ライフサイエンスコーパス: sciatic

1 and SB216763) in Schwann cells as well as in sciatic and facial nerves.

2 ber diameter and myelin thickness within the sciatic and phrenic nerves.

3 rmalities are likely to reflect pathology in sciatic and tibial nerve fibers.

4 athy, demonstrating strong associations with sciatic and tibial nerve findings.

5 FA of the sciatic and tibial nerves was lowest in the sDPN group.

6 anti-inflammatory mechanism mediated by the sciatic and vagus nerves that modulates the production o

7 second from the spinal cord; and (2) cortico-sciatic associative (CSA) protocol, in which the first s

8 associative protocols were tested: (1) spino-sciatic associative (SSA) protocol, in which the first s

9 rn identical to those of triplet proteins in sciatic axons and colocalizes with NFL on single neurofi

10 e dramatically reduces peripherin content in sciatic axons.

11 nal cord injuries with spasticity, spinal-to-sciatic DCS reduced transit and steady stretch-induced n

12 from spinal cord to sciatic nerve [spinal-to-sciatic direct current stimulation (DCS)] would inhibit

13 lleviated hind limb digital sensory, but not sciatic motor, nerve conduction slowing and thermal and

14 high-frequency electrical stimulation of the sciatic nerve (HFES) or injection of AICAR, an activator

15 rsisted in the dorsal root ganglia (DRG) and sciatic nerve (SN) for up to 72 hours.

16 egrated proteomics and metabolomics from the sciatic nerve (SN), the lumbar 4/5 dorsal root ganglia (

17 Diabetic wild-type mice displayed increased sciatic nerve 12/15-lipoxygenase and 12(S)-hydroxyeicosa

18 ) direct current flowing from spinal cord to sciatic nerve [spinal-to-sciatic direct current stimulat

19 Here, we report that sciatic nerve activation with electroacupuncture control

20 treatment also promoted remyelination of the sciatic nerve after crush.

21 A conditioning lesion of the sciatic nerve allows the central processes of dorsal roo

22 ne; (2) application of lidocaine in the left sciatic nerve alone significantly increased BP ipsilater

23 egeneration in dorsal root ganglia (DRG) and sciatic nerve and abundance of Schwann cells.

24 ions in axonal processes in the spinal cord, sciatic nerve and brain, but no excess of multivesicular

25 ere found in increased concentrations in the sciatic nerve and dorsal root ganglia of oxaliplatin tre

26 ve conduction velocity, nerve amplitude, and sciatic nerve and dorsal root ganglion morphology at 0.2

27 lso displayed a unique prolonged exposure in sciatic nerve and DRG.

28 ally accumulates in LRs in the TWI brain and sciatic nerve and in samples from brains of human Krabbe

29 ory components was evaluated in axons of the sciatic nerve and in spinal nerve axons after in vivo el

30 ssociated with a persistent M1 milieu in the sciatic nerve and in the regional and systemic lymphatic

31 having reliable signal acquisitions from the sciatic nerve and its branches such as the peroneal nerv

32 a small proportion of fibers that constitute sciatic nerve and its branches, but importantly breaks t

33 nction, intraepidermal nerve fiber loss, and sciatic nerve and spinal cord oxidative-nitrative stress

34 nduced by chronic constriction injury of the sciatic nerve and suppressed nerve injury-induced activa

35 rature on both the in-vitro stability of rat sciatic nerve and the response of the nerve to acute hyp

36 lly well in the lymph node, infection of the sciatic nerve and the rest of the nervous system was imp

37 which the first stimulus originated from the sciatic nerve and the second from the motor cortex.

