戻る
「早戻しボタン」を押すと検索画面に戻ります。

今後説明を表示しない

[OK]

コーパス検索結果 (1語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 ions of increased thalamic connectivity with sensory cortex.
2 hanism for the control of motor decisions by sensory cortex.
3  in supragranular pyramidal cells in primary sensory cortex.
4 motor cortex influence sensory processing in sensory cortex.
5 ivity across neuronal populations in primary sensory cortex.
6 N computations might be a generic feature of sensory cortex.
7 little is known about how stress affects the sensory cortex.
8 ulations such as those found in layer 2/3 of sensory cortex.
9 tations of sounds are formed at the level of sensory cortex.
10 ough a spatially global recruitment of early sensory cortex.
11 representation, even at the level of primary sensory cortex.
12 ation of specific sensory content in primary sensory cortex.
13 orking memory processes in PFC and posterior sensory cortex.
14  fields, again has more of the appearance of sensory cortex.
15 ypical and granular areas of association and sensory cortex.
16 ey to determining the response properties in sensory cortex.
17  characterized as a higher-order, multimodal sensory cortex.
18  SP and NO signaling pathways in rat somatic sensory cortex.
19  a basic organizational feature of mammalian sensory cortex.
20 nsidered an enteroceptive area within limbic sensory cortex.
21 including abnormal neural signaling in human sensory cortex.
22 e phase-locked between primary and secondary sensory cortex.
23 lly in the whisker barrel region of parietal sensory cortex.
24 e role for cross-sensory influences in early sensory cortex.
25 nt to generate attractor dynamics in primary sensory cortex.
26 creases in beta band power in prefrontal and sensory cortex.
27 that this information is shared early in the sensory cortex.
28 completely understood, especially outside of sensory cortex.
29  that allows for greater flexibility than in sensory cortex.
30 apping thalamic representations than primary sensory cortex.
31 omputational role of inhibitory cells in the sensory cortex.
32  interactions with the sensory thalamus, not sensory cortex.
33 is stored via synaptic plasticity in primary sensory cortex.
34 ntoparietal regions and travelled to FEF and sensory cortex.
35 ral, somatotopic organization of the sheep's sensory cortex.
36 s affect the responses of neurons throughout sensory cortex.
37 sed principally postnatally, and enriched in sensory cortex.
38 naptic plasticity via ECM removal within the sensory cortex.
39 odel originally formulated to describe early sensory cortex.
40 in the primary motor cortex (-25%; p<0.001), sensory cortex (-15%, p<0.001) and frontal cortex (-12%;
41 ive coding, high-level cortical areas inform sensory cortex about incoming sensory signals, a compari
42 r study is the first to show that postreward sensory cortex activity meets these two key criteria of
43    These results support the idea that early sensory cortex activity reflects perceptual experience,
44 ration and learning; they receive input from sensory cortex and excite deep layer neurons, which cont
45                         Although there is no sensory cortex and hence no conscious vision, some corti
46            Furthermore, replay events in the sensory cortex and hippocampus were coordinated to refle
47 o GAP-43 is present in primary and secondary sensory cortex and more so in secondary areas.
48 ed coordinates, broadcast throughout primary sensory cortex and provides strong modulation of signals
49 essing of attended acoustic stimuli in early sensory cortex and reduced processing of distracting inp
50 reveal distinct interactions between primary sensory cortex and rIFC in humans and suggest that synch
51 ular recording to reveal connections between sensory cortex and striatum.
52 n neural spiking activity in primary somatic sensory cortex and the frequency of whisker stimulation.
53 s (SEP) were recorded from the contralateral sensory cortex and the sensory nerve action potential (S
54 effects of dopaminergic neurotransmission in sensory cortex and their possible roles in perception, l
55 encoding is subsequently reinstated in early sensory cortex and what the role of the hippocampus is i
56  or overlapping representations in a primary sensory cortex and whether learning can modulate these r
57 trinsic properties of FS interneurons in the sensory cortex, and a deficit in the formation of excita
58 c transmission in the hippocampus and in the sensory cortex, and are found at somatodendritic as well
59  less activation of forebrain, inhibition of sensory cortex, and behavioral arrest.
60 rophy, cortical atrophy of primary motor and sensory cortex, and cortical reorganization of the senso
61 ebral cortex, in neuronal development in the sensory cortex, and in reward.
62 ory input can remodel representations in the sensory cortex, and this effect is heavily influenced by
63                        Neuronal responses of sensory cortex are highly variable, and this variability
64 Growing evidence indicates that responses in sensory cortex are modulated by factors beyond direct se
65            One hypothesis is that regions of sensory cortex are reactivated during retrieval of senso
66 nt evidence suggests that neurons in primary sensory cortex arrange into competitive groups, represen
67  stimulus-specific patterns of activation in sensory cortex as a result of expectation, but this meth
68  supported by postencoding interactions with sensory cortex associated with reward.
