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1 for 70% or more of male cases of primary and secondary syphilis.
2 eral blood mononuclear cells from women with secondary syphilis.
3 tages; 24 (38%) were diagnosed in primary or secondary syphilis.
4 ported cases of tuberculosis and primary and secondary syphilis.
5 e and seed new disseminated lesions to cause secondary syphilis.
6 tly different trends in rates of primary and secondary syphilis: Absolute increases in rates among bl
7 f sex partners that describe how primary and secondary syphilis affects men who have sex with men (MS
8 oint, compared with 76%-89% of patients with secondary syphilis and 44%-79% with latent syphilis.
9 n and peripheral blood from 23 patients with secondary syphilis and 5 healthy control subjects recrui
10 thin 3-6 months after therapy for primary or secondary syphilis, and within 12-24 months for latent s
11 treponemes to resolve lesions of primary and secondary syphilis, but cannot clear the treponemes that
12           Among HIV-positive MSM, decreasing secondary syphilis correlated with increasing testing co
13                           Guttate psoriasis, secondary syphilis, cutaneous lupus erythematosus, capil
14                         Rates of primary and secondary syphilis disproportionately increased among bl
15  diagnoses were repeatedly missed, including secondary syphilis, eczema herpeticum, gram-negative fol
16 ic infectious syphilis and relative falls in secondary syphilis for both HIV-positive and HIV-negativ
17 Although the annual incidence of primary and secondary syphilis has dropped to the lowest rate record
18                        Women with primary or secondary syphilis, herpes simplex virus type 1 (HSV-1)
19 rend for increasing frequency of primary and secondary syphilis in developed countries, especially in
20            Repeated epidemics of primary and secondary syphilis infection in the United States over t
21 e profiles of cells infiltrating primary and secondary syphilis lesions, reverse transcription and po
22 phocytes (CTL) are found in both primary and secondary syphilis lesions.
23                        Women with primary or secondary syphilis or with HSV-1 or HSV-2 infection had
24 ntibodies exclusively against TprC(C), while secondary syphilis patients fail to mount a detectable a
25 rrel portions of TP0326, whereas humans with secondary syphilis respond predominantly to POTRA.
26  from epidermal suction blisters raised over secondary syphilis skin lesions.
27            This study explores the nature of secondary syphilis, specifically, the contribution of an
28                                       During secondary syphilis, T. pallidum simultaneously elicits l
29               After treatment for primary or secondary syphilis, the HIV-infected patients responded
30 ina who had lesions suggestive of primary or secondary syphilis were evaluated using molecular techni

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