ライフサイエンスコーパス: semantic dementia

1 ith those areas known to be most affected in semantic dementia.

2 ormed markedly better than the patients with semantic dementia.

3 face and name knowledge in 15 patients with semantic dementia.

4 vanced interpretations of the impairments in semantic dementia.

5 pposed to the 'storage' deficits observed in semantic dementia.

6 ts of picture-naming data from patients with semantic dementia.

7 that is sensitive to semantic impairment in semantic dementia.

8 of a more severe generic semantic deficit in semantic dementia.

9 visual deficits as compared to patients with semantic dementia.

10 its, but sparing of phonology and fluency in semantic dementia.

11 the widely accepted diagnostic criteria for semantic dementia.

12 ad criteria, excluding only those with clear semantic dementia.

13 tices and nucleus accumbens in patients with semantic dementia.

14 ), progressive non-fluent aphasia (PNFA) and semantic dementia.

15 t mortem cases meeting clinical criteria for semantic dementia.

16 al levels: progressive nonfluent aphasia and semantic dementia.

17 h progressive non-fluent aphasia; eight with semantic dementia].

18 on this test of a group of 12 patients with semantic dementia (10 male, mean age: 64.4 years) correl

19 2% bvFTD/motor neuron disease cases), 2 with semantic dementia (5.9% of patients with semantic dement

20 t frontotemporal dementia (71% of cases) and semantic dementia (65% of cases) and in association with

21 ic cases) and heightened responsiveness with semantic dementia (73% of symptomatic cases) and Alzheim

22 er of central conceptual knowledge arises in semantic dementia, a degenerative condition associated w

23 The study of patients with semantic dementia, a variant of frontotemporal lobar deg

24 sotropy--from 10 patients with mild/moderate semantic dementia and 21 matched controls.

25 Semantic dementia and Alzheimer's disease groups did not

26 expert musicians with clinical diagnoses of semantic dementia and Alzheimer's disease, in comparison

27 the distribution of temporal lobe atrophy in semantic dementia and Alzheimer's disease.

28 tify the global and temporal lobe atrophy in semantic dementia and Alzheimer's disease.

29 l eating behavior in patients with bvFTD and semantic dementia and are likely responsible for the dif

30 heimer's disease, the focal lobar atrophies (semantic dementia and dementia of frontal type) and thre

31 t the performance of three patients-two with semantic dementia and focal temporal lobe atrophy and th

32 teria for behavioural variant FTD (bvFTD) or semantic dementia and had characteristic brain atrophy.

33 asting doubt over the conclusions drawn from semantic dementia and linked basic neuroscience studies.

34 h clinical diagnoses of Alzheimer's disease, semantic dementia and non-fluent primary progressive aph

35 osia is one of the clinical presentations of semantic dementia and not a separate clinical entity.

36 oral lobe epilepsy and the expectations from semantic dementia and other neuroscience findings.

37 h those observed in a group of patients with semantic dementia and predominant left-sided temporal lo

38 ic prominence, particularly for diagnosis in semantic dementia and prognosis in behavioural syndromes

39 ible for deteriorating semantic knowledge in semantic dementia and separate from 'classic' language a

40 All patients had a semantic dementia and the clinical details of two of the

41 psychological studies of surface dyslexia in semantic dementia and the connectionist triangle model o

42 s an examination of the relationship between semantic dementia and the focal clinical syndrome of pro

43 totemporal dementia with motoneuron disease, semantic dementia and, with one exception, progressive n

44 tients (five with svPPA and two with 'right' semantic dementia) and 12 healthy controls underwent pos

45 ith semantic dementia (5.9% of patients with semantic dementia), and none with progressive nonfluent

46 tients with dementia (19 with bvFTD, 15 with semantic dementia, and 15 with Alzheimer disease) were r

47 f primary progressive aphasia, also known as semantic dementia, and Alzheimer's disease have deficits

48 d in both progressive non-fluent aphasia and semantic dementia, and deficits of semantic processing a

49 clear palsy set), anterior temporal lobes in semantic dementia, and hippocampus and posterior cingula

50 ding of the anatomical changes that occur in semantic dementia, and may further help to explain the d

51 These cases most commonly presented as semantic dementia, and they had longer disease duration

52 h the volume of right temporal structures in semantic dementia, and with subcallosal gyrus volume in

53 rment of semantic knowledge in patients with semantic dementia appears to influence performance in a

54 ant primary progressive aphasia (also called semantic dementia) are two clinical variants of frontote

55 s suggested by observations on patients with semantic dementia, as well as posterior regions describe

56 a striking literary depiction of collective semantic dementia before the syndrome was recognized in

57 hat differed between patients with bvFTD and semantic dementia but included the cingulate cortices, t

58 In Alzheimer's disease and semantic dementia, by contrast, group level and individu

59 Insofar as the diagnosis of semantic dementia can have these two different meanings,

60 ecent findings indicate that the syndrome of semantic dementia can inform us about the organisation o

61 a visual decision task in four patients with semantic dementia compared with six age-matched normal c

62 longitudinal bundle, where abnormalities in semantic dementia did not extend caudal to the atrophic/

63 [(18)F]AV-1451, the pathological regions in semantic dementia do not normally contain significant le

64 [(18)F]AV-1451 binding potential, separated semantic dementia from controls with 86% sensitivity and

65 It has been reported that patients with semantic dementia function well in everyday life and som

66 ilateral anterior temporal lobe damage (e.g. semantic dementia), functional neuroimaging and repetiti

67 ative threshold, tensor abnormalities in the semantic dementia group mapped onto the tractographies f

68 temporal lobe atrophy patient group than the semantic dementia group.

