1 ope-specific Th responsiveness during murine schistosomiasis mansoni.

2 rincipal cause of morbidity and mortality in schistosomiasis mansoni.

3                                    In murine schistosomiasis mansoni, CD4(+) Th1 and Th2 cells partic

4  divided into group-I: patients with chronic schistosomiasis mansoni, group-II: HCV patients without

5                                           In schistosomiasis mansoni, helminth eggs secrete soluble e

6                                           In schistosomiasis mansoni, hepatic granulomatous inflammat

7 rleukin (IL)-13 is required at all stages of Schistosomiasis mansoni infection to induce fibrosis.

8 D in distinct or concomitant chronic HCV and schistosomiasis mansoni infections.

9 ndamental obstacle to vaccine development in schistosomiasis mansoni is a lack of understanding of wh

10                                              Schistosomiasis mansoni is characterized by hepatic gran

11 Immune cells within the granulomas of murine schistosomiasis mansoni make the neuropeptide substance

12                                           In schistosomiasis mansoni, parasite eggs cause hepatointes

13                                           In schistosomiasis mansoni, parasite eggs precipitate an in

14                                    In murine schistosomiasis mansoni, schistosome worms produce ova t

15                                           In schistosomiasis mansoni, soluble egg antigens of the wor

16 ine the role of the Th2-type response during schistosomiasis mansoni we compared disease progression

17 response, C57BL/6 strain animals with patent schistosomiasis mansoni were orally infected with the cy

18                        Such is the case with schistosomiasis mansoni, where chronic granulomatous inf