (JA), ethylene (ET) and salicylic acid (SA) signaling.

224 is mutants impaired in jasmonic acid (JA) or salicylic acid (SA) signaling.

225 In nox1 plants both salicylic acid (SA) synthesis and signalling were suppre

226 sochorismate synthase 1 (ICS1), required for salicylic acid (SA) synthesis, compromised gall formatio

227 provides controlled and sustained release of salicylic acid (SA) that locally resolves inflammation.

228 njugates both indole-3-acetic acid (IAA) and salicylic acid (SA) to modulate auxin and pathogen respo

229 lent pathogens, and elevated accumulation of salicylic acid (SA) upon infection.

230 Salicylic acid (SA), a hormone essential for defense aga

231 to result from antagonism between auxin and salicylic acid (SA), a major regulator of plant defenses

232 Salicylic acid (SA), an essential regulator of plant def

233 ation, represses accumulation of the hormone salicylic acid (SA), an established regulator of plant i

234 and systemically induces the accumulation of salicylic acid (SA), an important activator of defense,

235 xpression of tomato defence genes related to salicylic acid (SA), and TD itself strongly induced the

236 odiphosphate (MEcPP) and the defense hormone salicylic acid (SA), as well as the high MEcPP but SA de

237 that includes those mediated by the hormones salicylic acid (SA), ethylene (ET), jasmonic acid (JA),

238 signalling components and in particular with salicylic acid (SA), hydrogen peroxide (H2O2), 6-benzyla

239 ENE RESPONSE FACTOR 1 (ERF1)] clustered into salicylic acid (SA), jasmonic acid (JA), and ethylene (E

240 The plant hormones, salicylic acid (SA), jasmonic acid, and ethylene have em

241 ced by exogenous application of phytohormone salicylic acid (SA), methyl jasmonate (MeJA), phytopatho

242 xic acid (NA) and flumequine (FLU), but also salicylic acid (SA), natural organic matter (humic acid,

243 monas syringe pv. tomato (Pst) DC3000 in the salicylic acid (SA)- and nitric oxide (NO)-dependent pat

244 stance, biotrophic pathogens are resisted by salicylic acid (SA)- and reactive oxygen species (ROS)-d

245 nds in systemic acquired resistance (SAR), a salicylic acid (SA)-associated, broad-spectrum immune re

246 ses of Phi on Hpa infection was nullified in salicylic acid (SA)-defective plants (sid2-1, NahG) and

247 ED DISEASE SUSCEPTIBILITY1 (EDS1) to promote salicylic acid (SA)-dependent and SA-independent defense

248 ciated with changes in cell wall defence and salicylic acid (SA)-dependent gene expression.

249 endent formation of lesions on leaves due to salicylic acid (SA)-dependent PCD, revealing roles for m

250 endent formation of lesions on leaves due to salicylic acid (SA)-dependent PCD.

251 We propose that PRR2 contributes to salicylic acid (SA)-dependent responses when challenged

252 florescences mainly involves accumulation of salicylic acid (SA)-inducible defense genes (ZmNAC, ZmHS

253 d Arabidopsis (P(1)) were primed to activate salicylic acid (SA)-inducible defense genes and were mor

254 The expression levels of salicylic acid (SA)-inducible genes were higher, but tho

255 Plants deploy salicylic acid (SA)-mediated defences against biotrophs.

256 expression are associated with constitutive salicylic acid (SA)-mediated defense responses.

257 y been identified to play important roles in salicylic acid (SA)-mediated defense signaling.

258 usceptibility could be rescued by activating salicylic acid (SA)-mediated defense.

259 To determine this, decoding of the salicylic acid (SA)-mediated plant immunity signalling n

260 regulated by jasmonic acid (JA), whereas the salicylic acid (SA)-responsive pathogenesis-related gene

261 y cross-talk between jasmonic acid (JA)- and salicylic acid (SA)-signaling pathways.

262 signalling is associated with a decrease in salicylic acid (SA)-triggered immunity (SATI) in Arabido

263 r by the phytohormones jasmonic acid (JA) or salicylic acid (SA).

264 l, and the generation of the signal molecule salicylic acid (SA).

265 sitive feedback loop with the defense signal salicylic acid (SA).

266 accumulation of the key plant immune signal salicylic acid (SA).

267 and the stress-related hormones ethylene and salicylic acid (SA).

268 d that the interaction can be deregulated by salicylic acid (SA).

269 acid (AzA), glycerol-3-phosphate (G3P), and salicylic acid (SA).

270 ich are receptors for the signaling molecule salicylic acid (SA).

271 t on the accumulation of the defense hormone salicylic acid (SA).

272 iented approach that transforms a benzofuran salicylic acid scaffold into a highly potent (IC(50) = 3

273 For instance, salicylic acid showed positive correlations with species

274 affected in nrt2, which displays priming of salicylic acid signaling and concomitant irregular funct

275 d resistance response to Pst is dependent on salicylic acid signaling and not on jasmonic acid and et

276 Several salicylic acid signaling components, such as pathogenesi

277 f downstream responses mediated by jasmonate-salicylic acid signaling cross talk, is involved in the

278 transcripts of defense-related genes in the salicylic acid signaling pathway and enhanced disease re

279 piration and gas exchange, as well as better salicylic acid signaling.

280 roach, three strongest inhibitors namely are salicylic acid, tannic acid and trans-cinnamaldehyde hav

281 The ref8 mutant accumulates higher levels of salicylic acid than the wild type, but depletion of this

282 ng two bioactive molecules, either niacin or salicylic acid, to an omega-3 fatty acid.

283 ccelerated by up to threefold in response to salicylic acid treatment and challenges with mannitol.

284 pt level of OsGR3 was greatly increased with salicylic acid treatment but was not significantly affec

285 The effect of acetyl salicylic acid treatment on mortality of patients with s

286 de, low temperature, heat, abscisic acid and salicylic acid treatments.

287 licitations in the form of methyl jasmonate, salicylic acid, ultraviolet B light, and wounding, chose

288 analysis showed a 10.9% mortality for acetyl salicylic acid users and 17.2% mortality in the propensi

289 on of the primary metabolite chorismate into salicylic acid via isochorismate.

290 mation of micelles of SDS in the presence of salicylic acid was a thermodynamically spontaneous proce

291 saul1 mutants, and application of exogenous salicylic acid was indeed sufficient to trigger saul1 se

292 Moreover, we show that salicylic acid was not involved in the defense against P

293 Salicylic acid was readily halogenated, which is evident

294 their probability of being prescribed acetyl salicylic acid was undertaken.

295 , active cytokinins, active gibberellin, and salicylic acid were detected in the root tips of these p

296 Concentration of sinapic and salicylic acids were highest phenolic acids of pearl mil

297 Subsequently, substituted salicylic acids were prepared by deprotection of the est

298 Furthermore, salicylic acid, which is the major plant defense hormone

299 SNC1 protein accumulation is independent of salicylic acid, whose effects are often antagonized by A

300 a punched hole of 1.5mm diameter to release salicylic acid with minor influence on continuous growth