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1 to facilitate placement of sutureless aorto-saphenous anastomoses during off-pump coronary artery by
2 ression through Rho-kinase, we exposed human saphenous and pulmonary artery endothelial cells to hypo
3 mean caliber of myelinated axons in both the saphenous and sciatic nerves was reduced in galactose-in
5 d mouse skeletal muscle arterioles and mouse saphenous arteries were isolated, pressurized, and subje
7 s 30% for popliteal arteries, 45% vs 28% for saphenous arteries; P < .001 for both comparisons) and e
8 eparin to inhibit intimal hyperplasia in the saphenous artery of the baboon after balloon angioplasty
11 usion or significant stenoses of preexisting saphenous grafts were thought to be the major cause of r
12 patients (3.51%) (25.1% mediastinitis, 32.6% saphenous harvest site, 35.0% septicemia, 0.5% thoracoto
15 nd hairy hindpaw skin at various times after saphenous nerve axotomy suggested multiple changes in ne
16 reventing movement-induced afferent input by saphenous nerve block before, but not after, hindlimb mo
17 cancer learn to prefer a context paired with saphenous nerve block to elicit pain relief (i.e., condi
18 ment with systemic morphine abolished CPP to saphenous nerve block, demonstrating control of ongoing
19 Injection of Sox11 siRNAs into the mouse saphenous nerve caused a transient knockdown of Sox11 mR
20 ation of capsaicin to skin innervated by the saphenous nerve increased mitochondrial traffic in both
21 tron microscopy, we recently showed that the saphenous nerve of the naked mole-rat (Heterocephalus gl
25 s of hairy hindpaw skin and L2/L3 DRGs after saphenous nerve regeneration suggested that inhibition o
28 ctional class of afferent C fibre in the rat saphenous nerve, and are not found in the rabbit sapheno
32 ive adult axons in vivo, confocal imaging of saphenous nerves in anaesthetised mice was combined with
34 ed in the galactose group, while sciatic and saphenous sensory NCVs were not significantly changed.
35 d 20 and 16% reductions in sciatic motor and saphenous sensory nerve conduction velocity, which were
36 ammary artery (6.2+/-0.3 pmol/mg protein) or saphenous vein (1.4+/-0.2 pmol/mg protein, both P<0.05).
37 ammary artery (3.5+/-1.3 pmol/mg protein) or saphenous vein (1.4+/-0.3 pmol/mg protein, both P<0.0001
41 cantly augmented BH4 levels in plasma and in saphenous vein (but not internal mammary artery) but als
42 utive patients with primary unilateral great saphenous vein (GSV) reflux undergoing endovenous treatm
43 Standard harvest and preparation of human saphenous vein (HSV) for autologous coronary and periphe
46 ion of endothelial cells cultured from human saphenous vein (HSVECs) has identified a voltage-gated N
47 cation to the carotid artery (high shear) or saphenous vein (lower shear); hyperfibrinogenemia signif
48 mutation in FOXC2 showed reflux in the great saphenous vein (n=18), compared with only 1 of 12 refere
49 radial artery, internal mammary artery, and saphenous vein (n=24 patients) were examined by use of o
51 - 0.2% for the RA, and 55.0 +/- 0.2% for the saphenous vein (p = 0.002 for RA vs. saphenous vein, 0.1
52 trial comparing treatment options for small saphenous vein (SSV) incompetence exists, and there is n
53 trial comparing treatment options for small saphenous vein (SSV) incompetence exists, and there is n
56 have significantly better patency rates than saphenous vein (SV) grafts at 5 years, but the physiolog
58 profiles of primary cultured ECs from human saphenous vein (SVEC) and coronary artery (CAEC) exposed
63 ynthase (eNOS) uncoupling were quantified in saphenous vein and internal mammary artery segments.
