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1 ated on free-ranging otariids (fur seals and sea lions).
2 ity to assess the prevalence of ZcAV in live sea lions.
3 losely related to those adapted to seals and sea lions.
4 pi were obtained from control and chronic DA sea lions.
5 his mixing zone region have been optimal for sea lions.
6 since the last common ancestor of seals and sea lions.
7 ith thick myelin sheaths (elephant seal: 9%, sea lion: 7%) and thin myelin sheaths (elephant seal: 91
9 is the first description of coxiellosis in a sea lion and suggests that exposure to sea lions may be
10 and a naturally occurring condition in wild sea lions and simultaneously advance general knowledge o
12 rvational and experimental studies of seals, sea lions, and walruses reveal elements of vocal develop
16 sites in 48 healthy dolphins and 18 healthy sea lions, as well as those of adjacent seawater and oth
18 rmine if the spatial distribution of Steller sea lions at sea displayed similar scaling properties to
19 dent patterns in the distribution of Steller sea lions at sea or linkages with SST may have been appa
20 s indicate that the distributions of Steller sea lions at sea were more influenced by bathymetry than
22 rom 2000-2010 to estimate probabilities of a sea lion born in one DPS being seen within the range of
25 olphin, which evolved independently from the sea lion but displays similar feeding behavior, also has
26 d serum albumin concentration, but only in a sea lion colony exposed to anthropogenic environmental i
27 ntibody concentration during early Galapagos sea lion development were higher in a colony exposed to
28 a lions previously exposed to DA (chronic DA sea lions) display hippocampal neuropathology similar to
31 n an effort to find linkages between Steller sea lions (Eumetopias jubatus) and their environment, th
32 ion trends support the separation of Steller sea lions (Eumetopias jubatus) into a western distinct p
34 This ELISA provides a tool for testing live sea lions for ZcAV exposure and is valuable for subseque
36 us, hippocampal neuropathology of chronic DA sea lions is similar to that of human patients with temp
38 in a sea lion and suggests that exposure to sea lions may be a risk factor for contracting Q fever.
39 re deployed on 10 groups of juvenile Steller sea lions (n=52) at eight different locations within the
41 e decline of the endangered New Zealand (NZ) sea lion (Phocarctos hookeri) is linked to latent levels
46 n the brainstem, thalamus, and cortex in one sea lion pup and the external anatomy of the brain in a
48 x, we processed brain sections from seal and sea lion pups for Nissl substance, cytochrome oxidase, a
49 ity of bacteriophages was higher in unweaned sea lion pups than in juveniles and animals in rehabilit
55 th the northern elephant seal and California sea lion spend most of their lives at sea, but each also
57 to the inactivation of Tas1r2, we found that sea lion Tas1r1 and Tas1r3 are also pseudogenized, consi
58 ed serum and lung samples (n = 96) from wild sea lions that stranded along the California coast were
60 r receptor function is not restricted to the sea lion: the bottlenose dolphin, which evolved independ
61 dimension were calculated for each group of sea lions using a unit square box-counting method, where
62 nt capsid of Parkville virus, and San Miguel sea lion virus serotype 4 (SMSV4), which are representat
64 to human patients, hippocampal sclerosis in sea lions was unilateral in 79% of cases, mossy fiber sp
65 f wild marine carnivore, three seals and one sea lion, we find that Ly49 and KIR are each represented
66 ained opportunistically postmortem from wild sea lions with and without chronic clinical signs of tox
67 eal (Mirounga angustirostris) and California sea lion (Zalophus californianus) are members of a diver
71 ira interrogans serovar Pomona in California sea lions (Zalophus californianus) as a case study to il
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