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1 on and treatment of patients with idiopathic scoliosis.
2 ties, weight changes, hind limb atrophy, and scoliosis.
3 and small joint laxity and early progressive scoliosis.
4 ght than normal littermates and many develop scoliosis.
5 in infancy, early spinal rigidity, and early scoliosis.
6  of girls and 0.3% of boys) were treated for scoliosis.
7 had Marfan's disease, and one had congenital scoliosis.
8 mented medical or chiropractic evaluation of scoliosis.
9             Most commonly seen is idiopathic scoliosis.
10 ition, provide new opportunities to evaluate scoliosis.
11  therapeutic intervention may prevent severe scoliosis.
12 eptibility has been implicated in congenital scoliosis.
13 characteristic of TBX6-associated congenital scoliosis.
14 mutation is insufficient to cause congenital scoliosis.
15 dactyly of the hands, vertebral fusions, and scoliosis.
16 gularities, and vertebral anomalies, such as scoliosis.
17 derlying heart defect, surgical history, and scoliosis.
18 heir age, skeletal immaturity, and degree of scoliosis.
19 fragmented in some juveniles with idiopathic scoliosis.
20 cant weakness, respiratory insufficiency, or scoliosis.
21 , and horizontal gaze palsy with progressive scoliosis.
22 rgery in patients with adolescent idiopathic scoliosis.
23 sion for patients with adolescent idiopathic scoliosis.
24 camptocormia, antecollis, Pisa syndrome, and scoliosis.
25 sis (HGPPS), a rare disease marked by severe scoliosis.
26 ssful in all but one patient, who had severe scoliosis.
27 ine, such as those observed in patients with scoliosis.
28  in the 161 persons with sporadic congenital scoliosis (11%); we did not observe any null mutations i
29 Han Chinese persons with sporadic congenital scoliosis, 166 Han Chinese controls, and 2 pedigrees, fa
30                               Minor residual scoliosis after fusion does not adversely affect outcome
31                        Adolescent idiopathic scoliosis (AIS) affects 1-3% of children in the at-risk
32                        Adolescent idiopathic scoliosis (AIS) and pectus excavatum (PE) are common ped
33                        Adolescent idiopathic scoliosis (AIS) causes spinal deformity in 3% of childre
34                        Adolescent idiopathic scoliosis (AIS) is a complex inherited spinal deformity
35                        Adolescent idiopathic scoliosis (AIS) is a complex spine deformity, affecting
36                        Adolescent idiopathic scoliosis (AIS) is an unexplained and common spinal defo
37                        Adolescent idiopathic scoliosis (AIS) is the most common spinal deformity affe
38 r posterior fusion for adolescent idiopathic scoliosis, although outcomes are adversely affected if p
39 es of 76 Han Chinese persons with congenital scoliosis and a multicenter series of 42 persons with 16
40 tients who undergo surgery for correction of scoliosis and craniofacial anomalies should have serum m
41  joint contractures at birth and progressive scoliosis and fractures, but there is remarkable variabi
42                       The rapidly developing scoliosis and its associated pelvic obliquity can even c
43 that Nf1(Col2)(-/-) mice display progressive scoliosis and kyphosis, tibial bowing and abnormalities
44  advances in understanding tibial dysplasia, scoliosis and malignant peripheral nerve sheath tumors.
45 al-dominant SMD characterized by significant scoliosis and mild metaphyseal abnormalities in the pelv
46 l dysplasias characterized by a short trunk, scoliosis and mild short stature.
47                                              Scoliosis and pectus excavatum were significantly more p
48 ubset of control subjects were evaluated for scoliosis and pectus excavatum.
49 eletal manifestations, such as osteoporosis, scoliosis and short statures.
50           Respiratory failure was common and scoliosis and spinal rigidity were significant in some o
51 hyseal injuries; breast and pelvic injuries; scoliosis and spondylolysis; multidirectional shoulder i
52 ate treatment of fractures, the treatment of scoliosis and the use of intramedullary rods.
53 t careful attention be paid to screening for scoliosis and tibial dysplasia.
54                                   Dystrophic scoliosis and tibial pseudoarthrosis are the most severe
55 n with a slender body morphotype, often with scoliosis and/or pectus excavatum.
56 itional fusions between skeletal structures, scoliosis, and altered cartilage in the intervertebral j
57 phenotype of developmental delay, hypotonia, scoliosis, and cerebellar atrophy in three families.
58 nditions characterized by multiple pterygia, scoliosis, and congenital contractures of the limbs.
