ライフサイエンスコーパス: sciatic nerve

1 of circulating CX3CR1(+) monocytes into the sciatic nerve.

2 s following extraction of the Xenopus laevis sciatic nerve.

3 mes greater at the spinal column than at the sciatic nerve.

4 ronic constriction injury (CCI) model of the sciatic nerve.

5 nance of myelination and nodes of Ranvier in sciatic nerve.

6 , as occurs with conditioning lesions of the sciatic nerve.

7 of a polyethylene cuff placed around in the sciatic nerve.

8 he dorsal root ganglion (DRG) and the distal sciatic nerve.

9 ll proliferation is upregulated in Chd4-null sciatic nerve.

10 following chronic constriction injury of the sciatic nerve.

11 r after a chronic constriction injury of the sciatic nerve.

12 athologic changes in dorsal root ganglia and sciatic nerve.

13 s created by spared-nerve injury of the left sciatic nerve.

14 s recorded from isolated preparations of rat sciatic nerve.

15 les throughout the endoneurium of the mutant sciatic nerve.

16 f living or fixed myelinated fibers in mouse sciatic nerve.

17 d with ubiquitin loss in the spinal cord and sciatic nerve.

18 onents of syd vesicles within axons of mouse sciatic nerve.

19 was axonally transported to the DRG via the sciatic nerve.

20 transection and surgical repair of the mouse sciatic nerve.

21 esponding dorsal root ganglia (DRGs) and the sciatic nerve.

22 Ricinus communis agglutinin (RCA) I into the sciatic nerve.

23 xplaining pain along the distribution of the sciatic nerve.

24 econdary to compression or irritation of the sciatic nerve.

25 ecorded spike activity from the regenerating sciatic nerve.

26 lammatory macrophages, was upregulated in KO sciatic nerve.

27 in response to electrical stimulation of the sciatic nerve.

28 implant is sutured between the stumps of the sciatic nerve.

29 n in the conduction velocity along the adult sciatic nerves.

30 ssion of the T cell chemoattractant IP-10 in sciatic nerves.

31 sed numbers of large-diameter axons in their sciatic nerves.

32 doxycycline-treated compared with untreated sciatic nerves.

33 cell co-cultures and in vivo, in developing sciatic nerves.

34 preferentially and durably engrafted in the sciatic nerves.

35 transplantation on microsurgically repaired sciatic nerves.

36 -N-SH cells and on axonal transport in mouse sciatic nerves.

37 b weakness and impaired axonal conduction in sciatic nerves.

38 r roots, as well as in the femoral motor and sciatic nerves.

39 Diabetic wild-type mice displayed increased sciatic nerve 12/15-lipoxygenase and 12(S)-hydroxyeicosa

40 Injection of MMP-9-PEX into sciatic nerves, 24 h after crush injury, robustly increa

41 Here, we report that sciatic nerve activation with electroacupuncture control

42 treatment also promoted remyelination of the sciatic nerve after crush.

43 A conditioning lesion of the sciatic nerve allows the central processes of dorsal roo

44 ne; (2) application of lidocaine in the left sciatic nerve alone significantly increased BP ipsilater

45 egeneration in dorsal root ganglia (DRG) and sciatic nerve and abundance of Schwann cells.

46 ions in axonal processes in the spinal cord, sciatic nerve and brain, but no excess of multivesicular

47 ere found in increased concentrations in the sciatic nerve and dorsal root ganglia of oxaliplatin tre

48 ve conduction velocity, nerve amplitude, and sciatic nerve and dorsal root ganglion morphology at 0.2

49 lso displayed a unique prolonged exposure in sciatic nerve and DRG.