69 m inclusive of the medial geniculate; 3) the sensory cortex barrel field and cell bodies of the ventr
70 f plaques in the vibrissae-receptive primary sensory cortex (barrel cortex), in which the cortical co
71 have revealed traveling waves of activity in sensory cortex, both following sensory stimulation and d
72 cause linear changes in activity patterns in sensory cortex but cause dynamic, nonlinear changes in b
73 ctivity between thalamus and lateral primary sensory cortex but reduced connectivity between thalamus
74 ses, not only in their corresponding primary sensory cortex, but in other primary sensory cortices.
75 rmance depends on the activity of neurons in sensory cortex, but little is known about the brain's ca
76 te and chronic investigations of the sheep's sensory cortex by characterizing its exact position, its
77 ents in the SC similar to those generated in sensory cortex by specific thalamic afferents.
78  or developmental lesions, responsiveness of sensory cortex can be converted from the deprived modali
79 hared variability of neuronal populations in sensory cortex can be largely explained by two factors t
80 ve shown that the firing of neurons in early sensory cortex can be modulated by multisensory interact
81 ical or transcranial magnetic stimulation of sensory cortex can temporarily disrupt these phantom sen
82 d the groundwork for the notion of a modular sensory cortex, canonical cortical circuits and an under
83 oral choice means that even neurons in early sensory cortex carry information about an upcoming decis
84 the patterns of neural activity decoded from sensory cortex change as a function of load, as one woul
85 d past measurements, which show that primary sensory cortex codes the whisking envelope as a motor co
86 kness with lower values in primary motor and sensory cortex compared with association cortex.
87 ing in the leg area of the primary motor and sensory cortex compared with controls.
88 reased incidence of SDs in parietal/temporal sensory cortex compared with other regions.
89 al extracellular current in vibrissa primary sensory cortex contained oscillatory components at the s
90                                          The sensory cortex contains a wide array of neuronal types,
91 isrupted synaptic and circuit development in sensory cortex contributes to these deficits.
92 Y, n=9) in the frontal cortex, motor cortex, sensory cortex, corpus callosum, hippocampus, thalamus,
93 and spatial extent of activation of vibrissa sensory cortex critically depend on behavioral context a
94                  The neural circuitry within sensory cortex determines its functional properties, and
95 on became progressively worse, including the sensory cortex, dorsal striatum and thalamus.
96     These results illustrate that changes in sensory cortex during associative learning extend to the
97 level dependent functional MR imaging of rat sensory cortex during forepaw stimulation.
98 re selectivity within specific subregions of sensory cortex (e.g., orientation selectivity in primary
99                                For instance, sensory cortex encodes stimuli that fluctuate over few t
100 tion about stimulus occurrence is encoded in sensory cortex, evidence from neuronal recordings has no
101 ances states of high, persistent activity in sensory cortex evoked by behaviorally relevant stimuli.
102 reorganization has not yet been found in any sensory cortex except AI of the gerbil.
103                 We conclude that the primary sensory cortex exerts a significant influence on both sp
104   Different subtypes of GABAergic neurons in sensory cortex exhibit diverse morphology, histochemical
105 etwork in top-down modulation of activity in sensory cortex, expectation-related activity in several
106 th increased activations in the left primary sensory cortex face area due to median nerve stimulation
107 eg area during handgrip and the left primary sensory cortex face area during median nerve stimulation
108 e same set of animals, since most studies of sensory cortex focus on a single sensory modality.
109 e demonstrated the importance of the primary sensory cortex for the detection, discrimination, and aw
110 lators in the spinal cord, motor cortex, and sensory cortex from clinically and neuropathologically d
111 a different role for spontaneous activity in sensory cortex: gating of sensory inputs.
112 o explains why a transient thalamic input to sensory cortex gives rise to responses with amplitudes i
113 es in connectivity between motor centers and sensory cortex guide subsequent sensorimotor learning.
114                                          The sensory cortex has been interpreted as coding informatio
115  influences on neuronal responses in primary sensory cortex has been observed previously using severa
116                       Recently, the study of sensory cortex has focused on the context-dependent evol
117                           Neurons in primary sensory cortex have diverse response properties, whereas
118 ocorticography arrays implanted on motor and sensory cortex, high-frequency power (65-95 Hz) was extr
119 lling these attention-related modulations of sensory cortex, however, are poorly understood.
120 cement and suppression of neural activity in sensory cortex (i.e., top-down modulation).
