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1 disorder would be more prevalent than summer seasonal affective disorder.
2 was significantly more prevalent than summer seasonal affective disorder.
3 seasonal affective disorder and subsyndromal seasonal affective disorder.
4 cts (80%) were not aware of the existence of seasonal affective disorder.
5 ate effects of season and light treatment on seasonal affective disorder.
6 effects of change of season on patients with seasonal affective disorder.
7 ening in the treatment of most patients with seasonal affective disorder.
8 as specific antidepressants in patients with seasonal affective disorder.
9 operation appears abnormal in patients with seasonal affective disorder.
10 brighter, 10,000-lux exposure when treating seasonal affective disorder.
11 by serotonin 1A receptors appears normal in seasonal affective disorder.
12 a promising nonpharmacological treatment for seasonal affective disorder.
13 nt in the subsequent summer in patients with seasonal affective disorder.
14 of the Rosenthal et al. diagnosis of winter seasonal affective disorder.
15 toms predict response to light treatment for seasonal affective disorder.
16 terize the long-term course of patients with seasonal affective disorder.
17 ome, the polar T3 syndrome, and subsyndromal seasonal affective disorder.
19 tude, but that awareness of the existence of seasonal affective disorder, a condition with safe and e
22 new understanding of human diseases such as seasonal affective disorder and delayed sleep phase synd
23 ry function may differ between patients with seasonal affective disorder and healthy control subjects
25 ial similarities in cognitive impairments in seasonal affective disorder and major depressive disorde
26 refore, should be considered when diagnosing seasonal affective disorder and selecting treatment.
27 ality score and to estimate the frequency of seasonal affective disorder and subsyndromal seasonal af
28 by a ratio of 3:2; estimated rates of summer seasonal affective disorder and subsyndromal seasonal af
29 effects of light therapy in the treatment of seasonal affective disorder are reversed by both tryptop
30 successfully treat depression, in particular seasonal affective disorder, but the neural pathways and
32 uency, and pattern (winter versus summer) of seasonal affective disorder in African American college
34 s assessed for major depressive disorder and seasonal affective disorder in late autumn and completed
35 ma melatonin was sampled in 42 patients with seasonal affective disorder, in the evening or overnight
38 seasonal depressions in patients with winter seasonal affective disorder may be associated with great
40 ng Scale, the authors rated 15 patients with seasonal affective disorder on three occasions: during w
41 othesis for patients with winter depression (seasonal affective disorder, or SAD) uses low-dose melat
42 sults of several recent follow-up studies of seasonal affective disorder, providing support for the p
45 e central public health challenge for winter seasonal affective disorder (SAD) is recurrence preventi
47 varies with clinical state in patients with seasonal affective disorder (SAD), becoming elevated dur
48 e evidence supports light therapy for winter seasonal affective disorder (SAD), data on cognitive-beh
53 These results reveal that individuals with seasonal affective disorder showed cognitive impairments
54 rtain features of the group with complicated seasonal affective disorder suggested that they were mor
60 mmer versus winter pattern of seasonality of seasonal affective disorder was compared by using multin
63 seasonal affective disorder and subsyndromal seasonal affective disorder were 4.4% and 8.0%, respecti
64 The effects of gender and the awareness of seasonal affective disorder were evaluated with a two-wa
68 es were also evident among participants with seasonal affective disorder, with more women qualifying
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