ライフサイエンスコーパス: scleral

1 cidence of corneal (60% vs. 40%; P = 0.007), scleral (19% vs. 9%; P = 0.044), and orbital (13% vs. 3%

2 scence (5.3%), suture exposure (5.3%) (trans-scleral 9-0 polypropylene), and vitreous strand at infer

3 The difference between scleral and corneal IOP (IOPs - IOPk) correlated positiv

4 o evaluate reliability and agreement between scleral and corneal measurements of IOP.

5 Bland-Altman analysis for agreement between scleral and corneal pneumatonometry measurements showed

6 Scleral and corneal pneumatonometry showed nearly 1:1 li

7 nto global (GL) and orbital (OL) layers with scleral and pulley insertions, respectively.

8 ssed rapidly to develop corneal infiltrates, scleral and uveal tissue necrosis with hyphema, brownish

9 patients with histopathologically confirmed scleral and/or intraocular invasion of SCC at Wills Eye

10 tumors and with higher incidence of corneal, scleral, and orbital invasion.

11 S2 had more vitrectomies and scleral bands and fewer explants and associated cataract

12 ntal displacements to extract unique sets of scleral biomechanical properties.

13 Previous work has suggested a major role of scleral biomechanics in the pathogenesis of glaucoma.

14 ains to be shown whether and how the altered scleral biomechanics may affect the rate of glaucoma pro

15 er orientation is needed to fully understand scleral biomechanics.

16 (stromal choroidal thickness, SCT), or inner scleral border (total choroidal thickness, TCT) showed n

17 choroid, including the shape of the choroid-scleral border, location of the thickest point of choroi

18 Clinical features correlated with posterior scleral bowing included reduced distance to the optic di

19 Choroidal nevus can show focal posterior scleral bowing on EDI-OCT in 5% of cases.

20 Recognition of posterior scleral bowing with choroidal nevus is essential to avoi

21 All cases demonstrated posterior scleral bowing with mean scleral excavation of 398 micro

22 emonstrated the EDI-OCT feature of posterior scleral bowing.

23 who underwent vitrectomy with a supplemental scleral buckle (n = 488) had an increased failure rate c

24 tly lower failure rate versus treatment with scleral buckle (P = 7x10(-8)).

25 R, pars plana vitrectomy (PPV), and combined scleral buckle (SB) plus PPV (SB+PPV).

26 In addition, an encircling scleral buckle (SB) was used in 2 cases.

27 my (PPV), 413 (12.1%) were retinopexy with a scleral buckle (SB), and 297 (8.7%) were PPV with an SB

28 akic, and aphakic groups, those treated with scleral buckle alone (n = 1341) had a significantly lowe

29 tly lower failure rate versus treatment with scleral buckle alone (P = 0.0015).

30 thout scleral buckle, and those treated with scleral buckle alone (P = 0.7).

31 failure rate was higher when treated with a scleral buckle alone versus vitrectomy (P = 0.0017).

32 grees underwent PPV combined with encircling scleral buckle and 360 degrees laser retinopexy of the p

33 the risks and benefits of vitrectomy versus scleral buckle and keep in mind that the single-surgery

34 Scleral buckle and vitrectomy combined with belt buckle

35 pars plana vitrectomy (1/19, 5.3%), and post-scleral buckle exposure (1/19, 5.3%).

36 er in the vitrectomy group compared with the scleral buckle group (P = 3x10(-8)).

37 atients, final failure rate was lower in the scleral buckle group compared with those who had vitrect

38 outside institution before referral, or if a scleral buckle had been placed.

39 ted phakic retinal detachments, repair using scleral buckle may be a good option.

40 c retinopexy was found to be comparable with scleral buckle when a retinal hole was present (P = 0.65

41 vice (intraocular lens, glaucoma implant, or scleral buckle).

42 ined pars plana vitrectomy (PPV), encircling scleral buckle, 360 degrees Laser endophotocoagulation,

43 ely treated with PPV coupled with encircling scleral buckle, 360 degrees laser retinopexy and silicon

44 , surgical techniques, including vitrectomy, scleral buckle, and pneumatic retinopexy, are the only m

45 ose treated with vitrectomy, with or without scleral buckle, and those treated with scleral buckle al

46 egular corneal astigmatism in keratoconus or scleral-buckle-induced regular astigmatisms can be equal

47 L implantation in two cases - a patient with scleral-buckle-induced regular corneal astigmatism and a

48 r of toric IOL implantation in patients with scleral-buckle-induced regular corneal astigmatism.