50 ally accumulates in LRs in the TWI brain and sciatic nerve and in samples from brains of human Krabbe

51 ory components was evaluated in axons of the sciatic nerve and in spinal nerve axons after in vivo el

52 ssociated with a persistent M1 milieu in the sciatic nerve and in the regional and systemic lymphatic

53 having reliable signal acquisitions from the sciatic nerve and its branches such as the peroneal nerv

54 a small proportion of fibers that constitute sciatic nerve and its branches, but importantly breaks t

55 nction, intraepidermal nerve fiber loss, and sciatic nerve and spinal cord oxidative-nitrative stress

56 nduced by chronic constriction injury of the sciatic nerve and suppressed nerve injury-induced activa

57 rature on both the in-vitro stability of rat sciatic nerve and the response of the nerve to acute hyp

58 lly well in the lymph node, infection of the sciatic nerve and the rest of the nervous system was imp

59 which the first stimulus originated from the sciatic nerve and the second from the motor cortex.

60 which the first stimulus originated from the sciatic nerve and the second from the spinal cord; and (

61 ing required to prevent axonal damage of the sciatic nerve and to terminate the P0106-125-specific im

62 mmunohistological staining were performed in sciatic nerve and/or L4-L5 DRG tissue for galanin, CGRP

63 uated the levels of sulfasalazine targets in sciatic nerves and dorsal root ganglia (DRG) of treated

64 Along with the behavioral findings, sciatic nerves and DRG from sulfasalazine-treated diabet

65 ceptive neurons, increased CGRP release from sciatic nerves and DRGs, and a reduction in mechanical a

66 se reduces NMN accumulation in injured mouse sciatic nerves and preserves some axons for up to three

67 amined pathology and cell differentiation in sciatic nerves and ventral roots of the laminin-alpha2-d

68 DTI-MRN covered proximal (sciatic nerve) and distal (tibial nerve) nerve segments

69 e neurons (CSNs) in DRGs contributing to the sciatic nerve, and a decrease in their cold temperature

70 al day 7 onward in motoneurons, axons in the sciatic nerve, and axon terminals of the neuromuscular j

71 assays, immunohistochemistry with teased rat sciatic nerve, and immunoabsorption experiments.

72 ior cervical and dorsal root ganglion cells, sciatic nerve, and in adrenal glands, but its expression

73 uff is well-tolerated, delivers light to the sciatic nerve, and optically stimulates muscle in freely

74 e the location of the FAAH in adult rat DRG, sciatic nerve, and spinal cord.

75 duced at protein levels in both Erbin mutant sciatic nerves, and ErbB2 became unstable and NRG1 signa

76 In sciatic nerves, application of extracellular QX-314 with

77 nastomosed to recipient femoral vessels, the sciatic nerve approximated end-to-end and osteosynthesis

78 We used a chronic constriction injury of sciatic nerve as a model of neuropathic pain in male Spr

79 tivity, and morphological alterations in the sciatic nerve as evidenced by decrease in g-ratio; it al

80 r3 knockdown attenuates myelination in mouse sciatic nerves as well as in zebrafish.

81 e points in CMT2D mouse muscles, retina, and sciatic nerve, as well as in embryonic hindbrain.

82 Analysis of the sciatic nerve at 11 d after nerve crush showed that the

83 cord, and, during their apoptosis induced by sciatic nerve avulsion, nuclear and cytoplasmic 5-methyl

84 otyping after selectively silencing TRPV1(+) sciatic nerve axons by perineural injections of QX-314 a

85 se, live imaging of endosomal trafficking in sciatic nerve axons reveals disease-induced deficits in

86 , degenerating) nerve stumps on day 1 in the sciatic nerve axotomy model in rats.

87 After sciatic nerve axotomy, there was a rightward shift in th

88 combinations of the above compounds produced sciatic nerve blockade lasting up to 7.5 days (with STX

89 d lamellar inclusion material in the kidney, sciatic nerve, brain and spinal cord tissues.