64 ivo release were quantified in perivascular (saphenous vein and internal mammary artery) subcutaneous
65 ent human smooth muscle cells, cultured from saphenous vein and internal mammary artery, were exposed
70 he angiographic patency of radial artery and saphenous vein aortocoronary bypass grafts at 5 years af
73 nous enhancement (99 HU) was observed in the saphenous vein at the ankle, with all other venous stati
75 The use of aortic connectors for proximal saphenous vein bypass graft anastomoses eliminates the n
78 els and studies in patients (coronary artery saphenous vein bypass grafts, lesions of restenosis afte
84 complications during stenting of degenerated saphenous vein coronary bypass grafts are reduced, but n
85 nstable angina, peripheral arterial disease, saphenous vein coronary bypass grafts, and diabetic reti
88 ere incubated with IFN-gamma-activated human saphenous vein endothelial cells (HSVEC), but not with r
89 in bovine aortic endothelial cells and human saphenous vein endothelial cells in vitro and in adult m
90 to human erythroleukemia cells and to human saphenous vein endothelial cells was mediated by both al
92 ls of the hH1 isoform are expressed in human saphenous vein endothelium and that the presence of thes
99 aft on long-term outcomes after percutaneous saphenous vein graft (SVG) intervention is currently unk
100 t of creatine kinase (CK-MB) elevation after saphenous vein graft (SVG) intervention is high, its pro
101 use of embolic protection devices (EPD) for saphenous vein graft (SVG) intervention; however, studie
103 sealing intermediate nonobstructive coronary saphenous vein graft (SVG) lesions with drug-eluting ste
105 et effect of aspirin, or both, contribute to saphenous vein graft (SVG) occlusion after coronary arte
106 pose of this study was to present radial and saphenous vein graft (SVG) occlusion results more than 5
107 assess disease progression in nonintervened saphenous vein graft (SVG) segments to determine the nat
109 ectiveness of gamma-irradiation ((192)Ir) in saphenous vein graft (SVG) versus native coronary artery
112 25 women) underwent stenting of 212 vessels (saphenous vein graft [53%], left anterior descending cor
113 ) or conventional treatment (n = 395) in the Saphenous Vein Graft Angioplasty Free of Emboli Randomiz
117 lower FitzGibbon A patency for arterial and saphenous vein graft conduits and less effective revascu
119 e use of a radial artery graft compared with saphenous vein graft did not result in greater 1-year pa
120 sis, bifurcation lesions, left main disease, saphenous vein graft disease, and acute coronary syndrom
121 ve coronary arteries, inhibit the process of saphenous vein graft disease, and improve vein graft pat
131 ative coronary artery stenoses (CAVEAT-I) or saphenous vein graft lesions (CAVEAT-II) were randomized
132 undergoing percutaneous intervention of 682 saphenous vein graft lesions were prospectively randomiz
133 ocation (78.6% in left main lesion, 69.7% in saphenous vein graft lesions, 42.4% in circumflex lesion
136 h platelet activation (deep vein thrombosis; saphenous vein graft occlusion after coronary bypass sur
142 otection filter devices during uncomplicated saphenous vein graft, carotid, renal, and superficial fe
147 e center to have either the radial artery or saphenous vein grafted to a stenosed branch of the nativ
148 of patients undergoing a nonstaged multiple saphenous vein grafting (SVG) intervention with stents a
150 at the feasibility and safety of CTO PCI via saphenous vein grafts (19% of post-CABG cases) versus co
152 l conduits (85.8% versus 91.4%; P=0.003) and saphenous vein grafts (72.7% versus 80.4%; P<0.001).
153 ernal thoracic artery (LITA) supplemented by saphenous vein grafts (LITA+SVG) has been demonstrated i
155 enosis rate of 15.1%, compared with 5.9% for saphenous vein grafts (P=0.0003) and 4.8% for left inter
157 ception of patients treated with degenerated saphenous vein grafts (risk with placebo 16.3% vs. risk
159 l outcome after percutaneous intervention of saphenous vein grafts (SVG) and to identify the predicto
163 tomy prior to stent implantation in diseased saphenous vein grafts (SVGs) and thrombus-containing nat
167 Percutaneous coronary intervention (PCI) of saphenous vein grafts (SVGs) has historically been assoc
170 s related to successful stenting of diseased saphenous vein grafts (SVGs) using a novel filter-based
171 ercutaneous coronary interventions (PCIs) in saphenous vein grafts (SVGs) with thrombus have a high f
172 coronary interventions (PCIs) in degenerated saphenous vein grafts (SVGs) without distal embolic prot
173 before CABG would improve the redox state in saphenous vein grafts (SVGs), independently of low-densi
177 gnificantly better than the patency rate for saphenous vein grafts and comparable to reported patency
178 er superior long-term survival compared with saphenous vein grafts and should be considered in patien
179 erosclerotic progression among patients with saphenous vein grafts and that aggressive lipid lowering
180 nct to percutaneous intervention of diseased saphenous vein grafts and, compared with distal protecti
183 nary stenting of stenoses in old (> 9 years) saphenous vein grafts can be successfully performed, wit
184 rafts are thought to be better conduits than saphenous vein grafts for coronary artery bypass graftin
187 after percutaneous intervention in diseased saphenous vein grafts is reduced by distal microcirculat
188 placebo on progression of atherosclerosis in saphenous vein grafts of patients who had had CABG surge
189 placebo on progression of atherosclerosis in saphenous vein grafts of patients who had had CABG surge
191 enever possible during treatment of diseased saphenous vein grafts produced outcomes similar to those
194 rates of drug-eluting stents, which outlive saphenous vein grafts to non-left anterior descending ve
195 Medical) was developed to rapidly anastomose saphenous vein grafts to the aorta during coronary bypas
198 l of 594 patients undergoing stenting of 639 saphenous vein grafts were prospectively randomized, usi
200 year clinical outcomes of patients receiving saphenous vein grafts with multiple (m-SVG) versus singl
201 ger vessels, in the right coronary artery or saphenous vein grafts, and for unfavorable lesion charac
202 xus and Cardiac Surgery) score, treatment of saphenous vein grafts, ostial lesions, and in-stent rest
203 anterior descending, right coronary artery, saphenous vein grafts, ostial lesions, or in-stent reste
212 , or ostial; total occlusions; bifurcations; saphenous vein grafts; and multivessel interventions) fr
213 up reported significantly more pain than the saphenous vein group 3 months after surgery; however, si
215 operative baseline between radial artery and saphenous vein groups after adjusting for covariates (P
216 e consequences of radial artery harvest with saphenous vein harvest in patients undergoing elective c
217 hundred forty-two patients with sternal and saphenous vein harvest wounds had half of each wound clo
220 function in normal and atherosclerotic human saphenous vein imply a role for the peptide in the progr
223 reduced the average time to hemostasis after saphenous vein incision, and the time to occlusion after
224 ure base, centered on the treatment of great saphenous vein incompetence cannot simply be extrapolate
226 was to assess thrombus age in patients with saphenous vein insufficiency treated with sclerotherapy.