49 hment and grade C PVR after primary encircle scleral buckling (SB group - 12 eyes), or pars plana vit

50 Surgical repair was done either by scleral buckling (SB) or pars plana vitrectomy (PPV) acc

51 index studies) of pneumatic retinopexy (PR), scleral buckling (SB), pars plana vitrectomy (PPV), and

52 Initial surgery using scleral buckling alone was performed in most (8 of 13, 6

53 idwest demonstrated a greater preference for scleral buckling compared to all other regions (P < .01)

54 Scleral buckling declined from 6502 procedures in 2000 t

55 atism (>2.5 diopter) were caused by previous scleral buckling in one case and by keratoconus in the o

56 enous RD in BD can be effectively treated by scleral buckling in selected cases and PPV in more compl

57 Novel surgical tools, including bioerodible scleral buckling materials and artificial vitreous subst

58 Previous scleral buckling or pars plana vitrectomy seem to have n

59 ) and silicone oil tamponade with or without scleral buckling procedure (SBP) for recurrent RD due to

60 The patient underwent a scleral buckling procedure with a small segmental buckle

61 External drainage of subretinal fluid during scleral buckling procedures is considered by many ophtha

62 Scleral buckling sharply declined, and preference for re

63 samples obtained from patients who underwent scleral buckling surgery for primary rhegmatogenous reti

64 my with a distribution of 83% vitrectomy, 5% scleral buckling, and 12% pneumatic retinopexy in 2014.

65 detachment repair by pars plana vitrectomy, scleral buckling, and pneumatic retinopexy was analyzed.

66 were performed between pneumatic retinopexy, scleral buckling, and vitrectomy.

67 retinal detachment with surgical treatment (scleral buckling, vitrectomy, or pneumatic retinopexy).

68 ne peeling, tamponade choice, and concurrent scleral buckling, were constructed to assess association

69 f stage 4A ROP in the right eye and received scleral buckling.

70 cted the lamina cribrosa displacement (LCD), scleral canal expansion (SCE), and the stresses (forces)

71 e introduction of peripheral iridectomy with scleral cautery (thermal sclerostomy) in the 1950s and t

72 lped" in conjunctival, corneal, retinal, and scleral cells, similar to the behavior observed in macro

73 r pathway appeared to be responsible for the scleral changes during myopia development or recovery.

74 sclera was evident in the TPAF channel; the scleral collagen fibers showed no organization and appea

75 DT lumen appeared as scleral collagen second harmonic generation signal voids

76 Drug Administration-approved gas-permeable, scleral contact lens.

77 meibomian gland heating and expression, and scleral contact lenses are some of the latest options av

78 Among the humans, in the older eyes the scleral contour bowed inward in the region of the limbus

79 It has yet to be established whether scleral cross-linking is safe in humans and that it conf

80 Both assessments support trans-scleral delivery of posterior sub-Tenon's triamcinolone.

81 date the cellular and molecular mechanism of scleral diseases such as scleritis and myopia.

82 onstrated posterior scleral bowing with mean scleral excavation of 398 microm (median, 377 microm; ra

83 We hypothesize that scleral expansion at the location of these perforating v

84 ring PPV are male sex, advancing age, RRD, a scleral explant, a dropped lens fragment, and the use of

85 myopia there is regulated remodeling of the scleral extracellular matrix (ECM) that controls the ext

86 prelaminar/laminar optic nerve invasion, or scleral/extrascleral infiltration.

87 omechanics, experimental characterization of scleral fiber orientation is needed to fully understand

88 the posterior and peripapillary region, the scleral fibers were mostly circumferential but less alig

89 cated in the mouse TM, but not in corneal or scleral fibroblast cells.

90 f GAG was found in corneal stromal cells and scleral fibroblasts but not in corneal epithelium, endot

91 ameter that could represent small vessels or scleral fibroblasts.

92 At 15 weeks, thickness of scleral fibrosis was greater in GS (246 +/- 47 mum) and

93 Glued trans-scleral fixated posterior chamber IOL exchange for AC IO

94 eratoplasty (DMEK) in patients with existing scleral-fixated and iris-fixated intraocular lenses (sf-

95 nt of claw lenses, angle-supported IOLs, and scleral-fixated IOLs by means of an objective, repeatabl

96 antations is a valid alternative strategy to scleral-fixated or angle-supported IOL implantation.

