ライフサイエンスコーパス: semilunar

1 ion fraction and EF Z score were lowest, and semilunar and atrioventricular (AV) valve regurgitation

2 ed NCC persistence and contributions to both semilunar and atrioventricular (AV) valves in the mature

3 EGF(-/-) newborns had enlarged and malformed semilunar and atrioventricular heart valves, and hypopla

4 truncal valve stenosis and regurgitation; to semilunar and atrioventricular valve regurgitation, and

5 ology of widefield amacrine cells (stellate, semilunar, and thorny amacrine cells).

6 w tract regions and in the atrioventricular, semilunar, and venous valves.

7 hp2, we discovered that Egfr is required for semilunar, but not atrioventricular, valve development.

8 with Fog2(-/-) embryos as well as additional semilunar cardiac valve defects and a double-outlet righ

9 of the meibomian glands, and defects in the semilunar cardiac valves.

10 Furthermore, semilunar cell dendrites were reduced by unilateral depr

11 or Golgi staining for reconstruction of aPCX semilunar cell dendrites.

12 iven by feedforward excitation from layer II semilunar cells.

13 A modified semilunar coronally advanced flap is described for the t

14 positioned pedicle than previously described semilunar coronally advanced flap procedures.

15 flap repositioning than previously described semilunar coronally advanced flaps.

16 shows the possibility of applying a combined semilunar coronally repositioned flap with a frenectomy

17 (97%), fornix in 9 (28%), tarsus in 3 (9%), semilunar fold in 10 (31%), and caruncle in 7 (22%).

18 uronal class in the dentate molecular layer, semilunar granule cell (SGC), has been proposed to regul

19 found that synaptic barrages originate from semilunar granule cells (SGCs), glutamatergic neurons in

20 iny, granule-like neurons in the IML, termed semilunar granule cells (SGCs).

21 Semilunar granule cells appear to represent a distinct e

22 Autologous semilunar heart valves were then created in vitro using

23 treat the areas of recession involved making semilunar incisions over the maxillary central incisors

24 Semilunar incisions were made apical to the recession de

25 lar, tonsil, biventer, declive, and inferior semilunar lobules.

26 cortex (APC) can be divided into 2 subtypes: semilunar (SL) and superficial pyramidal (SP) cells.

27 Atrioventricular and semilunar valve abnormalities are common birth defects,

28 ries from veins, and direct formation of the semilunar valve and atrioventricular valves.

29 icular septal defects and a low incidence of semilunar valve defects, atrioventricular valve defects

30 The elucidation of mechanisms in semilunar valve development might enable the development

31 rta, double outlet right ventricle, aberrant semilunar valve development, bicuspid aortic valve, vent

32 ic Egfr allele waved-2 (Egfrwa2/wa2) exhibit semilunar valve enlargement resulting from over-abundant

33 f persistent truncus arteriosus and impaired semilunar valve formation in humans.

34 and increased OFT mesenchyme with failure of semilunar valve formation.

35 ptal defects, double outlet right ventricle, semilunar valve hyperplasia and aortic arch artery patte

36 es and genes repressed by VEGF between human semilunar valve leaflets from first and second trimester

37 In situ analysis of fetal semilunar valve leaflets has revealed cells coexpressing

38 al crest can result in functionally abnormal semilunar valves and concomitant aortic arch artery abno

39 stal truncus that was patterned into the two semilunar valves and in the proximal conotruncus where m

40 e is active throughout the developing AV and semilunar valves at E16.5, with more localized expressio

41 In addition, semilunar valves do not form in mutants, while the atrio

42 Results with semilunar valves have generally been better than the atr

43 al numbers of labeled cells persisted in the semilunar valves in late fetal, neonatal, and adult hear

44 titative histological assessment of 91 human semilunar valves obtained from fetuses at 14 to 19 and 2

45 endocardial cushions (the precursors of the semilunar valves) is required for late gestation valvula

46 homeostasis in HDAC3-null outflow tracts and semilunar valves, and pharmacological inhibition of TGF-

47 rdium of the developing atrioventricular and semilunar valves, the muscular interventricular septum,

48 in the remodeling atrioventricular (AV) and semilunar valves.

49 f Tgf-beta1 in HDAC3-null outflow tracts and semilunar valves.

50 re either atretic or connected distal to the semilunar valves.

51 signalling pathway required specifically for semilunar valvulogenesis, support the hypothesis that Sh