ライフサイエンスコーパス: sedentary

1 r memory recall as animals that had remained sedentary.

2 Pacific Ocean, whereas most from Crozet are sedentary.

3 ng, although it was modestly reduced in aged sedentary (-7%) and running (-19%) mice without satellit

4 MTG content in type I fibres (trained: +62%, sedentary: +79%; P < 0.05) but did not affect PLIN prote

5 from increasingly sophisticated marketing of sedentary activities and energy-dense, nutrient-poor foo

6 al activity, vigorous physical activity, and sedentary activity and change in total accelerometer cou

7 passive dispersal of gametes, or sluggish or sedentary adult life habits in the absence of gamete vec

8 Compared with less-sedentary adults (6 sedentary h/d), those who spent 10 s

9 Participants were sedentary adults aged 70 to 89 years who were at risk fo

10 -month randomized controlled trial among 278 sedentary adults with abdominal obesity (75%) or dyslipi

11 Forty-five males (30 athletes and 15 sedentary age-matched healthy controls) underwent compre

12 hat likely drove the Neolithic transition to sedentary agrarian societies in the Fertile Crescent mor

13 rchaeological human populations representing sedentary agriculturalists and mobile foragers.

14 ulbospinal catecholaminergic (C1) neurons in sedentary and active rats after chronic voluntary wheel-

15 f those from Crozet, especially females, are sedentary and breed in consecutive calendar years.

16 ic acid, and 2-ketoglutaric acid between the sedentary and fatigued mice.

17 s for mortality associations with time spent sedentary and in light- and moderate-to-vigorous-intensi

18 andy, and sweetened beverages and time spent sedentary and in moderate-to-vigorous physical activity.

19 iability through its differential effects on sedentary and migratory individuals.

20 e been associated with obesity phenotypes in sedentary and obese populations, but rarely with skeleta

21 smaller marine animals, and especially among sedentary and sessile species living at low densities, w

22 ntify intermediary metabolism fluxes in both sedentary and treadmill-running mice.

23 Male offspring were chow fed, sedentary, and studied at 8, 12, 24, 36, and 52 weeks of

24 Reduction of GLUT4 protein in sedentary animals upon treatment with rapamycin revealed

25 Compared with sedentary animals, the highest number of doublecortin-po

26 the association between objectively measured sedentary behavior (its total volume and accrual in prol

27 Information on sedentary behavior (television watching and computer tim

28 ntions and future quantitative guidelines on sedentary behavior and cardiovascular health outcomes.

29 haviors (for example, reduce tobacco use and sedentary behavior and improve diet) of workers, but eff

30 cipation in domains of physical activity and sedentary behavior at 7 years of age were reported.

31 nd specific domains of physical activity and sedentary behavior at the age of 7 years were associated

32 s that include accelerometer-assessed PA and sedentary behavior can contribute to these recommendatio

33 indings emphasize the importance of reducing sedentary behavior for the prevention of cardiometabolic

34 nce advisory reviews the current evidence on sedentary behavior in terms of assessment methods, popul

35 e find that objectively logged reductions in sedentary behavior in the following month are linked to

36 ulating that indicates greater time spent in sedentary behavior is associated with all-cause and card

37 possible deleterious consequences of excess sedentary behavior is rapidly evolving, with the epidemi

38 Sedentary behavior is recognized as a distinct construct

39 th increasing physical activity and reducing sedentary behavior may attenuate genetic associations wi

40 et, few studies have examined the effects of sedentary behavior on cognition or the long-term role of

41 Accelerometer-assessed sedentary behavior was generally not associated with all

42 tudy, we explored the associations of PA and sedentary behavior with all-cause and cardiovascular dis

43 Associations of physical activity and sedentary behavior with cognition appear to be context-s

44 the association of prolonged, uninterrupted sedentary behavior with glycemic biomarkers in a cohort

45 oking, alcohol consumption, body mass index, sedentary behavior, diabetes mellitus, cardiovascular di

46 interaction on obesity, yet the influence of sedentary behavior, distinct from a lack of physical act

47 udinal associations of physical activity and sedentary behavior, measured both objectively and via se

48 Parity was associated with sedentary behavior, weight gain, and higher triglyceride

49 howed significant interactions with MVPA and sedentary behavior, with effects on BMI and other adipos

50 commendations regarding the risks related to sedentary behavior.

