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1 h p16 silencing in the absence of p16 coding sequence deletion and MAX induction restores p16 express
2 he A genome include retroelement insertions, sequence deletions, and mutations causing in-frame stop
3           Furthermore, we identified a large sequence deletion ( approximately 0.58 kb) in the C-term
4  that transfected RNAs harboring 5'-terminal sequence deletions are able to direct the synthesis of v
5                          PolyA simple repeat sequence deletions are common in tumors with microsatell
6 of the break points associated with all four sequenced deletions are consistent with this model.
7 ated error-prone end-joining and restricting sequence deletion at the break junction during repair.
8                               53BP1 supports sequence deletion during MMEJ consistent with a putative
9 d deletions in terms of the size and rate of sequence deletion, evolutionary factors can reconcile th
10                                   Additional sequence deletion in order to shorten the internal stand
11 relia, mating type O is determined by coding-sequence deletions in a different gene, mtB, which is sp
12 siological effects of in-frame actin-binding sequence deletions in the context of a highly functional
13  for known coreceptor polymorphisms or large sequence deletions in the nef gene associated with delay
14    Experiments with antibody competition and sequence deletion indicated that His657-Lys676 of FAD-AP
15                                   The signal sequence deletion mutant, phoZDeltass, was used to ident
16  nucleus, as the nuclear localization signal sequence-deletion mutant of MDM2, which localized in the
17 p1-binding site in HEG1 contains a 9 residue sequence, deletion of which abrogates HEG1's ability to
18 ty arises in the LMP-1 gene: point mutation, sequence deletion or duplication, and homologous recombi
19  deletions or duplications within the coding sequence, deletion or duplication frameshifts, insertion
20 ever, we did identify the 21-bp MEF2A coding sequence deletion originally implicated in adCAD1 in 1 o
21                                        Small sequence deletions resulted in a novel class of heteroch
22 However, deviations from the model for short sequence deletions suggest that RAM contains sequence-sp
23  constructed a series of point mutations and sequence deletions throughout Cdc19p, which allow us to
24 T domain, as a G18A/G20A double mutant and a sequence deletion within the conserved motif impaired Ad

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