ライフサイエンスコーパス: sensory loss

1 complaint (balance disturbance, headache and sensory loss).

2 characterized by distal muscle weakness and sensory loss.

3 tions that are specifically uncovered during sensory loss.

4 of adult primates reactivate over time after sensory loss.

5 onsidered, rather than a model primarily for sensory loss.

6 ce and six times more than the subgroup with sensory loss.

7 icity related to chronic, persistent partial sensory loss.

8 ay be restored in individuals suffering from sensory loss.

9 quality of life of the individual with dual sensory loss.

10 em of sound localization in people with dual sensory loss.

11 kle joint pain, tenderness to palpation, and sensory loss.

12 ned, accompanied by hypotonia, and motor and sensory loss.

13 o pain, paralysis, autonomic involvement and sensory loss.

14 to upper extremity paresis, paresthesia, and sensory loss.

15 sses of hearing and vision function, or dual sensory loss, affect a large number of individuals of al

16 After sensory loss and deafness, the brain's effective connect

17 tentials (CMAPs and SNAPs), distal weakness, sensory loss and decreased reflexes.

18 ng sensory information to motor cortex, such sensory loss and representational reorganization could a

19 The region of sensory loss and visual analog pain scale improved in th

20 th-dependent axonal degeneration with distal sensory loss and weakness, deep-tendon-reflex abnormalit

21 distribution of both large- and small-fiber sensory loss and which approaches and techniques may be

22 f neurotoxic chemotherapy resulting in pain, sensory loss, and decreased quality of life.

23 akness and atrophy, foot deformities, distal sensory loss, as well as diminished tendon reflexes.

24 e used as a surrogate measure of small-fiber sensory loss but appear not to correlate closely with se

25 Following sensory loss, compensatory crossmodal reorganization occ

26 As a result, congenital sensory loss could be thought of as a connectome disease

27 es for which this explanation of the partial sensory loss could not be maintained.

28 osensory deficits in OA, since the extent of sensory loss directly correlated with the radiographic s

29 changes, as well as patterns of weakness and sensory loss due to transverse myelitis or peripheral ne

30 The pattern of sensory loss following transection of the anterolateral

31 'asleep numbness' in 20, mild pain in 13 and sensory loss in 23.

32 pmental synesthesia and plasticity following sensory loss in acquired synesthesia.

33 bacute meningomyeloradiculitis was left with sensory loss in the feet.

34 Sensory loss induces cross-modal plasticity, often resul

35 Prominent distal sensory loss involved all modalities, resulting in neuro

36 The area of partial sensory loss is due to transection of the fibres that ar

37 anding cross-modal plasticity in response to sensory loss is essential to maximize patient susceptibi

38 This pattern of sensory loss is explained as follows.

39 Dual sensory loss may present at any age as a result of genet

40 primary motor cortex (M1), especially if the sensory loss occurs early in development.

41 portunity to prevent progressive symptoms of sensory loss, pain, autonomic dysfunction, ulcerations,

42 e reactivation of somatosensory cortex after sensory loss produced by spinal cord lesions in the comm

43 The TRPA1-dependent sensory loss produced by STZ occurs before the onset of

44 leading to progressive gait and limb ataxia, sensory loss, reduced tendon reflexes, dysarthria, absen

45 t the functional reorganization that follows sensory loss results from changes in synaptic strength a

46 stations of the neuropathic state-allodynia, sensory loss, shooting pains, etc, that can manifest lon

47 professionals can meet the challenge of dual sensory loss that awaits us with the aging of the popula

48 d autonomic neuropathy type 1 (HSAN1) causes sensory loss that predominantly affects the lower limbs,

49 imilar, including distal muscle weakness and sensory loss, their molecular pathogenesis is likely to

50 atic, neurological examination showed distal sensory loss to pain, or vibration or distal loss of ref

51 ual variability in the brain's adaptation to sensory loss underpinning much of the observed variation

52 Prominent distal sensory loss was a consistent feature in one family, res

53 oa mutation as a mouse model for mixed motor-sensory loss when the entire neuraxis is considered, rat

54 d at a later age, we find these mice develop sensory loss with a distal small fiber neuropathy and pe

55 ave slowly progressive weakness, wasting and sensory loss, with an axonal neuropathy typical of CMT2,

56 Blindness is a common cause of major sensory loss, with an estimated 39 million people worldw