ライフサイエンスコーパス: secondhand smoke

1 e--is exquisitely sensitive to the toxins in secondhand smoke.

2 tion of the public from unwanted exposure to secondhand smoke.

3 valence of cigarette smoking and exposure to secondhand smoke.

4 smoking, bidi smoking, chewing tobacco, and secondhand smoke.

5 sation treatment, and studies on exposure to secondhand smoke.

6 o stop smoking or are chronically exposed to secondhand smoke.

7 urportedly has fewer traditional toxins than secondhand smoke.

8 tions, in the urine of nonsmokers exposed to secondhand smoke.

9 o stop were reviewed: industry manipulation, secondhand smoke, addiction, cessation, youth access, sh

10 Any exposure to secondhand smoke adversely affects both cross-sectional

11 from increasing awareness of the dangers of secondhand smoke among nonsmokers and smokers.

12 n active smoking (AS) or passive exposure to secondhand smoke and allergic conditions.

13 ion between objectively measured exposure to secondhand smoke and biomarkers of heart disease risk ha

14 The combination of secondhand smoke and hypercholesterolemia resulted in in

15 Both secondhand smoke and hypercholesterolemia were associate

16 e stress, we hypothesized that risk factors (secondhand smoke and hypercholesterolemia) are associate

17 uit smoking to reduce children's exposure to secondhand smoke and their risk of ADHD.

18 al smoking during the in utero period and to secondhand smoke are associated with occurrence of child

19 The effects of secondhand smoke are substantial and rapid, explaining t

20 nts indicated that industry manipulation and secondhand smoke are the most effective strategies for d

21 ntial carcinogen for malignancies induced by secondhand smoking are not fully understood yet.

22 likely than current smokers to be exposed to secondhand smoke at home (prevalence 39.1% [95% CI 36.6-

23 o smoking among non-smokers, and exposure to secondhand smoke at home and in public places.

24 spectively on smoking status and exposure to secondhand smoke based on questionnaires and biochemical

25 They not only protect bystanders from secondhand smoke but also contribute to less smoking amo

26 eveloping fetuses, and postnatal exposure to secondhand smoke causes adverse health effects during ch

27 Secondhand smoke causes cardiovascular and respiratory d

28 % vs 78%; OR = 4.5; 95% CI, 2.8 to 7.2), and secondhand smoke counseling (80% vs 63%; OR = 2.6; 95% C

29 Secondhand smoke exposure (SHSe) is a significant modifi

30 We examined trends in and predictors of secondhand smoke exposure among nonsmoking adults to det

31 Analyses using reported secondhand smoke exposure and cotinine-measured exposure

32 the relationship between active smoking and secondhand smoke exposure and critical illness.

33 d beyond the underlying long-term decline in secondhand smoke exposure and demonstrates the positive

34 multivariate regression analyses to examine secondhand smoke exposure as measured by the proportion

35 and exposure to lead, children with reported secondhand smoke exposure at home were more likely to ha

36 As the magnitude of secondhand smoke exposure declines because of proportion

37 Secondhand smoke exposure during pregnancy increased the

38 ng history and detected a high prevalence of secondhand smoke exposure in a critically ill population

39 Our findings suggest that secondhand smoke exposure in children is strongly associ

40 Secondhand smoke exposure in the home was associated wit

41 Secondhand smoke exposure increased EPCs and plasma vasc

42 Secondhand smoke exposure increases cardiovascular risk.

43 Secondhand smoke exposure increases the risk of acute my

44 Prenatal tobacco and postnatal secondhand smoke exposure is consistently associated wit

45 factors considered in this study, preventing secondhand smoke exposure may be the most effective way

46 inos, suggests that the important effects of secondhand smoke exposure occur acutely.

47 tatus did not confound the adverse effect of secondhand smoke exposure on lung function.

48 Interaction between gene variants and secondhand smoke exposure resulted in significant percen

49 impact of England's smokefree legislation on secondhand smoke exposure was above and beyond the under

50 Secondhand smoke exposure was captured in 2.6% of trials

51 Secondhand smoke exposure was higher among those exposed

52 rinogen and homocysteine seen in relation to secondhand smoke exposure were equivalent to approximate

53 ed, and examine the association of postnatal secondhand smoke exposure with ADHD using both reported

54 The association of secondhand smoke exposure with ADHD was examined by two

55 rature examines the association of postnatal secondhand smoke exposure with attention deficit hyperac

56 (86%) "seldom" or "never" ask patients about secondhand smoke exposure, 245 (84%) "always" or "period

57 Because of the strong relationship among secondhand smoke exposure, bacterial infection, and sinu

58 tigated the cross-sectional relation between secondhand smoke exposure, measured objectively as cotin

59 for lifetime number of cigarettes smoked and secondhand smoke exposure.

60 rons at risk for adverse events triggered by secondhand smoke exposure.

61 ciation is robust using both measurements of secondhand smoke exposure.