90 thic pain in the hind paw upon injury to the sciatic nerve, but the abnormal pain states were short l

91 r was generated and delivered into the mouse sciatic nerve by a single injection immediately distal t

92 vivo chromatin immunoprecipitation (ChIP) of sciatic nerve cells revealed a Sox10 binding site upstre

93 ng techniques, we have used a rodent partial sciatic nerve chronic constriction injury model (n = 5-8

94 Sciatic nerve conditioning at A-delta fiber strength, kn

95 elinated nerve fibers, specifically sural or sciatic nerve conduction velocities, but significantly i

96 rmance, quadriceps muscle contractility, and sciatic nerve conduction velocities.

97 mechanical hyperalgesia, cold allodynia, and sciatic nerve conduction velocity.

98 Electroacupuncture at the sciatic nerve controls systemic inflammation by inducing

99 O mice were subjected to neuropathic pain by sciatic nerve crash injury (SNI).

100 tion was impaired in CLU(-/-) mice following sciatic nerve crush and impaired regeneration nerve fibe

101 upregulated via MEK/ERK signaling and after sciatic nerve crush and Neto2(-/-) neurons from adult mi

102 nd conduction velocities consequent to acute sciatic nerve crush compared with wild-type control anim

103 kine IL-10 in terminating inflammation after sciatic nerve crush injury and promoting regeneration.

104 ution of inflammation and regeneration after sciatic nerve crush injury in mice.

105 Sciatic nerve crush injury in rats induced expression of

106 One day sciatic nerve crush injury triggered a robust increase i

107 nd sensory recovery occurred in mice after a sciatic nerve crush injury, there was little return of m

108 in, retarded early axonal regeneration after sciatic nerve crush injury.

109 Male Sprague Dawley rats were subjected to a sciatic nerve crush under anesthesia and mechanical thre

110 Adult rats with sciatic nerve crush were immediately and systemically in

111 genic mice enhanced locomotor recovery after sciatic nerve crush, associated to an improvement in key

112 After sciatic nerve crush, functional recovery in vivo was ret

113 The resulting mice were challenged with sciatic nerve crush.

114 changes in wild-type and fat-1 mice after a sciatic nerve crush.

115 ation to augment neuroregeneration in both a sciatic nerve cut-and-repair and rat hindlimb transplant

116 Rats undergoing sciatic nerve cut-and-repair and treated with either loc

117 Sciatic nerve CuZnSOD content in SynTgSod1(-/-) mice was

118 contribute to cellular reorganization after sciatic nerve damage.

119 ponse to the loss of CCR2, injured Ccr2(-/-) sciatic nerves demonstrate prolonged expression of neutr

120 jected to unilateral partial ligation of the sciatic nerve developed significant mechanical and therm

121 TAB at a s.c. injection site remote from the sciatic nerve did not result in prolonged sensory-specif

122 erformed miRNA expression profiling of mouse sciatic nerve distal segment after crush injury.

123 d region (3'UTR) landscapes after unilateral sciatic nerve entrapment (SNE) injury in rats.

124 Sciatic nerve entrapment (SNE) injury was used to develo

125 ng in a rat neuropathy induced by unilateral sciatic nerve entrapment (SNE).

126 was injected directly into crush-injured rat sciatic nerves, ERK1/2 phosphorylation was observed in m

127 Both autoradiography analysis of sciatic nerves excised from injured rats as well as whol

128 ties vanished, and the ultrastructure of the sciatic nerve exhibited numerous tomacula and remyelinat

129 The sciatic nerve exhibits reduced numbers of large-diameter

130 muscle with and was seen tracking along the sciatic nerve, explaining pain along the distribution of

131 l proteins at the nodes of Ranvier of teased sciatic nerve fibers.

132 ntractions were evoked by stimulation of the sciatic nerve for 5 min at 4 Hz and 40 Hz, respectively.

