ライフサイエンスコーパス: sedentary time

1 whilst also scoring poorly in another (e.g., sedentary time).

2 y activity, vigorous intensity activity, and sedentary time).

3 associated with larger increases in weekend sedentary time.

4 d for age, sex, body mass index and baseline sedentary time.

5 wards preventing the age-related increase in sedentary time.

6 ; 0.37, 1.66) and weekend (1.42; 0.65, 2.18) sedentary time.

7 time and 3.6 cm (95% CI, 2.8-4.3 cm) for low sedentary time.

8 lic risk factors regardless of the amount of sedentary time.

9 igher and lower MVPA were greater with lower sedentary time.

10 diometabolic risk factors across tertiles of sedentary time.

11 ants were stratified by tertiles of MVPA and sedentary time.

12 etabolic risk factors that is independent of sedentary time.

13 ntake, habitual physical activity (HPA), and sedentary time.

14 east moderate intensity rather than reducing sedentary time.

15 sociated with reduced activity and increased sedentary time.

16 ies and sleep duration when reallocated from sedentary time.

17 min/day, steps 3111+/-2290 vs. 7996+/-2649, sedentary time 1383+/-42 vs. 1339+/-44 min/day, p<0.01).

18 nts: 689.7, 746.5, and 799.4 min/d for total sedentary time; 7.7, 9.6, and 12.4 min/bout for sedentar

19 ifferent levels of sedentary time (vs lowest sedentary time) across studies.

20 nt-hours/day) was strongest for occupational sedentary time (adjusted CCC = 0.76, 95% CI: 0.64, 0.85)

21 Medium sedentary time also conveyed risk (hazard ratio, 1.13 [9

22 PA were 5.6 cm (95% CI, 4.8-6.4 cm) for high sedentary time and 3.6 cm (95% CI, 2.8-4.3 cm) for low s

23 We determined measurement properties of the Sedentary Time and Activity Reporting Questionnaire (STA

24 he relationship between objectively measured sedentary time and cardiometabolic biomarkers are sparse

25 data showed deleterious associations between sedentary time and cardiometabolic biomarkers, independe

26 No association was found between sedentary time and cardiorespiratory fitness (r = -.13,

27 ntitative, dose-response association between sedentary time and CVD risk is not known.

28 Except for sedentary time and diet, all examined health-related beh

29 th sedentary characteristics (ie, high total sedentary time and high sedentary bout duration) had the

30 us analyses, a nonlinear association between sedentary time and incident CVD was found (P for nonline

31 older that reported the association between sedentary time and incident CVD were included.

32 Both the total volume of sedentary time and its accrual in prolonged, uninterrupt

33 1) described the mortality dose-response for sedentary time and light- and moderate-to-vigorous-inten

34 The association between sedentary time and LTL was evaluated using multiple line

35 nsistency, and manner of association between sedentary time and outcomes independent of physical acti

36 Hazard ratios associated with sedentary time and outcomes were generally more pronounc

37 Sedentary time and PA were also mutually adjusted for on

38 ildren (49.1% males; 50.9% females) in which sedentary time and physical activity were measured with

39 ty in research designs and the assessment of sedentary time and physical activity.

40 Evaluation of the joint association of total sedentary time and sedentary bout duration showed that p

41 tantial validity for estimating occupational sedentary time and strenuous activity and fair validity

42 ve associations between objectively measured sedentary time and subcomponents of physical activity wi

43 The association between sedentary time and the risk for CVD is nonlinear with an

44 The mean biases were relatively small for sedentary time and vigorous PA: 0.7 +/- 2.8 h/d and -12

45 ctively measured time spent being sedentary (sedentary time) and obesity indicators.

