ライフサイエンスコーパス: salubrinal

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1 plasmic reticulum (ER) stress, we identified salubrinal, a selective inhibitor of cellular complexes

2 A receptor blocker AP-5 or by treatment with salubrinal, a selective inhibitor of eukaryotic translat

3 Furthermore, treatment with salubrinal, a small chemical compound that specifically

4 Salubrinal also blocks eIF2alpha dephosphorylation media

5 Salubrinal also protected osteoblast development by upre

6 ype PC12 cells, activation of this module by salubrinal ameliorates the response to Abeta.

7 erference to knock down caspase-12 levels or salubrinal (an ER stress inhibitor) partially protected

8 y, we explored the underlying means by which salubrinal, an activator of eIF2alpha signaling, enhance

9 ly, treatment of alphaS transgenic mice with Salubrinal, an anti-ER stress compound that delays the o

10 More important, treatment with Salubrinal, an anti-ERS compound, significantly attenuat

11 Sublethal dosage of salubrinal, an eIF2alpha phosphatase inhibitor, increase

12 In contrast, salubrinal, an inhibitor of eIF2alpha-P dephosphorylatio

13 Salubrinal, an inhibitor of eIF2alpha-P phosphatase PP1c

14 Furthermore, salubrinal, an inhibitor of ER stress-induced apoptosis,

15 neous or sequential combination therapy with salubrinal, an inhibitor of GADD34-PP1C phosphatase comp

16 mutation was more affected by treatment with salubrinal, an inhibitor of the ER-stress pathway.

17 induction of the PERK-eIF2alpha pathway with salubrinal attenuated the inflammatory response to both

18 More broadly, salubrinal demonstrates the feasibility of selective pha

19 or increasing eIF2alpha phosphorylation with salubrinal did not affect Nrf2 elevation by H(2)O(2).

20 tcome in the surgery-induced ONFH model, and salubrinal improves ONFH symptoms by enhancing angiogene

21 he specific eIF2alpha phosphatase inhibitor, salubrinal, induces resistance against oxidative glutama

22 Salubrinal is a synthetic chemical that inhibits de-phos

23 with an inhibitor of eIF2alpha phosphatase, salubrinal, led to improved photoreceptor survival.

24 nd reduction of adult hemoglobin as possible salubrinal mechanisms.

25 ion of translational arrest and ER stress by salubrinal or of MAPK signaling pathways attenuate cytok

26 wn inhibitor of eIF2alpha dephosphorylation, salubrinal, revealed that elevated eIF2alpha phosphoryla

27 se 1 a (PP1a)-specific phosphatase inhibitor Salubrinal (SAL) synergized with TG to induce EBV lytic

28 We then determined that salubrinal selectively increased the number of actively

29 t the ER stress was associated with ONFH and salubrinal significantly improved ONFH-induced symptoms

30 demise by co-treatment with calpastatin and salubrinal suggests co-activation of the calpain and ER-

31 osphorylation with systemically administered salubrinal throughout hypoxia/reoxygenation exposure pre

32 Intriguingly, salubrinal treatment of differentiating human CD34(+) ce

33 urgery-induced animal model was employed and salubrinal was administered to evaluate the role of ER s

34 g eIF2alphaP signaling by the small molecule salubrinal, which inhibits dephosphorylation of eIF2alph

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