ライフサイエンスコーパス: scalp

1 uman brain non-invasively through the intact scalp.

2 ally affects actinically damaged skin of the scalp.

3 han cryotherapy for thin AKs on the face and scalp.

4 small, constant electric current through the scalp.

5 en-dependent, progressive hair loss from the scalp.

6 pain, bilateral plantar burns, and a frozen scalp.

7 lving the head and neck and particularly the scalp.

8 lographic recordings from the surface of the scalp.

9 by applying weak electrical currents at the scalp.

10 nes from balding (BAB) and non-balding (BAN) scalp.

11 for the treatment of AKs on the forehead and scalp.

12 -69 years) were 2-fold higher for SCC on the scalp (0.38 [95% CI, 0.00-0.81] vs 1.07 [95% CI, 0.75-1.

13 were associated with unsuccessful culture in scalp (all p<0.05), but not in dura.

14 cross-analyzed with our laboratory's facial/scalp alloflap data for similar evaluation of various co

15 s a novel, low-risk therapeutic approach for scalp alopecias.

16 d eyedrops for glaucoma, may be relevant for scalp alopecias.

17 s, ingenol mebutate gel (0.015% for face and scalp and 0.05% for trunk and extremities).

18 red with baseline were 87.2% for the face or scalp and 86.8% for the trunk or extremities.

19 ata (AA) involving hair loss over the entire scalp and body and is often difficult to treat.

20 emia developed widespread alopecia involving scalp and body hair within weeks after starting nilotini

21 Lesions are often localized to the scalp and can result in permanent scarring, disfiguratio

22 via 4 transducer arrays placed on the shaved scalp and connected to a portable medical device.

23 of four cases, we found that both postmortem scalp and dura could be successfully reprogrammed into i

24 ve tumor densities of KCs were higher on the scalp and ear in men compared with women, and on the upp

25 th stage T2 multilesion angiosarcomas of the scalp and face that are larger than 10 cm demonstrate a

26 These scalp and intracranial data hence show that power-law sc

27 hat, based on a correspondence between human scalp and intracranial nonhuman primate recordings, thei

28 The scalp and retroauricular regions are always affected, an

29 us, scaly, and crusted, always affecting the scalp and retroauricular regions.

30 lesions involving >/=3 areas, including the scalp and retroauricular regions; (2) recurring nature o

31 with major features of aplasia cutis of the scalp and terminal transverse limb defects.

32 Here, we report through-scalp and through-skull fluorescence imaging of mouse ce

33 ily of aplasia cutis congenita of the vertex scalp and transverse terminal limb defects.

34 Concomitant face/scalp and UExt transplants contain between 1000 and 2800

35 fibroblast cell cultures generated from 146 scalp and/or 53 dura mater samples from 146 postmortem h

36 n times to recurrence were 365 days (face or scalp) and 274 days (trunk or extremities).

37 Of these, 100 patients (face or scalp) and 71 patients (trunk or extremities) completed

38 blindness, and necrotic plaques on her face, scalp, and hands.

39 ety of the qualified anatomical sites (face, scalp, and upper extremities) twice daily for 4 consecut

40 ash involving the body, extremities, face or scalp, and/or funisitis, presenting in the first week (<

41 ension placed on the pericranial muscles and scalp aponeurosis secondary to the underlying cervical s

42 trusion probe was more negative over frontal scalp (approximately 500ms).

43 16(INK4a), suggesting that DPCs from balding scalp are more sensitive to environmental stress than no

44 stmortem dura mater, and to a lesser extent, scalp, are viable sources of living fibroblasts for cult

45 decoding to find substantial involvement of scalp areas over the sensorimotor cortex contralateral t

46 well-demarcated hairless fatty nevus on the scalp, benign ocular tumors, and central nervous system

47 biomarkers that will eliminate the need for scalp biopsies.

48 ng suffices to capture neural signals on the scalp, but recent studies posit that increasing sensor d

49 ata to show that population responses on the scalp can capture choice-predictive activity that builds

50 While scalp cells proliferated more and grew more rapidly than

51 Cutaneous scalp changes were present in 5 patients.

52 icolor, folliculitis, atopic dermatitis, and scalp conditions such as dandruff.

