ライフサイエンスコーパス: sensory deficit

1 Hereditary hearing loss is the most common sensory deficit.

2 Hearing impairment is the most common sensory deficit.

3 physiological examination showed no motor or sensory deficit.

4 ds readily replace HCs and recover from such sensory deficits.

5 s show great potential for alleviating major sensory deficits.

6 nsory areas offers a promising treatment for sensory deficits.

7 ase, or genetic mutation, leads to permanent sensory deficits.

8 of the observers, coupled with long-standing sensory deficits.

9 uals with DS may be predominantly related to sensory deficits.

10 rbation leads to both isolated and syndromic sensory deficits.

11 n cause hair cell loss and lead to permanent sensory deficits.

12 neurons, mice lacking PHR1 do not have overt sensory deficits.

13 ol syndrome is a major cause of learning and sensory deficits.

14 s in dendritic arborization with concomitant sensory deficits.

15 urity-onset obesity, insulin resistance, and sensory deficits.

16 been shown previously to be born with severe sensory deficits.

17 mbness and pain and also the most pronounced sensory deficits.

18 d (VPT) is a reliable measure used to assess sensory deficits and is amenable to testing multiple bod

19 rder that causes intellectual disability and sensory deficits and is the most common known cause of a

20 d that glia-ablated animals exhibit profound sensory deficits and that glia provide activities that a

21 ronic disease prevalence and severity, pain, sensory deficits, and mobility difficulty across four ca

22 tes, cancer, heart disease, arthritis, pain, sensory deficits, and mobility difficulty than did those

23 , admission function, cognition, depression, sensory deficits, and mobility impairments.

24 Mental retardation and motor and sensory deficits are among the many debilitating symptom

25 These sensory deficits are not associated with any abnormality

26 promised: these mutants exhibit a variety of sensory deficits as well as the mislocalization of signa

27 and mammals lack the ability to restore the sensory deficits associated with their loss.

28 se with high anti-MAG titers and most severe sensory deficits at baseline.

29 Differences in sensory deficits between the two patient groups with the

30 ng dysaesthesia and six from patients with a sensory deficit but no dysaesthesia.

31 ed by sensory stimuli, it is unknown whether sensory deficits contribute to the development of anxiet

32 Mild to moderate sensory deficits develop in a minority of patients.

33 ic strategies for individuals who experience sensory deficits during development.

34 by asymmetric rigidity and apraxia, cortical sensory deficits, dystonia and myoclonus.

35 eroid injections are useful for leg pain and sensory deficits early in the course of sciatica seconda

36 Self-reported auditory-vestibular-visual sensory deficits, focal neurologic dysfunction, seizures

37 CI, 7.0 to 15.3); auditory-vestibular-visual sensory deficits (HR, 2.3; 95% CI, 1.3 to 4.0); focal ne

38 Hearing loss is the most common sensory deficit in humans, with some estimates suggestin

39 Deafness is the most common sensory deficit in humans.

40 Hearing loss is the most common sensory deficit in humans.

41 Hearing loss is the most common sensory deficit in humans.

42 It is possible that in dyslexia a persistent sensory deficit in monitoring the frequency of incoming

43 y the nature of the relationship between the sensory deficit in the demyelinated visual pathway and m

44 imeline of such reorganization events during sensory deficits in adulthood.

45 The sensory deficits in Egfr null dorsal skin were not assoc

46 owledge, this is the first study to evaluate sensory deficits in hip OA and to demonstrate that there

47 s a simple and reliable technique to measure sensory deficits in subjects with OA of the knee.

48 ed PLCbeta3 expression may contribute to the sensory deficits in the late-stage diabetic db(-)/db(-)

49 econdary etiological factor in the motor and sensory deficits in the rabbit model of cerebral palsy.

50 Lower extremity sensory deficits, including reduced proprioception, join

51 athic INS (n = 84) or INS with an associated sensory deficit: INS and albinism (n = 71), bilateral op

52 In contrast to the permanent sensory deficits observed in control mice after dorsal r

53 lassically described "onion skin" pattern of sensory deficits observed in patients after trigeminal t

54 d changes in learning are not the product of sensory deficits or state-dependent learning.

55 In addition, sensory deficits predicted impaired cognitive ERP genera

56 develop nerve conduction velocity (NCV) and sensory deficits prior to impaired glucose tolerance.

57 of electrical stimulation of the leg with a sensory deficit, protocols yielded increases in heart ra

58 sk for late-onset auditory-vestibular-visual sensory deficits (rate ratio [RR], 1.8; 95% CI, 1.5 to 2

59 d in MCI, which parallels the most prominent sensory deficit seen in AD.

60 nce with orientation information processing (sensory deficit) versus with the routing of information

61 Patients with baseline sensory deficits went on to develop more symptom complai

62 Increased sensory deficits were associated with increased activati

63 Motor or sensory deficits were present on clinical examination in

64 A high frequency of cognitive and sensory deficits with decreasing gestational age suggest