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1 ted into Xenopus embryos, CHL2 RNA induced a secondary axis.
2 on of CHL RNA into Xenopus embryos induced a secondary axis.
3 arly Xenopus embryo, Cngsc induces a partial secondary axis.
4 salize the ventral mesoderm, thus inducing a secondary axis.
5 1 class to induce goosecoid expression and a secondary axis.
7 ought + heat treatments by 19-57 days, while secondary axis branches were less likely to produce need
8 form side-by-side conjoined twins, with the secondary axis developing as either the left or right si
9 n of xfz8 in ventral cells leads to complete secondary axis formation and can synergize with Xwnt-8 w
10 onical Wnt signaling and blocked Wnt-induced secondary axis formation in a dose-dependent manner, but
12 type Jade-1 in reducing beta-catenin-induced secondary axis formation in Xenopus laevis embryos in vi
13 nopus embryos, Gdf3 misexpression results in secondary axis formation, and induces morphogenetic elon
18 servation that NvNoggin1 is able to induce a secondary axis in Xenopus embryos argues that N. vectens
22 ence; also, the presence of Tsg enhances the secondary axis-inducing activity of two products of chor
23 talytic subunit of PP2A (PP2A-C) potentiated secondary axis induction and Siamois reporter gene activ
29 initiated twin formation and on whether the secondary axis is on the left or right side of the prima
30 n misexpressed ventrally, derriere induces a secondary axis lacking a head, an effect that is due to
32 g BMP-4 signaling, leading to induction of a secondary axis on the ventral side of intact embryos and
34 piralian organizer to this general aspect of secondary axis patterning but highlight the significant
35 e Dpp/BMP2-4 pathway plays a central role in secondary axis patterning in many animals [1-11], but it
36 , the reconstituted embryos failed to form a secondary axis, suggesting that both 2d and 4d are requi
37 ed that neuroectoderm and mesoderm along the secondary axis were derived from the transplanted D quad
38 ntimorph, causing the formation of a partial secondary axis when expressed on the ventral side of the
39 bservations, ALK-4* is capable of inducing a secondary axis when injected into the ventral side of 32
40 enin-deficient micromeres failed to induce a secondary axis when transplanted to the animal pole of u
41 ation gradient, i.e. the potential to form a secondary axis, which is maximal in the head and is grad
42 s grafted into the body column, it induces a secondary axis, while the adjacent Cngsc(-) region has m
43 F hyperactivates Wnt signaling, developing a secondary axis with beta-catenin target gene upregulatio
44 which results in the formation of a complete secondary axis with head and eyes, did not cause the veg
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