ライフサイエンスコーパス: sensory deprivation

1 es in homeostatic gene expression related to sensory deprivation.

2 nificant functional reorganisation following sensory deprivation.

3 with SHF are selectively preserved following sensory deprivation.

4 development, or reinstated in adulthood, by sensory deprivation.

5 d limb range of motion, crowding, and visual sensory deprivation.

6 city and its genetic underpinnings following sensory deprivation.

7 targeted synaptic protein degradation under sensory deprivation.

8 cells, a previously unappreciated effect of sensory deprivation.

9 ecovery of cortical responsiveness following sensory deprivation.

10 recovery of retinal function after prolonged sensory deprivation.

11 he cortical reorganization in a rat model of sensory deprivation.

12 other common feature and results in combined sensory deprivation.

13 es in glutamatergic input synapses caused by sensory deprivation.

14 spectrum disorders, mental retardation, and sensory deprivation.

15 NMDA receptor currents were unaffected by sensory deprivation.

16 ir use to facilitate recovery from trauma or sensory deprivation.

17 ted by PV-positive basket cells is pruned by sensory deprivation.

18 mouse visual cortex in vivo with and without sensory deprivation.

19 this polymodal area is modified after early sensory deprivation.

20 ressed in NM neurons and are not affected by sensory deprivation.

21 rge-scale changes in synaptic dynamics after sensory deprivation.

22 rding how the deaf brain in humans adapts to sensory deprivation: (1) is meaning extracted and integr

23 GABA(A) receptors could underlie effects of sensory deprivation, [3H]muscimol binding was assessed i

24 Sensory deprivation after new GCs had differentiated ind

25 Here we report that sensory deprivation alters short-term synaptic dynamics

26 Sensory deprivation alters the properties of synaptic pl

27 Furthermore, P2 asymmetry is modified by sensory deprivation and abolished by decreased BDNF leve

28 es two powerful drivers for brain plasticity-sensory deprivation and altered use.

29 Both sensory deprivation and blockade of gamma-aminobutyric a

30 ical neurons are bidirectionally modified by sensory deprivation and experience, but the synaptic bas

31 However, research has largely focused on sensory deprivation and maladaptive change.

32 3 (L2/3) of adult mouse barrel cortex during sensory deprivation and recovery.

33 l blindness, the brain develops under severe sensory deprivation and undergoes remarkable plastic cha

34 Moreover, duplicate S1 showed plasticity to sensory deprivation, and duplicate V1 responded to visua

35 deaf white cat represents an animal model of sensory deprivation because it mimics a form of human de

36 contacts within the IPL also decreased with sensory deprivation but required at least 6 weeks to rec

37 aptic proteins whose levels were affected by sensory deprivation but whose synaptic roles have not ye

38 endent homeostatic plasticity in response to sensory deprivation, but IB cells were capable of a much

39 restore cortical activity in vivo following sensory deprivation, but it is unclear whether this reco

40 plasticity was preserved under conditions of sensory deprivation, but was rapidly lost by sensory exp

41 Congenital sensory deprivation can lead to reorganization of the de

42 In contrast to experience, sensory deprivation caused homeostatic synaptic enhancem

43 These results suggest that sensory deprivation causes synaptic depression by revers

44 For instance, sensory deprivation causes the cortical area representin

45 Deafferentation or sensory deprivation decreases TH expression but it is no

46 gs have extra axon branches, suggesting that sensory deprivation disrupts axon outgrowth.

47 3 encompasses a critical period during which sensory deprivation disrupts central mechanisms that sup

48 Sensory deprivation during a critical period reduces spi

49 ic proteomes are altered in barrel cortex by sensory deprivation during synaptogenesis.

50 However, sensory deprivation during the critical period degraded

51 animals reared with certain types of visual sensory deprivation during their first few months of lif

52 Sensory deprivation during these periods permanently com

53 to mark the close of the critical period and sensory deprivation during this epoch disrupts developme

54 plasticity in the central nervous system and sensory deprivation during this period significantly imp

55 II function, such as occurred in rats during sensory deprivation, elevated the generation and propaga

56 Sensory deprivation experiments in adult rats also have

57 Classically, sensory deprivation first drives rapid Hebbian weakening

58 Sensory deprivation greatly influences the development o

59 Sensory deprivation, if initiated before P14, disrupted

60 homeostatic changes in spine size following sensory deprivation in a subset of inhibitory (layer 2/3

61 somatosensory cortex after a brief period of sensory deprivation in adulthood.