227 f stenotic coronary arteries with autologous saphenous vein is an established treatment for ischemic
232 recipients: 3 of 3 organ recipients, 1 of 2 saphenous vein recipients, 1 of 3 tendon recipients, and
234 rior descending artery, and radial artery or saphenous vein segments are used to graft the lateral an
236 nical significance was investigated by using saphenous vein segments from non-coronary heart disease
238 growth factor-stimulated migration of human saphenous vein SMCs and decrease phosphorylation of the
239 tudy, transient transfection assays in human saphenous vein smooth muscle cells (HSVSMC) and pulmonar
240 od or from adult peripheral blood, and human saphenous vein smooth muscle cells (HSVSMCs) as a source
241 n was increased in carotid atherectomies and saphenous vein specimens from diabetic versus nondiabeti
244 ncRNAs whose expression was altered in human saphenous vein vascular smooth muscle cells following st
246 uccessful ablation of the main trunks of the saphenous vein was less common in the foam group than in
248 Segments of internal mammary artery and saphenous vein were obtained during coronary artery bypa
249 nts, segments of internal mammary artery and saphenous vein were obtained from five patients who rece
250 = 65 years) in mammary artery (no change in saphenous vein), accompanied by increased alpha(1b)>alph
252 for the saphenous vein (p = 0.002 for RA vs. saphenous vein, 0.11 for RA vs. ITA, and p < 0.001 for I
253 89%; 95% confidence interval [CI], 86%-93%; saphenous vein, 239/269; 89%; 95% CI, 85%-93%; adjusted
254 ments from either internal mammary artery or saphenous vein, both forskolin and 8-Br-cAMP inhibited l
255 reduced the adhesion of these cells to human saphenous vein, whereas PBM adhesion in the presence of
269 easured by thromboelastography and prolonged saphenous-vein bleeding times, which are consistent with
272 of 120 patients with in-stent restenosis in saphenous-vein grafts, the majority of whom had diffuse
274 number of arterial grafts into the LIMA plus saphenous veins (LIMA/SV) group (n=7435) or the MultArt
275 s of culture, the I/M ratio increased in the saphenous veins (P=0.03, P=0.04 versus 0 day, respective
277 tylcholine and bradykinin were determined in saphenous veins and internal mammary arteries from 117 p
278 superoxide production was quantified in both saphenous veins and internal mammary arteries from 45 di
279 F, and total homocysteine were determined in saphenous veins and internal mammary arteries obtained d
283 henous veins before organ culture and in pig saphenous veins before interposition grafting into carot
284 essed TIMP-3 at the luminal surface of human saphenous veins before organ culture and in pig saphenou
286 Limitations in the long-term patency of saphenous veins for bypass grafts have encouraged intere
287 factor (VWF) by immunofluorescence in great saphenous veins harvested at cardiac bypass surgery.
288 P)H oxidase activity was determined in human saphenous veins obtained from 110 patients with coronary
289 peroxide production were determined in human saphenous veins obtained from 133 patients with coronary
290 Adenovirus-mediated gene transfer to human saphenous veins resulted in functional transgene express
292 3.8+/-0.8% in the internal mammary arteries, saphenous veins, and normal coronary arteries, respectiv
293 istance arteries, internal mammary arteries, saphenous veins, and small subcutaneous veins were studi
294 f 100 sternotomy CABG patients using IMA and saphenous veins, both treating equivalent number of targ
295 l of intimal hyperplasia, we incubated human saphenous veins, internal mammary arteries, and radial a
297 -C protein was not detected in control human saphenous veins; however, it was uniformly and strongly
298 nts provide effective treatment for stenotic saphenous venous aorto-coronary bypass grafts, but their
299 mal proliferation, sclerosis of arterialized saphenous venous graft, and fibromuscular dysplasia) rev
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