53 Participants were randomized to scalp cooling (n = 119) or control (n = 63).

54 m baseline to chemotherapy cycle 4 among the scalp cooling and control groups.

55 A positive association between scalp cooling and reduced risk of hair loss would be dem

56 To assess whether a scalp cooling device is effective at reducing chemothera

57 Scalp cooling was done using a scalp cooling device.

58 Although scalp cooling devices have been used to prevent this alo

59 t of mild headache and 3 (2.8%) discontinued scalp cooling due to feeling cold.

60 monstrated if 50% or more of patients in the scalp cooling group achieved treatment success, with the

61 nthracycline and taxane (106 patients in the scalp cooling group and 16 in the control group; 14 matc

62 No participants in the scalp cooling group received anthracyclines.

63 , 56.2%-75.4%) evaluable for alopecia in the scalp cooling group vs 0 of 16 patients (0%) in the cont

64 Of the 106 patients in the scalp cooling group, 4 (3.8%) experienced the adverse ev

65 1 month after the end of chemotherapy in the scalp cooling group.

66 To evaluate whether use of a scalp cooling system is associated with a lower amount o

67 Use of a scalp cooling system.

68 In previous studies of scalp cooling to prevent chemotherapy-induced alopecia,

69 py for early-stage breast cancer, the use of scalp cooling vs no scalp cooling was associated with le

70 reast cancer, the use of scalp cooling vs no scalp cooling was associated with less hair loss at 4 we

71 Scalp cooling was done using a scalp cooling device.

72 Scalp cooling was initiated 30 minutes prior to each che

73 anthracycline, or both, those who underwent scalp cooling were significantly more likely to have les

74 Of patients who underwent scalp cooling, 27.3% (95% CI, 18.0%-36.6%) reported feel

75 py cycle compared with those who received no scalp cooling.

76 s to identify several distinct foci of skull-scalp currents and to analyse their individual time-cour

77 Laplacian method was used to calculate skull-scalp currents corresponding to the measured scalp poten

78 characterized by congenital limb defects and scalp cutis aplasia.

79 Scalp data from these records were used to train a scalp

80 We propose that the limb and scalp defects might also be due to a vasculopathy in NOT

81 multiple scaly patches of alopecia underwent scalp dermoscopy, direct microscopic examinations, and m

82 mized, single-blind study, employing a split-scalp design, comparing the effectiveness and adverse ef

83 n D2 (PGD2), which is upregulated in balding scalp, differentially impacts on the proliferation of di

84 ted whether the hitherto not well-determined scalp distribution of the EDAN matches the well-establis

85 ion of the EDAN matches the well-established scalp distribution of the N2pc.

86 asked repetition priming exhibited a frontal scalp distribution that was most pronounced for particip

87 issociable in terms of latency, duration, or scalp distribution.

88 of oxidative stress on balding and occipital scalp DPCs.

89 Patients with scalp dysesthesia also had abnormal cervical spine image

90 Scalp dysesthesia is characterized by abnormal sensation

91 of medical records as having been seen with scalp dysesthesia, 14 patients had cervical spine diseas

92 al spine disease may lead to the symptoms of scalp dysesthesia.

93 Scalp-ear-nipple (SEN) syndrome is a rare, autosomal-dom

94 widespread, crusted, pruritic papules on the scalp, ears, and face and a purpuric and targetoid painf

95 entials in the subthalamic nucleus (STN) and scalp EEG (modified 10/20 montage) during sleep in human

96 Unilateral IEDs on scalp EEG (P = .001) and complete resection of brain reg

97 ere that KCs can be quasi-synchronous across scalp EEG and across much of the cortex using electrocor

98 Unilateral-only IEDs on preoperative scalp EEG and complete resection of IEDs on baseline ECo

99 High density scalp EEG and subdural ECoG recordings provide an opport

100 s over a 38 year age range (15-53 years) and scalp EEG data from healthy younger (20-30 years) and ol

101 We addressed this hypothesis by recording scalp EEG during a stop task while modulating STN activi

102 (ECoG; subdural electrodes) and noninvasive scalp EEG during intensive care.

103 vapor and similar in size and flexibility to scalp EEG electrodes.