46 Physical activity, sedentary time, and behavioral covariates were obtained

47 etween physical activity (PA) subcomponents, sedentary time, and body composition in preschoolers rem

48 odels estimated replacement associations for sedentary time, and separate models were fit for low- (<

49 e; (3) self-reported or objectively measured sedentary time; and (4) an outcome measure of metabolic

50 vigorous physical activity (MVPA) intensity; sedentary time; and body composition were analyzed by us

51 The association between birth weight and sedentary time appears partially mediated by central adi

52 easing light-intensity activity and reducing sedentary time are also important, particularly for inac

53 lf-reporting to evaluate the total volume of sedentary time as a prognostic risk factor for mortality

54 , 0.40), and WC (0.44; 0.23, 0.66) predicted sedentary time at follow-up after adjustment for sex, ba

55 seline was associated with higher amounts of sedentary time at follow-up.

56 95% CI: 0.0, 0.9), and objectively measured sedentary time (B = 0.8, 95% CI: 0.1, 1.4) at age 7 year

57 circumference was positively associated with sedentary time (B = 0.82, P < 0.001).

58 Birth weight was positively associated with sedentary time (B = 4.04, P = 0.006) and waist circumfer

59 r adjustment for sex, baseline age, baseline sedentary time, baseline physical activity energy expend

60 BMI, FM, and WC may predict sedentary time, but our results do not suggest that sede

61 edian, 2.5 h/d), participants in the highest sedentary time category (median, 12.5 h/d) had an increa

62 tegorical analyses, compared with the lowest sedentary time category (median, 2.5 h/d), participants

63 alcohol intake, diet, physical activity, and sedentary time), comorbid conditions (diabetes, hyperten

64 ncrease physical activity level and decrease sedentary time could help reduce mortality risk in black

65 Daily unsupervised physical activity and sedentary time did not change in any exercise group vers

66 Sedentary time did not predict any of the obesity indica

67 Understanding the determinants of sedentary time during childhood contributes to the devel

68 igh for both sedentary characteristics (high sedentary time [>/=12.5 h/d] and high bout duration [>/=

69 e highest quartile of accelerometer-measured sedentary time had significantly shorter LTL than those

70 ysical activity were significantly lower and sedentary time higher in the stroke group compared to co

71 e associations were not independent of total sedentary time; however, a significant interaction betwe

72 risk associated with intermediate levels of sedentary time (HR for 7.5 h/d, 1.02; 95% CI, 0.96-1.08)

73 multivariable-adjusted models, greater total sedentary time (HR, 1.22 [95% CI, 0.74 to 2.02]; HR, 1.6

74 Whether birth weight predicts later sedentary time in humans is uncertain.

75 This highlights the importance of reducing sedentary time in order to improve metabolic health, pos

76 Accruing sedentary time in prolonged, uninterrupted bouts may be

77 cise fall far short of replacing most of the sedentary time in the modern lifestyle, because both the

78 amined the relation between birth weight and sedentary time in youth and examined whether this associ

79 and abdominal adiposity may be correlates of sedentary time in youth.

80 s, low levels of physical activity, and high sedentary time increase the risk of cardiovascular disea

81 es have examined whether the manner in which sedentary time is accrued (in short or long bouts) carri

82 ave not examined whether the manner in which sedentary time is accrued (in short or long bouts) carri

83 Prior studies suggest that higher sedentary time is associated with a greater risk for car

84 Excessive sedentary time is ubiquitous in developed nations and is

85 Excessive sedentary time is ubiquitous in Western societies.

86 adjusting for WC.Physical activity, but not sedentary time, is prospectively associated with cardiom

87 Sedentary time (<100 counts/min) was standardized to 16

88 Both physical activity and sedentary time may be appropriate intervention targets f

89 basis of accelerometer measurements, higher sedentary time may be associated with shorter LTL among

90 collecting data regarding physical fitness, sedentary time, obesity measures (comprising body weight

91 Sedentary time (% of daytime hours) was measured by indi

92 High total sedentary time or high sedentary bout duration alone wer

93 There was no association with mortality for sedentary time or light or moderate-to-vigorous activity

94 diet, increased physical activity, decreased sedentary time, or a combination of these among adults w

95 (p = 0.04), and 13.2 (1.3, 25.2) min/d more sedentary time (p = 0.03).