97 chamber intraocular lens, 2 patients with a scleral-fixated posterior chamber intraocular lens (PCIO

98 The anterior chamber IOL was removed after scleral fixating the dislocated posterior chamber IOL in

99 ntly described techniques include sutureless scleral fixation and intraocular endoscopy-guided suture

100 e found between groups operated with Hoffman scleral fixation and Sharioth technique.

101 The scleral fixation suture was 9-0 polypropylene in 16 eyes

102 Hoffman haptic scleral fixation was performed in 31 eyes, Sharioth tech

103 area of LC insertion into the peripapillary scleral flange and into the pia, and computed the total

104 ential transconjunctival sutures through the scleral flap and connected them to the adjacent sclera i

105 Even 4 years after resuturing of the scleral flap through the intact conjunctiva, there is ev

106 l acuity (VA), IOP, number of sutures in the scleral flap, laser suture lysis, surgeon, and lateralit

107 ation of a GMS+ by means of a full-thickness scleral flap.

108 Neovascularization was measured on choroidal-scleral flat mounts using intercellular adhesion molecul

109 acuity, intraocular pressure, and trends in scleral grading.

110 In 8 cases, scleral grafting was performed as part of the initial su

111 oing treatment for ocular tumors followed by scleral grafts in a tertiary eye care center in the Unit

112 In this series, scleral grafts were well accepted when placed as part of

113 Sclera was reconstructed with allogenic scleral grafts.

114 ic cells may regulate both overall eye size (scleral growth) and the growth of the retina (proliferat

115 he majority of their dendritic arbors to the scleral half or "Off" sublamina of the inner plexiform l

116 erior chamber IOL implantation using Hoffman scleral haptic fixation and sutureless Sharioth techniqu

117 Scleral hypercompliance may precede stiffening or be a u

118 of the sclera and the presence or absence of scleral hyporeflective areas representing intrascleral e

119 We found that a history of scleral icterus may increase the odds of G6PD deficiency

120 cient were 2.1 times higher in children with scleral icterus than those without (p=0.0351).

121 revealed a palpable mass in the epigastrium; scleral icterus was absent.

122 During vitrectomy and under scleral indentation at 5-o'clock position, a cilium was

123 Examination with scleral indentation of the RE revealed 2 peripheral smal

124 idal and optic nerve invasion (n = 17, 12%), scleral infiltration (n = 20, 14%), and extrascleral inv

125 e or nodular scleritis with a high degree of scleral inflammation (>2+) (OR = 4.70, P = 0.001).

126 se or nodular scleritis with a low degree of scleral inflammation (</= 2+) (odds ratio [OR] = 2.89, P

127 Control of scleral inflammation and pain was achieved in all but 2

128 atment is usually effective in reducing both scleral inflammation and symptoms and possibly reduces t

129 e or nodular scleritis with a high degree of scleral inflammation may respond to SAIDs.

130 r scleritis (OR, 2.33; P = 0.042), degree of scleral inflammation of more than 2+ (range, 0-4+; OR, 3

131 necrotizing scleritis, posterior scleritis, scleral inflammation of more than 2+, anterior uveitis,

132 2-step reduction or reduction to grade 0 in scleral inflammation on a 0 to +4 scale according to a s

133 se or nodular scleritis with a low degree of scleral inflammation or without ocular complications may

134 cs (laterality, type of scleritis, degree of scleral inflammation, ocular complications, delay in pre

135 sirolimus leads to a short-term reduction in scleral inflammation, though relapses requiring reinject

136 e presence of AK with concurrent ipsilateral scleral inflammation.

137 nted with acute (85.7%) and unilateral (80%) scleral inflammation.

138 une, non-necrotizing anterior scleritis with scleral inflammatory grade of >/=1+ in at least 1 quadra

139 s, non-necrotizing anterior scleritis with a scleral inflammatory grade of +1 to +3 in at least 1 eye

140 eny the human brain detects social cues from scleral information even in the absence of conscious awa

141 r the ability to respond to social cues from scleral information without conscious awareness exists e

142 muscle margins oversewn to the poles of the scleral insertion, avoiding the anterior ciliary arterie

143 ), and hyporeflective gradation at the nevus-scleral interface (P = .33).