133 blished model of complete transection of the sciatic nerve for comparison, we observed similar but mo

134 ated a mouse model in which a portion of the sciatic nerve from one hind limb was transected at postn

135 and transmission electron microscopy of the sciatic nerve from one of the affected individuals revea

136 electron microscopy, we show that injury of sciatic nerves from mice of either sex triggers extensiv

137 f gene expression arrays of large myelinated sciatic nerves from pioglitazone-treated animals reveale

138 The spinal transplants may have effects on sciatic nerve function as well.

139 te was as effective as glucose in supporting sciatic nerve function, and was continuously released in

140 en assessed using von Frey filaments and the sciatic nerve functional index.

141 no significant long-term adverse effects on sciatic nerve functions.

142 supporting the re-innervation across a 10 mm sciatic nerve gap, with results close to that of the aut

143 concomitant delivery of HIV-gp120 to the rat sciatic nerve (gp120+ddC).

144 high-frequency electrical stimulation of the sciatic nerve (HFES) or injection of AICAR, an activator

145 l injection of QX-314 and capsaicin near the sciatic nerve; however, in contrast to the effect of lid

146 nociceptive C-fibers in the Remak bundles of sciatic nerves; however, there was no loss of NMSCs that

147 When injected proximal to the sciatic nerve in mice via intramuscular (i.m.) injection

148 by chronic constriction injury (CCI) of the sciatic nerve in mice, was related to both an increase i

149 Transection of the sciatic nerve in the M83 Tg mice significantly delayed t

150 P was expressed throughout the length of the sciatic nerve in up to 50% of Schwann cells starting 2 w

151 In response to crush injury, sciatic nerves in scLRP1(-/-) mice showed accelerated de

152 more than 100 O-GlcNAcylated proteins in rat sciatic nerve, including Periaxin (PRX), a myelin protei

153 sensory digital nerve conduction velocities, sciatic nerve indexes of oxidative stress, prostaglandin

154 Chronic constriction injury (CCI) of the sciatic nerve induced IL-33 production in the spinal cor

155 uency (0.2 or 3 Hz) stimulation (LFS) to the sciatic nerve induced long-term potentiation (LTP) of C-

156 ory neurons following direct intraganglionic sciatic nerve injection and intraperitoneal and intraven

157 ic tracer wheat germ agglutinin which, after sciatic nerve injection, labels all unmyelinated nocicep

158 orn would be altered by chronic constriction sciatic nerve injury (CCI).

159 peralgesia using a rat model of constriction sciatic nerve injury (CCI).

160 aviors in WKY rats with chronic constriction sciatic nerve injury (CCI).

161 model of the condition chronic constriction sciatic nerve injury (CCI).

162 ys a critical role in neuropathic pain after sciatic nerve injury and bone cancer in rodents.

163 action against ER stress, in mouse models of sciatic nerve injury and found that ablation of the tran

164 pes in vitro and ameliorate a critical-sized sciatic nerve injury and its associated defects in a mur

165 In this study, an in vivo rat sciatic nerve injury and regeneration model was combined

166 of dorsal root ganglia (DRGs) neurons after sciatic nerve injury in the rat.

167 were protected from hypersensitivity in two sciatic nerve injury models.

168 Sciatic nerve injury triggered generation of two-chain S

169 tage-dependent anion channel 1 (VDAC1) after sciatic nerve injury triggers Schwann cell demyelination

170 In animals with combined spinal cord and sciatic nerve injury, folate-mediated CNS axon regenerat

171 rol mice with anchored PrP, intracerebral or sciatic nerve inoculation resulted in rapid CNS neuroinv

172 ing the same population of neurons following sciatic nerve inoculation.

173 Glycogen was present in sciatic nerve, its concentration varying directly with a

174 Here, we show that peripheral sciatic nerve lesion in adult mice leads to elevated lev

175 e noxious stimuli, but subsequent to partial sciatic nerve ligation (PNL), KOs did not develop mechan

176 Partial sciatic nerve ligation (pSNL) markedly increased glial f

177 nitiation of neuropathic pain in the partial sciatic nerve ligation (PSNL) mouse model.

178 of behavioral hypersensitivity after partial sciatic nerve ligation (PSNL).