38 which the first stimulus originated from the sciatic nerve and the second from the spinal cord; and (

39 ing required to prevent axonal damage of the sciatic nerve and to terminate the P0106-125-specific im

40 mmunohistological staining were performed in sciatic nerve and/or L4-L5 DRG tissue for galanin, CGRP

41 nastomosed to recipient femoral vessels, the sciatic nerve approximated end-to-end and osteosynthesis

42 We used a chronic constriction injury of sciatic nerve as a model of neuropathic pain in male Spr

43 tivity, and morphological alterations in the sciatic nerve as evidenced by decrease in g-ratio; it al

44 Analysis of the sciatic nerve at 11 d after nerve crush showed that the

45 cord, and, during their apoptosis induced by sciatic nerve avulsion, nuclear and cytoplasmic 5-methyl

46 otyping after selectively silencing TRPV1(+) sciatic nerve axons by perineural injections of QX-314 a

47 se, live imaging of endosomal trafficking in sciatic nerve axons reveals disease-induced deficits in

48 , degenerating) nerve stumps on day 1 in the sciatic nerve axotomy model in rats.

49 After sciatic nerve axotomy, there was a rightward shift in th

50 combinations of the above compounds produced sciatic nerve blockade lasting up to 7.5 days (with STX

51 r was generated and delivered into the mouse sciatic nerve by a single injection immediately distal t

52 vivo chromatin immunoprecipitation (ChIP) of sciatic nerve cells revealed a Sox10 binding site upstre

53 ng techniques, we have used a rodent partial sciatic nerve chronic constriction injury model (n = 5-8

54 Sciatic nerve conditioning at A-delta fiber strength, kn

55 elinated nerve fibers, specifically sural or sciatic nerve conduction velocities, but significantly i

56 rmance, quadriceps muscle contractility, and sciatic nerve conduction velocities.

57 mechanical hyperalgesia, cold allodynia, and sciatic nerve conduction velocity.

58 Electroacupuncture at the sciatic nerve controls systemic inflammation by inducing

59 O mice were subjected to neuropathic pain by sciatic nerve crash injury (SNI).

60 tion was impaired in CLU(-/-) mice following sciatic nerve crush and impaired regeneration nerve fibe

61 upregulated via MEK/ERK signaling and after sciatic nerve crush and Neto2(-/-) neurons from adult mi

62 nd conduction velocities consequent to acute sciatic nerve crush compared with wild-type control anim

63 kine IL-10 in terminating inflammation after sciatic nerve crush injury and promoting regeneration.

64 ution of inflammation and regeneration after sciatic nerve crush injury in mice.

65 Sciatic nerve crush injury in rats induced expression of

66 One day sciatic nerve crush injury triggered a robust increase i

67 nd sensory recovery occurred in mice after a sciatic nerve crush injury, there was little return of m

68 in, retarded early axonal regeneration after sciatic nerve crush injury.

69 Male Sprague Dawley rats were subjected to a sciatic nerve crush under anesthesia and mechanical thre

70 Adult rats with sciatic nerve crush were immediately and systemically in

71 genic mice enhanced locomotor recovery after sciatic nerve crush, associated to an improvement in key

72 After sciatic nerve crush, functional recovery in vivo was ret

73 The resulting mice were challenged with sciatic nerve crush.

74 changes in wild-type and fat-1 mice after a sciatic nerve crush.

75 ation to augment neuroregeneration in both a sciatic nerve cut-and-repair and rat hindlimb transplant

76 Rats undergoing sciatic nerve cut-and-repair and treated with either loc

77 Sciatic nerve CuZnSOD content in SynTgSod1(-/-) mice was

78 jected to unilateral partial ligation of the sciatic nerve developed significant mechanical and therm

79 TAB at a s.c. injection site remote from the sciatic nerve did not result in prolonged sensory-specif

80 erformed miRNA expression profiling of mouse sciatic nerve distal segment after crush injury.

81 d region (3'UTR) landscapes after unilateral sciatic nerve entrapment (SNE) injury in rats.

82 Sciatic nerve entrapment (SNE) injury was used to develo

83 ng in a rat neuropathy induced by unilateral sciatic nerve entrapment (SNE).

