1 of 46 mg/d to prevent the deficiency disease scurvy.

2  4-hydroxylation, and a noncanonical form of scurvy.

3 ynthesize ASC (>95% ASC deficit) and develop scurvy.

4 el at sea with 12 seamen critically ill with scurvy.

5 esented the minimum amount needed to prevent scurvy.

6 act infections (19.4%), measles (15.7%), and scurvy (6.5%) caused most of the 108 deaths.

7         Vitamin deficiency syndromes such as scurvy and beriberi are uncommon in Western societies.

8  Lind evaluated six potential treatments for scurvy, and rapidly reached the conclusion that daily co

9  manifestations owing to l-ascorbic acid for scurvy as comparison to d-ascorbic acid and challenges o

10 ase associated with connective tissue (e.g., scurvy), improves cardiovascular and immune cell functio

11 te (vitamin C) is best known for its role in scurvy, in which the hydroxylation of collagen catalyzed

12  the amount of vitamin C required to prevent scurvy is not sufficient to optimally protect against th

13 r ascorbic acid (vitamin C) is in preventing scurvy, presumably because it is a cofactor for hydroxyl

14 the ancestors of living vertebrates were not scurvy prone and that the loss of gulonolactone oxidase

15          In the vitamin C deficient disease, scurvy, reduced activity of 2-OG oxygenases results in i

16 morrhage was unlikely to be simply a form of scurvy since Slc23a1(-/-) mice showed no hemorrhage in a

17 corbate deficiency, and clinical features of scurvy; these abnormalities were corrected by treatment

18      Although citrus fruits prevent and cure scurvy, they may not always be as good for you as you th