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1  in the remodeling atrioventricular (AV) and semilunar valves.
2 f Tgf-beta1 in HDAC3-null outflow tracts and semilunar valves.
3 re either atretic or connected distal to the semilunar valves.
4                         Atrioventricular and semilunar valve abnormalities are common birth defects,
5 ries from veins, and direct formation of the semilunar valve and atrioventricular valves.
6 al crest can result in functionally abnormal semilunar valves and concomitant aortic arch artery abno
7 stal truncus that was patterned into the two semilunar valves and in the proximal conotruncus where m
8 homeostasis in HDAC3-null outflow tracts and semilunar valves, and pharmacological inhibition of TGF-
9 e is active throughout the developing AV and semilunar valves at E16.5, with more localized expressio
10 icular septal defects and a low incidence of semilunar valve defects, atrioventricular valve defects
11             The elucidation of mechanisms in semilunar valve development might enable the development
12 rta, double outlet right ventricle, aberrant semilunar valve development, bicuspid aortic valve, vent
13                                 In addition, semilunar valves do not form in mutants, while the atrio
14 ic Egfr allele waved-2 (Egfrwa2/wa2) exhibit semilunar valve enlargement resulting from over-abundant
15 f persistent truncus arteriosus and impaired semilunar valve formation in humans.
16 and increased OFT mesenchyme with failure of semilunar valve formation.
17                                 Results with semilunar valves have generally been better than the atr
18 ptal defects, double outlet right ventricle, semilunar valve hyperplasia and aortic arch artery patte
19 al numbers of labeled cells persisted in the semilunar valves in late fetal, neonatal, and adult hear
20  endocardial cushions (the precursors of the semilunar valves) is required for late gestation valvula
21 es and genes repressed by VEGF between human semilunar valve leaflets from first and second trimester
22                    In situ analysis of fetal semilunar valve leaflets has revealed cells coexpressing
23 titative histological assessment of 91 human semilunar valves obtained from fetuses at 14 to 19 and 2
24 rdium of the developing atrioventricular and semilunar valves, the muscular interventricular septum,

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