84 ties vanished, and the ultrastructure of the sciatic nerve exhibited numerous tomacula and remyelinat

85 l proteins at the nodes of Ranvier of teased sciatic nerve fibers.

86 ntractions were evoked by stimulation of the sciatic nerve for 5 min at 4 Hz and 40 Hz, respectively.

87 blished model of complete transection of the sciatic nerve for comparison, we observed similar but mo

88 ated a mouse model in which a portion of the sciatic nerve from one hind limb was transected at postn

89 and transmission electron microscopy of the sciatic nerve from one of the affected individuals revea

90 The spinal transplants may have effects on sciatic nerve function as well.

91 te was as effective as glucose in supporting sciatic nerve function, and was continuously released in

92 en assessed using von Frey filaments and the sciatic nerve functional index.

93 no significant long-term adverse effects on sciatic nerve functions.

94 supporting the re-innervation across a 10 mm sciatic nerve gap, with results close to that of the aut

95 When injected proximal to the sciatic nerve in mice via intramuscular (i.m.) injection

96 by chronic constriction injury (CCI) of the sciatic nerve in mice, was related to both an increase i

97 Transection of the sciatic nerve in the M83 Tg mice significantly delayed t

98 P was expressed throughout the length of the sciatic nerve in up to 50% of Schwann cells starting 2 w

99 Chronic constriction injury (CCI) of the sciatic nerve induced IL-33 production in the spinal cor

100 uency (0.2 or 3 Hz) stimulation (LFS) to the sciatic nerve induced long-term potentiation (LTP) of C-

101 ory neurons following direct intraganglionic sciatic nerve injection and intraperitoneal and intraven

102 orn would be altered by chronic constriction sciatic nerve injury (CCI).

103 peralgesia using a rat model of constriction sciatic nerve injury (CCI).

104 aviors in WKY rats with chronic constriction sciatic nerve injury (CCI).

105 model of the condition chronic constriction sciatic nerve injury (CCI).

106 ys a critical role in neuropathic pain after sciatic nerve injury and bone cancer in rodents.

107 action against ER stress, in mouse models of sciatic nerve injury and found that ablation of the tran

108 pes in vitro and ameliorate a critical-sized sciatic nerve injury and its associated defects in a mur

109 In this study, an in vivo rat sciatic nerve injury and regeneration model was combined

110 of dorsal root ganglia (DRGs) neurons after sciatic nerve injury in the rat.

111 were protected from hypersensitivity in two sciatic nerve injury models.

112 Sciatic nerve injury triggered generation of two-chain S

113 tage-dependent anion channel 1 (VDAC1) after sciatic nerve injury triggers Schwann cell demyelination

114 In animals with combined spinal cord and sciatic nerve injury, folate-mediated CNS axon regenerat

115 rol mice with anchored PrP, intracerebral or sciatic nerve inoculation resulted in rapid CNS neuroinv

116 ing the same population of neurons following sciatic nerve inoculation.

117 Here, we show that peripheral sciatic nerve lesion in adult mice leads to elevated lev

118 e noxious stimuli, but subsequent to partial sciatic nerve ligation (PNL), KOs did not develop mechan

119 nitiation of neuropathic pain in the partial sciatic nerve ligation (PSNL) mouse model.

120 of behavioral hypersensitivity after partial sciatic nerve ligation (PSNL).

121 Neuropathic pain was induced by partial sciatic nerve ligation (which induces hypersensitivity t

122 s residue (pY12), we now report that partial sciatic nerve ligation increased pY12-K(ir)3.1-immunorea

123 lations of murine models, we have shown that sciatic nerve ligation induces a re-emergence of immatur

124 In the rat sciatic nerve ligation model (ip), (R)-10 (12 mg/kg) pro

125 R-neutralizing antibody to rats with partial sciatic nerve ligation resulted in a delayed onset of ne

126 We found that in mice with partial sciatic nerve ligation, TRPA1 silencing in nociceptors a

127 generate following a dorsal root injury or a sciatic nerve ligation-cut injury and that exposure in v

128 vant) or nerve injury (axotomy; AXO, partial sciatic nerve ligation; PSNL, spinal nerve ligation; SNL

129 Ki 60 nM) and also completely reversed mouse sciatic nerve mechanoallodynia (in vivo, 3 mumol/kg, po)

130 in vitro findings in vivo using a transected sciatic nerve model.