104 Elevated PAC is detectable using scalp EEG in PD patients off medications compared to on

105 Time and frequency domain analyses of scalp EEG recordings are widely used to track changes in

106 Previous research has suggested that human scalp EEG recordings contain signals that reflect the ne

107 In human scalp EEG recordings, both sustained potentials and alph

108 Scalp EEG was the most useful test for identifying patie

109 The classifications of scalp EEG were various, including non-localisible, later

110 observed in clinically standard, continuous scalp EEG, and underlying depolarizations can spread wid

111 To examine this, we recorded simultaneously scalp EEG, intracerebral EEG, and unit firing in multipl

112 For scalp EEG, there was greater beta power, around the time

113 We used simultaneous interictal scalp EEG-fMRI to evaluate its value for predicting long

114 ts high spatial resolution compared with the scalp EEG.

115 uestion using fMRI and concurrently acquired scalp EEG.

116 We analysed clinical semiology, the scalp EEG/SEEG findings and cortico-cortical evoked pote

117 hemorrhage who underwent continuous surface (scalp) EEG (sEEG) recording and multimodality monitoring

118 We recorded scalp-EEG (electrophysiology) in healthy participants an

119 For scalp-EEG, surprising events engage the same independent

120 alized interictal epileptiform discharges on scalp EEGs were associated with an excellent surgical ou

121 We recorded scalp electrical brain activity in 4-6 months infants vi

122 ed that these short EEEs are undetectable by scalp electrocorticography.

123 metal insert blocking the magnetic field and scalp electrodes that delivered matched somatosensory se

124 Bioelectrical activity from face and scalp electrodes was recorded from neonates during an ey

125 ented and ERPs were recorded from thirty-two scalp electrodes while participants watched a silent car

126 cal evoked potentials were recorded using 16 scalp electrodes, and antidromic ERGs were obtained usin

127 currents are applied to the human brain via scalp electrodes.

128 om rare simultaneous human intrathalamic and scalp electroencephalogram (EEG) recordings from eight v

129 e of gamma during fast stopping and recorded scalp electroencephalogram and local field potentials fr

130 focal mesial temporal lobe seizures based on scalp electroencephalogram coherence features, lends wei

131 of logistic regression classifiers that used scalp electroencephalogram coherence properties as input

132 ising from the mesial temporal lobe based on scalp electroencephalogram network connectivity measures

133 ing any obvious signs of seizure activity on scalp electroencephalogram.

134 recordings with foramen ovale electrodes and scalp electroencephalogram.

135 on, routine clinical interpretation of these scalp electroencephalograms failed to identify any of th

136 Here, we used pattern similarity analysis to scalp electroencephalographic (EEG) recordings during a

137 Using scalp electroencephalographic recordings and event-relat

138 d the electrical and vascular responses with scalp electroencephalography (EEG) and diffuse optical i

139 Scalp electroencephalography (EEG) and intraoperative el

140 tent of neural data acquired via noninvasive scalp electroencephalography (EEG) are insufficient to e

141 rdings while accessing global networks using scalp electroencephalography (EEG) in rhesus macaques.

142 Here, we assessed cortical excitability from scalp electroencephalography (EEG) responses to transcra

143 ty (FRN) is a commonly observed potential in scalp electroencephalography (EEG) studies related to th

144 We used scalp electroencephalography in groups of patients with

145 In this study, we used scalp electroencephalography to study the spectral featu

146 rain circuit models, magnetoencephalography, scalp electroencephalography, and even invasive electroe

147 Ictal patterns on scalp-electroencephalography are often visible only afte

148 re onset at sub-lobar/gyral level when ictal scalp-electroencephalography is not helpful.

149 multaneous epidural-electrocorticography and scalp-electroencephalography recordings in the rat demon

150 Here, we recorded scalp electrophysiological (EEG) brain responses in 5-y-

151 e human brain using various neuroimaging and scalp electrophysiological techniques.

152 vidence from a study employing pharmacology, scalp electrophysiology and computational modeling (N =

153 connectivity networks derived from multi-day scalp encephalogram (EEG) recordings in five healthy hum

154 Erosive pustular dermatosis of the scalp (EPDS) is an inflammatory skin condition that deve

155 eriment 1, independent component analysis of scalp ERP data revealed a common neural generator implic

156 oped at flexural regions, genitalia, and the scalp, especially the psoriasiform lesions.

