ライフサイエンスコーパス: sepal

1 ted to the pedicels (and, in some cases, the sepals).

2 in the variability of clones throughout the sepal.

3 t a continuous mechanical model of a growing sepal.

4 on throughout most of the development of the sepal.

5 and cell size patterning in the Arabidopsis sepal.

6 outer epidermis of the Arabidopsis thaliana sepal.

7 to four extra petals, and one or two missing sepals.

8 of all four SEP genes in the development of sepals.

9 nes are also expressed in the guard cells of sepals.

10 s and, possibly, palea and lemma as modified sepals.

11 oduce flowers in which all organs develop as sepals.

12 of the floral organs examined apart from the sepals.

13 the growth of clones of cells in Arabidopsis sepals.

14 neighboring cell growth was reduced in ftsh4 sepals.

15 may not control the identity of the petaloid sepals.

16 ased numbers of sepals and exhibit fusion of sepals.

17 Most wild-type Arabidopsis flowers have 4 sepals, 4 petals, 6 stamens, and 2 carpels.

18 y results in all flower organs developing as sepals [5].

19 ost commonly show a pentamerous pattern of 5 sepals, 5 petals 5 stamens, and 2 carpels.

20 ly growing trichome cells in the Arabidopsis sepal, a reproducible floral organ.

21 nes of 'Hayward' kiwifruit, at the "break of sepals", about one week before anthesis, to study its ef

22 In the sepal and carpel whorls the smallest sectors of marked a

23 marked tissue were found to be one half of a sepal and one half of a carpel, respectively.

24 We also show that the role of RBE in sepal and petal development is mediated in part through

25 fl) phenotype, where defects are observed in sepal and petal development, but leaf blades are apparen

26 jag alleles have their strongest effects on sepal and petal development, suggesting that JAG may act

27 The A-class gene APETALA2 (AP2) promotes sepal and petal identities in whorls 1 and 2 and restric

28 istem identity but function independently in sepal and petal identity (AP1) and in proper fruit devel

29 (AP1) specifies floral meristem identity and sepal and petal identity in Arabidopsis.

30 ly, the role of APETALA1 as an "A-function" (sepal and petal identity) gene is thought to be Brassica

31 est known for the role of AP1 in Arabidopsis sepal and petal identity, the canonical A function of th

32 , regulate developmental events required for sepal and petal organogenesis.

33 d later its expression becomes restricted to sepal and petal primordia.

34 also has subtle pleiotropic effects on both sepal and pistil morphology.

35 limited primarily to vascular tissue in the sepal and the silique.

36 of TAGL1 in tomato resulted in expansion of sepals and accumulation of lycopene, supporting the role

37 11 is uniquely expressed in the trichomes of sepals and ACS1 in the replum.

38 of flower development, including carpelloid sepals and an inhibition of petal development.

39 ant, principally after infusion of its dried sepals and calyces, which are usually discarded.

40 versions of floral organ whorls 2 and 3 into sepals and carpelloid structures, respectively, similar

41 We show that bnq3 mutants have pale-green sepals and carpels and decreased chlorophyll levels, sug

42 mutants, petals and stamens are missing and sepals and carpels develop in their place.

43 etals and stamens are partially converted to sepals and carpels, respectively.

44 lts in flowers consisting almost entirely of sepals and carpels.

45 aloid, that produces flowers comprising only sepals and carpels.

46 all biotypes examined and could extend into sepals and corolla.

47 t some rbe flowers have increased numbers of sepals and exhibit fusion of sepals.

48 Additionally, rin plants display enlarged sepals and loss of inflorescence determinacy.

49 elopmentally in young flowers, and in mature sepals and ovaries, whereas transcript for hmg3.3 accumu

50 t U1-70K is necessary for the development of sepals and petals and is an essential gene in plants.