62 tenance of cortical responsiveness following sensory deprivation in adulthood.

63 e been thought to limit reorganisation after sensory deprivation in adults.

64 indings indicate that a restricted period of sensory deprivation in early postnatal life (1) impairs

65 for the loss of synaptic strength caused by sensory deprivation in visual cortex.

66 resent long-term depression (LTD) induced by sensory deprivation in vivo.

67 n synaptic input caused by lesions or severe sensory deprivation induce marked sprouting or retractio

68 Sensory deprivation induced marked changes in the densit

69 Sensory deprivation, induced by unilateral naris occlusi

70 These results suggest that sensory deprivation-induced homeostatic down-regulation

71 that in the cortex, BC1 RNA is required for sensory deprivation-induced structural plasticity of den

72 These maps can be modified by sensory deprivation, injury and experience in both young

73 Recovery from sensory deprivation is slow and incomplete in adult visu

74 Sensory deprivation is the known causal mechanism for ac

75 The resulting sensory deprivation jeopardizes auditory cortex (AC) mat

76 alters the spatial pattern of apoptosis and sensory deprivation leads to exacerbated amounts of apop

77 There is substantial evidence that sensory deprivation leads to important cross-modal brain

78 Sensory deprivation markedly (approximately 40%) reduced

79 Sensory deprivation most likely results in AMPA receptor

80 ether the synaptic reorganization induced by sensory deprivation occurred differently in mature neona

81 y help to explain the unique consequences of sensory deprivation on plasticity in the developing vers

82 According to the effect of sensory deprivation on synaptic plasticity of developing

83 he structural and functional consequences of sensory deprivation on the establishment of parallel cir

84 kingly rescued independent of Mecp2 by early sensory deprivation or genetic deletion of the excitator

85 We demonstrate several scenarios, such as sensory deprivation or heightened plasticity, under whic

86 petition between thalamocortical axons using sensory deprivation or increasing the size of S1.

87 tances such as perinatal brain injury, early sensory deprivation or limb malformation may result in a

88 at analyses from prolonged periods of either sensory deprivation or stimulation during adulthood are

89 factory system is particularly vulnerable to sensory deprivation, owing to the widespread prevalence

90 Following sensory deprivation, primary somatosensory and visual co

91 l cortex neurons would fail to develop after sensory deprivation produced by bilateral whisker trimmi

92 Sensory deprivation reduced thalamocortical inputs on NG

93 cross-modal plasticity in the case of early sensory deprivation relates to the original functional s

94 Sensory deprivation reorganizes neurocircuits in the hum

95 We have recently shown that sensory deprivation results in large changes of the shor

96 Sensory deprivation shows competitive interactions betwe

97 Sensory deprivation started a few days earlier at P10, h

98 In contrast, when sensory deprivation started after synaptic formation was

99 Sensory deprivation started at P12-P13, but not at P16,

100 ggest that increased use of one sense due to sensory deprivation, such as touch in blind people, lead

101 Sensory deprivation, such as whisker deprivation, is one

102 process in a higher mammal model of complete sensory deprivation, the congenitally deaf cat.

103 how that in young adolescent mice, long-term sensory deprivation through whisker trimming prevents ne

104 pression (LTD) of cortical synapses, but how sensory deprivation triggers LTP and LTD in vivo is unkn

105 t plasticity after early restricted neonatal sensory deprivation was analyzed in barrel field cortex

106 Sensory deprivation was done by whisker plucking, and sy

107 Selective sensory deprivation was induced by trimming two whiskers

108 he greatest decrement in synchrony following sensory deprivation, while neurons with diverse inputs f

109 evel of Abeta-LTMR activity in rat models of sensory deprivation (whisker clipping, tail suspension,

110 somatosensory cortex remodels in response to sensory deprivation, with regions deprived of input inva