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1 in the remodeling atrioventricular (AV) and semilunar valves.
2 f Tgf-beta1 in HDAC3-null outflow tracts and semilunar valves.
3 re either atretic or connected distal to the semilunar valves.
6 al crest can result in functionally abnormal semilunar valves and concomitant aortic arch artery abno
7 stal truncus that was patterned into the two semilunar valves and in the proximal conotruncus where m
8 homeostasis in HDAC3-null outflow tracts and semilunar valves, and pharmacological inhibition of TGF-
9 e is active throughout the developing AV and semilunar valves at E16.5, with more localized expressio
10 icular septal defects and a low incidence of semilunar valve defects, atrioventricular valve defects
12 rta, double outlet right ventricle, aberrant semilunar valve development, bicuspid aortic valve, vent
14 ic Egfr allele waved-2 (Egfrwa2/wa2) exhibit semilunar valve enlargement resulting from over-abundant
18 ptal defects, double outlet right ventricle, semilunar valve hyperplasia and aortic arch artery patte
19 al numbers of labeled cells persisted in the semilunar valves in late fetal, neonatal, and adult hear
20 endocardial cushions (the precursors of the semilunar valves) is required for late gestation valvula
21 es and genes repressed by VEGF between human semilunar valve leaflets from first and second trimester
23 titative histological assessment of 91 human semilunar valves obtained from fetuses at 14 to 19 and 2
24 rdium of the developing atrioventricular and semilunar valves, the muscular interventricular septum,
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