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1 d 2, simian immunodeficiency virus, and Rous sarcoma virus.
2 n that is present in the Gag protein of Rous sarcoma virus.
3 .5 of herpes simplex virus and NL-S of avian sarcoma virus.
4 dermal sarcoma virus and salmon swim bladder sarcoma virus.
5 fied in the gag-specific protein p2b of Rous sarcoma virus.
6 irus with significant homology to the simian sarcoma virus.
7 ogue of v-src, the transforming gene of Rous Sarcoma virus.
8 sed by gamma-herpesviruses, such as Kaposi's sarcoma virus.
9 es of two in-vitro-assembled capsids of Rous sarcoma virus.
10 irus type 1, hepatitis C virus, and Kaposi's sarcoma virus.
11 of the naturally nonmyristoylated MA of Rous sarcoma virus.
12 The retroviral oncogene p3k (v-p3k) of avian sarcoma virus 16 (ASV16) codes for the catalytic subunit
13 en embryo fibroblasts (CEF) infected by Rous sarcoma virus against a confluent background of uninfect
14 brane subunit (TM) of the avian leukosis and sarcoma virus (ALSV) envelope glycoprotein (Env) contain
19 Despite sequence differences between avian sarcoma virus and HIV-1 IN and their recognition sequenc
20 eferred in vitro integration sites for avian sarcoma virus and human immunodeficiency virus-1 integra
23 outbreaks of disease, such as walleye dermal sarcoma virus and salmon swim bladder sarcoma virus.
25 (DR) sequences flanking the src gene in Rous sarcoma virus are essential posttranscriptional control
26 eriments reported here, using the avian Rous sarcoma virus as a model system, further define the natu
27 s seen in the integrase core domain of avian sarcoma virus as well as human immunodeficiency virus ty
29 d independently of catalysis with both avian sarcoma virus (ASV) and human immunodeficiency virus typ
30 the core domain of integrase (IN) from avian sarcoma virus (ASV) and its active-site derivative conta
31 The direct-repeat elements (dr1) of avian sarcoma virus (ASV) and leukosis virus have the properti
32 immunodeficiency virus type 1 (HIV-1), avian sarcoma virus (ASV) and their close orthologs from the L
33 Recent studies have demonstrated that avian sarcoma virus (ASV) can transduce cycle-arrested cells.
36 ubiquitin ligases bind the L domain in avian sarcoma virus (ASV) Gag and facilitate viral particle re
38 HIV-1, simian sarcoma virus (SIV), and avian sarcoma virus (ASV) INs predicted which of these residue
39 x was identified as an interactor with avian sarcoma virus (ASV) integrase (IN) in a yeast two-hybrid
40 ined the size and shape of full-length avian sarcoma virus (ASV) integrase (IN) monomers and dimers i
42 Here, we report the mapping of 226 avian sarcoma virus (ASV) integration sites in the human genom
43 arable to one previously described for avian sarcoma virus (ASV) that was stimulated by the presence
44 ntegrase protein of an oncoretrovirus, avian sarcoma virus (ASV), suggesting an active import mechani
45 repression and silencing of integrated avian sarcoma virus (ASV)-based vector DNAs in human HeLa cell
47 cell populations that harbored silent avian sarcoma virus-based green fluorescent protein (GFP) vect
48 egration catalyzed by HIV-1 to that of avian sarcoma virus by analyzing the effect of defined mutatio
49 allowed for the first-time formation of Rous sarcoma virus CA into structures resembling authentic ca
52 arged amino acids on the surface of the Rous sarcoma virus capsid protein in the assembly of appropri
53 quence similarity, the structure of the Rous sarcoma virus capsid protein is similar to the structure
54 that exist in the C-terminal domain of Rous sarcoma virus capsid relative to the other capsid protei
55 ly unrelated late domain sequences from Rous sarcoma virus (contained in its p2b sequence) or equine
56 kinase substrates and identified CRKL (v-Crk sarcoma virus CT10 oncogene-like protein), a substrate o
61 ted the modified MND LTR (myeloproliferative sarcoma virus enhancer, negative control region deleted,
62 ficiency virus type 1, visna virus, and Rous sarcoma virus exhibited distinct preferences for water o
63 ken embryos with replication-competent avian sarcoma virus expressing either FgfR2(C278F), a receptor
64 no acid level) to that of the avian leukosis-sarcoma virus family, it retains several sequences of de
65 ntisense to fau (Finkel-Biskis-Reilly murine sarcoma virus (FBR-MuSV)-associated ubiquitously express
66 that while deletion of the NC domain of Rous sarcoma virus Gag abolishes formation and budding of VLP
74 fied, slightly truncated version of the Rous sarcoma virus Gag protein, Delta MBD Delta PR, and DNA o
75 nce correlation spectroscopy-to examine Rous sarcoma virus Gag-Gag and -membrane interactions in livi
76 e indicated that in the context of the avian sarcoma virus genome, precise deletion of both ASV dr1 e
78 osed that dimerization of the Moloney murine sarcoma virus genomic RNAs relies upon the concentration
81 The low molecular weight G protein RhoA (rat sarcoma virus homolog family member A) serves as a node
83 anganate modification to show that the avian sarcoma virus IN catalytic domain is able to distort vir
84 ture of the three-domain integrase from Rous sarcoma virus in complex with viral and target DNAs.