62 HD using both reported and cotinine-measured secondhand smoke exposure.

63 ow that male smoking prevalence approximates secondhand smoke exposure.

64 e (TGFbeta1) amplify the negative effects of secondhand smoke exposure.

65 Temporal trends of secondhand smoke exposure: nonsmoking workers in the Uni

66 Evidence is less clear for passive (secondhand) smoke exposure.

67 idence to suggest that low-level exposure to secondhand smoke has a clinically important effect on su

68 Exposure to secondhand smoke has been associated with a disproportio

69 Secondhand smoke has detrimental effects on vascular smo

70 Exposure to secondhand smoke has plummeted.

71 cessation efforts Additional restriction of secondhand smoke in any places where the public may cong

72 Of 812 participants with data on secondhand smoke in the home, 188 (23.2%) were exposed.

73 the importance of investigating paternal and secondhand smoking in addition to maternal smoking in AS

74 Secondhand smoke increased EMPs and decreased FMD.

75 Secondhand smoke increases the risk of coronary heart di

76 Secondhand smoke increases the risk of vascular disease

77 Secondhand smoke-induced endothelial dysfunction has bee

78 rmally harmless Ag, and they may explain why secondhand smoke is a major risk factor for the developm

79 ogistic regression models that differ in the secondhand smoke measurement used.

80 active smokers (n = 6, 21%) or as exposed to secondhand smoke (n = 21, 75%).

81 search describing the mechanistic effects of secondhand smoke on the cardiovascular system, emphasizi

82 Interactions between diet and exposure to secondhand smoke on the prevalence of childhood obesity:

83 Although secondhand smoke or environmental tobacco smoke (ETS) ha

84 crease in the weekly duration of exposure to secondhand smoke (P<0.001 by the chi-square test for tre

85 osure to sidestream cigarette smoke (SS), or secondhand smoke, promoted nicotinic acetylcholine recep

86 men who reported smoking or being exposed to secondhand smoke (rho = 0.24).

87 gated the independent effects of exposure to secondhand smoke, road vehicle traffic, and dietary frui

88 , recent smokers or significantly exposed to secondhand smoke (SHS) (0.5-13.9 ng/mL), lightly exposed

89 n developed for the detection of cotinine, a secondhand smoke (SHS) biomarker.

90 the association between objectively measured secondhand smoke (SHS) exposure and incident cardiovascu

91 Secondhand smoke (SHS) exposure has been linked to the w

92 Secondhand smoke (SHS) exposure has been related to vari

93 Secondhand smoke (SHS) exposure is a known risk factor f

94 ing pregnancy (in utero smoking) and current secondhand smoke (SHS) exposure on asthma control is poo

95 study sought to analyze the effects of acute secondhand smoke (SHS) exposure on the number and functi

96 We sought to determine the effects of secondhand smoke (SHS) exposure on vascular reactivity i

97 Despite progress in tobacco control, secondhand smoke (SHS) exposure remains prevalent worldw

98 Secondhand smoke (SHS) has been associated with a variet

99 Children's exposure to secondhand smoke (SHS) has been causally linked to a num

100 The scientific consensus that secondhand smoke (SHS) increases cardiovascular disease

101 Secondhand smoke (SHS) is associated with a 30% increase

102 Exposure to secondhand smoke (SHS) may increase risk for obesity, bu

103 creased risk of dementia, but the effects of secondhand smoke (SHS) on dementia risk are not known to

104 oth active smoking and household exposure to secondhand smoke (SHS) on subsequent bladder cancer risk

105 characterized the exposure of nonsmokers to secondhand smoke (SHS) outside a restaurant and bar in A

106 uch intervention has not yet been applied to secondhand smoke (SHS) reduction programs that target sm

107 levels of individual VOCs in freshly emitted secondhand smoke (SHS) were in the range of 1-300 mug m(

108 alence have been associated with exposure to secondhand smoke (SHS), maternal smoking during pregnanc

109 nge of relatively low concentrations of aged secondhand smoke (SHS), similar to those encountered com

110 side of the home for nonsmokers' exposure to secondhand smoke (SHS).

111 effects of low levels of exposure including secondhand smoke (SHS).

112 njunction with the industry manipulation and secondhand smoke strategies.

113 High exposure to secondhand smoke suggests a need for countries to pass s

114 t ultraviolet radiation, 4-aminobiphenyl and secondhand smoke that are known to be strong, moderate a

115 -control messages emphasizing the dangers of secondhand smoke to smokers and nonsmokers undermine the

116 A public ordinance reducing exposure to secondhand smoke was associated with a decrease in AMI h

117 increase of surface-bound TSNAs when sorbed secondhand smoke was exposed to 60 ppbv HONO for 3 hours

118 Student exposure to secondhand smoke was high both at home (more than four i

119 nd apoE(-/-) mice exposed to filtered air or secondhand smoke were quantified.

120 ged 4-15 years who were exposed to postnatal secondhand smoke with prevalence in those who were not e

121 shed since 1995, and compared the effects of secondhand smoke with the effects of active smoking.