131 cam(20884) mutant is characterized by normal sciatic nerve morphology and a mild electrophysiological

132 cifically colocalize with SMIT1 and SMIT2 at sciatic nerve nodes of Ranvier and in axon initial segme

133 ed into partially denervated branches of the sciatic nerve of adult mice.

134 mary motoneurons in vitro and in vivo in the sciatic nerve of adult WT and mutant SBMA mice demonstra

135 pathic pain was induced by cuffing the right sciatic nerve of C57BL/6J mice.

136 tudied mitochondrial transport in the intact sciatic nerve of living mice and analyzed axonal mitocho

137 toxin B (CTB) into the gastrocnemius muscle/sciatic nerve of SOD1 rats before disease onset and also

138 ice after chronic constriction injury of the sciatic nerve of the left hindpaw and observed a strikin

139 ticularly in the spinal cord, but not in the sciatic nerve of the PNS.

140 nodal expression of selected isoforms in the sciatic nerve of the transgenic mouse Oct6(DeltaSCE/beta

141 The sciatic nerve of these cytNmnat1 transgenic mice was tra

142 sociated protein tau, in the spinal cord and sciatic nerve of wild-type (WT) and SBMA mice at various

143 ssion was upregulated in the spinal cord and sciatic nerve of WT mice.

144 The sciatic nerve or its branches was most commonly affected

145 Like acute hypoxia, stimulation of the sciatic nerve or the nasal mucosa evoked greater increas

146 ic with streptozotocin displayed less severe sciatic nerve oxidative-nitrative stress and peripheral

147 n, whereas wild-type mice developed complete sciatic nerve paralysis due to massive CPNI.

148 of Angiotensin II receptor types in the rat sciatic nerve pathway, including L(4)-L(5) spinal cord s

149 ice, and no evidence of CGRP upregulation in sciatic nerve post-CCI was found.

150 Transection of the sciatic nerve prior to hind-limb inoculation diminished

151 eported that myelin clearance in the injured sciatic nerve proceeds unhindered in the Ccr2(-/-) mouse

152 tion of 6 mul of 150 mum ATP into female rat sciatic nerve quadrupled the number of axons growing int

153 ined GSK3 activity show markedly accelerated sciatic nerve regeneration.

154 here that genetic deletion of BACE1 affects sciatic nerve remyelination.

155 e (M6P), a scar reducing agent, to a site of sciatic nerve repair.

156 roduced by placing a plastic cuff around the sciatic nerve resolved within several days when the cuff

157 Molecular interrogation of the sciatic nerve reveals that aged Schwann cells (SCs) fail

158 iation of pain along the distribution of the sciatic nerve should always be looked for if abnormaliti

159 t this time, ultrastructural analysis of the sciatic nerve showed de- and dysmyelination of fibers, w

160 sthetised, ventilated dogs were elicited via sciatic nerve stimulation (50 Hz; 200 ms duration; 1 con

161 TP) following conditioning by high-frequency sciatic nerve stimulation (HFS) at intensities recruitin

162 oration of direct muscle responses evoked by sciatic nerve stimulation to pretransection levels over

163 ns (50 Hz; 200 ms duration) via supramaximal sciatic nerve stimulation were used to manipulate metabo

164 -positive vesicular clusters in axons in the sciatic nerve suggest that this denervation results from

165 Local lidocaine was applied to the left sciatic nerve to block both orthodromic signals and anti

166 and its subsequent retrograde transport via sciatic nerve to DRG.