51 -function is required for the development of sepals and petals and to antagonize the C-function in th

52 a striking increase in the longevity of the sepals and petals as well as delays in a selected set of

53 roductive tissue, expression was observed in sepals and petals before anthesis, and in all floral org

54 whorls, where it specifies the identities of sepals and petals by restricting the expression of AGAMO

55 e organs surrounded by a sterile perianth of sepals and petals constitute the basic floral structure.

56 In many plants, including Arabidopsis, the sepals and petals form distinctive nanoridges in their c

57 clade in which perianth differentiation into sepals and petals has evolved multiple times.

58 The A1 and A3 genes are expressed in the sepals and petals of flowers as well as the outer layer

59 loral organ abscission, and produced leaves, sepals and petals with diminished blades, indicating a r

60 inct floral organs, including differentiated sepals and petals, and a perianth distinct from stamens

61 (mSlARF10) developed narrow leaflet blades, sepals and petals, and abnormally shaped fruit.

62 le flowers within one sepal whorl, fusion of sepals and petals, and conversion of sepals into carpel-

63 be expressed in all floral organs, including sepals and petals, arguing against the hypothesis that p

64 epetition of sepals and stamens, rather that sepals and petals, as is observed in agamous single muta

65 in floral organ number, particularly in the sepals and petals, correlating with an increase in the w

66 d sepal-to-petal transformations and/or more sepals and petals, suggesting interference with N. benth

67 ggum flowers contain more organs, especially sepals and petals, than found in wild-type flowers.

68 1 gene is required for normal development of sepals and petals.

69 vein pattern defects in cotyledons, leaves, sepals and petals.

70 t to floral meristems and the development of sepals and petals.

71 Mutants have fused sepals and reduced organ numbers in all four whorls, esp

72 lateral stipules from leaves and of lateral sepals and stamens from flowers.

73 nts and caused transformation of petals into sepals and stamens into carpels.

74 sults in homeotic conversions of petals into sepals and stamens into carpels.

75 nts exhibit partial conversions of petals to sepals and stamens to carpels.

76 indeterminate and consist of a repetition of sepals and stamens, rather that sepals and petals, as is

77 petals, stamens, and carpels in addition to sepals and that it plays an important role in meristem i

78 The first (outermost) whorl consists of four sepals and the fourth (innermost) whorl is made up of tw

79 tight cluster; it also has narrower leaves, sepals, and petals than either single mutant or wild-typ

80 ongation of floral organs, including petals, sepals, and siliques in Arabidopsis.

81 P10 Nkx2-5(+/R52G) mice demonstrated atrial sepal anomalies, with significant increase in the size o

82 However, the fact that sepals are converted into carpelloid organs in certain m

83 s plants is seen in flowers where individual sepals are fused along the lower part of their margins.

84 tem formation, coupled with slower growth of sepals at 15 degrees C, produced larger intersepal regio

85 (AtZFP2), was elevated in stamen, petal, and sepal AZs.

86 Sepals become swollen, red, and succulent, produce ethyl

87 e plant with expression in leaves, stems and sepals but not in petals, mesophyll cells or roots.

88 In vitro culture of VFNT Cherry tomato sepals (calyx) at 16-21 degrees C results in development

89 demonstrates that differential growth in the sepal can generate transverse tensile stress at the tip.

90 d whorl sepal-petal-stamens and fourth whorl sepal-carpels.

91 ent field accurately describes the growth of sepal cell lineages and reveals underlying trends within

92 s elongation or differentiation of petal and sepal cells.

93 to AGAMOUS gene, TAG1, in ripening, in vitro sepal cultures and other tissues from the plant and foun

94 ransitional staminodes, a staminal tube, and sepal cup can be viewed as prehypanthial, lacking only f

95 loral characters of the fossils, including a sepal cup, staminal tube, and apparently nectariferous s

96 king only fusion of the staminal tube to the sepal cup.

