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1 ophosphoglycan in the anterior midgut of the sandfly.
2 ations of two insect vectors: mosquitoes and sandflies.
4 tilize an arthropod vector (e.g., mosquitos, sandflies, and ticks) to spread throughout human populat
7 Leishmania infections specifically increased sandfly biting persistence and feeding on multiple hosts
8 e parasitic disease, is transmitted during a sandfly blood meal as the parasite is delivered into the
11 proper management include inadequate vector (sandfly) control, no vaccine, and insufficient access to
12 hlebovirus genus, distinct from the existing sandfly fever and Uukuniemi virus groups, in the family
13 other phleboviruses-i.e., Punta Toro virus, sandfly fever Sicilian virus, or Frijoles virus-has no e
14 te against the hydrolytic environment of the sandfly gut and/or the parasitophorous vacuole of host m
16 V), which is transmitted by Phlebotomus spp. sandflies, is a major etiologic agent of aseptic meningi
19 peat galectin expressed in the midgut of the sandfly Phlebotomus papatasi, is used by Leishmania majo
20 ed to their mammalian hosts when an infected sandfly probes in the skin for a bloodmeal and injects t
21 report that a single molecule, maxadilan, in sandfly saliva can exacerbate infection with L. major to
26 ully the passage of the parasite between its sandfly vector and different mammalian host species.
28 oa are introduced into a mammalian skin by a sandfly vector, whereupon they encounter increased tempe
29 . as promastigotes in the gut lumen of their sandfly vectors and as amastigotes in the phagolysosomal
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