167 pia and arachnoid meninges as well as in the sciatic nerve to mimic central and peripheral schwannoma

168 ent unilateral electrical stimulation of the sciatic nerve to mimic resistance exercise in the tibial

169 glycoprotein gp120 application onto the rat sciatic nerve to test the role of phosphorylated C/EBPbe

170 ontrol and injury conditions at 3 days after sciatic nerve transection (SNT).

171 d time course in a more severe injury model, sciatic nerve transection and reanastamosis.

172 OL and tibialis anterior (TA) function after sciatic nerve transection and repair.

173 Sciatic nerve transection induced upregulation of OPN an

174 ttern nor the number of neurons changed in a sciatic nerve transection model of neuropathic pain or i

175 as applied to bridge injured nerves in a rat sciatic nerve transection model.

176 As expected, sciatic nerve transection triggered WGA expression in NP

177 re was little return of motor function after sciatic nerve transection, because of the delay in motor

178 pacity to disseminate to the brain following sciatic nerve transection, indicating that wild-type reo

179 Sciatic nerve transections and repairs were performed in

180 After CCI, upregulation of CD11b in sciatic nerve was less in GFAP-IkappaBalpha-dn mice comp

181 hronic constriction injury (CCI) of the left sciatic nerve was performed on wild type (WT) and GFAP-I

182 following chronic constriction injury of the sciatic nerve was rapidly and dose-dependently reversed

183 In anaesthetised C57-Black-6 mice, the left sciatic nerve was sectioned and repaired using 4 epineur

184 After control data collection, the sciatic nerve was transected and repaired and the rat wa

185 unoprecipitation analysis of the myelinating sciatic nerve was used to show developmental association

186 ata for 24-week-old BKS db/db and db/+ mouse sciatic nerve were analyzed to define significantly diff

187 cultures of purified SCs from newborn mouse sciatic nerve were used to characterize both the role of

188 as confirmed by histological analysis of the sciatic nerve which showed predominantly axonal damage:

189 ignals were recorded simultaneously from the sciatic nerve with a 16-contact cuff electrode.

190 estigate this, we inoculated hamsters in the sciatic nerve with long-incubation-period strain 139H pr

191 l of strain interference, inoculation of the sciatic nerve with the drowsy (DY) strain of the transmi

192 We cared for a child with sciatic nerve zoster who had severe pain over the lower

193 DTI-MRN covered proximal (sciatic nerve) and distal (tibial nerve) nerve segments

194 ring rate and stimulus-evoked (brush, pinch, sciatic nerve) responses were markedly enhanced as were

195 e neurons (CSNs) in DRGs contributing to the sciatic nerve, and a decrease in their cold temperature

196 al day 7 onward in motoneurons, axons in the sciatic nerve, and axon terminals of the neuromuscular j

197 assays, immunohistochemistry with teased rat sciatic nerve, and immunoabsorption experiments.

198 ior cervical and dorsal root ganglion cells, sciatic nerve, and in adrenal glands, but its expression

199 uff is well-tolerated, delivers light to the sciatic nerve, and optically stimulates muscle in freely

200 e the location of the FAAH in adult rat DRG, sciatic nerve, and spinal cord.

201 e points in CMT2D mouse muscles, retina, and sciatic nerve, as well as in embryonic hindbrain.