85 tive sequence (MiDAS) for the Moloney murine sarcoma virus in the final dimer state using selective 2
86 ast, CEF cultures heavily infected with Rous sarcoma virus in the same medium underwent pervasive tra
87 putative dimer-dimer interface of the avian sarcoma virus IN with its analogue, loop188-194, from hu
88 ategy, the unique amino acids found in avian sarcoma virus IN, rather than HIV-1 or Mason-Pfizer monk
89 air (bp) laboratory-generated Moloney murine sarcoma virus, induced subcutaneous tumors in about 14%
91 rosine-phosphorylated when expressed in Rous sarcoma virus-infected chicken embryo fibroblasts (RSV-C
92 tudies of the catalytic core domain of avian sarcoma virus integrase (ASV IN) have provided the most
93 omplex of the catalytic core domain of avian sarcoma virus integrase (ASV IN) were solved at 1.9- to
94 positions in visna virus integrase and Rous sarcoma virus integrase changed the target site preferen
95 tudies revealed that truncated forms of Rous sarcoma virus integrase containing two of the three prot
96 an immunodeficiency virus-1 integrase, avian sarcoma virus integrase, and bacteriophage Mu transposas
99 A direct comparison demonstrates that Rous sarcoma virus is capable of infecting aphidicolin-arrest
100 Examination of the genomes of three Rous sarcoma virus isolates indicated that codon composition
101 fau) increased the tumorigenicity of a mouse sarcoma virus, it was proposed that fau might be a tumor
102 ed a small segment of the 3' end of the Rous sarcoma virus, just inside the poly(A) tail, at the same
103 ropic virus (HTLV-1) and Kaposi's associated sarcoma virus (KSHV) contribute to 10-15% of the cancers
104 estigated the features of the Moloney murine sarcoma virus leader sequence necessary for RNA packagin
105 susceptible mice with the myeloproliferative sarcoma virus leads to the formation of erythroid bursts
106 ing chicken Y-box protein that promotes Rous sarcoma virus long terminal repeat (RSV LTR)-driven tran
107 chimeric transcription factor on the Moloney sarcoma virus long terminal repeat and a synthetic promo
108 to the levels observed in vivo with the Rous sarcoma virus long terminal repeat constitutive promoter
109 iotic-resistance gene was driven by the Rous sarcoma virus long terminal repeat or the herpes simplex
110 in promoter activities (but not viral murine sarcoma virus-long terminal repeat promoter activity) we
111 shown to transcriptionally activate the Rous sarcoma virus-long terminal repeat promoter, which was f
112 virus, simian virus SV40, and Moloney murine sarcoma virus (MoMSV) long terminal repeat (LTR), were u
114 emia Virus (MMLV) and the Myeloproliferative Sarcoma Virus (MPSV) long terminal repeats (LTR) or by t
116 FB27 and FB29) under the control of a murine sarcoma virus (MSV) long terminal repeat (LTR) express t
117 long terminal repeat (LTR) promoters-murine sarcoma virus (MSV), MoMLV (MLV), and the LTR (termed Rh
118 the in vitro dimerization of Moloney murine sarcoma virus (MuSV) RNA in the context of these two mod
122 site phosphorylated by gamma-PAK in the Rous sarcoma virus nucleocapsid protein NC in vivo and in vit
123 coma viral oncogene homolog B (BRAF), or rat sarcoma virus oncogene (RAS) alterations], we determined
124 audin-4, secretin receptor, claudin-7, V-ros sarcoma virus oncogene homolog 1, cadherin 22, mucin-1,
125 an (HeLa) cells, mediated by either an avian sarcoma virus or a human immune deficiency virus type 1
126 sarcoma virus such as the myeloproliferative sarcoma virus or the target cells for the two viruses we
127 le late assembly domains carried by the Rous sarcoma virus p2b protein and human immunodeficiency vir
129 ting determinants involved in avian leukosis sarcoma virus packaging RNA binding to Gag protein.
