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1 filaments of rod-shaped cells that failed to septate.
2 enlarged regions are convoluted or partially septated.
3 ates toward the caudal midline as the cloaca septates.
4 h failing at nuclear division, these mutants septate and divide.
5 sted by the inability of plo1 Delta cells to septate and the prolific septation following plo1(+) ove
6 ogs of the BBB are occlusive (pleated-sheet) septate and tight junctions between perineurial cells, g
7 nd ZO-1, which are arranged symmetrically at septate and tight junctions, respectively.
8        Microscopically, hyphae were hyaline, septate, and branched and remained totally devoid of con
9 H resulted in a viable strain that failed to septate at any temperature.
10 ls formed a misshapen actin ring, but rarely septated at 36 degrees C.
11  the cells are arrested in the cell cycle as septated, binucleated cells with highly condensed chroma
12  tissue from lungs and brain showed hyaline, septate, branched hyphae with clamp connections.
13           Microscopically, their hyphae were septate, branched, and phaeoid and bore lateral and term
14       Histopathological examination revealed septate branching hyphae, suggesting a diagnosis of inva
15 arance of abnormal medial ring structures in septated cells that lack imp2.
16                                              Septating cells are attacked more readily than nonseptat
17 oskeleton shows the lack of a medial ring in septating cells that overexpress imp2, and the appearanc
18 (iii) a medial band in a small percentage of septating cells.
19 ineered mutants of HDC1 had smaller and less septate conidia and exhibited an approximately 50% reduc
20 haeoid and bore lateral and terminal, erect, septate conidiophores.
21                           Fifty patients had septated cysts; seven of these patients had borderline n
22  is in place before the machinery to degrade septated daughter cells is enabled.
23 tomy and excisional biopsy by observation of septate, dematiaceous hyphal elements 2 to 3 microm in w
24  Helotiales, a group that includes many dark septate endophytes known to associate positively with ro
25 amine silver stains showed numerous hyaline, septate, fungal hyphae of various lengths, many broken i
26 ted germination-structures and then produced septate germlings at a water-activity of just 0.585 ( id
27 of differentiated germination-structures and septate germlings, and subsequent development of myceliu
28                                    Branched, septate hyphae and moniliform hyphae consisting of chain
29             A black velvety mould with brown septate hyphae and tapered annellides was isolated from
30 fections due to molds characterized by thin, septate hyphae branching at acute angles, voriconazole s
31 eg lesion also noted on day 67 p.t. revealed septate hyphae consistent with Aspergillus species, and
32 olar lavage (BAL) fluid in order to identify septate hyphae noted by Gomori methenamine silver (GMS)
33                      At the time of autopsy, septate hyphae were present in heart, thyroid, and lung
34                   Filamentous fungi with non-septate hyphae were presumptively identified as agents o
35 yogranulomatous inflammation with branching, septate hyphae.
36 stinal wall showed infiltration by branching septate hyphae.
37 ori's methenamine silver strains showed many septate hyphal elements of various lengths, some exhibit
38  outflow tract (OFT) of the developing heart septates into the base of the pulmonary artery and aorta
39  Von Meyenberg complexes, usually present as septated intrahepatic cystic lesions.
40 t the Drosophila Na,K-ATPase is required for septate junction (SJ) formation and that of the three be
41       We also show that vari is an essential septate junction (SJ) gene encoding a membrane associate
42 pericardial cell adhesion also relies on the septate junction (SJ) proteins Neurexin-IV (Nrx-IV), Sin
43 elia, whereas in invertebrate epithelia, the septate junction (SJ) provides this function.
44 ar junction that is rare in vertebrates, the septate junction (SJ).
45  is localized on the cytoplasmic face of the septate junction and is required for the maintenance of
46 inantly required for correct localization of septate junction components, while Sinuous is predominan
47 nd the transmembrane protein Neurexin to the septate junction display an interdependent relationship,
48 ng that Kune-kune has a more central role in septate junction formation than either Sinuous or Megatr
49 ed for tracheal tube size control, including septate junction formation, deposition of a lumenal/apic
50 equirements for these claudins in epithelial septate junction formation.
51  of the Na+/K+ ATPase play a crucial role in septate junction function and that septate junctions hav
52 sinuous functions in the same pathway as the septate junction genes neurexin and scribble, but that n
53 ted that Coracle protein is localized to the septate junction in epithelial cells and is required for
54  is localized to the cytoplasmic face of the septate junction in epithelial cells.
55 tion in epithelial cells and is required for septate junction integrity.
56  is both necessary and sufficient for proper septate junction localization in transgenic embryos.
57 ion of production of their respective smooth septate junction membrane proteins located within the in
58 zes to septate junctions and is required for septate junction organization and paracellular barrier f
59 SF2, of the apical determinant Crumbs, or of septate junction protein Varicose.
60 ells become irregular in size and shape, and septate junction proteins are mislocalized to a more api
61 ly required for maintaining normal levels of septate junction proteins.
62 e is a necessary structural component of the septate junction required for the maintenance of the tra
63 markedly reduced gut acidification, atypical septate junction structure, depressed gut motility, and
64 sential membrane-organizing functions at the septate junction, and that these functions are carried o
65 ateral domain of epithelia, particularly the septate junction, functions in restricting apical membra
66         Scrib is localized to the epithelial septate junction, the analogue of the vertebrate tight j
67  Drosophila, this barrier is provided by the septate junction, which, despite being ultrastructurally
68  we report that the Drosophila transmembrane septate junction-specific protein Neurexin IV (Nrx IV) f
69  required for restricting the protein to the septate junction.
