ライフサイエンスコーパス: salt sensitivity

1 fectors has provided important insights into salt sensitivity.

2 ression lowers basal BP and decreases Ang II/salt sensitivity.

3 ween the clades and substrate specificity or salt sensitivity.

4 eceptor gene in mice increases BP and causes salt sensitivity.

5 s extrarenal collectrin on BP regulation and salt sensitivity.

6 arly flowering, low fertility, and increased salt sensitivity.

7 on disturbs ion status that is manifested as salt sensitivity.

8 s qualitatively defective and exhibited some salt sensitivity.

9 sion of a deregulated version of CAX1 caused salt sensitivity.

10 enefits in the treatment of hypertension and salt sensitivity.

11 evels of Mn(2+) significantly decreased this salt sensitivity.

12 In some cases stress unmasks the salt sensitivity.

13 d animal studies suggest a genetic basis for salt sensitivity.

14 he pressure-natriuretic relation and lead to salt sensitivity.

15 alt and a transition from salt resistance to salt sensitivity.

16 nique spectrum of antimicrobial activity and salt sensitivity against bacteria commonly causing sepsi

17 tudy indicate that AHK5 positively regulates salt sensitivity and contributes to resistance to the ba

18 on, and polymorphisms at this site relate to salt sensitivity and low plasma renin activity (PRA).

19 The salt sensitivity and moderate processivity of the isolat

20 ignificant risk factor in the development of salt sensitivity and subsequent hypertension.

21 component of ion homeostasis correlated with salt sensitivity and tolerance.

22 leads to endothelial dysfunction, augmented salt sensitivity, and hypertension.

23 lectrin results in hypertension, exaggerated salt sensitivity, and impaired pressure natriuresis.

24 tion of stored carbon, gravitropic response, salt sensitivity, and specific susceptibility to the fun

25 ng two techniques used for the assessment of salt sensitivity as well as several studies attempting t

26 sitivity of sos3-1 and also causes increased salt sensitivity by itself.

27 Salt sensitivity can also be inherited and ongoing studi

28 Na(+) export pumps and as a result displays salt sensitivity comparable with wheat.

29 Bile salt sensitivity could not be attributed to a generalize

30 3.13-fold increased odds (1.80-5.43) of high salt-sensitivity during the high-sodium intervention.

31 ld increased odds (95% CI 2.05-6.11) of high salt-sensitivity during the low-sodium and a 3.13-fold i

32 ating in the Genetic Epidemiology Network of Salt Sensitivity (GenSalt) Study, blood pressure (BP) re

33 rats, the specific genes contributing to the salt sensitivity have not yet been determined.

34 SlZF2 enhanced salt sensitivity in Arabidopsis, whereas SlZF2 delayed s

35 Salt sensitivity in Hsd11b2.BKO mice was not caused by i

36 acy of mineral intake and protection against salt sensitivity in humans provides an important opportu

37 sis salt overly sensitive (sos) mutants, and salt sensitivity in the K(+) transport mutants akt1 (Ara

38 on at this residue, glc7-R260P, confers only salt sensitivity, indicating that the glycogen and salt

39 type 1 channels may contribute to increased salt sensitivity, inflammation, and end organ damage.

40 In many species, salt sensitivity is associated with the accumulation of

41 Salt sensitivity is characterized by an alteration of ki

42 Altogether, these data suggest that salt sensitivity is determined by intrarenal collectrin,

43 Thus, the complex syndrome of salt sensitivity may be a function of the endothelium, w

44 als exert their effect and (2) suggests that salt sensitivity might be an important marker for biolog

45 This report discusses the hypothesis that salt sensitivity need not be demonstrated exclusively by

46 Despite this discrepancy in salt sensitivity, NMR and CD data indicate that neither

47 and was Ca2+-dependent, consistent with the salt sensitivity observed in a phospholipid-independent

48 ndent ENA1 gene promoter and compensated the salt sensitivity of a mutant deficient in TCN1--a transc

49 ignificantly depressed pK(a) values, and the salt sensitivity of all histidine pK(a) values was subst

50 Salt sensitivity of arterial pressure (salt-sensitive hy

51 n users had lower blood pressure and reduced salt sensitivity of blood pressure (both P<0.001).

52 ysis to identify the genetic determinants of salt sensitivity of blood pressure (BP) in a large famil

53 Salt sensitivity of blood pressure (BP) is influenced by

54 This paper reviews the evidence that salt sensitivity of blood pressure is related both to th

55 e association between metabolic syndrome and salt sensitivity of blood pressure.

56 tly, independently, and inversely related to salt sensitivity of BP and may be particularly effective

57 These findings show a novel mechanism for salt sensitivity of BP and the significance of tubuloglo

58 Salt sensitivity of BP varies widely, but certain subgro

59 Salt sensitivity of BP was defined as mean BP change fro

60 4.1 might harbor functional variants for the salt sensitivity of BP.

61 thaliana orthologue, AtSLT1, also suppressed salt sensitivity of cnb delta but only when expressed wi

62 04), but not full-length protein, suppressed salt sensitivity of cnb1.

63 NtSLT1 partially suppressed the salt sensitivity of ena1-4 indicating that NtSLT1 has bo

64 of a mutant called enh1-1 that enhances the salt sensitivity of sos3-1 and also causes increased sal

65 nd to further characterize the magnitude and salt sensitivity of surface electrostatic interactions i

66 he multivariable-adjusted odds ratio of high salt sensitivity of systolic BP was 0.66 (95% CI: 0.49,

67 ied a central region that contributes to the salt sensitivity of TFB activity, and deleting residues

68 with a truncation fragment and the observed salt sensitivity of the assembly process are explained b

69 d DNA binding is responsible for the extreme salt sensitivity of the endonuclease activity of the enz

70 We have investigated the salt sensitivity of the hexagonal-to-cholesteric phase t

71 The low salt sensitivity of the mitochondrial DNA helicase is mi

72 alone that is responsible for the increased salt sensitivity of the sos2-2 ndpk2 double mutant.

73 We conclude that salt-sensitivity of blood pressure in essential hyperten

74 tantial amount of new information concerning salt-sensitivity of blood pressure in humans has appeare

75 ndings demonstrate a novel pathway mediating salt-sensitivity of blood pressure.

76 The observed weak salt-sensitivity of the ratio of specific and non-specif

77 to acquired forms of hypertension caused by salt sensitivity or diabetes mellitus.

78 l characterization of subunit stoichiometry, salt sensitivity, processivity, and stimulation by proli

79 se in nitric oxide are impaired or absent in salt sensitivity, promoting an increase in BP in these i

80 Their striking salt sensitivity reflects screening of weak, repulsive,

81 The second stage, characterized by salt-sensitivity, renal arteriolar disease, and blunted

82 d bile salt resistance, suggesting that bile salt sensitivity requires an active system for F pilin s

83 Suppression of salt sensitivity requires two ion transporters, the Gef1

84 Additionally, risk of salt sensitivity rose with increasing numbers of risk fa

85 On the basis of salt sensitivity studies carried out with various fragme

86 ndothelial surface layer as a contributor to salt sensitivity, the consequences of a perturbed endoth

87 High salt sensitivity was defined as a decrease in mean arter

88 Salt sensitivity was defined as BP responses to low- and

89 Bcl-2 suppression of cnb1Delta salt sensitivity was ENA1 (P-type ATPase gene)-dependent