132 sig-mEndo expression was driven by the Rous sarcoma virus promoter had moderately high serum levels
133 om the beta-galactosidase gene when the Rous sarcoma virus promoter is used to drive transgene expres
137 ive substitutions in this region of the Rous sarcoma virus protein were lethal due to a severe defici
139 ution of a fragment of the integrase of Rous sarcoma virus (residues 49-286) containing both the cons
142 ction in the total amount of HIV-1 and avian sarcoma virus retroviral vector DNA that is joined to ho
149 ane binding, we fused the MA domains of Rous sarcoma virus (RSV) and HIV-1 to the chemically inducibl
150 Gag protein; however, recent studies of Rous sarcoma virus (RSV) and human immunodeficiency virus hav
154 odeficiency virus type 1 (HIV-1) and of Rous sarcoma virus (RSV) are morphologically distinct when vi
156 ted to investigate the initial steps of Rous sarcoma virus (RSV) assembly by examining the associatio
160 te NMR (ssNMR) resonance assignments of Rous sarcoma virus (RSV) CA, assembled into hexamer tubes tha
161 y incompetent by testing the ability of Rous sarcoma virus (RSV) CA-SP to assemble in vitro into icos
163 cture of the N-terminal domain (NTD) of Rous sarcoma virus (RSV) capsid protein (CA), with an upstrea
165 munodeficiency virus type 1 (HIV-1) and Rous sarcoma virus (RSV) capsid proteins form a beta-hairpin.
166 appears to be modular, as the unrelated Rous sarcoma virus (RSV) Env can be made Vpu sensitive by rep
167 We have previously described a mutant Rous sarcoma virus (RSV) Env protein, Env-mu26, with an L165R
168 model, CA proteins from both HIV-1 and Rous sarcoma virus (RSV) form similar hexagonal lattices.
169 e have identified an assembly-defective Rous sarcoma virus (RSV) Gag mutant that retains significant
170 in vitro-assembled, immature virus-like Rous sarcoma virus (RSV) Gag particles and have determined th
175 he NC domain in assembly of VLPs from a Rous sarcoma virus (RSV) Gag protein and have characterized t
178 reported that nuclear transport of the Rous sarcoma virus (RSV) Gag protein is intrinsic to the viru
183 n was the 11-amino-acid p2b sequence of Rous sarcoma virus (RSV) Gag, which could fully restore HIV-1
184 tial steps in understanding the chicken Rous sarcoma virus (RSV) genome association with a nonpermiss
185 vely short, 82 nucleotide region of the Rous sarcoma virus (RSV) genome, called muPsi, was shown to b
188 e L domains of oncoretroviruses such as Rous sarcoma virus (RSV) have a more N-terminal location and
189 We produced kinetically stabilized Rous sarcoma virus (RSV) intasomes with human immunodeficienc
190 ite-directed mutagenesis of recombinant Rous sarcoma virus (RSV) integrase (IN) allowed us to gain in
191 ed when the serine at amino acid 124 of Rous sarcoma virus (RSV) integrase is replaced by alanine, va
194 ve examined whether the alpharetrovirus Rous sarcoma virus (RSV) is susceptible to inhibition by a ra
199 rus (CMV) immediate-early promoter, the Rous sarcoma virus (RSV) long terminal repeat, and the adenov
200 ation of HIV-1 Gag, as well as purified Rous sarcoma virus (RSV) MA and Gag, depends strongly on the
201 tro flotation assay to directly measure Rous sarcoma virus (RSV) MA-membrane interaction in the absen
202 repeat (IR) within the U5 region of the Rous sarcoma virus (RSV) mRNA forms a structure composed of a
206 The late assembly domain (L-domain) of Rous sarcoma virus (RSV) or human immunodeficiency virus type
207 f Gag in in vivo and in vitro assembled Rous sarcoma virus (RSV) particles and to compare these featu
208 er, in the presence of budding HIV-1 or Rous sarcoma virus (RSV) particles, some glycoproteins, such
210 deficiency virus type 1 (HIV-1) PTAP or Rous sarcoma virus (RSV) PPPY L domain in the p9 protein or b
211 enzymatic and structural properties of Rous sarcoma virus (RSV) PR are exquisitely sensitive to muta
212 e gene under the control of a truncated Rous sarcoma virus (RSV) promoter (AdRSVpHyde) was generated.