179 Neuropathic pain was induced by partial sciatic nerve ligation (which induces hypersensitivity t

180 s residue (pY12), we now report that partial sciatic nerve ligation increased pY12-K(ir)3.1-immunorea

181 lations of murine models, we have shown that sciatic nerve ligation induces a re-emergence of immatur

182 In the rat sciatic nerve ligation model (ip), (R)-10 (12 mg/kg) pro

183 R-neutralizing antibody to rats with partial sciatic nerve ligation resulted in a delayed onset of ne

184 However, sciatic nerve ligation studies reveal that inhibition of

185 We found that in mice with partial sciatic nerve ligation, TRPA1 silencing in nociceptors a

186 generate following a dorsal root injury or a sciatic nerve ligation-cut injury and that exposure in v

187 vant) or nerve injury (axotomy; AXO, partial sciatic nerve ligation; PSNL, spinal nerve ligation; SNL

188 Ki 60 nM) and also completely reversed mouse sciatic nerve mechanoallodynia (in vivo, 3 mumol/kg, po)

189 in vitro findings in vivo using a transected sciatic nerve model.

190 cam(20884) mutant is characterized by normal sciatic nerve morphology and a mild electrophysiological

191 We demonstrate that in sciatic nerve, myocilin is expressed in Schwann cells wi

192 In mouse sciatic nerve, myosin-1d is expressed along the axon and

193 cifically colocalize with SMIT1 and SMIT2 at sciatic nerve nodes of Ranvier and in axon initial segme

194 ed into partially denervated branches of the sciatic nerve of adult mice.

195 mary motoneurons in vitro and in vivo in the sciatic nerve of adult WT and mutant SBMA mice demonstra

196 pathic pain was induced by cuffing the right sciatic nerve of C57BL/6J mice.

197 tudied mitochondrial transport in the intact sciatic nerve of living mice and analyzed axonal mitocho

198 toxin B (CTB) into the gastrocnemius muscle/sciatic nerve of SOD1 rats before disease onset and also

199 ice after chronic constriction injury of the sciatic nerve of the left hindpaw and observed a strikin

200 ticularly in the spinal cord, but not in the sciatic nerve of the PNS.

201 nodal expression of selected isoforms in the sciatic nerve of the transgenic mouse Oct6(DeltaSCE/beta

202 The sciatic nerve of these cytNmnat1 transgenic mice was tra

203 sociated protein tau, in the spinal cord and sciatic nerve of wild-type (WT) and SBMA mice at various

204 ssion was upregulated in the spinal cord and sciatic nerve of WT mice.

205 elow the incisures in the single mutants, in sciatic nerves of caspr(-/-)/caspr2(-/-) mice, these cha

206 oid precursor protein accumulated in ligated sciatic nerves of control and eribulin-treated mice, but

207 s found to be carbonylated and aggregated in sciatic nerves of dbdb mice.

208 e treatment also increased inosine levels in sciatic nerves of diabetic rats.

209 We found that, compared with wild-type, sciatic nerves of Lama2(-/-) mice had a significant incr

210 vels of Th1 cytokines, CXCL10, and RANTES in sciatic nerves of mice that developed SAP.

211 Sciatic nerves of myocilin null mice express reduced lev

212 Attenuated HSV (NV1023) injected to sciatic nerves of nude mice had no toxic effect on nerve

213 y implanting prostate carcinoma cells in the sciatic nerves of nude mice.

214 Mtmr13 loss leads to axonal degeneration in sciatic nerves of older mice.

215 Electron microscopy of sciatic nerves of paclitaxel-treated mice showed reduced

216 flow, and capillary density were reduced in sciatic nerves of streptozotocin-induced diabetic mice b

217 The sciatic nerve or its branches was most commonly affected

218 Like acute hypoxia, stimulation of the sciatic nerve or the nasal mucosa evoked greater increas

219 ic with streptozotocin displayed less severe sciatic nerve oxidative-nitrative stress and peripheral

220 n, whereas wild-type mice developed complete sciatic nerve paralysis due to massive CPNI.