121 ion biases stimulus representations in early sensory cortex, i.e., whether the integration of prior k
122 ssible explanation for these results is that sensory cortex implements attractor dynamics, although t
123 pyramidal neurones of layer II/III of somato-sensory cortex in acutely isolated slices obtained from
124 vides a foundation to understand the role of sensory cortex in combining sensory and cognitive variab
125 ol operates in part by biasing processing in sensory cortex in favor of expected target stimuli.
126 describe the neuroanatomical organization of sensory cortex in four rodents: laboratory Norway rats (
127 elium may be a functional feature of primary sensory cortex in general.
128  SDs are inducible preferentially in primary sensory cortex in mice and most likely in humans.
129 cubic millimeter regions of vibrissa primary sensory cortex in mouse.
130  image tissue oxygenation in the rat primary sensory cortex in response to sensory stimulation.
131 and/or disturbed organization of the primary sensory cortex in schizophrenia.
132 e sensory/motor divide by showing a role for sensory cortex in the selection of motor behavior.
133 s temporal rate is represented by neurons in sensory cortex, in this issue of Neuron, new evidence fr
134 asticity have been well characterized within sensory cortex, in which the ability of altered sensory
135 ous stimuli may result from neural biases in sensory cortex induced by recent perceptual history.
136 ntracortical recurrent circuits in mammalian sensory cortex influence dynamics of sensory representat
137                                   Neurons in sensory cortex integrate multiple influences to parse ob
138  expansion of trunk motor cortex and forepaw sensory cortex into the deafferented hindlimb cortex, as
139                                              Sensory cortex is able to encode a broad range of stimul
140 s on post-perceptual changes (integration in sensory cortex is adult-like, but higher-level decision
141 e maturation of the GABAergic circuit in the sensory cortex is altered during a critical developmenta
142 ctional organization and plasticity of adult sensory cortex is derived from animals housed in standar
143            We show, in mouse and human, that sensory cortex is more susceptible to SDs.
144 population responses and network activity in sensory cortex is not well understood.
145 ontribute to map plasticity and stability in sensory cortex is not well-understood.
146                                              Sensory cortex is ordered into columns, each tuned to a
147                               Although early sensory cortex is organized along dimensions encoded by
148                                  The primary sensory cortex is positioned at a confluence of bottom-u
149 l variability in the responses of neurons in sensory cortex is shared between pairs of neurons.
150 lore the possibility that a core function of sensory cortex is the generation of an internal simulati
151 e response properties that define columns in sensory cortex is thought to begin early in cortical mat
152                                        Early sensory cortex is typically investigated in response to
153 ual neurons and local population activity in sensory cortex is unclear.
154 ty so that changes could occur in regions of sensory cortex lacking activity.
155                                           In sensory cortex, layer 4 (the granular layer) is the targ
156 l hyperexcitation in supragranular layers in sensory cortex, likely through reduced inhibition.
157 by an integrated neural system involving the sensory cortex, limbic system, and striatum.
158 Such expectancy-driven modulation of primary sensory cortex may affect perceptions of external events
159 e, the nature of aura symptoms suggests that sensory cortex may be preferentially susceptible.
160 al learning, suggest that changes in primary sensory cortex may mediate learning.
161 ore likely stimulus modality and the primary sensory cortex may participate in the redistribution of
162                     Our results suggest that sensory cortex may play a previously unrecognized role i
163  neuronal representation as early as primary sensory cortex mediate the perceptual advantage conferre
164  in IDBs, it remains largely unclear whether sensory cortex modulates IDBs and what the underlying ne
165                            Spike trains from sensory cortex neurons can predict trial-to-trial variab
166           Action Potential (APs) patterns of sensory cortex neurons encode a variety of stimulus feat
167 ure selectivity is a fundamental property of sensory cortex neurons, yet the mechanisms underlying it
168                                          For sensory cortex, neurophysiological and psychophysical fi
169 of NR2A and NR2B (NR2A/B) protein in somatic sensory cortex of adult rats.
170 e analyzed multineuron word distributions in sensory cortex of anesthetized rats and cats, and found
171 iphery may shape the organization of primary sensory cortex of other modalities as well.
172 of neuronal activity in the vibrissa primary sensory cortex of rat, a region that receives intrinsic
173                          In the thalamus and sensory cortex of the anesthetized rats, where the lCBF
174 ation and spontaneous neural activity in the sensory cortex of the awake monkey.
175  circuit function have been uncovered in the sensory cortex of the mouse model of FXS (Fmr1 KO).
176 e of alterations in neuronal circuits in the sensory cortex of the mouse model of FXS (Fmr1 KO).
177 ic resonance imaging (fMRI) studies of early sensory cortex often measure stimulus-driven increases i
178  neuronal oscillations in stimulus-receiving sensory cortex) only accounts for the accuracy time cour
179 Due to the diversity of tuning properties in sensory cortex, only a fraction of neurons are engaged i
180 nocellular visual pathway beginning in early sensory cortex or even subcortically.