144 vessels (P = 1), visualization of the nevus-scleral interface (P = .6), and hyporeflective gradation

145 Visualization of the complete nevus-scleral interface was significantly (P = .02) more appar

146 Scleral intraocular lens (IOL) fixation is an accepted t

147 ea, and tumors with local invasion (corneal, scleral, intraocular or orbital invasion) were associate

148 all cases with residual tumor demonstrating scleral invasion (n = 15) and/or anterior chamber invasi

149 ificantly associated with lower DFS included scleral invasion by the TNM system and massive choroidal

150 es (accuracy, 93.3%; specificity, 95.6%) and scleral invasion in 5 out of 6 eyes (accuracy, 98.7%; sp

151 In eyes with residual tumor showing scleral invasion or intraocular involvement, enucleation

152 ting postlaminar optic nerve, choroidal, and scleral invasion showed sensitivities of 59% (95% CI, 37

153 oidal and 13 with scleral invasion], 12 with scleral invasion without postlaminar optic nerve invasio

154 ary body, optic nerve, choroidal, and (extra)scleral invasion.

155 th concomitant massive choroidal and 13 with scleral invasion], 12 with scleral invasion without post

156 tive alternative to enucleation for residual scleral-invasive conjunctival SCC following resection.

157 the resection margin of the optic nerve and scleral involvement, but only the former was independent

158 To now, no long-term data on scleral IOL fixation in MFS exist.

159 the following equation: corneal IOP = 1.04 x scleral IOP - 10.37.

160 Main outcome measures were scleral IOP and corneal IOP.

161 d for determination of whether the choroidal-scleral junction (CSJ) could be visualized and for measu

162 outer choroidal vessel (OCV), and choroidal-scleral junction (CSJ) visualization in inverted versus

163 phy (EDI-OCT) require a well-defined choroid-scleral junction (CSJ), which may appear in some eyes as

164 he retinal pigment epithelium to the choroid-scleral junction at 500-mum intervals up to 2500 mum nas

165 (0.750-0.869), even in eyes where choroidal-scleral junction visibility was <75%.

166 I-OCT images were obtained and the choroidal-scleral junction was analyzed through semiautomated segm

167 Two graders marked the choroidal-scleral junction with segmentation software using differ

168 ve afferent pupillary defect (APD), old age, scleral laceration, and retinal detachment.

169 t to follow-up, 4 eyes abandoned wearing the scleral lens because of an inability to handle the lense

170 investigate the success and failure rates of scleral lens correction in severe keratoconus.

171 The scleral lens fitting process can be completed efficientl

172 vey mailed to all patients who completed the scleral lens fitting process to evaluate the long-term s

173 s, 115 (188 eyes) successfully completed the scleral lens fitting process, and therapeutic goals (imp

174 Scleral lens fitting was completed in an average of 3 vi

175 Scleral lens fitting was proposed for the 75 eyes includ

176 of 27 eyes of 17 MFS patients that underwent scleral lens fixation at our clinic between 1999 and 201

177 Scleral lens fixation in MFS patients achieves satisfyin

178 2 patients (346 eyes) who were evaluated for scleral lens therapy for the management of ocular surfac

179 process to evaluate the long-term success of scleral lens therapy in the management of ocular surface

180 The most common indications for scleral lens therapy were undifferentiated ocular surfac

181 ee patients experienced complications during scleral lens wear that resolved without loss of visual a

182 Visual acuity improved with scleral lens wear, from 0.32 +/- 0.37 logarithm of the m

183 nge, 0-8) other forms of intervention before scleral lens wear.

184 oss of visual acuity, enabling resumption of scleral lens wear.

185 ery were successfully treated with long-term scleral lens wear.

186 0.19 logMAR (Snellen equivalent, 20/26) with scleral lenses (P<0.001).

187 Commercially available scleral lenses can be successfully used in the managemen

188 n addition to protecting the ocular surface, scleral lenses improve visual acuity in patients whose s

189 Scleral lenses were prescribed in 51 of 75 eyes.

190 Differences between serial scleral measurements reflect differences between serial

191 cytosis (P = .02), and 1.9 times higher with scleral melanocytosis (P < .001).