157 Here, we recorded high-density scalp event-related potentials (ERPs) while participants

158 anti-CD133 antibody treatment of human fetal scalp explants depresses beta-catenin and E-cadherin mem

159 th face" to "bilateral elbow-level with face/scalp") extended from 1010 (+/- 81) to 2766 (+/- 202) cm

160 laxation time T1 of brain tissue, blood, and scalp fat at B0 and Bp, and cerebrospinal fluid at B0.

161 on host status and body sites, including the scalp, feet, and groin.

162 concentrations, increased hair synthesis in scalp follicle organ culture and advanced mouse pelage h

163 remain unaffected by androgens that inhibit scalp follicles and stimulate many others.

164 les in vivo, mirroring eyelash behavior, and scalp follicles contain bimatoprost-sensitive prostamide

165 Thus, bimatoprost stimulates human scalp follicles in culture and rodent pelage follicles i

166 ysis identified 3 relevant receptor genes in scalp follicles in vivo.

167 is hypothesis, we analyzed bald and non-bald scalp from AGA individuals for the presence of hair foll

168 we also show that its expression in lesional scalp from patients with AA is markedly upregulated in t

169 n over 6,000 Latin Americans for features of scalp hair (shape, colour, greying, balding) and facial

170 Human scalp hair follicles (hHF) harbour several epithelial st

171 air follicle response to chemotherapy, human scalp hair follicles cultured ex vivo were treated with

172 Confocal microscopy of human fetal and adult scalp hair follicles demonstrated a cytoplasmic pattern

173 et al. report that transplantation of human scalp hair follicles onto chemotherapy-treated immunodef

174 f2 in protecting human organ function (i.e., scalp hair follicles) against redox insult.

175 he epithelial stem cell (eSC) niche of human scalp hair follicles, during the inflammatory permanent

176 n a uniquely accessible human (mini-) organ: scalp hair follicles.

177 Human scalp hair has attractive features in clinical studies b

178 I and III, which are characterized by sparse scalp hair in addition to craniofacial and skeletal abno

179 Amino acid profiles of scalp hair of 27 Jordanian subjects (15 diabetes mellitu

180 ial amino acid Ile were more abundant in the scalp hair of diabetic patients compared to the hair of

181 Incomplete scalp hair recovery was more frequent in the EC-T than E

182 Keratin protein is the major component of scalp hair shaft material and it is composed of 21 amino

183 n spectroscopy in vivo that human gray/white scalp hair shafts accumulate hydrogen peroxide (H(2)O(2)

184 These include novel loci for scalp hair shape and balding, and the first reported loc

185 EDAR370A has been associated with increased scalp hair thickness and changed tooth morphology in hum

186 urbance in childhood and had lifelong sparse scalp hair with normal facial hair.

187 data presented here, obtained directly from scalp hair, implies escalating coca and alcohol ingestio

188 e with central visual disturbance and sparse scalp hair.

189 strophies by the universally thin and sparse scalp hair.

190 tagen transformation of microdissected human scalp HFs can be observed in organ culture, it permits t

191 mis, we investigated in organ-cultured human scalp HFs whether TRH (30 nM), TSH (10 mU ml(-1)), thyro

192 by transfecting normal, organ-cultured human scalp HFs with lipofectamine and CDH3-specific or scramb

193 nterventions, except for skin redness on the scalp in active tDCS (P = .03).

194 ent of seborrheic dermatitis of the face and scalp in adults?

195 characterized by abnormal sensations of the scalp in the absence of any other unusual physical exami

196 Spindles are highly synchronous across the scalp in the electroencephalogram (EEG) but have low spa

197 ggesting that deficiency of DPC from balding scalps in fostering vascularization around the hair foll

198 ng filamentous bacteria, was isolated from a scalp infection in a patient from Pondicherry, India.

199 Greater scalp involvement (>25%) was associated with greater imm

200 angiosarcoma (>/=20 cm) involving 80% of the scalp, left forehead, and left cheek, with no evidence o

201 y exposing them to various chemicals through scalp lesions and burns.