181 such disturbances are present in any primary sensory cortex other than the auditory.
182                 Models of learning-dependent sensory cortex plasticity require local activity and rei
183                      Neuronal variability in sensory cortex predicts perceptual decisions.
184 ndent functional connectivity within the MDT/sensory cortex/prefrontal cortex network.
185 ggered increases of activity in the auditory sensory cortex prior to the occurrence of an auditory ta
186                           Lesions of primary sensory cortex produce impairments in brain function as
187 bgranular layers (layers 5 and 6) of primary sensory cortex provide corticofugal output to thalamus a
188 elative impairment of potassium clearance in sensory cortex, providing a potential mechanism for the
189 at D1/D5-mediated dopaminergic modulation in sensory cortex regulates positive recurrent corticoeffer
190 n conscious awareness associated with sleep, sensory cortex remains highly active during the differen
191 onsistent with growing evidence that primary sensory cortex remains plastic into adulthood, and they
192                      Neuronal populations in sensory cortex represent time-changing sensory input thr
193               The fine-tuning of circuits in sensory cortex requires sensory experience during an ear
194 bottom-up signals arise from the frontal and sensory cortex, respectively, and different modes of att
195                         Psilocybin increased sensory cortex responses during emotional recollection,
196 ation of the body part somatotopy in primary sensory cortex (S1 complex, hereafter S1) [1, 2], and th
197 erior cingulate cortex (ACC) and the primary sensory cortex (S1) in rats with inflammatory pain.
198             However, we show here that, like sensory cortex (S1), mouse M1 also has the canonical cir
199  the mouse compare with those in neighboring sensory cortex (S1).
200                             Neurons in early sensory cortex show weak but systematic correlations wit
201 ects represent an efficient process by which sensory cortex simultaneously enhances relevant informat
202                                              Sensory cortex, such as the auditory cortex, is involved
203 um concentrations showed larger increases in sensory cortex, suggesting a mechanism of susceptibility
204 ression of multiple types of gamma rhythm in sensory cortex suggests a mechanistic substrate for comb
205  pharmacological inactivation of rat somatic sensory cortex suppresses peripheral information transmi
206 th glutamatergic inputs from the association sensory cortex (Te3) that drive BLA projection neurons,
207 nclear why there are so many more neurons in sensory cortex than in the sensory periphery.
208 known translaminar inhibitory circuit in the sensory cortex that acts to enhance the feature selectiv
209 sponse to an instruction to attend, areas of sensory cortex that code the attributes of the expected
210      Layer (L)2 is a major output of primary sensory cortex that exhibits very sparse spiking, but th
211 aring them to the functional organization of sensory cortex that is activated by naturalistic stimuli
212 ng accounts for the resting hyperactivity in sensory cortex that leads to hallucinations.
213 al circuit for pursuit to identify a part of sensory cortex that provides instructive signals for mot
214 vity distributed across regions of low-level sensory cortex that univariate methods cannot detect.
215 ow the gamma oscillations are constrained to sensory cortex, that they occur independently in auditor
216 put is necessary to maintain organisation in sensory cortex, thereby reopening the question what happ
217 sociated with circuit-specific plasticity in sensory cortex, this switch in LTS cell synaptic inhibit
218 ning reconfigures neural circuits in primary sensory cortex to "learn" associative attributes of a st
219          We performed a study of rat somatic sensory cortex to confirm and extend these observations,
220 ex parallels the retinotopic organization of sensory cortex to enable an efficient interface between
221                            It spreads beyond sensory cortex to frontoparietal association areas, whic
222 l circuits that transmit behavioral state to sensory cortex to produce this modulation are unknown.
223                   The response of neurons in sensory cortex to repeated stimulus presentations is hig
224 directly impacts the piriform, the olfactory sensory cortex, to mediate social learning.
225 specificity of endocannabinoid signalling in sensory cortex using whole-cell recordings from layer 2/
226 cal areas, secondary and cross-modal, of the sensory cortex (visual, auditory, and somatosensory), as
227  (visual or auditory) induces LTP within the sensory cortex (visual/auditory, respectively) and can b
228 , we examined neuronal activity in vibrissal sensory cortex, vS1, together with vibrissal motor corte
229 ated with neural codes particularly in early sensory cortex, we have so far no understanding of the n
230  We propose that this regime is relevant for sensory cortex when it extracts complex features from li
231  calcium-binding proteins are upregulated in sensory cortex when thalamocortical afferents arrive.
232 rocess (preparatory excitability increase in sensory cortex), whereas the effect on RT is explained b
233 ntion can act directly on neural activity in sensory cortex without involving attentional modulation

WebLSDに未収録の専門用語(用法)は "新規対訳" から投稿できます。
 
Page Top