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1 the presence of Leishmania in the gut of the sand fly.
2 sruption of transmission by the phlebotomine sand fly.
3 rasite patches that govern infectiousness to sand flies.
4 y blood-feeding female Lutzomyia longipalpis sand flies.
5 an hosts by the bites of bloodsucking vector sand flies.
6 long-term reservoir of infection for vector sand flies.
7 g-term reservoir of infection back to vector sand flies.
8 ania mexicana-infected Lutzomyia longipalpis sand flies.
9 eficient mice challenged by bite of infected sand flies.
10 Leishmania major metacyclic promastigotes by sand flies.
11 vaccinated dogs that are also infectious to sand flies.
12 d are transmitted by bites from phlebotomine sand flies.
13 ckettsia were reported for the first time in sand flies.
14 Fabaceae family was detected in 94.7% of the sand flies.
15 ism of Leishmania transmissibility to vector sand flies.
16 nated papers was determined with wild-caught sand flies.
17 ansmitted to the vertebrate host by infected sand flies.
18 All patients infected sand flies.
19 cted long term when challenged with infected sand flies.
20 were used to coinfect Lutzomyia longipalpis sand flies.
21 following a natural challenge with infected sand flies.
22 st bite in volunteers exposed to colony-bred sand flies.
23 isceral leishmaniasis (VL) is transmitted by sand flies.
24 distribution reduces the expected number of sand flies acquiring parasites, it increases the infecti
27 phoglycans (lpg2-) were unable to survive in sand flies and macrophages, but retained the ability to
28 ternate between flagellated promastigotes in sand flies and nonflagellated amastigotes in mammals, ca
29 life cycle involving transmission by biting sand flies and replication within mammalian macrophage p
30 n of molecules present in the midgut of this sand fly and the transcripts potentially modulated by bl
31 rast to bloodsucking Nematocera (mosquitoes, sand flies, and black flies), appear to concentrate a go
32 is study, we show that salivary Ags from the sand fly, and specifically the LJM11 salivary protein, a
36 e, we demonstrate that gut microbes from the sand fly are egested into host skin alongside Leishmania
40 athogen, and the Leishmania that utilize the sand fly as a vehicle for transmission between mammalian
42 ave identified the first representative of a sand fly-associated flavivirus, Ecuador Paraiso Escondid
43 icantly more attractive to 50% of the female sand flies at the end of infection compared to before in
45 vade and exploit the innate host response to sand fly bite in order to establish and promote disease.
48 are introduced into mammalian skin through a sand fly bite, but different species cause distinct clin
52 te the durability and T(H)1 nature of DTH to sand fly bites in humans living in a cutaneous leishmani
59 replication of vesicular stomatitis virus in sand fly cells and resulted in strains that initially re
60 s, isolated from mosquitoes and phlebotomine sand flies collected in Brazil, Peru, the United States,
61 rior exposure of mice to bites of uninfected sand flies conferred powerful protection against Leishma
63 cted dogs and household spraying to kill the sand fly) could be effective, but have proven hard to ma
65 ration of immune homeostasis in phlebotomine sand flies during the growth of bacterial and Leishmania
66 profile of certain midgut transcripts in the sand fly during blood meal digestion and that this modul
68 greatly facilitate the understanding of the sand fly ecology, which would provide critical informati
72 rasites, it increases the infection load for sand flies feeding on a patch, increasing their potentia
74 cales provide the best fit with experimental sand fly feeding data, pointing to the importance of the
75 r organisms, <4% were identical to described sand fly genes, and 42% had no match to any database seq
81 have demonstrated that the activation of the sand fly immune system, via depletion of a single gene,
84 ngs suggest that the ecological diversity of sand fly in Sichuan and Henan may contribute to shaping
90 phoglycan-containing molecules in Leishmania-sand fly interactions were tested by using mutants speci
92 nfected mice to transmit parasites to vector sand flies, it was observed that following low-dose chal