70 h one another at the cytoplasmic face of the septate junction.
71 he electron microscopic observations of the "septate" junction conform to a honeycomb structure, with
72                    We imaged a member of the septate-junction complex that was used to outline the th
73  of the Drosophila cardiac system depends on septate-junction proteins even though the heart lacks di
74 es and destabilization of paranodal axoglial septate junctions (AGSJs) as early as postnatal day 30.
75 elia where tight junctions in vertebrates or septate junctions (SJ) in invertebrates from three cells
76 tion by glial cells and the establishment of septate junctions (SJs) between glial cell membranes.
77 incipally tight junctions in vertebrates and septate junctions (SJs) in invertebrates.
78                          Here we report that septate junctions (SJs), the vertebrate analogs of TJs,
79 the tight junctions analogous structure, the septate junctions (SJs).
80 ode membrane proteins associated with smooth septate junctions (SSJ) which are required for intestina
81                   Immature Diptera have such septate junctions (without tight junctions) while both j
82 ophila discs large (dlg) disrupts epithelial septate junctions and causes overgrowth of imaginal disc
83 ophila claudin, Kune-kune, that localizes to septate junctions and is required for junction organizat
84 e Dlg protein is a critical component of the septate junctions and is required for maintaining apicob
85  sinuous encodes a claudin that localizes to septate junctions and is required for septate junction o
86  regulation of the transport pathway through septate junctions and the reversible assembly of proton
87 he paracellular barriers formed by arthropod septate junctions and vertebrate tight junctions have a
88 nents identified at the vertebrate axo-glial septate junctions are also present at the Drosophila sep
89 now provide genetic evidence that Drosophila septate junctions are part of the gut immune barrier, a
90          Collectively, our data suggest that septate junctions are required to maintain the subtle ba
91 s gene, the Dlg protein, is localized at the septate junctions between epithelial cells, and that mut
92 eripheral nervous system where pleated-sheet septate junctions bond cells of the nascent (embryonic)
93                           Mutations in known septate junctions genes cause the same tracheal tube-siz
94                      In the tracheal system, septate junctions have a barrier-independent function th
95 ATPalpha and nrv2 mutations, indicating that septate junctions have a previously unidentified role in
96  addition, new roles for axo-glial paranodal septate junctions have emerged, which suggest that the p
97 l role in septate junction function and that septate junctions have multiple distinct functions that
98 ing vertebrate tight junctions and arthropod septate junctions in epithelia.
99 ed at and required for the formation of both septate junctions in epithelial cells and synaptic junct
100 t analyses suggest that tube-size control by septate junctions is mediated by at least two discernabl
101 tion of SPG polyploidy caused rupture of the septate junctions necessary for the blood-brain barrier.
102 ch in turn is necessary for the formation of septate junctions of sufficient length to achieve proper
103 ates, it has recently been demonstrated that septate junctions play an essential role in axonal insul
104 exin IV, a molecular component of Drosophila septate junctions, has been shown to be essential for ax
105 a midgut, BBG is present at the level of the septate junctions, on the apical side of the enterocytes
106               The mutant mice lack paranodal septate junctions, resulting in the diffusion of Caspr a
107 2 mutations also disrupt stable formation of septate junctions, structures with some functional and m
108 target an epitope-tagged Dlg to pre-existing septate junctions.
109 ateralisation at a stage before formation of septate junctions.
110 eins even though the heart lacks discernable septate junctions.
111 sium-rich hemolymph by forming intercellular septate junctions.
112 junctions are also present at the Drosophila septate junctions.
113 nd Malpighian tubules have junctions of the "septate" kind.
114 actin enables the formation of the paranodal septate-like axo-glial junctions in myelinated periphera
115                  Our results indicate that a septate-like junction provides structural support to cal
116 med paranodin--has been shown to localize to septate-like junctional structures.These vertebrate junc
117 are not clustered in md spinal cords, and no septate-like junctions between oligodendrocyte processes
118 myelin loops, and focal absence of paranodal septate-like junctions between the terminal loops and ax
119  At the ultrastructural level, the paranodal septate-like junctions immediately adjacent to the node
120 odes, but the glial and axonal components of septate-like junctions remain colocalized.
121  demonstrated that Caspr is localized to the septate-like junctions that form between axons and the p
122 al cells closely appose and form specialized septate-like junctions with axons.
123 sis of the junctions showed intact paranodal septate-like junctions.
124                         All three had large, septate low-attenuation areas at computed tomography con
125 d, namely tight (occludin) and pleated-sheet septate (neurexin IV).
126 the mutants had smooth colony morphology and septated normally, but all were lysozyme sensitive.
127 xtensive screen for this novel class of sns (septated, not in S-phase) mutants.
128         The lack of Ltbp1L in the ECM of the septating OFT and associated vessels results in altered
129  of an extensive network of brown-pigmented, septate, profusely branched hyphae.
130 planate divided bicellular forms or multiply septated sclerotic bodies in post-log phase, when the G1
131  cells and insufficient mesenchymal cells to septate the cardiac outflow tract.
132 hyme and hypoplastic cushions that failed to septate the ventricular inlet.
133 erian duct anomaly which is characterised by septate uterus with obstruction of a one-sided cavity an
134 ngle ventricular chamber or two incompletely septated ventricles?

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