ライフサイエンスコーパス: septohippocampal

1 t this GABA/peptidergic projection modulates septohippocampal activity and hippocampal theta rhythm r

2 ssfully accounts for changes in behavior and septohippocampal activity observed in studies of the acq

3 Our septohippocampal and caudocentral divisions are topograp

4 topographically comparable to the mammalian septohippocampal and septofimbrial nuclei, respectively,

5 d cholinergic neurons that contribute to the septohippocampal and septohabenular pathways exhibit som

6 ve studied the time course of changes in the septohippocampal and the hippocampal commissural project

7 eural circuitry of fear and anxiety, but the septohippocampal axis also plays an important role.

8 Eighteen MS-DB neurons were identified as septohippocampal by antidromic activation.

9 ne fibres were found in the vicinity of both septohippocampal cholinergic and GABAergic cell types.

10 loss would also obtund histamine effects on septohippocampal cholinergic and GABAergic functions and

11 The relationship between the septohippocampal cholinergic and GABAergic systems and w

12 eficits that, in part, reflect disruption of septohippocampal cholinergic function.

13 rom a direct histamine-induced activation of septohippocampal cholinergic neurones and a subsequent i

14 rodegenerative disorders involving a loss of septohippocampal cholinergic neurones as such a loss wou

15 chanisms whereby acetylcholine released from septohippocampal cholinergic neurons acts to modulate hi

16 gonists in the MSDB involve an excitation of septohippocampal cholinergic neurons and a subsequent in

17 nation, there is age-related degeneration of septohippocampal cholinergic neurons and astrocytic hype

18 Adult septohippocampal cholinergic neurons are dependent on tr

19 hat are associated with a loss or atrophy of septohippocampal cholinergic neurons cannot be attribute

20 ling technique to selectively visualize live septohippocampal cholinergic neurons in rat brain slices

21 ize that epilepsy-related neuroplasticity of septohippocampal cholinergic neurons is capable of incre

22 Degeneration of septohippocampal cholinergic neurons results in memory d

23 trophysiological effects of Hcrt peptides on septohippocampal cholinergic neurons that were identifie

24 Hcrt activation of septohippocampal cholinergic neurons was reversible, rep

25 Both Hcrt1 and Hcrt2 activated septohippocampal cholinergic neurons with similar EC(50)

26 found that muscarinic agonists do not excite septohippocampal cholinergic neurons, instead they inhib

27 locally, presumably via axon collaterals of septohippocampal cholinergic neurons.

28 The septohippocampal cholinergic neurotransmission has long

29 The group with lesions of the septohippocampal cholinergic pathway displayed performan

30 lved in working/episodic memory but that the septohippocampal cholinergic pathway is either not contr

31 The contribution of the septohippocampal cholinergic pathway to performance of a

32 e MSDB does not decrease impulse flow in the septohippocampal cholinergic pathway; instead, it decrea

33 The septocingulate and septohippocampal cholinergic pathways were selectively d

34 roprotective role through maintenance of the septohippocampal cholinergic phenotype and preservation

35 tal, low-level lead exposure induces loss of septohippocampal cholinergic projection neurons in neona

36 e placed in the FF lesion cavity and induced septohippocampal cholinergic regeneration or sympathetic

37 details of structural reorganization of the septohippocampal cholinergic system in experimental epil

38 haracterized model paradigm that engages the septohippocampal cholinergic system.

39 in the aged mouse, possibly by acting on the septohippocampal cholinergic system.

40 en NGF gene expression and experience on the septohippocampal circuitry was assessed by counting retr

41 ergic neurons and influences both septal and septohippocampal circuitry.

42 etically encoded calcium indicators in mouse septohippocampal cocultures, we found interactive but in

43 term memory but suggest that the cholinergic septohippocampal component of this pathway is not requir

44 tion gene expression analysis at the time of septohippocampal deficits in a trisomic mouse model shed

45 activity of protein kinase C (PKC) following septohippocampal denervation of the rat hippocampus.

46 aced into lateral, medial, caudocentral, and septohippocampal divisions, with the lateral and medial

47 There is also cholinergic septohippocampal dysfunction in the PTD model.

48 eling studies, 20% of parvalbumin-containing septohippocampal GABA neurons colocalized the mu recepto

49 Restoration of impulse flow in the septohippocampal GABA pathway, possibly via M(3) recepto

50 mechanism may be the concomitant decrease in septohippocampal GABA release and a subsequent disruptio

51 zes direct and indirect mechanisms to excite septohippocampal GABA-type neurones in a reversible, rep

52 led neurons, confirmed to be noncholinergic, septohippocampal GABA-type neurons using retrograde mark

53 and a subsequent indirect activation of the septohippocampal GABAergic neurones.