59 arpal/tarsal coalition, conductive deafness, scoliosis, and craniosynostosis.
60 , previous thoracotomy/ies, body mass index, scoliosis, and diaphragm palsy.
61 haracterized by camptodactyly, tall stature, scoliosis, and hearing loss (CATSHL syndrome) to chromos
62 cal manifestations of horizontal gaze palsy, scoliosis, and intellectual disability.
63 phy predominantly of the distal lower limbs, scoliosis, and mild distal sensory involvement.
64  cases can cause spinal deformities, such as scoliosis, and result in disability and distress of affe
65 83) likely to have spontaneous pneumothorax, scoliosis, and striae but were comparable in revised Ghe
66 astia, cleft palate/bifid uvula, progressive scoliosis, and structural brain abnormalities.
67 screening programs for adolescent idiopathic scoliosis are mandated in 26 states in the United States
68                   Although Pisa syndrome and scoliosis are sometimes used interchangeably to describe
69 xhibited stereotypical positioning of limbs, scoliosis, arthrogryposis, pulmonary hypoplasia, and res
70 ration pre- and intraoperatively, or type of scoliosis between those who developed SIADH and those wh
71 oration of cilia motility after the onset of scoliosis blocks spinal curve progression.
72 g identified 5 of the 9 children treated for scoliosis but resulted in referrals for another 87 child
73 ificant medical morbidity and deformity that scoliosis can insidiously inflict.
74 nificant p-values for most of the idiopathic scoliosis candidate loci, and for some loci, the estimat
75 f TBX6 accounted for up to 11% of congenital scoliosis cases in the series that we analyzed.
76 ic manifestations of Marfan syndrome include scoliosis, chest wall deformity, dural ectasia, joint hy
77 by severe intellectual disability, epilepsy, scoliosis, choreoathetosis, dysmorphic facial features a
78  with osteopenia, severe osteoarthritis, and scoliosis compared with previously described techniques.
79 ance: Horizontal gaze palsy with progressive scoliosis, Congenital mirror movements, and Congenital f
80            Surgical management of idiopathic scoliosis continues to evolve, and now a thoracoscopic e
81  patients with SMA-II undergoing surgery for scoliosis correction.
82           Skeletal defects include kyphosis, scoliosis, crooked tails and curvature and overgrowth of
83 es caused by malformed vertebrae (congenital scoliosis (CS)) are apparent at birth.
84 n frontal radiographs and the measurement of scoliosis curves as important tools in the management of
85  hypertriglyceridemia, hypercholesterolemia, scoliosis, developmental delay and pulmonary and urologi
86 cles, progressive joint contractures, severe scoliosis, elevated serum creatine kinase level, myopath
87 alizations, inability to walk, bradykinesia, scoliosis, gastrostomy feeding, age of seizure onset, an
88   Only a quarter of patients with structural scoliosis had evidence of bony fusion on the side of the
89 ron distribution and metabolism and frequent scoliosis have also been associated with an allele of in
90           Prognostic indicators for juvenile scoliosis have been identified.
91     Previous long-term studies of idiopathic scoliosis have included patients with other etiologies,
92 cause horizontal gaze palsy with progressive scoliosis (HGPPS), a rare disease marked by severe scoli
93  with horizontal gaze palsy with progressive scoliosis (HGPPS).
94 ed in horizontal gaze palsy with progressive scoliosis (HGPPS).
95                     Other conditions such as scoliosis, hip instability, patellar instability and foo
96 ly, short stature, mild spasticity, thoracic scoliosis, hyperextendable MCP joints, rocker-bottom fee
97             There was hearing loss in 21 and scoliosis in 17.
98                        Adolescent idiopathic scoliosis in humans is often associated with vestibulomo
99 the impact of primary myopathy on idiopathic scoliosis in mice and man.
100                                   The severe scoliosis in most individuals and rare developmental cox
101 of Ptk7 in motile ciliated lineages prevents scoliosis in ptk7 mutants, and mutation of multiple inde
102 sufficient to generate vertebral fusions and scoliosis in the adult spine.
103        The cumulative incidence of diagnosed scoliosis in this population was 1.8% (95% confidence in
104  hyperextensible joints (in 68 percent), and scoliosis (in 76 percent of patients 16 years of age or
105                   The finding of 'structural scoliosis' in Pisa syndrome should not preclude interven
106 eous conclusion that all types of idiopathic scoliosis inevitably end in disability.