51 Sedentary behaviors and physical inactivity are not only

52 Meal contexts and sedentary behaviors may represent important intervention

53 iseases, body mass index, diabetes, smoking, sedentary behaviors, and alcohol intake.

54 ssages encouraging exercise and discouraging sedentary behaviors.

55 d broad guidelines that recommend minimizing sedentary behaviors.

56 or 7 d, and self-reported meal practices and sedentary behaviors.

57 behaviors in adults; interventions to reduce sedentary behaviors; and the harms of behavioral counsel

58 We examined the associations of sedentary behaviour and physical activity with all-cause

59 ility (C. cinereum and S. magellanicus), and sedentary behaviour by two terrestrial-foraging species

60 High amounts of sedentary behaviour have been associated with increased

61 al activity is inversely associated with and sedentary behaviour is positively (and independently) as

62 The lifestyle factors of physical activity, sedentary behaviour, and diet are increasingly being stu

63 omising use of strategies that aim to reduce sedentary behaviours as therapy to improve metabolic fle

64 c flexibility, while physical inactivity and sedentary behaviours trigger a state of metabolic 'infle

65 social isolation, low physical activity, and sedentary behaviours.

66 yer in the transition between planktonic and sedentary biofilm-associated bacterial lifestyles.

67 Cereal cyst nematodes are sedentary biotrophic endoparasites that maintain a compl

68 60 to 4.30]; P for trend < 0.001) and longer sedentary bout duration (HR, 1.03 [CI, 0.67 to 1.60]; HR

69 High total sedentary time or high sedentary bout duration alone were not associated with d

70 ; however, a significant interaction between sedentary bout duration and total sedentary time was obs

71 oint association of total sedentary time and sedentary bout duration showed that participants in the

72 derate to vigorous physical activity, longer sedentary bout duration was dose-dependently associated

73 tics (ie, high total sedentary time and high sedentary bout duration) had the highest levels of homeo

74 entary time; 7.7, 9.6, and 12.4 min/bout for sedentary bout duration) in models that included moderat

75 errupted sedentariness was expressed as mean sedentary bout length.

76 Females were bred with chow-fed sedentary C57BL/6 males.

77 10 weeks of exercise training or remained as sedentary, cage-controls.

78 Twenty-seven sedentary Cameroonian volunteer men participated in the

79 hat participants classified as high for both sedentary characteristics (high sedentary time [>/=12.5

80 participants in the upper quartile for both sedentary characteristics (ie, high total sedentary time

81 esistance (P<0.001 versus low group for both sedentary characteristics) and 2-hour glucose (P=0.002 v

82 r glucose (P=0.002 versus low group for both sedentary characteristics).

83 Average DERs of sedentary, clinically stable patients receiving MHD are

84 Compared with sedentary conditions, exercise performed immediately aft

85 e expression were evaluated in exercised and sedentary control mice.

86 in young competitive athletes compared with sedentary controls, but rarely >40 mm.

87 e reached a value similar to that in healthy sedentary controls, the increase in peak arteriovenous o

88 rved in exercised rodent muscles compared to sedentary controls; however, exercise-induced Mdm2 phosp

89 High fat-fed sedentary dams produced female offspring with impaired g

90 High-fat feeding of sedentary dams resulted in impaired glucose tolerance, i

91 pose tissue (scWAT) from exercise-trained or sedentary donor mice was transplanted into the visceral

92 state switching between active (roaming) and sedentary (dwelling) states; however, when provided with

93 xercise training leads to similar changes in sedentary elderly men.

94 Cyst nematodes are sedentary endoparasites that induce the formation of mul

95 Far from being unsophisticated compared with sedentary endoparasites, migratory endoparasites are exq

96 ilarities in lifestyle between migratory and sedentary endoparasites.

97 n roots is less obvious than infestations of sedentary endoparasites; nevertheless, in many instances

98 d molecular mechanisms between migratory and sedentary endoparasitic nematodes.