192 e to biomechanical changes; (2) glaucomatous scleral modulus associated with an IOP of 10 mm Hg decre

193 imal chronic IOP elevation; (3) glaucomatous scleral modulus associated with IOPs of 30 and 45 mm Hg

194 Associated features included dermal (n = 6), scleral (n = 9), iris (n = 3), and palate (n = 1) melano

195 Scleral necrosis after plaque radiotherapy of uveal mela

196 tions included corneal epitheliopathy in 6%, scleral necrosis in 3%, cataract in 53%, elevated intrao

197 of factors that predicted clinically evident scleral necrosis included ciliary body (P = 0.0001) and

198 Factors predictive of scleral necrosis included increasing tumor thickness, ci

199 Treatment of scleral necrosis included observation in 81% of patients

200 Scleral necrosis occurred in <1% of patients (3/1140) wh

201 Scleral necrosis remained stable in 48% of patients (35/

202 ime interval between plaque radiotherapy and scleral necrosis was 32 months (median, 23 months; range

203 The mean basal dimension of scleral necrosis was 4 mm (median, 3 mm; range, 1-15 mm)

204 On the basis of tumor location, scleral necrosis was detected after plaque radiotherapy

205 Another patient developed scleral necrosis, secondary infectious scleritis, and in

206 noma, 73 (1%) developed radiotherapy-induced scleral necrosis.

207 ve conjunctival squamous cell carcinoma with scleral necrosis.

208 eyes of 30 patients with unilateral anterior scleral or episcleral inflammation.

209 heme of these anomalies is the presence of a scleral (or lamina cribrosa) defect permitting anomalous

210 ed therapeutic keratoplasty, combined with a scleral patch graft in 1 eye, 1 eye was eviscerated afte

211 uded observation in 81% of patients (59/73), scleral patch graft in 14% of patients (10/73), and enuc

212 in 48% of patients (35/73), or progressed to scleral perforation in 4% of patients (3/73) over a mean

213 Intraoperative scleral perforation or retinal redetachment related to t

214 ery, and 11% of cases develop intraoperative scleral perforation or retinal redetachment.

215 l membrane, vitreoretinal traction, optic or scleral pit, or advanced glaucomatous optic nerve change

216 l pachymetry and serial corneal and temporal scleral pneumatonometry (baseline, immediately after, an

217 nts included corneal pneumatonometry (IOPk), scleral pneumatonometry (IOPs), axial length (AL), spher

218 Scleral pneumatonometry averaged 9.0 mmHg higher than co

219 Scleral pneumatonometry correlates positively with corne

220 When adjusted for age, scleral pneumatonometry may be an adequate alternative i

221 One-time baseline corneal and scleral pneumatonometry readings were obtained in the no

222 Scleral pneumatonometry should be considered as an alter

223 owed nearly 1:1 linear correlation, although scleral pneumatonometry was biased toward higher values

224 The difference between corneal and scleral pneumotonometry was correlated between the two e

225 Scleral proteins were isolated and resolved by DIGE.

226 s been implicated in the local regulation of scleral proteoglycan synthesis in vivo.

227 Scleral proteoglycan synthesis was measured in vitro by

228 d, and the effect of choroid-derived atRA on scleral proteoglycan synthesis.

229 ID), corneoscleral junction angle (CSJ), and scleral radius (SR) were extracted from multiple OCT ima

230 Strikingly, the superior scleral region stood out as showing the strongest and mo

231 every 1% decrease in CPT code 67255 payment, scleral reinforcement with graft service volume increase

232 For scleral reinforcement with graft, the payment-volume ela

233 sease, likely as an extension of myopia-like scleral remodeling triggered by deprivation of a retinal

234 management despite synchronous radiotherapy, scleral resection, or cryotherapy.

235 enticule inserted under a "recipient" corneo-scleral rim mounted on an artificial anterior chamber.

236 nsertion area under the Bruch's membrane and scleral rim.

237 n the periphery beneath the neuroretinal and scleral rims or vascular structures.

238 On the basis of an analysis of scleral ring and orbit morphology in 33 archosaurs, incl

239 rchosaurs using the anatomy of the orbit and scleral ring.

240 Finite element (FE) models of each scleral shell were constructed that incorporated stretch

241 The intact posterior scleral shells were pressurized from 5 to 45 mm Hg while

242 Scleral specimens from 22 donors with no history of glau

243 s, and antigens were detected in corneal and scleral specimens, the iris, the ciliary body, and oculo

244 gle opening distance 500 mum anterior to the scleral spur (AOD500) were compared among the quadrants

245 IA), angle opening distance 500 mum from the scleral spur (AOD500), and iridotrabecular contact lengt

246 rabeculo-iris space area at 500 mum from the scleral spur (AOD500, TISA-500), anterior chamber angle,

247 ngle opening distance (AOD) 500 mum from the scleral spur (median DeltaAOD500 = 103 mum; interquartil

248 ot visible and open if the angle was open to scleral spur and beyond.

249 and 750 mum anterior from scleral spur), and scleral spur angle.

250 pening distance at 750 mum (AOD750) from the scleral spur as the 2 dependent angle width variables.

