ライフサイエンスコーパス: selenium deficiency

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1 sts that patients with cirrhosis do not have selenium deficiency.

2 o severe neurodegeneration and death in mild selenium deficiency.

3 hepatocyte selenium to peripheral tissues in selenium deficiency.

4 worsening clinical manifestations of dietary selenium deficiency.

5 repressor of selenoprotein synthesis during selenium deficiency.

6 in, zinc, vitamin A, kwashiorkor, biotin and selenium deficiencies, along with the clinical entities

7 Although selenium deficiency also affects the activity of several

8 Selenium deficiency, an independent predictor of mortali

9 man plasma has shown that it is depressed by selenium deficiency and by cirrhosis.

10 Both selenium deficiency and toxicity are serious problems wo

11 wn-regulated by MsrB1 small interfering RNA, selenium deficiency, and selenocysteine tRNA mutations;

12 This effect was not explained by uniform selenium deficiency because methionine sulfoxide reducta

13 oped severe neurodegeneration caused by mild selenium deficiency, brain selenium was approximately 35

14 It responds to selenium deficiency by curtailing excretion and secretin

15 In addition, selenium deficiency down-regulated the mRNA of the most

16 During selenium deficiency, eIF4a3 binds SECIS elements from no

17 Selenium deficiency has not been reported to occur in he

18 In extreme selenium deficiency, however, brain selenium of approxim

19 In studies of coxsackievirus infection and selenium deficiency in mice, we found that mice fed a se

20 that severe cirrhosis causes mild functional selenium deficiency in some patients that is associated

21 trate that deletion of Sepp1, and presumably selenium deficiency in the brain, produce both neuronal

22 cquired immunodeficiency syndrome (AIDS) and selenium deficiency, indicating a protective role for th

23 Selenium deficiency lowered liver thioredoxin reductase

24 Selenium deficiency (plasma selenium < 0.070 microg/mL)

25 The data provide evidence that dietary selenium deficiency shortens, while supplementation of t

26 al dysfunction and death under conditions of selenium deficiency, suggesting an interaction of apoER2

27 ther cirrhosis is associated with functional selenium deficiency (the lack of selenium for the proces

28 During selenium deficiency, there is a hierarchy of selenoprote

29 luding liver can be substantially reduced by selenium deficiency without any apparent change in pheno

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