99 five groups (n = 8-14): C (control diet and sedentary), F (fed the fructose-rich diet and sedentary)

100 edentary), F (fed the fructose-rich diet and sedentary), FA (fed the fructose-rich diet and subject t

101 iles for 362 rainforest hunter-gatherers and sedentary farmers.

102 Sedentary farming communities emerged in parts of the Fe

103 ological documentation of the development of sedentary farming societies in Anatolia is not yet mirro

104 et of plant root cells and must sustain this sedentary feeding site for 2-4 weeks.

105 Sedentary female flower mantises dramatically increased

106 behaviour) with respect to wheel running and sedentary female Wistar rats at 8 and 14 weeks of age.

107 ntary (third tertile) versus less time spent sedentary (first tertile) (beta = 0.73 kg/m(2) [SE, 0.10

108 oup conflict was rare until the emergence of sedentary foraging and complex sociopolitical organizati

109 in per day, 5 days a week, for 6 weeks) or a sedentary group that was not exercised and analysed lymp

110 not statistically different from that of the sedentary group.

111 ns/week of aerobic activities >/=30 minutes: sedentary (group 1), <2 sessions; casual (group 2), 2 to

112 adults (6 sedentary h/d), those who spent 10 sedentary h/d had 29% greater risk (HR: 1.29; 95% CI: 1.

113 Compared with less-sedentary adults (6 sedentary h/d), those who spent 10 sedentary h/d had 29%

114 s to have long-term stable distributions and sedentary habits.

115 rs) athletes with HCM were compared with 101 sedentary HCM patients.

116 .4 versus 10.7+/-3.9, P<0.001) compared with sedentary HCM patients.

117 d intensive endurance training in previously sedentary healthy young individuals could induce cardiac

118 heel running (exercised mice) or remained in sedentary housing (sedentary mice).

119 iomarkers of cardiovascular health in young, sedentary humans.

120 m, in some instances cultivation began among sedentary hunter-gatherers whereas more often it was ini

121 example, increasing physical activity among sedentary individuals affects a wide range of important

122 The inherent inaccessibility of sweat in sedentary individuals in large volume (>/=10 microL) for

123 lipid storage in muscle between trained and sedentary individuals is unknown.

124 lar to published estimated values for normal sedentary individuals of similar age and sex, there was

125 By contrast, in sedentary individuals, lipid infusion only increased the

126 lipid storage in muscle between trained and sedentary individuals.

127 ry artery calcium (CAC) scores compared with sedentary individuals.

128 content in type I fibres of both trained and sedentary individuals.

129 in content was higher in trained compared to sedentary individuals.

130 S/NADP(H)oxidase protein ratio in previously sedentary lean and obese young men.

131 e of smoking (42.6% to 36.5%; RR = 0.86) and sedentary lifestyle (43.4% to 29.0%; RR = 0.69) declined

132 (OR, 1.12 [95% CI, 1.08-1.17]; P < .001), a sedentary lifestyle (OR, 2.99 [95% CI, 1.62-5.55]; P = .

133 idemic is largely secondary to an increasing sedentary lifestyle and highly prevalent overweight and

134 lic syndrome, smoking, hyperlipidemia, and a sedentary lifestyle are the major modifiable risk factor

135 nd cardiorespiratory fitness compared with a sedentary lifestyle during similar weight loss.

136 Self-reported sedentary lifestyle was associated with less diary use i

137 hy young Danish men, we investigated whether sedentary lifestyle was associated with testicular funct

138 ood production, but the transition to a more sedentary lifestyle was gradual, extending through later

139 with low intake of fruit and vegetables and sedentary lifestyle was performed.

140 2)): 27-37; age: 18-40 y] who had a normally sedentary lifestyle were randomly allocated to 1 of 2 in

141 essive alcohol intake, smoking, obesity, and sedentary lifestyle) in populations with low socioeconom

142 egative health behaviors (e.g., poor diet, a sedentary lifestyle) may act as mediating pathways that

143 ors including body mass index, healthy diet, sedentary lifestyle, alcohol consumption, smoking, and u

144 ase due to high rates of overweight/obesity, sedentary lifestyle, and other factors.

