ライフサイエンスコーパス: self-stimulation

1 es into brain areas implicated in electrical self-stimulation.

2 ons in anhedonia as measured by intracranial self-stimulation.

3 of other opioids measured with intracranial self-stimulation.

4 ohol reward were examined using intracranial self-stimulation.

5 effect on locomotor activity or intracranial self-stimulation.

6 lso capable of communicating, but must avoid self-stimulation.

7 e internal capsule (IC), which also supports self-stimulation.

8 ossible anatomical substrates supporting MFB self-stimulation.

9 ndle electrodes supported intense electrical self-stimulation.

10 reward-seeking behaviour measured by optical self-stimulation.

11 ariable effects on the rewarding efficacy of self-stimulation.

12 avior normalized quickly in the intracranial self-stimulation and 5-choice serial reaction time task

13 Using intracranial self-stimulation and fast scan cyclic voltammetry, we fo

14 or NMDA receptor antagonists blocked optical self-stimulation and place preference.

15 nd developmentally equivalent cells to avoid self-stimulation and to coordinate their behavior to ach

16 l systems subserving positive reinforcement (self-stimulation) and incentive motivation (relapse).

17 n, shifts sucrose preference, drives optical self-stimulation, and directs sensory discrimination lea

18 generate positive reinforcement, as shown by self-stimulation, and negative reinforcement shown by st

19 hold-lowering effect on lateral hypothalamic self-stimulation, and whether any such effect can be att

20 elements in the anatomical substrate for MFB self-stimulation are discussed.

21 iable excitatory synaptic responses, optical self-stimulation behaviour was not observed by activatio

22 motivation and reinforcement of intracranial self-stimulation but have not assigned these effects to

23 ts from the lateral hypothalamus (LH), where self-stimulation can be induced.

24 own that lateral hypothalamic stimulation or self-stimulation can release dopamine in the nucleus acc

25 ng that experiential differences in auditory self-stimulation cannot explain the perceptual change.

26 es project, is activated by vaginal-cervical self-stimulation (CSS) in such women, as visualized by f

27 y neurons proved to be capable of supporting self-stimulation, demonstrating that behavioral reinforc

28 An intracranial self-stimulation discrete trial procedure that provides

29 ion among other controls, we show that NKG2D self-stimulation has tumor-promoting capacity.

30 Intracranial self-stimulation (ICS) is a motivated behavior that resu

31 hat supports this hypothesis is intracranial self-stimulation (ICS), during which animals repeatedly

32 Intracranial self-stimulation (ICSS) activates the neural pathways th

33 ts brain reward processes using intracranial self-stimulation (ICSS) and inducible bitransgenic mice

34 aminergic neurons contribute to intracranial self-stimulation (ICSS) and other reward-seeking behavio

35 the CEA and examined effects on intracranial self-stimulation (ICSS) as an index of hedonic state, an

36 atergic neurons produced robust intracranial self-stimulation (ICSS) behavior, which was dose-depende

37 Intracranial self-stimulation (ICSS) can be utilized in rodents (rats

38 ate midazolam preference and an intracranial self-stimulation (ICSS) paradigm to evaluate the impact

39 was trained on a discrete trial intracranial self-stimulation (ICSS) procedure interpreted to assess

40 The intracranial self-stimulation (ICSS) procedure was used to investigat

41 or activation and antagonism on intracranial self-stimulation (ICSS) reward using a discrete-trial cu

42 ty discounting paradigm wherein intracranial self-stimulation (ICSS) serves as the positive reinforce

43 The intracranial self-stimulation (ICSS) test is sensitive to the functio

44 ts of benzodiazepines using the intracranial self-stimulation (ICSS) test, a procedure with which the

45 pendent) sessions and had daily intracranial self-stimulation (ICSS) thresholds assessed.

46 rebrain bundle, as reflected by intracranial self-stimulation (ICSS) thresholds in rats.

47 the forced swim test (FST), and intracranial self-stimulation (ICSS) thresholds.

48 conditioned stimuli would lower intracranial self-stimulation (ICSS) thresholds.

49 Intracranial self-stimulation (ICSS) was used to compare the effects

50 Intracranial self-stimulation (ICSS) was used to quantify CSDS-induce

51 bundle (for behavior studies of intracranial self-stimulation (ICSS)) or with cannulae for microdialy

52 eward-seeking behaviors such as intracranial self-stimulation (ICSS), although its precise role remai

53 ate with reward learning during intracranial self-stimulation (ICSS).

54 mined the effects on mood using intracranial self-stimulation (ICSS).

55 thalamus and trained to perform intracranial self-stimulation (ICSS).

