ライフサイエンスコーパス: schizophrenics

1 levels are elevated in postmortem brains of schizophrenics.

2 groups; CA1 only showed changes in the SO of schizophrenics.

3 on 1 meta-analysis and 2 studies of chronic schizophrenics.

4 gh-frequency synchronization in the brain of schizophrenics.

5 iction and the high prevalence of smoking in schizophrenics.

6 eceptor subunits in the prefrontal cortex of schizophrenics.

7 echanisms producing olfactory dysfunction in schizophrenics.

8 receptor complex, may also be upregulated in schizophrenics.

9 n relation to those reported for later-onset schizophrenics.

10 Some unaffected first-degree relatives of schizophrenics also manifest cortical gray-matter defici

11 in the left and right hippocampus between 11 schizophrenics and 13 controls.

12 ause current therapy is ineffective for many schizophrenics and because treatment is often accompanie

13 By contrast, the cerebellar cortex of both schizophrenics and controls contained very high levels o

14 ignificantly different between first-episode schizophrenics and controls over the posterior sagittal

15 loci in a large sample of unrelated Scottish schizophrenics and controls, but failed to replicate the

16 In the cerebral cortex of both schizophrenics and controls, mRNAs for NR1, NR2A, NR2B,

17 ix pairs of individually matched brains from schizophrenics and controls, we measured the volumes and

18 mporal, and cerebellar cortex of brains from schizophrenics and from neuroleptic-treated and nonmedic

19 xamined in dorsolateral prefrontal cortex of schizophrenics and matched controls by using in situ hyb

20 Both the familial schizophrenics and their first-degree relatives (but not

21 caused by retinoid dysfunction are found in schizophrenics and their relatives; (ii) those loci that

22 se found in the cerebrospinal fluid (CSF) of schizophrenics, antipsychotic drugs augment NMDA activit

23 al deficits were present in the offspring of schizophrenics at adolescence, to examine their stabilit

24 eased P50 inhibition occurs not only in most schizophrenics, but also in many of their nonschizophren

25 ic mutations in mGlu1 have been described in schizophrenics creating interest in this receptor as a t

26 First-episode schizophrenics displayed smaller amplitudes over the lef

27 es were divided into 2 groups: 32 studies of schizophrenics enrolled at various illness points (25 57

28 Despite many schizophrenics' extremely heavy nicotine use, nicotinic

29 In schizophrenics, gamma2S transcripts had fallen to 51.7%

30 Previous studies have shown that schizophrenics have decreased expression of alpha7-nicot

31 both mRNAs were significantly reduced in the schizophrenics, having controlled for the effects of bra

32 s points (25 578 subjects) and 29 studies of schizophrenics identified at either illness onset or fir

33 ctivity of dorsolateral prefrontal cortex in schizophrenics is well known.

34 In human schizophrenics, NR2A is selectively reduced in fast-firi

35 (BG) were well visualized in normals and in schizophrenics off medication.

36 sagittal thalamic area was also seen for the schizophrenics (P = .03), which was unchanged at rescan

37 vuII) were more frequent in African American schizophrenics (P = 0.01-0.001).

38 All 4 offspring of schizophrenics receiving schizophrenia spectrum diagnose

39 ermined to have altered expression levels in schizophrenics relative to controls are involved in a nu

40 Childhood schizophrenics showed a significantly greater increase i

41 While schizophrenics showed poorer performance than HC in the

42 verlapped with the subregions and laminae in schizophrenics showing the most marked increases of GABA

43 re broadly consistent with the proposal that schizophrenics suffer from enhanced signaling of salienc

44 ted allele had a somewhat lower frequency in schizophrenics than in controls.

45 gnificantly lower relative metabolic rate in schizophrenics than in controls.

46 he frequency of this oscillation is lower in schizophrenics than in healthy individuals.

47 In schizophrenics, the alterations in expression of NR2 sub

48 In schizophrenics, the BG/CE ratio off medication was 1.54

49 For schizophrenics, the distribution differed significantly

50 tributes to clozapine's clinical activity in schizophrenics through modulation of both muscarinic and

51 en-activated protein (MAP) kinase pathway in schizophrenics using postmortem brain samples.

52 nscripts in prefrontal cortex of brains from schizophrenics were reduced by 28.0%, although this redu

53 This study estimates that 4.9% of schizophrenics will commit suicide during their lifetime

54 Adolescent offspring of schizophrenics with poor neurobehavioral functioning had

55 Treatment of schizophrenics with some antipsychotic drugs has been as

56 Schizophrenics with the 'at risk' PvuII genotype had low

57 eversible hyperactivity was not a feature of schizophrenics without passivity.