145 This review concludes that a sedentary lifestyle, obesity and ageing impair the vasod

146 Previous research has shown that a sedentary lifestyle, obesity and ageing lead to impairme

147 ates of relative hyperinsulinemia, including sedentary lifestyle, obesity, and increased dietary glyc

148 o reduce liver diseases that are linked to a sedentary lifestyle, overeating, and genetic factors.

149 Other modifiable risk factors include sedentary lifestyle, smoking, obesity, diabetes mellitus

150 Here, compared with a sedentary lifestyle, we report a very modest effect of H

151 smoke cigarettes, consume alcohol, and have sedentary lifestyle.

152 isk factors are obesity, family history, and sedentary lifestyle.

153 d to prevent falls, and reduce smoking and a sedentary lifestyle.

154 Such an extreme "sedentary" lifestyle is consistent with the proposed rol

155 dominantly calcific plaques (72.7%), whereas sedentary males showed predominantly mixed morphology pl

156 ston units or coronary plaques compared with sedentary males with a similar risk profile.

157 (44.3% versus 22.2%; P=0.009) compared with sedentary males, and only male athletes showed a CAC >/=

158 Sedentary MCK-PGC1alpha mice were more susceptible to di

159 Overweight, sedentary men and women (n = 52; aged 45-65 y) were rand

160 We randomized 1635 sedentary men and women aged 70 to 89 years with a Short

161 isms for plaque formation in athletic versus sedentary men.

162 s were preserved post METH administration in sedentary mice and were associated with the development

163 We tested this hypothesis in male sedentary mice by experimentally depleting satellite cel

164 to quantitate OAs in quadriceps muscle from sedentary mice compared to fatigued mice subjected to ei

165 High-fat diet in sedentary mice led to endoplasmic reticulum stress and a

166 ised mice) or remained in sedentary housing (sedentary mice).

167 whole-body insulin sensitivity compared with sedentary mice, insulin action was not affected by AraC

168 cyte gap junctions, a change not observed in sedentary mice.

169 abelled and ablated satellite cells in adult sedentary mice.

170 ventricular (RV) phenotype in nonexercised (sedentary) mice.

171 Trained (n = 11) and sedentary (n = 10) subjects, matched for age, sex and bo

172 Compared with sedentary nonjoggers, 1 to 2.4 h of jogging per week was

173 d moderate joggers have lower mortality than sedentary nonjoggers, whereas strenuous joggers have a m

174 tients receiving MHD are similar to those of sedentary normal individuals.

175 signature by gene-chip profiling tissue from sedentary normal subjects who reached 65 years of age in

176 Sedentary nutrient fluxes in the postabsorptive state we

177 additional WD-fed OLETFs were randomized to sedentary (O-SED), food restriction (O-FR; approximately

178 uscle metabolism between overweight-to-obese sedentary (Ob/Sed) and lean active (L/Ac) individuals th

179 We found that sedentary obese mice had markedly decreased collecting l

180 ly improved lymphatic function compared with sedentary obese mice.

181 biopsies were taken from eight Trained, Lean sedentary, Obese and T2DM subjects.

182 rcise training and detraining, 12 previously sedentary older (65 +/- 3 years) men and women underwent

183 Among sedentary older adults, a 24-month moderate-intensity ph

184 pared with mice transplanted with scWAT from sedentary or sham-treated mice.

185 ted cell-to-cell communication to survive as sedentary organisms.

186 The largest benefits occurred between sedentary patient groups and between those with the high

187 indices of diastolic function compared with sedentary patients.

188 l feasibility studies in extremely unfit and sedentary people.

189 ods, monkey age, and possibly a postexercise sedentary period but no direct effect of exercise was fo

190 Obese and sedentary persons have an increased risk for cancer, but

191 Participants: 1635 sedentary persons, aged 70 to 89 years, who had function

192 pine density was lower in 14- vs. 8-week-old sedentary rats (P = 0.03).

193 In aged sedentary rats, compared to young sedentary rats, a 42% increase in IVRT, a 64% decrease i

194 In aged sedentary rats, compared to young sedentary rats, a 42%

195 alysed by quantitative RT-PCR in age-matched sedentary rats.

196 ins from 8 to 14 weeks of age in running and sedentary rats.

197 Transplantation of trained scWAT in sedentary recipient mice results in striking improvement

198 was transplanted into the visceral cavity of sedentary recipients.