107                                   Congenital scoliosis is a common type of vertebral malformation.
108                        Adolescent idiopathic scoliosis is a complex disease with unclear etiopathogen
109                                              Scoliosis is a complex genetic disorder of the musculosk
110                                   Congenital scoliosis is a deformity of the developing spine that re
111                                   Idiopathic scoliosis is a diagnosis of exclusion and the approach t
112                                   Idiopathic scoliosis is a lateral curvature of the spine greater th
113 tection and close surveillance of congenital scoliosis is critical, as a rapidly progressive curve ma
114 ibes a patient who lists to the side whereas scoliosis is defined by spinal curvature and rotation an
115                                   Idiopathic scoliosis is more clearly understood recently and is rec
116                                              Scoliosis is more common in patients with missense mutat
117                                              Scoliosis is traditionally evaluated by measuring the Co
118                                   Idiopathic scoliosis (IS) affects 3% of children worldwide, yet the
119                                   Idiopathic scoliosis (IS) is a common paediatric musculoskeletal di
120                                   Idiopathic scoliosis (IS) is a complex pediatric disease of unknown
121                                   Idiopathic scoliosis (IS) is a spine deformity that affects approxi
122                                   Idiopathic scoliosis (IS) is the most common spinal deformity in ch
123 underlying vertebral abnormality (idiopathic scoliosis (IS)) most commonly manifest during adolescenc
124                        Late-onset idiopathic scoliosis (LIS) is a distinct entity with a unique natur
125                       In those patients with scoliosis necessitating treatment, bracing should be the
126                      Nineteen had idiopathic scoliosis, nine had neuromuscular scoliosis, one had Mar
127 an adult man with a gracile build and severe scoliosis of the thoracic spine.
128                             All patients had scoliosis on standing radiographs, and 12 had scoliosis
129 idiopathic scoliosis, nine had neuromuscular scoliosis, one had Marfan's disease, and one had congeni
130 ch encompass contractures of hands and feet, scoliosis, ophthalmoplegia, and ptosis.
131  abnormalities of the thoracic cage, such as scoliosis or flail chest, interfere with the action of t
132 ssues include electrocardiographic findings, scoliosis, osteopenia, and motor control.
133 gilant monitoring and thorough evaluation of scoliosis patients can steer patients toward appropriate
134 ll and lean body habitus and higher rates of scoliosis, pectus excavatum, mitral valve prolapse, and
135 coliosis on standing radiographs, and 12 had scoliosis persisting in the supine position.
136 numerous ciliary genes are associated with a scoliosis phenotype, among ciliopathies and knockout ani
137                               They can cause scoliosis, pressure on the neighboring lung parenchyma a
138                                              Scoliosis progression is influenced by growth rate, but
139         Sim2 mutants also develop congenital scoliosis, reflected by the unequal sizes of the left an
140          Although the etiology of idiopathic scoliosis remains unclear, there continues to be a searc
141 mean age 15.5 [11-26] years) with late-onset scoliosis requiring corrective surgery were enrolled.
142  increased frequency of perinatal mortality, scoliosis, resting tremors and ptosis.
143            Egr3-/- mice develop gait ataxia, scoliosis, resting tremors, and ptosis, suggesting a def
144                   In this population, school scoliosis screening identified some children who went on
145 sidered in making decisions regarding school scoliosis screening.
146                                              Scoliosis severity in AIS cases was associated with FBN1
147 exclusion and the approach to a patient with scoliosis should aim toward ruling out other possible ca
148 everal medical complications can occur after scoliosis surgery in children and adolescents.
149 ticularly pulmonary complications related to scoliosis surgery, embolic complications of joint arthro
150 to recent literature specifically related to scoliosis surgery.
151 and skeletal symptoms, including progressive scoliosis, that did not conform to standard diagnostic c
152 ng program for the identification of treated scoliosis was 0.05 (95% CI, 0.048-0.052), with 448 child
153                                              Scoliosis was noted in 28% (340).
154 ents who had undergone laminectomy developed scoliosis, whereas spinal deformities were only detected
155  syndactyly, clinodactyly, cleft palate, and scoliosis, which, together with cardiodysrhythmic period
156 acing in patients with adolescent idiopathic scoliosis who are at risk for curve progression and even
157 involving additional persons with congenital scoliosis who carried a deletion affecting TBX6 confirme
158 atropic dysplasia present with a progressive scoliosis, widespread metaphyseal involvement of the app
159 llmarks of AIS, including postnatal onset of scoliosis without malformations of vertebral units.

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