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1 o had been treated for ailments unrelated to scarlet fever.
2 muscle fibers, the inner ear, leukaemia, and scarlet fever.
3 were not higher than those in patients with scarlet fever.
4 to identify periods of sudden escalation of scarlet fever.
5 tococcal SAgs are known virulence factors in scarlet fever and toxic shock syndrome, mechanisms by ho
6 om acute rheumatic fever (ARF) and untreated scarlet fever and/or pharyngitis patients were reacted w
8 early periodicity in population incidence of scarlet fever but of consistently lower magnitude than t
10 ments associated with the expansion of emm12 scarlet fever clones in the M1T1 genomic background.
19 ed surveillance study, we analysed statutory scarlet fever notifications held by Public Health Englan
22 ive tuberculosis test results, strep throat, scarlet fever, pneumonia, bacterial meningitis, yeast in
23 urrence of multidrug-resistant ssa-harboring scarlet fever strains should prompt heightened surveilla
24 ncluding pharyngitis, tonsillitis, impetigo, scarlet fever, streptococcal toxic shock syndrome, necro
25 ncluding streptococcal pyrogenic exotoxin A (scarlet fever toxin) and two uncharacterized pyrogenic e
26 to disentangle the enigma: The 116 cases of scarlet fever were compared with 117 "controls" selected
28 and is experiencing an unprecedented rise in scarlet fever with the highest incidence for nearly 50 y
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