199 ecifically, we examined whether total HRV at sedentary rest (measured as the SD of normal-to-normal i

200 Young rats assigned to either sedentary (SED) or running exercise (RE) groups were fir

201 The adoption of farming, stock breeding and sedentary societies during the Neolithic may have result

202 the transition from hunting and gathering to sedentary societies, laying the foundation for the devel

203 istence of warfare before the development of sedentary societies.

204 species is dramatically larger than for more sedentary species.

205 n was maintained in the syncytium during all sedentary stages of nematode development.

206 ed with running wheels during gestation), or sedentary (static cages).

207 Twenty young, sedentary subjects were assigned to participate in 8 wee

208 r greater than what is typically observed in sedentary subjects with exercise training.

209 on biomarkers of vascular function in young, sedentary subjects.

210 = 0.005), and in those with more time spent sedentary (third tertile) versus less time spent sedenta

211 Sedentary time (<100 counts/min) was standardized to 16

212 95% CI: 0.0, 0.9), and objectively measured sedentary time (B = 0.8, 95% CI: 0.1, 1.4) at age 7 year

213 circumference was positively associated with sedentary time (B = 0.82, P < 0.001).

214 Birth weight was positively associated with sedentary time (B = 4.04, P = 0.006) and waist circumfer

215 risk associated with intermediate levels of sedentary time (HR for 7.5 h/d, 1.02; 95% CI, 0.96-1.08)

216 multivariable-adjusted models, greater total sedentary time (HR, 1.22 [95% CI, 0.74 to 2.02]; HR, 1.6

217 xamined associations of objectively measured sedentary time (via Actical accelerometers for 7 days) a

218 dent CVD associated with different levels of sedentary time (vs lowest sedentary time) across studies

219 igh for both sedentary characteristics (high sedentary time [>/=12.5 h/d] and high bout duration [>/=

220 he relationship between objectively measured sedentary time and cardiometabolic biomarkers are sparse

221 data showed deleterious associations between sedentary time and cardiometabolic biomarkers, independe

222 ntitative, dose-response association between sedentary time and CVD risk is not known.

223 Except for sedentary time and diet, all examined health-related beh

224 th sedentary characteristics (ie, high total sedentary time and high sedentary bout duration) had the

225 us analyses, a nonlinear association between sedentary time and incident CVD was found (P for nonline

226 older that reported the association between sedentary time and incident CVD were included.

227 Both the total volume of sedentary time and its accrual in prolonged, uninterrupt

228 1) described the mortality dose-response for sedentary time and light- and moderate-to-vigorous-inten

229 The association between sedentary time and LTL was evaluated using multiple line

230 nsistency, and manner of association between sedentary time and outcomes independent of physical acti

231 Hazard ratios associated with sedentary time and outcomes were generally more pronounc

232 ildren (49.1% males; 50.9% females) in which sedentary time and physical activity were measured with

233 ty in research designs and the assessment of sedentary time and physical activity.

234 Evaluation of the joint association of total sedentary time and sedentary bout duration showed that p

235 ve associations between objectively measured sedentary time and subcomponents of physical activity wi

236 The association between sedentary time and the risk for CVD is nonlinear with an

237 The association between birth weight and sedentary time appears partially mediated by central adi

238 easing light-intensity activity and reducing sedentary time are also important, particularly for inac

239 lf-reporting to evaluate the total volume of sedentary time as a prognostic risk factor for mortality

240 edian, 2.5 h/d), participants in the highest sedentary time category (median, 12.5 h/d) had an increa

241 tegorical analyses, compared with the lowest sedentary time category (median, 2.5 h/d), participants

242 Daily unsupervised physical activity and sedentary time did not change in any exercise group vers

243 e highest quartile of accelerometer-measured sedentary time had significantly shorter LTL than those

244 Whether birth weight predicts later sedentary time in humans is uncertain.

245 Accruing sedentary time in prolonged, uninterrupted bouts may be

246 cise fall far short of replacing most of the sedentary time in the modern lifestyle, because both the

247 amined the relation between birth weight and sedentary time in youth and examined whether this associ

248 and abdominal adiposity may be correlates of sedentary time in youth.

