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1 s and modulate their sensitivity directly in sensory nerve endings.
2 tivating TRPA1, an excitatory ion channel on sensory nerve endings.
3 activating capsaicin-sensitive perivascular sensory nerve endings.
4 tussive mediators and activation of afferent sensory nerve endings.
5 e-1 (COX-1) or COX-2 produce hyperalgesia in sensory nerve endings.
6 activating specific (vanilloid) receptors on sensory nerve endings.
7 Capsaicin is a specific activator of sensory nerve endings.
8 ng at CB1 cannabinoid receptors localized on sensory nerve endings.
9 ds, arrector pili muscles, Merkel cells, and sensory nerve endings.
10 been reported to be released from cutaneous sensory nerve endings after hapten application, we deter
11 mily of ion channels, which are expressed in sensory nerve endings and in skin, respond to distinct t
12 estored the branching of diabetes-suppressed sensory nerve endings and regeneration in the diabetic c
13 there is increased pulpal NGF, sprouting of sensory nerve endings, and increased immunoreactivity fo
14 In knockout mice lacking synapsin I and II, sensory nerve endings are normally developed but not sti
17 TRPM3, and indicate that TRPM3 activation in sensory nerve endings can contribute to neurogenic infla
18 alpha-motor axons in SOD1(G93A) mice, Ia/II sensory nerve endings degenerate in the absence of obvio
19 ent with capsaicin, which depletes SP in the sensory nerve endings, eliminates stress-control differe
20 nates following the activation of peripheral sensory nerve endings following damage or exposure to in
22 ation results from the excitation of primary sensory nerve endings in the skin, but the underlying mo
25 suggest that endogenous CGRP concentrated in sensory nerve endings may regulate locally the immune re
26 el, DRASIC, in several different specialized sensory nerve endings of skin, suggesting it might parti
28 dermal epidermal junction next to peripheral sensory nerve endings, suggesting that viral reactivatio
29 A raises blood pressure by stimulating renal sensory nerve endings that contain synapsin-positive mic
30 hiocyanate) to the skin activates underlying sensory nerve endings, thereby producing pain, inflammat
31 ent strategies that target TRPA1 channels on sensory nerve endings to achieve chemical deterrence.
33 another group, we blocked 5-HT3 receptors on sensory nerve endings with tropisetron (300 microg kg(-1
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