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1 a region with genes linked to familial focal segmental glomerulosclerosis.
2 ly in podocytes, in human kidneys with focal segmental glomerulosclerosis.
3 membrane splitting, and secondary focal and segmental glomerulosclerosis.
4 ssure did correlate with the degree of focal segmental glomerulosclerosis.
5 ith renal failure caused by idiopathic focal segmental glomerulosclerosis.
6 ized by skeletal changes, glaucoma and focal segmental glomerulosclerosis.
7 e, but chronic overactivation leads to focal segmental glomerulosclerosis.
8 ion mutations cause autosomal dominant focal segmental glomerulosclerosis.
9 -associated nephropathy and idiopathic focal segmental glomerulosclerosis.
10 ic kidney disease that resembles human focal segmental glomerulosclerosis.
11 se and membranous nephropathy, but not focal segmental glomerulosclerosis.
12 essed to renal failure associated with focal segmental glomerulosclerosis.
13 erlying disease mechanism in inherited focal segmental glomerulosclerosis.
14 for hypertension-attributable ESRD and focal segmental glomerulosclerosis.
15 s in INF2 cause the kidney disease focal and segmental glomerulosclerosis.
16 in sclerosing glomerulopathies such as focal segmental glomerulosclerosis.
17 l cells in diabetic nephropathy and in focal segmental glomerulosclerosis.
18 ain showed histology characteristic of focal segmental glomerulosclerosis.
19 ting in progressive kidney failure and focal segmental glomerulosclerosis.
20 e numbers decrease in association with focal segmental glomerulosclerosis.
21 embly assay identified alpha-actinin-4/focal segmental glomerulosclerosis 1 (FSGS1) as an essential f
22 s-linking approach, we have identified focal segmental glomerulosclerosis 3/CD2-associated protein (F
23 was a surprisingly high prevalence of focal segmental glomerulosclerosis (50%) and global glomerulos
24 es of fetal development results in focal and segmental glomerulosclerosis, a reduced number of nephro
25 ls of both obstructive nephropathy and focal segmental glomerulosclerosis (adriamycin nephropathy), w
27 iated nephropathy with collapsing-type focal segmental glomerulosclerosis and characteristic tubulocy
28 is mutated in two human diseases, focal and segmental glomerulosclerosis and Charcot-Marie-Tooth dis
29 mal at birth but developed progressive focal segmental glomerulosclerosis and died in terminal renal
31 with congenital (onset 0 to 3 months) focal segmental glomerulosclerosis and five patients with poss
32 sed histologically for evidence of focal and segmental glomerulosclerosis and hyalinosis (FSGS), and
33 annel TRPC6 lead to autosomal dominant focal segmental glomerulosclerosis and podocyte expression of
34 ptor (suPAR) have been associated with focal segmental glomerulosclerosis and poor clinical outcomes
35 abnormalities, including albuminuria, focal segmental glomerulosclerosis and progressive kidney dise
36 n, we briefly review new insights into focal segmental glomerulosclerosis and the role of podocytes i
37 syndrome in evaluation of females with focal segmental glomerulosclerosis and to consider their risk
38 had significantly lower baseline eGFR, more segmental glomerulosclerosis and total glomerulosclerosi
39 iltration, glomerular hypertrophy, focal and segmental glomerulosclerosis, and intimal vascular hyper
40 he spectrum of minimal change disease, focal segmental glomerulosclerosis, and related disorders.
41 ephrosis and why some patients develop focal segmental glomerulosclerosis are less striking, but rece
42 es with normal genital development and focal segmental glomerulosclerosis associated with a WT1 intro
43 arly pathomechanism in mice developing focal-segmental glomerulosclerosis associated with functional
44 and active caspase-3 increased in focal and segmental glomerulosclerosis biopsies, and both proteins
45 ciated with kidney diseases, including focal segmental glomerulosclerosis, characterized by proteinur
46 y has been associated with a secondary focal segmental glomerulosclerosis coined obesity-related glom
48 es near the K255E mutation that causes focal segmental glomerulosclerosis) demonstrated increased act
51 ystic kidney disease (ADPKD), familial focal segmental glomerulosclerosis (FSG), hypomagnesemia with
52 report describes five patients who had focal segmental glomerulosclerosis (FSGS) (four with recurrent
53 inistration to patients with recurrent focal segmental glomerulosclerosis (FSGS) after transplantatio
54 e, we show that, in African Americans, focal segmental glomerulosclerosis (FSGS) and hypertension-att
55 thy, minimal change disease (MCD), focal and segmental glomerulosclerosis (FSGS) and membranous nephr
57 ons in INF2 lead to the kidney disease focal segmental glomerulosclerosis (FSGS) and the neurological
58 fold higher odds (95% CI 11 to 26) for focal segmental glomerulosclerosis (FSGS) and twenty-nine-fold
62 duals with the proteinuric disease focal and segmental glomerulosclerosis (FSGS) compared to control
63 e histopathologic diagnosis of primary focal segmental glomerulosclerosis (FSGS) has come to include
65 nephropathy (IgM) in 14 patients, and focal segmental glomerulosclerosis (FSGS) in five patients.
