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1 on and treatment of patients with idiopathic scoliosis.
2 ties, weight changes, hind limb atrophy, and scoliosis.
3 and small joint laxity and early progressive scoliosis.
4 ght than normal littermates and many develop scoliosis.
5 in infancy, early spinal rigidity, and early scoliosis.
6 of girls and 0.3% of boys) were treated for scoliosis.
7 had Marfan's disease, and one had congenital scoliosis.
8 mented medical or chiropractic evaluation of scoliosis.
9 Most commonly seen is idiopathic scoliosis.
10 ition, provide new opportunities to evaluate scoliosis.
11 therapeutic intervention may prevent severe scoliosis.
12 eptibility has been implicated in congenital scoliosis.
13 characteristic of TBX6-associated congenital scoliosis.
14 mutation is insufficient to cause congenital scoliosis.
15 dactyly of the hands, vertebral fusions, and scoliosis.
16 gularities, and vertebral anomalies, such as scoliosis.
17 derlying heart defect, surgical history, and scoliosis.
18 heir age, skeletal immaturity, and degree of scoliosis.
19 fragmented in some juveniles with idiopathic scoliosis.
20 cant weakness, respiratory insufficiency, or scoliosis.
21 , and horizontal gaze palsy with progressive scoliosis.
22 rgery in patients with adolescent idiopathic scoliosis.
23 sion for patients with adolescent idiopathic scoliosis.
24 camptocormia, antecollis, Pisa syndrome, and scoliosis.
25 sis (HGPPS), a rare disease marked by severe scoliosis.
26 ssful in all but one patient, who had severe scoliosis.
27 ine, such as those observed in patients with scoliosis.
28 in the 161 persons with sporadic congenital scoliosis (11%); we did not observe any null mutations i
29 Han Chinese persons with sporadic congenital scoliosis, 166 Han Chinese controls, and 2 pedigrees, fa
38 r posterior fusion for adolescent idiopathic scoliosis, although outcomes are adversely affected if p
39 es of 76 Han Chinese persons with congenital scoliosis and a multicenter series of 42 persons with 16
40 tients who undergo surgery for correction of scoliosis and craniofacial anomalies should have serum m
41 joint contractures at birth and progressive scoliosis and fractures, but there is remarkable variabi
43 that Nf1(Col2)(-/-) mice display progressive scoliosis and kyphosis, tibial bowing and abnormalities
44 advances in understanding tibial dysplasia, scoliosis and malignant peripheral nerve sheath tumors.
45 al-dominant SMD characterized by significant scoliosis and mild metaphyseal abnormalities in the pelv
51 hyseal injuries; breast and pelvic injuries; scoliosis and spondylolysis; multidirectional shoulder i
56 itional fusions between skeletal structures, scoliosis, and altered cartilage in the intervertebral j
57 phenotype of developmental delay, hypotonia, scoliosis, and cerebellar atrophy in three families.
58 nditions characterized by multiple pterygia, scoliosis, and congenital contractures of the limbs.
61 haracterized by camptodactyly, tall stature, scoliosis, and hearing loss (CATSHL syndrome) to chromos
64 cases can cause spinal deformities, such as scoliosis, and result in disability and distress of affe
65 83) likely to have spontaneous pneumothorax, scoliosis, and striae but were comparable in revised Ghe
67 screening programs for adolescent idiopathic scoliosis are mandated in 26 states in the United States
69 xhibited stereotypical positioning of limbs, scoliosis, arthrogryposis, pulmonary hypoplasia, and res
70 ration pre- and intraoperatively, or type of scoliosis between those who developed SIADH and those wh
72 g identified 5 of the 9 children treated for scoliosis but resulted in referrals for another 87 child
74 nificant p-values for most of the idiopathic scoliosis candidate loci, and for some loci, the estimat
76 ic manifestations of Marfan syndrome include scoliosis, chest wall deformity, dural ectasia, joint hy
77 by severe intellectual disability, epilepsy, scoliosis, choreoathetosis, dysmorphic facial features a
78 with osteopenia, severe osteoarthritis, and scoliosis compared with previously described techniques.
79 ance: Horizontal gaze palsy with progressive scoliosis, Congenital mirror movements, and Congenital f
84 n frontal radiographs and the measurement of scoliosis curves as important tools in the management of
85 hypertriglyceridemia, hypercholesterolemia, scoliosis, developmental delay and pulmonary and urologi
86 cles, progressive joint contractures, severe scoliosis, elevated serum creatine kinase level, myopath
87 alizations, inability to walk, bradykinesia, scoliosis, gastrostomy feeding, age of seizure onset, an
88 Only a quarter of patients with structural scoliosis had evidence of bony fusion on the side of the
89 ron distribution and metabolism and frequent scoliosis have also been associated with an allele of in
92 cause horizontal gaze palsy with progressive scoliosis (HGPPS), a rare disease marked by severe scoli
96 ly, short stature, mild spasticity, thoracic scoliosis, hyperextendable MCP joints, rocker-bottom fee
101 of Ptk7 in motile ciliated lineages prevents scoliosis in ptk7 mutants, and mutation of multiple inde
104 hyperextensible joints (in 68 percent), and scoliosis (in 76 percent of patients 16 years of age or
113 tection and close surveillance of congenital scoliosis is critical, as a rapidly progressive curve ma
114 ibes a patient who lists to the side whereas scoliosis is defined by spinal curvature and rotation an
123 underlying vertebral abnormality (idiopathic scoliosis (IS)) most commonly manifest during adolescenc
129 idiopathic scoliosis, nine had neuromuscular scoliosis, one had Marfan's disease, and one had congeni
131 abnormalities of the thoracic cage, such as scoliosis or flail chest, interfere with the action of t
133 gilant monitoring and thorough evaluation of scoliosis patients can steer patients toward appropriate
134 ll and lean body habitus and higher rates of scoliosis, pectus excavatum, mitral valve prolapse, and
136 numerous ciliary genes are associated with a scoliosis phenotype, among ciliopathies and knockout ani
141 mean age 15.5 [11-26] years) with late-onset scoliosis requiring corrective surgery were enrolled.
147 exclusion and the approach to a patient with scoliosis should aim toward ruling out other possible ca
149 ticularly pulmonary complications related to scoliosis surgery, embolic complications of joint arthro
151 and skeletal symptoms, including progressive scoliosis, that did not conform to standard diagnostic c
152 ng program for the identification of treated scoliosis was 0.05 (95% CI, 0.048-0.052), with 448 child
154 ents who had undergone laminectomy developed scoliosis, whereas spinal deformities were only detected
155 syndactyly, clinodactyly, cleft palate, and scoliosis, which, together with cardiodysrhythmic period
156 acing in patients with adolescent idiopathic scoliosis who are at risk for curve progression and even
157 involving additional persons with congenital scoliosis who carried a deletion affecting TBX6 confirme
158 atropic dysplasia present with a progressive scoliosis, widespread metaphyseal involvement of the app
159 llmarks of AIS, including postnatal onset of scoliosis without malformations of vertebral units.
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