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1 ophosphoglycan in the anterior midgut of the sandfly.
2 ations of two insect vectors: mosquitoes and sandflies.
3 nments encountered during transition between sandfly and vertebrate hosts.
4 tilize an arthropod vector (e.g., mosquitos, sandflies, and ticks) to spread throughout human populat
5 ria-infected mosquitoes, Leishmania-infected sandflies, and Trypanosoma-infected tsetse flies.
6 s that are recruited as a normal response to sandfly bites.
7 Leishmania infections specifically increased sandfly biting persistence and feeding on multiple hosts
8 e parasitic disease, is transmitted during a sandfly blood meal as the parasite is delivered into the
9                 As before, epidemics of this sandfly-borne disease occur periodically in India and el
10  and Uukuniemi groups) and from the mosquito/sandfly-borne phleboviruses.
11 proper management include inadequate vector (sandfly) control, no vaccine, and insufficient access to
12 hlebovirus genus, distinct from the existing sandfly fever and Uukuniemi virus groups, in the family
13  other phleboviruses-i.e., Punta Toro virus, sandfly fever Sicilian virus, or Frijoles virus-has no e
14 te against the hydrolytic environment of the sandfly gut and/or the parasitophorous vacuole of host m
15  infectious dogs (17%) accounted for >80% of sandfly infections.
16 V), which is transmitted by Phlebotomus spp. sandflies, is a major etiologic agent of aseptic meningi
17 s that it encounters during residence in the sandfly, its insect vector.
18 aniasis is only transmitted by blood-feeding sandflies, not through human remains.
19 peat galectin expressed in the midgut of the sandfly Phlebotomus papatasi, is used by Leishmania majo
20 ed to their mammalian hosts when an infected sandfly probes in the skin for a bloodmeal and injects t
21 report that a single molecule, maxadilan, in sandfly saliva can exacerbate infection with L. major to
22                                      Indeed, sandfly saliva markedly enhances the infectivity of L. m
23 se phleboviruses, namely, Rift Valley fever, sandfly Sicilian, and Toscana viruses.
24 a are kinetoplastid parasites that cause the sandfly-transmitted disease leishmaniasis.
25         Phlebotomus ariasi is one of the two sandflies transmitting the causative agent of zoonotic l
26 ully the passage of the parasite between its sandfly vector and different mammalian host species.
27 essed through time by xenodiagnosis with the sandfly vector, Lutzomyia longipalpis.
28 oa are introduced into a mammalian skin by a sandfly vector, whereupon they encounter increased tempe
29 . as promastigotes in the gut lumen of their sandfly vectors and as amastigotes in the phagolysosomal
30 ough the adaptive manipulation of one of its sandfly vectors, Lutzomyia longipalpis.

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