221 of Angiotensin II receptor types in the rat sciatic nerve pathway, including L(4)-L(5) spinal cord s

222 ice, and no evidence of CGRP upregulation in sciatic nerve post-CCI was found.

223 Transection of the sciatic nerve prior to hind-limb inoculation diminished

224 eported that myelin clearance in the injured sciatic nerve proceeds unhindered in the Ccr2(-/-) mouse

225 tion of 6 mul of 150 mum ATP into female rat sciatic nerve quadrupled the number of axons growing int

226 ined GSK3 activity show markedly accelerated sciatic nerve regeneration.

227 here that genetic deletion of BACE1 affects sciatic nerve remyelination.

228 e (M6P), a scar reducing agent, to a site of sciatic nerve repair.

229 roduced by placing a plastic cuff around the sciatic nerve resolved within several days when the cuff

230 ring rate and stimulus-evoked (brush, pinch, sciatic nerve) responses were markedly enhanced as were

231 Molecular interrogation of the sciatic nerve reveals that aged Schwann cells (SCs) fail

232 ion in the leg along the distribution of the sciatic nerve, secondary to compression or irritation of

233 iation of pain along the distribution of the sciatic nerve should always be looked for if abnormaliti

234 t this time, ultrastructural analysis of the sciatic nerve showed de- and dysmyelination of fibers, w

235 Electron microscopic analysis of sciatic nerves showed a reduction in the number of neuro

236 rsisted in the dorsal root ganglia (DRG) and sciatic nerve (SN) for up to 72 hours.

237 egrated proteomics and metabolomics from the sciatic nerve (SN), the lumbar 4/5 dorsal root ganglia (

238 Western blot analysis of 1 month compressed sciatic nerve specimens.

239 ) direct current flowing from spinal cord to sciatic nerve [spinal-to-sciatic direct current stimulat

240 n response was identified in neural tissues (sciatic nerve, spinal cord) of streptozotocin diabetic r

241 sthetised, ventilated dogs were elicited via sciatic nerve stimulation (50 Hz; 200 ms duration; 1 con

242 TP) following conditioning by high-frequency sciatic nerve stimulation (HFS) at intensities recruitin

243 at engage EAA afferents to the LC, including sciatic nerve stimulation and auditory stimuli and the t

244 oration of direct muscle responses evoked by sciatic nerve stimulation to pretransection levels over

245 ns (50 Hz; 200 ms duration) via supramaximal sciatic nerve stimulation were used to manipulate metabo

246 -positive vesicular clusters in axons in the sciatic nerve suggest that this denervation results from

247 sed by approximately 50% in Mtmr13-deficient sciatic nerves, suggesting a mode of Mtmr2 regulation.

248 Four weeks after axonal crush of sciatic nerve, TDP-43 transgenic mice remained paralyzed

249 In developing perinatal rat sciatic nerve, the kinetics of p65 phosphorylation at S2

250 Zoster of the sciatic nerve, the longest nerve in the human body, is a

251 The infection spread to the sciatic nerve, the spinal cord, and the brain, causing p

252 After ligation of the sciatic nerve, there was a high accumulation of AT(1) re

253 K3-mediated CRMP2 inhibition was detected in sciatic nerves, thus revealing a fundamental difference

254 e intensity of lipid ions in spinal cord and sciatic nerve tissues from rats exposed to DCA.

255 Local lidocaine was applied to the left sciatic nerve to block both orthodromic signals and anti

256 and its subsequent retrograde transport via sciatic nerve to DRG.

