ライフサイエンスコーパス: scrapie agent

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1 persistently infected with the mouse-adapted scrapie agent.

2 multiple doses of feed contaminated with the scrapie agent.

3 nucleating the biological replication of the scrapie agent.

4 ction by sheep-derived Chandler (Ch) and 22L scrapie agents.

5 ormation upon exposure of cells to different scrapie agents.

6 udied by making 4 serial passages of hamster scrapie agent (263K) in mice.

7 ally and orally infected sheep with clinical scrapie agent and orally infected preclinical and infect

8 While most anti-scrapie agent compounds inhibit PrP-res formation in vit

9 In addition, sheep with no exposure to the scrapie agent did not contain any measurable prions with

10 play a key role in the translocation of the scrapie agent from the gut lumen to the GALT from which

11 ransmitted naturally through exposure to the scrapie agent in wasted placentas of infected ewes.

12 icating that PrP expression was required for scrapie agent induction of other cytokines detected.

13 PrP(sc) was detected in the blood of 55% of scrapie agent-infected animals (n = 80) and 71% of anima

14 nses, primary glial cultures were exposed to scrapie agent-infected brain homogenates.

15 to analyze protein levels of 24 cytokines in scrapie agent-infected C57BL/10 mouse brains, we observe

16 sistant prion protein (PrP-res) formation in scrapie agent-infected cells, we tested other antimalari

17 ve compound tested against RML and 22L mouse scrapie agent-infected cells, with 50% inhibitory concen

18 c, potently inhibits PrP-res accumulation in scrapie agent-infected neuroblastoma cells (50% inhibito

19 e the onset of clinical signs in a subset of scrapie agent-infected sheep, followed from 3 months of

20 Scrapie agent-infected wild-type mice and transgenic mic

21 rodegenerative disease by 160 days after RML scrapie agent infection.

22 Scrapie agent infectivity was present in the PNS at low

23 and Creutzfeldt-Jakob agents as well as with scrapie agent isolated from sheep or mice.

24 n vivo in GPI(-/-) PrP tg mice infected with scrapie agent may likely involve the GABAergic pathway.

25 stigated the contribution of CD11c(+) DCs in scrapie agent neuroinvasion through use of CD11c-dipther

26 Using two distinct scrapie agent strains (ME7 and 139A scrapie agents), we

27 Initial work with scrapie agents suggested that B cells were central vecto

28 ay the onset of intraperitoneally inoculated scrapie agent, the result previously observed with quina

29 After infection with RML murine scrapie agent, transgenic (tg) mice expressing prion pro

30 distinct scrapie agent strains (ME7 and 139A scrapie agents), we show that when CD11c(+) DCs were tra

31 cted with the 263K strain of hamster-adapted scrapie agent were placed in covered quartz-glass crucib

32 oradic CJD and BSE agents and representative scrapie agents were clearly different from kuru in incub

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