202 thic pain in the hind paw upon injury to the sciatic nerve, but the abnormal pain states were short l

203 muscle with and was seen tracking along the sciatic nerve, explaining pain along the distribution of

204 more than 100 O-GlcNAcylated proteins in rat sciatic nerve, including Periaxin (PRX), a myelin protei

205 Glycogen was present in sciatic nerve, its concentration varying directly with a

206 We demonstrate that in sciatic nerve, myocilin is expressed in Schwann cells wi

207 In mouse sciatic nerve, myosin-1d is expressed along the axon and

208 ion in the leg along the distribution of the sciatic nerve, secondary to compression or irritation of

209 n response was identified in neural tissues (sciatic nerve, spinal cord) of streptozotocin diabetic r

210 Four weeks after axonal crush of sciatic nerve, TDP-43 transgenic mice remained paralyzed

211 Zoster of the sciatic nerve, the longest nerve in the human body, is a

212 The infection spread to the sciatic nerve, the spinal cord, and the brain, causing p

213 After ligation of the sciatic nerve, there was a high accumulation of AT(1) re

214 to transection and repair of the entire rat sciatic nerve, to attempt to influence the misdirection

215 of perineural HIV gp120 application onto the sciatic nerve, we found that pC/EBPbeta was triggered by

216 onal transport in single axons in the intact sciatic nerve, we have identified clear axonal transport

217 l neurons and peripheral nerves, such as the sciatic nerve, which have provided most of our informati

218 ort the concept that HFS conditioning of the sciatic nerve, which leads to spinal LTP, is associated

219 r perianal abscess causing irritation of the sciatic nerve, which was diagnosed on MRI of the lumbosa

220 we observed galanin upregulation in DRG and sciatic nerve, which was less in GFAP-IkappaBalpha-dn mi

221 ster of differentiation 45 expression in the sciatic nerve, with no difference between genotypes.

222 of circulating CX3CR1(+) monocytes into the sciatic nerve.

223 s following extraction of the Xenopus laevis sciatic nerve.

224 mes greater at the spinal column than at the sciatic nerve.

225 ronic constriction injury (CCI) model of the sciatic nerve.

226 nance of myelination and nodes of Ranvier in sciatic nerve.

227 , as occurs with conditioning lesions of the sciatic nerve.

228 of a polyethylene cuff placed around in the sciatic nerve.

229 he dorsal root ganglion (DRG) and the distal sciatic nerve.

230 ll proliferation is upregulated in Chd4-null sciatic nerve.

231 following chronic constriction injury of the sciatic nerve.

232 r after a chronic constriction injury of the sciatic nerve.

233 athologic changes in dorsal root ganglia and sciatic nerve.

234 s created by spared-nerve injury of the left sciatic nerve.

235 s recorded from isolated preparations of rat sciatic nerve.

236 les throughout the endoneurium of the mutant sciatic nerve.

237 f living or fixed myelinated fibers in mouse sciatic nerve.

238 d with ubiquitin loss in the spinal cord and sciatic nerve.

239 onents of syd vesicles within axons of mouse sciatic nerve.

240 was axonally transported to the DRG via the sciatic nerve.

241 transection and surgical repair of the mouse sciatic nerve.

242 esponding dorsal root ganglia (DRGs) and the sciatic nerve.

243 xplaining pain along the distribution of the sciatic nerve.

244 econdary to compression or irritation of the sciatic nerve.

245 ecorded spike activity from the regenerating sciatic nerve.

246 lammatory macrophages, was upregulated in KO sciatic nerve.

247 in response to electrical stimulation of the sciatic nerve.

248 implant is sutured between the stumps of the sciatic nerve.

249 uated the levels of sulfasalazine targets in sciatic nerves and dorsal root ganglia (DRG) of treated

250 Along with the behavioral findings, sciatic nerves and DRG from sulfasalazine-treated diabet

251 ceptive neurons, increased CGRP release from sciatic nerves and DRGs, and a reduction in mechanical a

252 se reduces NMN accumulation in injured mouse sciatic nerves and preserves some axons for up to three

253 amined pathology and cell differentiation in sciatic nerves and ventral roots of the laminin-alpha2-d

254 r3 knockdown attenuates myelination in mouse sciatic nerves as well as in zebrafish.