66 amilial and sporadic steroid-resistant focal segmental glomerulosclerosis (FSGS) in the pediatric pop
82 who displayed elevated TNF levels, and focal segmental glomerulosclerosis (FSGS) patients, whose TNF
85 ntial markers of steroid-resistance in focal segmental glomerulosclerosis (FSGS) we evaluated intra-g
86 ells from 15 (41%) of 36 patients with focal segmental glomerulosclerosis (FSGS), 2 (10%) of 20 patie
87 forms of glomerulonephritis seen were focal segmental glomerulosclerosis (FSGS), 57; immunoglobulin
88 alpha-actinin 4 (ACTN4), are linked to focal segmental glomerulosclerosis (FSGS), a chronic kidney di
89 actinin-4 have been linked to familial focal segmental glomerulosclerosis (FSGS), a common renal diso
90 ACTN4) are linked to familial forms of focal segmental glomerulosclerosis (FSGS), a kidney disease ch
92 L), seen in some patients with primary focal segmental glomerulosclerosis (FSGS), comprises prolifera
93 r a dominantly inherited form of human focal segmental glomerulosclerosis (FSGS), evidence supporting
94 tes with increased risk for idiopathic focal segmental glomerulosclerosis (FSGS), HIV-1-associated ne
95 with the nondiabetic kidney diseases, focal segmental glomerulosclerosis (FSGS), HIV-associated neph
96 s a susceptibility gene for idiopathic focal segmental glomerulosclerosis (FSGS), HIV-associated neph
98 iopathic nephrotic syndrome, including focal segmental glomerulosclerosis (FSGS), minimal change dise
99 et of adults and children with primary focal segmental glomerulosclerosis (FSGS), proteinuria and ren
102 ing to idiopathic and HIV-1-associated focal segmental glomerulosclerosis (FSGS), we carried out an a
122 s in all examined glomerular diseases (focal segmental glomerulosclerosis [FSGS], minimal-change dise
124 To determine whether patients with focal segmental glomerulosclerosis have a circulating factor c
125 tor) and kidney diseases including focal and segmental glomerulosclerosis, HIV-associated nephropathy
126 ividuals without such variants to have focal segmental glomerulosclerosis, HIV-associated nephropathy
127 erent) and lower for all other groups: focal segmental glomerulosclerosis (HR, 0.80; 95% CI, 0.77-0.8
128 hy (HRa, 0.47; 95% CI, 0.29-0.75), and focal segmental glomerulosclerosis (HRa, 0.69; 95% CI, 0.45-1.
129 osclerosis in idiopathic and secondary focal segmental glomerulosclerosis, hypertensive nephroscleros
131 five families with autosomal dominant focal segmental glomerulosclerosis in which disease segregated
134 ing factor found in some patients with focal segmental glomerulosclerosis is associated with recurren
136 sive nephrotic syndrome, in particular focal segmental glomerulosclerosis, its pathogenesis and alter
137 to induce spontaneous proteinuria and focal segmental glomerulosclerosis-like glomerular damage in m
139 lthy control samples and patients with focal-segmental glomerulosclerosis, membranous glomerulonephri
140 nsion, capsular adhesions (synechiae), focal segmental glomerulosclerosis, mild persistent proteinuri
141 ed from patients with IgA nephropathy, focal segmental glomerulosclerosis, minimal change disease, id
142 ve nephrosclerosis (n = 7), idiopathic focal segmental glomerulosclerosis (n = 11), focal sclerosis s
143 diffuse proliferative lupus nephritis, focal segmental glomerulosclerosis (not collapsing glomerulopa
144 ogic entity that includes proteinuria, focal segmental glomerulosclerosis often of the collapsing var
145 odocytes in glomeruli from humans with focal segmental glomerulosclerosis or diabetic nephropathy exh
146 ent progress in the physiopathology of focal segmental glomerulosclerosis recurrence after transplant
148 ent membrane glomerulonephritis, and a focal segmental glomerulosclerosis that is characteristic of s
149 associated with morphologic changes of focal segmental glomerulosclerosis together with interstitial
150 ortional hazards regression, the presence of segmental glomerulosclerosis was the only factor that si
151 eases such as diabetic nephropathy and focal segmental glomerulosclerosis, we observed upregulation o
152 y idiopathic membranous nephropathy or focal segmental glomerulosclerosis were confirmed by gene sequ
156 ad transplant arteriosclerosis and focal and segmental glomerulosclerosis, while animals treated with
157 nd a renal biopsy specimen that showed focal segmental glomerulosclerosis with abnormal podocytes con
158 ), is an aggressive form of collapsing focal segmental glomerulosclerosis with accompanying tubular a
159 Histologically, HIVAN is a collapsing focal segmental glomerulosclerosis with prominent tubular dama
160 is review discusses the development of focal segmental glomerulosclerosis, with particular attention
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