202 patients with complete clearance of face or scalp lesions in the original trial and 76 patients with

203 igher likelihood of PsA were nail dystrophy, scalp lesions, and intergluteal/perianal psoriasis.

204 radiological features of IPEH involving the scalp, localized on the left side of the skull and in th

205 The short latency and posterior scalp location of the effect suggest that perceptual his

206 EG responses were found in a centro-parietal scalp location, whose slope depended on change size, con

207 ent predictors were ulceration, mitoses, and scalp location.

208 ctroencephalogram (EEG) was recorded from 19 scalp locations from 371 subjects ranging in age from 5

209 creases in sleep slow wave activity power at scalp locations matching their seizure focus.

210 -old girl presenting with a 6-cm protuberant scalp mass, which had doubled in size since birth.

211 In humans, recordings of the scalp middle-latency AEPs have confirmed early ( approxi

212 mplitude at frontal and central sites at the scalp midline.

213 sorder characterized by cutis aplasia of the scalp; minor anomalies of the external ears, digits, and

214 es showing that weak currents applied to the scalp modulate neural processing.

215 nstrate that a powerful magnet placed on the scalp modulates normal brain activity and induces behavi

216 absent TILs]), and anatomic site other than scalp/neck (0.1 [0.01-0.6] for scalp/neck vs trunk/pelvi

217 te other than scalp/neck (0.1 [0.01-0.6] for scalp/neck vs trunk/pelvis), and BRAF+ melanoma was asso

218 We identified 25 scalp-negative mesial temporal lobe seizures in 10 patie

219 the detector raised seizure alarms, 80% had scalp-negative mesial temporal lobe seizures.

220 Our scalp-negative seizure detector has clear clinical utili

221 data from these records were used to train a scalp-negative seizure detector, which consisted of a pa

222 As such, little is known about scalp-negative seizures and their role in the natural hi

223 a novel approach to non-invasively identify scalp-negative seizures arising from the mesial temporal

224 formance, this detector correctly identified scalp-negative seizures in 40% of patients, and correctl

225 al, currently, the only way to detect these 'scalp-negative seizures' is with intracranial recordings

226 encephalograms failed to identify any of the scalp-negative seizures.

227 Carlo (MC) simulations to densely cover the scalp of a well-characterized, adult male template brain

228 ctrical brain activity was recorded from the scalps of healthy men and women while they performed an

229 consecutive days for lesions on the face or scalp or for 2 consecutive days for the trunk or extremi

230 nodular tumors, male sex, tumor site on the scalp or neck, or tumor invasion of the entire papillary

231 tients with actinic keratoses on the face or scalp or on the trunk or extremities to receive ingenol

232 ectrical signals that can be recorded at the scalp or to surrogates of electrical activity, namely ma

233 al culture was almost two-fold compared with scalp (OR = 1.95, 95% CI: [1.01, 3.9], p = 0.047).

234 r culture in dura was 16-fold as compared to scalp (OR = 16.0, 95% CI: [2.1-120.6], p = 0.0007).

235 has remained unclear whether the infra-slow scalp potential fluctuations in full-band electroencepha

236 mparable effect occurred at the level of the scalp potential in humans.

237 ionship of infra-slow fluctuations (ISFs) in scalp potentials and BOLD signals.

238 ecific association indicates that infra-slow scalp potentials are directly associated with the endoge

239 Interestingly, at time of choice, scalp potentials continue to appear parametrically modul

240 mics of fast (1-100 Hz) oscillations and the scalp potentials per se exhibit fluctuations in the same

241 scalp currents corresponding to the measured scalp potentials.

242 s of trustworthiness perception by employing scalp recorded event related potentials and evaluating e

243 a initiative, we used source localization of scalp-recorded EEG resting data to examine the neural co

244 it unlikely to be a direct generator of the scalp-recorded EEG, these covariational patterns appear

245 The auditory brainstem response (ABR), a scalp-recorded electrical potential, is known for its ab

246 may be useful for identifying generators of scalp-recorded event related potentials and for examinin

247 Evidence suggests that scalp-recorded occipital alpha-band (8-13 Hz) oscillatio

248 eature-selective information processing with scalp recordings of electroencephalography (EEG).