255 ponse to the loss of CCR2, injured Ccr2(-/-) sciatic nerves demonstrate prolonged expression of neutr

256 Both autoradiography analysis of sciatic nerves excised from injured rats as well as whol

257 electron microscopy, we show that injury of sciatic nerves from mice of either sex triggers extensiv

258 f gene expression arrays of large myelinated sciatic nerves from pioglitazone-treated animals reveale

259 In response to crush injury, sciatic nerves in scLRP1(-/-) mice showed accelerated de

260 elow the incisures in the single mutants, in sciatic nerves of caspr(-/-)/caspr2(-/-) mice, these cha

261 oid precursor protein accumulated in ligated sciatic nerves of control and eribulin-treated mice, but

262 s found to be carbonylated and aggregated in sciatic nerves of dbdb mice.

263 We found that, compared with wild-type, sciatic nerves of Lama2(-/-) mice had a significant incr

264 vels of Th1 cytokines, CXCL10, and RANTES in sciatic nerves of mice that developed SAP.

265 Sciatic nerves of myocilin null mice express reduced lev

266 Attenuated HSV (NV1023) injected to sciatic nerves of nude mice had no toxic effect on nerve

267 Mtmr13 loss leads to axonal degeneration in sciatic nerves of older mice.

268 Electron microscopy of sciatic nerves of paclitaxel-treated mice showed reduced

269 flow, and capillary density were reduced in sciatic nerves of streptozotocin-induced diabetic mice b

270 Electron microscopic analysis of sciatic nerves showed a reduction in the number of neuro

271 ication of its target mRNAs in vivo in mouse sciatic nerves using ribonomics showed an enrichment of

272 appeared that myelin sheath thickness in the sciatic nerves was not increased in BACE1(-/-)/Akt-DD mi

273 ted tomography (CT)-guided IRE of 11 porcine sciatic nerves was performed in nine pigs, and histopath

274 In addition, acute RFA of six porcine sciatic nerves was performed in six pigs that were harve

275 conduction velocities of the trigeminal and sciatic nerves were decreased.

276 lidate the model experimentally, excised rat sciatic nerves were subjected to stretching, which induc

277 Injection of MMP-9-PEX into sciatic nerves, 24 h after crush injury, robustly increa

278 duced at protein levels in both Erbin mutant sciatic nerves, and ErbB2 became unstable and NRG1 signa

279 In sciatic nerves, application of extracellular QX-314 with

280 was injected directly into crush-injured rat sciatic nerves, ERK1/2 phosphorylation was observed in m

281 K3-mediated CRMP2 inhibition was detected in sciatic nerves, thus revealing a fundamental difference

282 n in the conduction velocity along the adult sciatic nerves.

283 ssion of the T cell chemoattractant IP-10 in sciatic nerves.

284 sed numbers of large-diameter axons in their sciatic nerves.

285 doxycycline-treated compared with untreated sciatic nerves.

286 cell co-cultures and in vivo, in developing sciatic nerves.

287 preferentially and durably engrafted in the sciatic nerves.

288 -N-SH cells and on axonal transport in mouse sciatic nerves.

289 b weakness and impaired axonal conduction in sciatic nerves.

290 r roots, as well as in the femoral motor and sciatic nerves.

291 nociceptive C-fibers in the Remak bundles of sciatic nerves; however, there was no loss of NMSCs that

292 When joints were immobilized by sciatic neurectomy, regulation of selected genes was abr

293 ilized, either by prolonged anesthesia or by sciatic neurectomy.

294 a single injection immediately distal to the sciatic notch.

295 ts (n = 258) with lumbar disc herniation and sciatic pain, all European-Caucasian, were recruited fro

296 at eight standardized sites adjacent to the sciatic, peroneal, and tibial nerves of one leg.

297 sphincter dysfunction, partially resembling sciatic symptoms.

298 Sciatic-to-spinal DCS caused opposite effects.

299 t current flowing in the opposite direction (sciatic-to-spinal DCS) would excite spinal motor neurons

300 anglia, an increase in the peripheral nerve (sciatic) yet no change in the central root.