97 gen allocation to female whorls (pistils and sepals) decreased under high density, whereas nitrogen a

98 more, in non-embryonic tissues (true leaves, sepals), DEK1 is required for epidermis differentiation

99 f a conserved petal identity program between sepal-derived and stamen-derived petaloid organs in the

100 We found that neither sepal-derived nor stamen-derived petaloid organs exhibit

101 are not expressed during the development of sepal-derived petals and are not implicated in petal ide

102 utant that exhibits defects in whorl 2 where sepals develop in place of petals, but third whorl stame

103 egulates ripening, whereas LeMADS-MC affects sepal development and inflorescence determinacy.

104 ts, aberrant phyllotaxy of flowers, aberrant sepal development, floral buds that open precociously, a

105 ng, we show that during Arabidopsis thaliana sepal development, fluctuations in the concentration of

106 ut are lost progressively at later stages of sepal development, indicating that CUS2 is crucial for t

107 d TAGL1 act together to repress fourth whorl sepal development, most likely through the MACROCALYX ge

108 tterns suggested roles in fruit ripening and sepal development, respectively.

109 Sepals enclose and protect the flower bud, while petals

110 The sepal epidermis of Arabidopsis (Arabidopsis thaliana) co

111 reduction in cuticular ridges on the mature sepal epidermis, but only a moderate effect on petal con

112 mutant with ectopic giant cells covering the sepal epidermis.

113 nescent tissues: leaves, cotyledons, petals, sepals, filaments, stigmas, nectaries and siliques.

114 alyze the epidermal cells of the Arabidopsis sepal, focusing on cortical microtubule arrays, which al

115 , individual cell lineages in the developing sepal follow similarly shaped growth curves.

116 uticular ridge formation progresses down the sepal from tip to base as the sepal grows.

117 th cuticular ridge formation, descending the sepal from tip to base.

118 AGAMOUS in sepals, TAG1 RNA levels in green sepals from greenhouse-grown plants is detectable, its c

119 esses down the sepal from tip to base as the sepal grows.

120 both ridge formation and maintenance as the sepal grows.

121 y meristem growth and in floral meristem and sepal identity and that they also play a key role in fru

122 euAP1 genes function in floral meristem and sepal identity, whereas euFUL genes control phase transi

123 y step leading to growth arrest in the whole sepal in response to its own growth.

124 pels because these organs are converted into sepals in sep1 sep2 sep3 triple mutants.

125 The sepals in the transgenic 35S-NAG plants also support eff

126 sion of sepals and petals, and conversion of sepals into carpel-like structures that grew into fruits

127 tions, leading to the homeotic conversion of sepals into petals, carpels, or stamens, depending on th

128 resh flower mass, petal length, petal width, sepal length, sepal width, long stamen length, short sta

129 ies, exhibit numerous instances of petal and sepal loss, transference of petal function between flora

130 the basal left ventricular summit (25%) and sepal LVOT.

131 lular organisation during the development of sepal margins and ovule integument outgrowth.

132 omical analysis indicates that the fusion of sepal margins precludes shedding even though abscission,

133 3 expression was strongest in the petals and sepals of developing flowers and declined after flower o

134 nt in epidermal tissue of roots, petals, and sepals of flower buds, papillae cells of flowers, siliqu

135 ter activity of AtBSMT1 was localized to the sepals of flowers, and also to leaf trichomes and hydath

136 Sepals of fruit on transgenic tomato plants that express

137 observed within organ primordia, such as the sepals of terminal flowers produced by centroradialis mu

138 al tissues, and TTS mRNAs are induced in the sepals of these plants.

139 0-kDa molecular mass range accumulate in the sepals of these transgenic, 35S-NAG plants.

140 ons, increased lateral root density, delayed sepal opening, elongated pistils, and reduced fertility

141 e expressed specifically or predominantly in sepals or petals.

142 at the summit and the first whorl transverse sepal pair at the base.

