ライフサイエンスコーパス: scald

1 nflammation, as typified by murine models of scald and arthritis.

2 fluid balance were measured before and after scalding and resuscitation.

3 SCALD appears to be the mouse ortholog of the human prot

4 t mMCP-5 or human neutrophil elastase to the scalded area increases epidermal injury with subsequent

5 d a 60% total body surface area third-degree scald burn and were randomly divided to receive either 4

6 d a 60% total body surface area third-degree scald burn and were randomly divided to receive either r

7 muscles of rats after a full-thickness skin scald burn covering 20% of the total body surface area,

8 A second-degree epidermal scald burn in mice elicits an inflammatory response medi

9 o sham or burn injury consisting of a dorsal scald burn injury covering 30% of total body surface are

10 very of myofibre area and volume following a scald burn injury in mice.

11 Alcohol administered 30 minutes before a 15% scald burn injury.

12 Using a scald burn model in mice, we find that this part of the

13 om male rats subjected to a 40% third-degree scald burn or sham burn.

14 A 20% full-thickness scald burn was used in C57BL/6 mice.

15 show decreased injury after a second-degree scald burn, whereas mice lacking the MC-restricted trypt

16 d subjected to a 30% total body surface area scald burn.

17 ged in age from 3 to 18 years, with flame or scald burns covering more than 40% of their body surface

18 The scald caused an initial 30% reduction in cardiac output,

19 n Xanthomonas albilineans, which causes leaf scald disease of sugarcane and is also pathogenic to cor

20 In testis, SCALD expression is restricted to pachytene spermatocyte

21 se two extremes - intense food-poor cold and scalding food-rich oases.

22 es a membrane-bound enzyme that we designate SCALD, for short-chain aldehyde reductase.

23 We expressed SCALD in bacteria, purified it extensively, and studied

24 line dextran) were infused 30 mins after the scald injury at a rate to restore and maintain the basel

25 Using a well-established murine scald injury model with BALB/c and BALB/c CD1d knockout

26 reased mortality was explored using a murine scald injury model.

27 mice indicated a significant reduction after scald injury whereas mMCP4(-/-) mice showed no significa

28 The initial baseline period was followed by scald injury, and three different treatment regimens wer

29 g a standardized rat model of full thickness scald injury, left ventricular pressures were recorded i

30 SCALD is also expressed in four layers of the retina, in

31 Northern blot analysis indicates that SCALD is expressed most abundantly in mouse liver and te

32 In the liver of mice, SCALD is suppressed by fasting and induced by refeeding,

33 des in addition to retinals, we propose that SCALD may be induced by SREBP in liver and other tissues

34 of human MC chymase restores burn injury to scalded mMCP-4-deficient mice but not to mMCP-5-deficien

35 days and 14 days after 50% body surface area scald or sham injury.

36 ent as a burn model with full-thickness skin scald over 30% total body surface area.

37 owed some disruption of the epidermis at the scald site in the protected strains suggesting the possi

38 Staphylococcal scalded skin syndrome (SSSS) is an exfoliative dermatiti

39 an, and its generalized form, staphylococcal scalded skin syndrome (SSSS), is a frequent manifestatio

40 wn to be a causative agent of staphylococcal scalded skin syndrome (SSSS).

41 s (DRESS), eczema herpeticum, staphylococcal scalded skin syndrome and inherited ichthyosis.

42 oliative toxin A (ETA) causes staphylococcal scalded skin syndrome which is characterized by a specif

43 xfoliative toxins (ETs) cause staphylococcal scalded skin syndrome, a disease characterized by specif

44 pneumonia, burn infections, bacteremia, and scalded skin syndrome.

45 the symptoms associated with staphylococcal scalded skin syndrome.

46 Staphylococcal scalded-skin syndrome is an exfoliative dermatitis chara

47 diseases bullous impetigo and staphylococcal scalded-skin syndrome, pathogenesis depends on cleavage

48 d are the causative agents of staphylococcal scalded-skin syndrome.

49 ering in bulbous impetigo and staphylococcal scalded-skin syndrome.