69 Patients with bvFTD and semantic dementia had a strong sucrose preference compar

70 The patients with semantic dementia had the expected deficit in object ide

71 While research on semantic dementia has provided evidence for a critical r

72 Cognitive deficits in semantic dementia have been attributed to anterior tempo

73 For example, patients with semantic dementia have prominent atrophy in anterolatera

74 performed on a wider cohort of patients with semantic dementia, in which the patients with more exten

75 Semantic dementia, in which there is progressive deterio

76 INTRODUCTION: Semantic dementia, including the semantic variant of pri

77 d the hypothesis that concept degradation in semantic dementia involves a combination of these pan-mo

78 Semantic dementia is a focal clinical syndrome, resultin

79 The core clinical feature of semantic dementia is a progressive yet selective degrada

80 Semantic dementia is a syndrome of progressive deteriora

81 This study shows that semantic dementia is associated with anatomical damage t

82 Although it is well-known that semantic dementia is associated with temporal lobe degen

83 on between progressive nonfluent aphasia and semantic dementia is strengthening.

84 In patients with semantic dementia, it appears that both the left tempora

85 the Alzheimer disease (mean, 710 calories), semantic dementia (mean, 573 calories), and control grou

86 f progressive nonfluent aphasia (n = 23) and semantic dementia (n = 15).

87 ral variant FTD (n = 26), language variants (semantic dementia, n = 9; and progressive nonfluent apha

88 havioral variant frontotemporal dementia and semantic dementia, often respond to treatment with selec

89 dial temporal lobe lesions and patients with semantic dementia on nine tests of semantic knowledge an

90 al dementia, progressive non-fluent aphasia, semantic dementia or mixture of these syndromes for muta

91 longitudinal fasciculus were tracked in five semantic dementia patients and eight healthy controls.

92 Semantic dementia patients had significantly higher mean

93 ', strategies that resonate with attempts by semantic dementia patients to cope with their disease.

94 The semantic dementia patients were profoundly impaired on b

95 erall profile masked individual differences: semantic dementia patients with predominant left tempora

96 Three groups of 10 subjects were studied: semantic dementia patients, Alzheimer's disease patients

97 the tests of new learning, the patients with semantic dementia performed markedly better than the amn

98 r each FTD subtype (behavioural variant FTD, semantic dementia, progressive non-fluent aphasia, and F

99 drome that includes frontotemporal dementia, semantic dementia, progressive nonfluent aphasia and pro

100 Semantic dementia refers to the variant of frontotempora

101 essing in progressive non-fluent aphasia and semantic dementia, respectively.

102 Parallel studies of semantic dementia, rTMS in normal participants, and neur

103 wo groups suffering from different diseases: semantic dementia (SD) and herpes simplex virus encephal

104 sy exists regarding the relationship between semantic dementia (SD) and progressive aphasia.

105 with frontotemporal dementia (FTD), 19 with semantic dementia (SD) and six with progressive non-flue

106 A), progressive nonfluent aphasia (PNFA), or semantic dementia (SD) based on language testing.

107 Semantic dementia (SD) is a neurodegenerative disease ch

108 diverse cortical regions in semantic memory: semantic dementia (SD) is characterized by atrophy of th

109 ed patients with Alzheimer's disease (AD) or semantic dementia (SD) on a visual oddity judgment task

110 ral atrophy of the anterior temporal lobe in semantic dementia (SD) produces a gradual degradation of

111 herpes simplex virus encephalitis (HSVE) and semantic dementia (SD) typically affect anterior tempora

112 Semantic dementia (SD), one of the main clinical variant

113 Solitude to that exhibited by patients with semantic dementia (SD).

114 uent aphasia (PNFA) (or a mixed aphasia) and semantic dementia (SD); and to compare the age of onset,

115 onfluent progressive aphasia (NFPA; n = 11), semantic dementia (SD; n = 10), and a third variant term

116 18 progressive nonfluent aphasia [PNFA], 16 semantic dementia [SD]), 22 progressive supranuclear pal

117 The patient with semantic dementia showed relatively preserved recognitio

118 FA), progressive logopenic aphasia (PLA) and semantic dementia (SMD).

119 In addition, the pattern of atrophy in semantic dementia suggests that semantic memory is subse

120 rment found across different patient groups (semantic dementia, temporal lobe epilepsy, glioma and st

121 The cortical anatomy of 6 patients with semantic dementia (the temporal lobe variant of frontote

122 convincingly demonstrated that patients with semantic dementia (the temporal variant of frontotempora

123 Our results line up with evidence from semantic dementia, the convergence zone framework and me

124 The study of patients with semantic dementia, the temporal variant of frontotempora

125 In semantic dementia, there is profound progressive and rel

126 In semantic dementia, there was asymmetrical temporal lobe

127 est to evaluate the ability of patients with semantic dementia to use recollection-based memory proce

128 e left language-related cerebral pathways in semantic dementia using diffusion tensor imaging-based t

129 Semantic dementia was predominantly associated with TDP-

130 The term "semantic dementia" was originally introduced to designat

131 To explore this phenomenon in nine cases of semantic dementia, we designed a set of semantic tests r

132 Behavioral symptoms and semantic dementia were associated with a range of pathol

133 uced fractional anisotropy for patients with semantic dementia were spatially concordant with each ot

134 frontotemporal dementia alone, one had mixed semantic dementia with frontal features and three had pr

135 or temporal lobe structures were affected in semantic dementia, with the entorhinal cortex, amygdala,

136 ntext of their implications for the place of semantic dementia within the classification of progressi