56 sufficient to support vigorous intracranial self-stimulation (ICSS).

57 n rewarded behaviors, including intracranial self-stimulation (ICSS).

58 Intracranial self-stimulation in rats showed that elongating the N-al

59 In addition, we performed intracranial self-stimulation in rats to understand how the chemical

60 activation of these terminals did not induce self-stimulation in the absence of an external reward.

61 ation by itself failed to support behavioral self-stimulation in the absence of any paired external f

62 anges in threshold pulse frequency (pps) for self-stimulation in the ventral tegmental area (VTA).

63 ffect on thresholds for lateral hypothalamic self-stimulation (LHSS) and did not alter the cocaine do

64 r, the effect of ADX on lateral hypothalamic self-stimulation (LHSS) and its facilitation by cocaine

65 of D-amphetamine in the lateral hypothalamic self-stimulation (LHSS) paradigm.

66 erone acetate) decrease lateral hypothalamic self-stimulation (LHSS) reward if rats are denied access

67 curve-shift analysis of lateral hypothalamic self-stimulation (LHSS) to evaluate whether the elevated

68 ion of cocaine reinforcing effects, and MPFC self-stimulation (MPFCSS) is mediated by a neural substr

69 Optogenetic self-stimulation of acid-sensing TRCs in thirsty animals

70 roposed hypothesis regarding the role in MFB self-stimulation of ascending cholinergic input from the

71 Mice displayed robust intracranial self-stimulation of LH to VTA fibers, an operant behavio

72 o complement the presence of its ligands for self-stimulation of parameters of tumorigenesis.

73 flies could implement behavior that induces self-stimulation of specific neurons in their brains.

74 nged access to a running wheel on electrical self-stimulation of the lateral hypothalamus (LHSS), a m

75 measured using taste reactivity, and optical self-stimulation of the rostral and caudal shells was al

76 o complement the presence of its ligands for self-stimulation of tumor growth and presumably malignan

77 We used the intracranial self-stimulation paradigm to assess the effects of cocai

78 ard function in rats (using the intracranial self-stimulation procedure) and protein levels of brain-

79 threshold) required to maintain intracranial self-stimulation responding in male and female rats, a d

80 ee of negative affect in our model predicted self-stimulation responding.

81 frequency required to maintain half-maximal self-stimulation response rates whereas injecting compar

82 reference scores were highly correlated with self-stimulation responses.

83 on of the dMHb in vivo supports intracranial self-stimulation, showing that dMHb activity is intrinsi

84 tal area reward neurons, the distribution of self-stimulation sites in this structure was mapped in 2

85 opulation of CB1 receptors modulates optical self-stimulation sustained by activation of PFC afferent

86 neurochemistries along the trajectory of the self-stimulation system has stronger effects on appetiti

87 NAc in the rat brain during an intracranial self-stimulation task in which a cue predicted lever ava

88 s motivation as measured in the intracranial self-stimulation test.

89 red before both the 8 and 12 hr post-cocaine self-stimulation tests, reversed the threshold elevation

90 reases in cocaine self-administration and LH self-stimulation that are reversed by dynorphin antagoni

91 ding termination and satiety, locomotion and self-stimulation, the modulation of anxiety-like behavio

92 In general, self-stimulation thresholds obtained from lateral sites

93 ated CREB produced increases in intracranial self-stimulation thresholds, a depressive-like sign refl

94 of nicotine-induced lowering of intracranial self-stimulation thresholds.

95 ted plus maze test and elevated intracranial self-stimulation thresholds; both of these effects were

96 "rate-frequency" procedure for intracranial self-stimulation to determine the frequency at which sti

97 properties of nicotine, we used intracranial self-stimulation to measure alterations in the threshold

98 lt Sprague-Dawley rats, we used intracranial self-stimulation to measure effects of the KOR agonist (

99 proximated the human SRT, using intracranial self-stimulation to promote rapid continuous responding

100 The animals would perform both self-stimulation to turn the current on and stimulation-

101 ctly investigates the role of the RRF in MFB self-stimulation using transient lidocaine-induced inact

102 ABA neurons, the degree of responding for IC self-stimulation was proportional to the magnitude of el

103 The threshold for responding for IC self-stimulation was the threshold for electrical coupli

104 red in response to a cue during intracranial self-stimulation were also attenuated by intra-VTA micro

105 lamic areas; of these, roughly 2/3 supported self-stimulation which was widely observed throughout th

106 ar, by measuring thresholds for intracranial self-stimulation with and without concurrent cocaine adm

107 d GJ blockers increased the threshold for IC self-stimulation without affecting performance.