96 x10(-)(2)(9)), and 3.5 (1.5, 5.5) min/d more sedentary time (p = 5.0x10(-)(4)).

97 ased risk observed for more than 10 hours of sedentary time per day (pooled HR, 1.08; 95% CI, 1.00-1.

98 to analyze the interrelation among obesity, sedentary time, physical fitness level, and asthma; a st

99 ry time, but our results do not suggest that sedentary time predicts future obesity.

100 This can explain why high amounts of sedentary time produce distinct metabolic and cardiovasc

101 (r = 0.70) and marginally insignificant for sedentary time (r = 0.27, P = 0.06).

102 lity benefits of light activity and risk for sedentary time remain uncertain.

103 , and the role of physical fitness level and sedentary time remains unexplored in the link between ob

104 Controlling for sedentary time, sociodemographics, hypertension, diabete

105 gnificant indirect effect of birth weight on sedentary time through waist circumference (B: 1.30; 95%

106 eir physical fitness levels and reduce their sedentary time to prevent central obesity-related asthma

107 ines should target reducing and interrupting sedentary time to reduce risk for death.

108 xamined associations of objectively measured sedentary time (via Actical accelerometers for 7 days) a

109 dent CVD associated with different levels of sedentary time (vs lowest sedentary time) across studies

110 The mean sedentary time was 11.9 hours/d (74% of accelerometer we

111 easured in hospitals or maternally reported, sedentary time was assessed by using accelerometry (<100

112 ctivity and confounding variables, prolonged sedentary time was associated with decreased high-densit

113 ontrolled for, the effect of birth weight on sedentary time was attenuated by 32% (B = 2.74, P = 0.06

114 Sedentary time was calculated as the heart rate observat

115 iduals meeting physical activity guidelines, sedentary time was detrimentally associated with several

116 Prolonged sedentary time was independently associated with deleter

117 Sedentary time was measured using a hip-mounted accelero

118 ured at baseline and at follow-up 7 mo later.Sedentary time was not associated with any of the indivi

119 Sedentary time was not associated with any outcome indep

120 Accelerometer-measured sedentary time was not associated with LTL in women abov

121 Self-reported sedentary time was not associated with LTL.

122 Sedentary time was objectively measured with a hip-mount

123 on between sedentary bout duration and total sedentary time was observed.

124 At follow-up, sedentary time was significantly correlated with BW (par

125 At baseline, sedentary time was significantly correlated with FM (par

126 in systolic blood pressure for high and low sedentary time were 0.7 mm Hg (95% CI, -0.07 to 1.6) and

127 Daily unsupervised physical activity and sedentary time were measured by accelerometer.

128 Physical activity and sedentary time were measured objectively by individually

129 Time spent in MVPA and sedentary time were measured using accelerometry after r

130 Energy intake, HPA, and sedentary time were measured with the use of a 3-d weigh

131 (N = 6413 at 2.1 years' follow-up), MVPA and sedentary time were not associated with waist circumfere

132 Smaller increases in after-school sedentary time were observed in children with more sibli

133 hort designs were used in all but 3 studies; sedentary times were quantified using self-report in all

134 en objectively measured PA subcomponents and sedentary time with body composition in 4-y-old children

135 d associations between physical activity and sedentary time with cardiometabolic risk factors in heal

136 In less-active adults, replacing 1 h of sedentary time with either light- or moderate-to-vigorou

137 Additionally, replacing 30 minutes/day of sedentary time with equal amounts of low-light or high-l

138 d with the reallocation of 30 minutes/day of sedentary time with equal time of either sleep (2.2% low

139 Few studies have assessed the association of sedentary time with leukocyte telomere length (LTL).

140 of accelerometer-measured and self-reported sedentary time with LTL in a sample of 1,481 older white

141 ugh safely replacing a large amount of daily sedentary time with physical activity in everyone, regar

142 mortality benefits associated with replacing sedentary time with physical activity, accounting for to

143 endent associations between time in MVPA and sedentary time, with outcomes, were examined using meta-