257 pia and arachnoid meninges as well as in the sciatic nerve to mimic central and peripheral schwannoma

258 ent unilateral electrical stimulation of the sciatic nerve to mimic resistance exercise in the tibial

259 glycoprotein gp120 application onto the rat sciatic nerve to test the role of phosphorylated C/EBPbe

260 to transection and repair of the entire rat sciatic nerve, to attempt to influence the misdirection

261 ontrol and injury conditions at 3 days after sciatic nerve transection (SNT).

262 d time course in a more severe injury model, sciatic nerve transection and reanastamosis.

263 OL and tibialis anterior (TA) function after sciatic nerve transection and repair.

264 Sciatic nerve transection induced upregulation of OPN an

265 ttern nor the number of neurons changed in a sciatic nerve transection model of neuropathic pain or i

266 as applied to bridge injured nerves in a rat sciatic nerve transection model.

267 As expected, sciatic nerve transection triggered WGA expression in NP

268 re was little return of motor function after sciatic nerve transection, because of the delay in motor

269 pacity to disseminate to the brain following sciatic nerve transection, indicating that wild-type reo

270 Following sciatic nerve transection, the expression of these prote

271 coexpressing neurons are lost 25 days after sciatic nerve transection.

272 Sciatic nerve transections and repairs were performed in

273 ication of its target mRNAs in vivo in mouse sciatic nerves using ribonomics showed an enrichment of

274 After CCI, upregulation of CD11b in sciatic nerve was less in GFAP-IkappaBalpha-dn mice comp

275 To visualize transport of SP, the sciatic nerve was ligated ipsilateral to formalin inject

276 hronic constriction injury (CCI) of the left sciatic nerve was performed on wild type (WT) and GFAP-I

277 following chronic constriction injury of the sciatic nerve was rapidly and dose-dependently reversed

278 In anaesthetised C57-Black-6 mice, the left sciatic nerve was sectioned and repaired using 4 epineur

279 After control data collection, the sciatic nerve was transected and repaired and the rat wa

280 unoprecipitation analysis of the myelinating sciatic nerve was used to show developmental association

281 appeared that myelin sheath thickness in the sciatic nerves was not increased in BACE1(-/-)/Akt-DD mi

282 ted tomography (CT)-guided IRE of 11 porcine sciatic nerves was performed in nine pigs, and histopath

283 In addition, acute RFA of six porcine sciatic nerves was performed in six pigs that were harve

284 of perineural HIV gp120 application onto the sciatic nerve, we found that pC/EBPbeta was triggered by

285 onal transport in single axons in the intact sciatic nerve, we have identified clear axonal transport

286 ata for 24-week-old BKS db/db and db/+ mouse sciatic nerve were analyzed to define significantly diff

287 cultures of purified SCs from newborn mouse sciatic nerve were used to characterize both the role of

288 conduction velocities of the trigeminal and sciatic nerves were decreased.

289 lidate the model experimentally, excised rat sciatic nerves were subjected to stretching, which induc

290 Rat sciatic nerves were transected followed by microsurgical

291 as confirmed by histological analysis of the sciatic nerve which showed predominantly axonal damage:

292 l neurons and peripheral nerves, such as the sciatic nerve, which have provided most of our informati

293 ort the concept that HFS conditioning of the sciatic nerve, which leads to spinal LTP, is associated

294 r perianal abscess causing irritation of the sciatic nerve, which was diagnosed on MRI of the lumbosa

295 we observed galanin upregulation in DRG and sciatic nerve, which was less in GFAP-IkappaBalpha-dn mi

296 ignals were recorded simultaneously from the sciatic nerve with a 16-contact cuff electrode.

297 ain targeting, we inoculated hamsters in the sciatic nerve with either the hyper (HY) or drowsy (DY)

298 estigate this, we inoculated hamsters in the sciatic nerve with long-incubation-period strain 139H pr

299 l of strain interference, inoculation of the sciatic nerve with the drowsy (DY) strain of the transmi

300 ster of differentiation 45 expression in the sciatic nerve, with no difference between genotypes.

301 We cared for a child with sciatic nerve zoster who had severe pain over the lower