249 s, low levels of physical activity, and high sedentary time increase the risk of cardiovascular disea

250 es have examined whether the manner in which sedentary time is accrued (in short or long bouts) carri

251 ave not examined whether the manner in which sedentary time is accrued (in short or long bouts) carri

252 Prior studies suggest that higher sedentary time is associated with a greater risk for car

253 Excessive sedentary time is ubiquitous in developed nations and is

254 Excessive sedentary time is ubiquitous in Western societies.

255 basis of accelerometer measurements, higher sedentary time may be associated with shorter LTL among

256 High total sedentary time or high sedentary bout duration alone wer

257 There was no association with mortality for sedentary time or light or moderate-to-vigorous activity

258 ased risk observed for more than 10 hours of sedentary time per day (pooled HR, 1.08; 95% CI, 1.00-1.

259 This can explain why high amounts of sedentary time produce distinct metabolic and cardiovasc

260 lity benefits of light activity and risk for sedentary time remain uncertain.

261 gnificant indirect effect of birth weight on sedentary time through waist circumference (B: 1.30; 95%

262 eir physical fitness levels and reduce their sedentary time to prevent central obesity-related asthma

263 ines should target reducing and interrupting sedentary time to reduce risk for death.

264 The mean sedentary time was 11.9 hours/d (74% of accelerometer we

265 easured in hospitals or maternally reported, sedentary time was assessed by using accelerometry (<100

266 ctivity and confounding variables, prolonged sedentary time was associated with decreased high-densit

267 ontrolled for, the effect of birth weight on sedentary time was attenuated by 32% (B = 2.74, P = 0.06

268 iduals meeting physical activity guidelines, sedentary time was detrimentally associated with several

269 Prolonged sedentary time was independently associated with deleter

270 Sedentary time was measured using a hip-mounted accelero

271 ured at baseline and at follow-up 7 mo later.Sedentary time was not associated with any of the indivi

272 Accelerometer-measured sedentary time was not associated with LTL in women abov

273 Self-reported sedentary time was not associated with LTL.

274 Sedentary time was objectively measured with a hip-mount

275 on between sedentary bout duration and total sedentary time was observed.

276 Daily unsupervised physical activity and sedentary time were measured by accelerometer.

277 Energy intake, HPA, and sedentary time were measured with the use of a 3-d weigh

278 In less-active adults, replacing 1 h of sedentary time with either light- or moderate-to-vigorou

279 Few studies have assessed the association of sedentary time with leukocyte telomere length (LTL).

280 of accelerometer-measured and self-reported sedentary time with LTL in a sample of 1,481 older white

281 ugh safely replacing a large amount of daily sedentary time with physical activity in everyone, regar

282 mortality benefits associated with replacing sedentary time with physical activity, accounting for to

283 ifferent levels of sedentary time (vs lowest sedentary time) across studies.

284 alcohol intake, diet, physical activity, and sedentary time), comorbid conditions (diabetes, hyperten

285 odels estimated replacement associations for sedentary time, and separate models were fit for low- (<

286 adjusting for WC.Physical activity, but not sedentary time, is prospectively associated with cardiom

287 diet, increased physical activity, decreased sedentary time, or a combination of these among adults w

288 east moderate intensity rather than reducing sedentary time.

289 sociated with reduced activity and increased sedentary time.

290 ies and sleep duration when reallocated from sedentary time.

291 etabolic risk factors that is independent of sedentary time.

292 ntake, habitual physical activity (HPA), and sedentary time.

293 nts: 689.7, 746.5, and 799.4 min/d for total sedentary time; 7.7, 9.6, and 12.4 min/bout for sedentar

294 e associations were not independent of total sedentary time; however, a significant interaction betwe

295 hort designs were used in all but 3 studies; sedentary times were quantified using self-report in all

296 al activity, regardless of dose ranging from sedentary to competitive marathon running, was not assoc

297 support a model in which the transition from sedentary to light activity is associated with an increa

298 The mean (+/-SD) time spent sedentary was 370 +/- 91 min/d.

299 rous physical activity (MVPA) and time spent sedentary with genetic variants on obesity among 9,645 U

300 ectiveness of TWH interventions for reducing sedentary work behavior in office workers over 16 to 52