54 NA, via alpha 1-adrenoceptors, excites MSDB septohippocampal GABAergic neurons and influences both s

55 e speculate that Hcrt2-induced activation of septohippocampal GABAergic neurons will, by engaging dis

56 sence of Hcrt receptor-2 immunoreactivity in septohippocampal GABAergic neurons, as well as the prese

57 gesting the presence of alpha 1-receptors on septohippocampal GABAergic neurons.

58 y; instead, it decreases impulse flow in the septohippocampal GABAergic pathway via M(3) muscarinic r

59 sms, caused by increased impulse flow in the septohippocampal GABAergic pathway, may underlie the cog

60 assessing animals for cognitive function and septohippocampal integrity.

61 Septohippocampal interactions determine how stimuli are

62 luate NGF gene-experience interaction on the septohippocampal neural circuitry in mice.

63 ts of muscarine on antidromically identified septohippocampal neurons (SHNs).

64 A had the reverse effect, reducing GABAergic septohippocampal neurons and sparing cholinergic neurons

65 Cholinergic septohippocampal neurons are affected by circulating est

66 the neurons, we propose that the slow firing septohippocampal neurons are cholinergic whereas the bur

67 gic whereas the burst firing and fast firing septohippocampal neurons are GABAergic.

68 Slow firing septohippocampal neurons had significantly slower conduc

69 scent-labeling techniques to identify living septohippocampal neurons in rat brain slices, we now rep

70 ently increased firing in a subpopulation of septohippocampal neurons with fast conducting fibres (me

71 ies, mu-opioids inhibited a subpopulation of septohippocampal neurons with high conduction velocity f

72 Of the septohippocampal neurons, four were slow firing neurons,

73 Using recordings from retrogradely labeled septohippocampal neurons, we found that Hcrt2-excited MS

74 olinergic neurons although sparing GABAergic septohippocampal neurons.

75 ulum, and parasubiculum), and in the septum (septohippocampal nucleus and lateral dorsal septum).

76 rnix, septal nuclei, hippocampal commissure, septohippocampal nucleus, fimbria, anteroventral thalami

77 mygdaloid nucleus and dentate gyrus, but the septohippocampal nucleus, lateral septal nuclei, amygdal

78 , the CA1-CA3 fields of the hippocampus, the septohippocampal nucleus, the diagonal band of Broca, th

79 al pathway was only mildly affected, and the septohippocampal pathway and the striatum were both pres

80 The septohippocampal pathway contains cholinergic, GABAergic

81 Cholinergic mechanisms in the septohippocampal pathway contribute to several cognitive

82 se seen following surgical disruption of the septohippocampal pathway in adult animals.

83 inergic and catecholaminergic markers in the septohippocampal pathway in rats through fourth month of

84 ion to changes in cholinergic markers in the septohippocampal pathway of the rat.

85 ats in tasks believed to be modulated by the septohippocampal pathway tended to be differentially aff

86 enetically in the adult rat nigrostriatal or septohippocampal pathway using recombinant adeno-associa

87 n the basal forebrain that contribute to the septohippocampal pathway were found to express m2, m3, a

88 In the septohippocampal pathway, GABAergic deficits are observe

89 iagonal band (MSDB), which gives rise to the septohippocampal pathway, is a critical locus for the mn

90 septum-diagonal band (MSDB) complex, via the septohippocampal pathway, is thought to be critical for

91 lues that were unchanged in the striatum and septohippocampal pathway, significantly lower in the ant

92 evealed a graded effect of experience on the septohippocampal pathway, with the largest change occurr

93 ibly by coordinating activity in the SCN and septohippocampal pathway.

94 ing and regulating normal functioning of the septohippocampal pathway.

95 Spatial learning requires the septohippocampal pathway.

96 ty of mRNA included the medial habenula; the septohippocampal, periventricular, suprachiasmatic, and

97 loid (Abeta) toxicity and maintenance of the septohippocampal phenotype.

98 that acetylcholine release from cholinergic septohippocampal projections causes a long-lasting GABAe

99 imbalance between GABAergic and cholinergic septohippocampal projections, as produced by septal infu

100 Cultured postnatal day-of-birth (P0) septohippocampal (SH) neurons that were labeled with 1,1

101 e growth factor (NGF) affect the survival of septohippocampal (SH) neurons.

102 M is, however, reciprocally connected to the septohippocampal system and there is strong evidence tha

103 The rodent septohippocampal system contains "theta cells," which bu

104 Recent studies have supported a role for the septohippocampal system in the online processing of inte

105 battery of behavioral tests modulated by the septohippocampal system including two versions of the Mo

106 The disruption of cholinergic septohippocampal system may be an important factor in la

107 rmation and provide further evidence for the septohippocampal system's involvement in processing inte

108 of two neural systems, the amygdala and the septohippocampal system, to examine possible interaction

109 the subcellular localization of TrkA within septohippocampal terminal fields, two rabbit polyclonal

110 of the 3 main cholinergic pathways-the Ch1 (septohippocampal), the Ch4 (innominatocortical), and the

111 Reduction of septohippocampal TrkA receptor expression selectively im