213 NA for human iNOS was cloned behind the Rous sarcoma virus (RSV) promoter to create adenovirus (Ad) 5
217 initiated by electroporation of cloned Rous sarcoma virus (RSV) proviral DNA into the developing chi
218 xpression of the Gag-Pol polyprotein of Rous sarcoma virus (RSV) requires a -1 ribosomal frameshiftin
221 g site in the 5' untranslated region of Rous sarcoma virus (RSV) RNA play an integral role in multipl
223 se monoclonal antibody directed against Rous sarcoma virus (RSV) subgroup A Env that will be useful i
224 uce milligram quantities of the soluble Rous sarcoma virus (RSV) synaptic complex that is kinetically
225 ibes new mutations in the CA protein of Rous sarcoma virus (RSV) that were designed to test whether t
226 chick embryo fibroblasts transformed by Rous sarcoma virus (RSV) the tyrosine phosphorylation of a ce
227 eline immunodeficiency virus (FIV), and Rous sarcoma virus (RSV) to critically address the role of in
229 ansport, the multidomain Gag protein of Rous sarcoma virus (RSV) undergoes importin-mediated nuclear
231 overy from Ray Erikson's group that the Rous sarcoma virus (RSV) v-Src-transforming protein had an as
232 ctor containing a constitutively active Rous sarcoma virus (RSV) viral promoter driving the luciferas
233 A PPPPY motif within the L domain of Rous sarcoma virus (RSV) was previously characterized as bein
234 ctions with purified CA proteins of the Rous sarcoma virus (RSV) were used to define factors that inf
235 We previously reported that a mutant Rous sarcoma virus (RSV) with an alternate polypurine tract (
238 1, Moloney murine leukemia virus (MLV), Rous sarcoma virus (RSV), and human T-cell lymphotropic virus
239 immunodeficiency virus type 1 (HIV-1), Rous sarcoma virus (RSV), and Mason-Pfizer monkey virus (MPMV
240 les of the prototypic avian retrovirus, Rous sarcoma virus (RSV), by using scanning transmission elec
241 In some orthoretroviruses, including Rous Sarcoma Virus (RSV), CA carries a short and hydrophobic
242 ous recovered a virus, now known as the Rous sarcoma virus (RSV), from a chicken sarcoma, which repro
244 based on the existence of MA mutants in Rous sarcoma virus (RSV), murine leukemia virus, human immuno
245 During assembly and morphogenesis of Rous sarcoma virus (RSV), proteolytic processing of the struc
248 ammaretrovirus, and the Alpharetrovirus Rous sarcoma virus (RSV), were susceptible to inhibition by R
249 ation-competent shuttle vector based on Rous sarcoma virus (RSV), with alternate retroviral PPTs and
250 utility of the system, we developed new Rous sarcoma virus (RSV)-based replication-incompetent vector
251 the PBS and the CA dinucleotide of the Rous sarcoma virus (RSV)-derived vector RSVP(A)Z to match the
252 ndogenous polypurine tract (PPT) of the Rous sarcoma virus (RSV)-derived vector RSVP(A)Z was replaced
255 or encoding a peptide inhibitor of PKA [Rous sarcoma virus (RSV)-protein kinase A inhibitor (PKI)].
259 well-established retroviral model-avian Rous sarcoma virus (RSV)-we analyzed changes in an RSV varian
266 parate structural alignment of HIV-1, simian sarcoma virus (SIV), and avian sarcoma virus (ASV) INs p
268 sequence of the Atlantic salmon swim bladder sarcoma virus (SSSV) provirus is 10.9 kb in length and s
269 also found for other retroviruses, the Rous sarcoma virus structural protein Gag is necessary and su
270 express TVA, the receptor for avian leukosis sarcoma virus subgroup A (ALSV-A), under the control of
271 uced with Friend virus suggested that either sarcoma virus such as the myeloproliferative sarcoma vir
272 gative regulator of splicing (NRS) from Rous sarcoma virus suppresses viral RNA splicing and is one o
273 RS) is a long cis-acting RNA element in Rous sarcoma virus that contributes to unspliced RNA accumula
274 ant of the viral matrix (MA) protein of Rous sarcoma virus that disrupts viral RNA dimerization.
276 nd nonreceptor tyrosine kinase Src from Rous sarcoma virus, these interactions are mediated by an N-t
279 1 (HcrtR1/OX(1)R) but not to Kirsten murine sarcoma virus transformed rat kidney epithelial (KNRK) c
281 subunit (Na,K-beta) in highly motile Moloney sarcoma virus-transformed Madin-Darby canine kidney (MSV
285 s containing the cellular receptors for Rous sarcoma virus (Tva) or ecotropic murine leukemia virus (
286 composed of a Tec-homology (TH) domain and a sarcoma virus tyrosine kinase (Src)-homology 3 (SH3) dom
287 yo fibroblasts (CEF) infected with the avian sarcoma virus UR2, encoding the oncogenic receptor prote
296 ounding member of this group, walleye dermal sarcoma virus (WDSV), induces benign skin tumors in the
298 including the prototypic oncoretrovirus Rous sarcoma virus, were synthesized on cytosolic ribosomes a
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