249 Scalp recordings pointed to statistically distinct MMN t

250 Yet, psychophysical studies, scalp recordings, and neurophysiological recordings sugg

251 using relatively inexpensive and noninvasive scalp recordings.

252 mulator signals, population responses on the scalp reflect the influence of other decision-related si

253 e between 20-150 ms over left fronto-central scalp region.

254 he occipital, lateral-occipital, and frontal scalp regions increased with the peak latency.

255 d lateral electrode sites were examined over scalp regions where auditory-somatosensory integration w

256 ye movement sleep over widespread, bilateral scalp regions.

257 ation processing, mainly over fronto-central scalp regions.

258 tion and larger abnormalities in nonlesional scalp samples (ie, CXCL10 and KRT85).

259 patients with AA for comparison with normal scalp samples (n = 6).

260 profiling of 27 lesional and 17 nonlesional scalp samples from patients with AA for comparison with

261 KRT15(hi) stem cells were maintained in bald scalp samples.

262 hen compared to those with intracortical and scalp seizures (50% and 25% death or severe disability,

263 ion that weak electric fields applied to the scalp significantly affect neural processing in the prim

264 ferentiated in terms of the EEG according to scalp site and bandwidth.

265 Alpha power at widespread scalp sites decreased with increasing acoustic detail (d

266 an the American listeners at left hemisphere scalp sites that probably index activity from the superi

267 mined the P3 component over central-parietal scalp sites that was elicited by the test probe at the e

268 i) the shape and timing of ERPs at different scalp sites, ii) the event-related desynchronization in

269 for sensory than motor words, over posterior scalp sites.

270 Human hair follicles can be dissected out of scalp skin and cultured in vitro in defined growth mediu

271 clinical human hair research purposes, human scalp skin can be xenografted onto immunocompromised mic

272 an skin cell populations and in normal human scalp skin in situ, introducing skin as a nonconventiona

273 r (layer 1) represents extracerebral tissue (scalp, skull, dura mater, subarachnoid space, etc.) and

274 The primary outcome was the change in scalp surface area with hair loss over 24 weeks followin

275 th alopecia areata affecting at least 10% of scalp surface area, were eligible; 1 declined to partici

276 By assessing scalp surface EEG in conjunction with LORETA source esti

277 tion rates can be further improved using the scalp tangential electric field and the surface Laplacia

278 nutes prior to each chemotherapy cycle, with scalp temperature maintained at 3 degrees C (37 degrees

279 cited a contralateral ERP over the occipital scalp that was localized to neural generators in extrast

280 sis of the two trials involving the face and scalp, the rate of complete clearance was higher with in

281 on technique that applies mA currents at the scalp to modulate cortical excitability.

282 Crucially, there were clear differences in scalp topographies of this alpha activity depending on t

283 iminating components with spatially distinct scalp topographies.

284 Furthermore, the scalp topography of this repetition effect differed sign

285 ontralateral delay activity component with a scalp topography over somatosensory cortex contralateral

286 We sought to define the AA scalp transcriptome and associated biomarkers with compa

287 Patients had half of their scalp treated with daylight photodynamic therapy and the

288 Two samples, a tea tree oil-based scalp treatment and a white lavender body lotion, exhibi

289 ions of varying sizes were identified on the scalp, trunk, and extremities and were reported to have

290 Scalp uptake yielded consistent results (over the brain,

291 EEG and localized current sources across the scalp using a surface Laplacian transform.

292 high-frequency activity is detectable at the scalp using electroencephalography (EEG).

293 ence of aplasia cutis congenita (ACC) of the scalp vertex and terminal limb-reduction defects.

294 In vivo, tDCS was delivered to intact mice scalp via surface electrodes.

295 Hair loss on the scalp was noted in 120 (96%), arms and legs in 52 (42%),

296 spatial distribution of VLF EEG power across scalp was similar to that seen in previous resting VLF E

297 ient-matched DPCs from balding and occipital scalp were cultured at atmospheric (21%) or physiologica

298 wsiness, hair loss, nausea, and dry or itchy scalp were reported while patients were receiving WBRT,

299 velocity from neural data acquired from the scalp with 55-channel EEG during a 3D center-out reachin

300 expression in vivo in full thickness dermal scalp wounds created in experimental K14.Cre (+) .Foxo1