93 udied plant feeding of Lutzomyia longipalpis sand flies, known vectors of Leishmania infantum/chagasi
94 fter transmission through the bite of female sand flies, Leishmania spp. can cause a broad spectrum o
95 ant for sand fly ecological adaptability and sand fly-Leishmania genetic co-variation could be helpfu
97 ries constructed from midgut tissue from the sand fly Lutzomyia longipalpis and analyzed the transcri
98 that salivary gland lysate of the New World sand fly Lutzomyia longipalpis markedly enhanced L. majo
99 istributed hematophagous insect vectors, the sand fly Lutzomyia longipalpis s.l., the mosquitoes Anop
100 n with Leishmania major is enhanced when the sand fly Lutzomyia longipalpis salivary peptide maxadila
101 isolated from salivary gland lysates of the sand fly Lutzomyia longipalpis, a vector of leishmaniasi
102 LJM11, an abundant salivary protein from the sand fly Lutzomyia longipalpis, belongs to the insect "y
107 w that the concentration of ROS increased in sand fly midguts after they fed on the insect pathogen S
108 gotes and procyclic LPG were able to bind to sand fly midguts in vitro whereas metacyclic parasites a
109 at Leishmania are transmitted exclusively by sand flies, none of the experimental models of leishmani
112 In this article, we investigate whether the sand fly Phlebotomus papatasi, known to produce a strong
115 e that prior exposure to bites of uninfected sand flies potentiates their ability to transmit infecti
116 e where it was discovered, was isolated from sand flies (Psathyromyia abonnenci, formerly Lutzomyia a
117 he blood or skin as a source of infection to sand flies remains unclear, and the possible effect of m
118 demonstrates a differential response of the sand fly ROS system to gut microbiota, an insect pathoge
120 experiments demonstrate that pre-exposure to sand fly saliva confers protection against leishmaniasis
122 mmune activation, oxidative stress, and anti-sand fly saliva IgG concentrations in dog sera with diff
124 e describe the duration and nature of DTH to sand fly saliva in humans from an endemic area of Mali.
129 the vertebrate host is also inoculated with sand fly saliva, which exerts powerful immunomodulatory
131 functional genomics approach to identify the sand fly salivary components that are responsible for th
133 as a vaccine expression system for LJM11, a sand fly salivary protein identified as a good vaccine c
134 umulative evidence that immunity to specific sand fly salivary proteins confers a significant level o
135 rmal immunization of mice with 500 ng of the sand fly salivary recombinant protein LJM11 (rLJM11) fro
136 ases that degrade the stomodeal valve of the sand fly; secretion of a neuropeptide that arrests midgu
140 of Leishmania within the alimentary canal of sand flies the parasites have to survive the hostile env
141 hroids, and susceptibility profile of Indian sand flies, the continued use of DDT in this IRS program
142 et of sequence data reported from a specific sand fly tissue and provides further information of the
144 rotected against cutaneous disease following sand fly transmission of Leishmania major in susceptible
146 eishmania major (L.m.) parasites early after sand fly transmission or needle inoculation, but phagocy
148 ocytic choriomeningitis virus (LCMV), or the sand fly-transmitted arbovirus Toscana virus (TOSV).
151 d controlled protective immune response to a sand fly-transmitted Leishmania somewhat mimicking "leis
152 (LPG) implicated in parasite survival in the sand fly vector and the initial stages of establishment
155 omes infected with Leishmania major when the sand fly vector injects parasites into skin along with s
157 that the infectiousness of patients for the sand fly vector of visceral leishmaniasis is linked to p
158 quence tag library has been generated from a sand fly vector of visceral leishmaniasis, Lutzomyia lon
159 the differentiation of parasites within the sand fly vector to the highly infective metacyclic proma
174 tion of parasites by the bite of an infected sand fly, we identified differences in the preexisting a
180 biota by pretreatment of Leishmania-infected sand flies with antibiotics or neutralizing the effect o
183 ur after exposure to the bite of an infected sand fly, yet only one is under evaluation in humans.
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