251 easured at both 500 mum and 750 mum from the scleral spur landmark.

252 hwork at 500 mum and 750 mum anterior to the scleral spur to the anterior iris surface (AOD500 and AO

253 ecular meshwork height was measured from the scleral spur to the Schwalbe line.

254 TISA, measured 500 and 750 mum anterior from scleral spur), and scleral spur angle.

255 (AOD, measured 500 and 750 mum anterior from scleral spur), the trabecular-iris-space area (TISA, mea

256 ult [LV], iris thickness at 750 mum from the scleral spur, and iris cross-sectional area) explain >80

257 r-iris space area (TISA500) 500 mum from the scleral spur, were made using the FD-OCT RTVue(R).

258 d iris thickness (IT750) at 750 mum from the scleral spur.

259 gle recess area at 750 mum (ARA750) from the scleral spur.

260 ers measured at different distances from the scleral spur: angle opening distance at 250 mum (AOD250)

261 gle recess area at 750 mum (ARA750) from the scleral spur; and trabecular-iris angle (TIA).

262 mum (TISA500) and 750 mum (TISA750) from the scleral spur; angle recess area at 750 mum (ARA750) from

263 mum (TISA500) and 750 mum (TISA750) from the scleral spur; angle recess area at 750 mum (ARA750) from

264 mm (CMT2), and 3 mm (CMT3) posterior to the scleral spur; maximum (CMTMAX) thickness was also assess

265 mum (AOD500), and 750 mum (AOD750) from the scleral spur; trabecular-iris space area at 500 mum (TIS

266 mum (AOD500), and 750 mum (AOD750) from the scleral spur; trabecular-iris space area at 500 mum (TIS

267 e lens and the horizontal line joining the 2 scleral spurs on horizontal AS-OCT scans.

268 of poor image quality or inability to locate scleral spurs, leaving 1465 subjects for analysis.

269 The scleral stem/progenitor cells (SSPCs) possessed clonogen

270 Increased scleral stiffness after glaucoma exposure in mice mimics

271 Longer eyes, greater scleral strain in some directions at baseline, and gener

272 Mid-peripheral scleral strains were lower but exhibited a similar secto

273 Scleral strips from ADAMTS10-mutant (affected) dogs and

274 om which the full strain tensor of the outer scleral surface was calculated.

275 o 45 mm Hg while the 3D displacements of the scleral surface were measured by speckle interferometry.

276 field three-dimensional displacements of the scleral surface were measured using laser speckle interf

277 es include anterior chamber lenses, iris- or scleral-sutured lenses, and iris-claw lenses.

278 re randomly assigned to IOL repositioning by scleral suturing (n = 54) or IOL exchange with a retropu

279 ts (104 eyes) either to IOL repositioning by scleral suturing (n = 54) or to IOL exchange with retrop

280 of the staphyloma and secondary to excessive scleral thickening.

281 erior and posterior laminar insertions), and Scleral Thickness (at the Anterior Sub-arachnoid space)

282 Normalization to scleral thickness abolished the species differences in s

283 The mean transconjunctival scleral thickness was 747 mum (SD +/- 68.97) with a rang

284 Underlying the nevus, the scleral thickness was not measurable; however, at the ne

285 Axial length and scleral thickness were measured after sacrifice in the C

286 Choroidal thickness, scleral thickness, and bulge height were positively corr

287 Retinal, choroidal, and scleral thicknesses and bulge height were measured on SD

288 some directions at baseline, and generalized scleral thinning after glaucoma were characteristic of C

289 vision-threatening complications, including scleral thinning, ulceration, and delayed conjunctival e

290 Scleral tissue from C57BL6/J mice was separated from the

291 tion of fine needle aspiration biopsy (FNAB) scleral tracts to determine the incidence of iatrogenic

292 may indicate an increased facilitated trans-scleral transport of nanoparticle carboplatin, with a su

293 ickness abolished the species differences in scleral transport.

294 ness accounts for the species differences in scleral transport.

295 nce demonstrated mild hyperautofluorescence (scleral unmasking) in hypopigmented choroid and no lipof

296 a, and the relationships between perforating scleral vessels and retinal-choroidal structures.

297 Perforating scleral vessels are often present beneath the site at wh

298 measures were the prevalence of perforating scleral vessels at the site of the lacquer crack, the po

299 ng minus lens wear may produce the increased scleral viscoelasticity that results in faster axial elo

300 photoreceptor loss and myopia with increased scleral wall elasticity.