143 r amounts in leaves, fruit coats, seeds, and sepals; Pap2 transcript was abundant only in the petals;

144 pocotyl, roots, various parts of the flower (sepals, pedicle, style, etc.) and in the stigmatic and a

145 However, like AP2, LIP genes play a role in sepal, petal and ovule development, although some of the

146 HUA2, leads to the production of third whorl sepal-petal-stamens and fourth whorl sepal-carpels.

147 uses and defects in the timely elongation of sepals, petals and stamens, similar to 35S::KNAT1 plants

148 In sepals, petals and stamens, the strongest defects are se

149 om transactivated lines failed to shed their sepals, petals, and anthers during pod expansion and mat

150 expression in the abscission zones where the sepals, petals, and stamens attach to the receptacle, at

151 ifferentiation of four distinct organ types (sepals, petals, stamens and carpels), each of which may

152 wers are organized into concentric whorls of sepals, petals, stamens and carpels, with each of these

153 , and cotyledons) and reproductive (pistils, sepals, petals, stamens, and floral buds) organs examine

154 on patterning was regulated by PeMYBs in the sepals/petals and lip.

155 In the sepals/petals, silencing of PeMYB2, PeMYB11, and PeMYB12

156 ssed at moderate levels in leaves, pedicels, sepals, pistils and petals, and at very low levels in ro

157 Sepal primordia had accelerated cell division, cell enla

158 pi-5 flowers, second whorl organs develop as sepals rather than petals, but third whorl stamens are n

159 role in developmental changes that result in sepal ripening.

160 In wild-type sepals, ROS accumulate in maturing cells and limit organ

161 to stress in katanin and spiral2 mutant made sepal shape dependent on trichome number, suggesting tha

162 no significant effect of trichome number on sepal shape in wild-type and lines with trichome number

163 model of the sepal, we predict an impact on sepal shape that is validated experimentally using mutan

164 recision, we designed a screen for disrupted sepal size and shape uniformity in Arabidopsis and ident

165 n TF binding elements--CArG-boxes, directing sepal specific expression in Arabidopsis--were accrued i

166 stic alterations, with one additional petal, sepal, stamen, and carpel at each of the four whorls, re

167 d that gene expression was restricted to the sepals, stigmas, anther filaments, and receptacles, reac

168 s of sepals with a diminished third whorl of sepals surrounding a fourth whorl of carpels, or three w

169 fourth whorl of carpels, or three whorls of sepals surrounding abnormal carpels.

170 ity, with flowers containing three whorls of sepals surrounding fertile carpels, two whorls of sepals

171 rary to reports on the absence of AGAMOUS in sepals, TAG1 RNA levels in green sepals from greenhouse-

172 ovary, and pollen grains, but low in petals, sepals, the epidermis of anthers, styles, and filaments.

173 However, at the sepal tip, where organ maturation initiates and growth s

174 ulator, channels the growth and shape of the sepal tip.

175 tions and align along maximal tension at the sepal tip.

176 the two compounds is restricted to glandular sepal tips; thus, differential expression analysis of co

177 GUS expression signals were also evident in sepal tissues of these double-transgenic plants.

178 es in the Arabidopsis (Arabidopsis thaliana) sepal to determine how the growth curves of individual c

179 type, in which homeotic transformations from sepals to carpels are found in flowers derived from old

180 Interestingly, 35S::MIR172 plants had sepal-to-petal transformations and/or more sepals and pe

181 tissues (pistil tips, developing anthers and sepal vasculature).

182 chanical feedback in our growth model of the sepal, we predict an impact on sepal shape that is valid

183 In both bgal10 and bgal10 xyl1, siliques and sepals were shorter, a phenotype that could be explained

184 ifferent flower organs (carpels, petals, and sepals) were profiled for the first time at a spatial re

185 rmed a gradient in the developing leaves and sepals, whereas low or no GUS activity was detected in t

186 ing of the cellular variability in wild-type sepals, which is disrupted in the less-variable cells of

187 re observed in siliques, carpels, petals and sepals while stemlike organs (filaments and pedicels) ha

188 ng production of multiple flowers within one sepal whorl, fusion of sepals and petals, and conversion

189 ss, petal length, petal width, sepal length, sepal width, long stamen length, short stamen length, an

190 s surrounding fertile carpels, two whorls of sepals with a diminished third whorl of sepals surroundi